Search results for "Colitis"

showing 10 items of 483 documents

Modulation of mucin 2 and mucin 3 in colitis induced by iodoacetamide and enteropathogenic bacteria in rats

2016

The iinate and acquired immune systems are both implicarted in the etiology of Inflammatory Bowel Disease (IBD) in addition to the genetic predisposition, the environmental factors and the intestinal flora covering the mucosa. A defect in the mucous covering will lead to an invasion of pathogens and stimulation of the immuune response with aberrations of mucin 2, the major mucin of the mucous layer. Aim: this study aims to assess the modulation of colonic MUC 2 and MUC 3 in a arat model of IBD induced by a combination of iodoacetamide and enteropatogenic E. Coli. Methods: 78 sprague-Dawley female rats were divided into 4 groups. Each group was subjeceted, on a basis, to a rectal injection o…

Settore MED/18 - Chirurgia GeneraleSettore BIO/16 - Anatomia UmanaIBD Inflammatory Bowel Disease Colitis induced in rat iodoacetamide Enteropatogenic bacteria
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NUTRITION IN IBD PATIENTS

2013

Inflammatory Bowel Disease (IBD) is a chronic disorder characterized by a relapsing-remitting course, which alternates between active and quiescent states, ultimately impairing a patients' quality of life.The two main types of IBD are Crohn's disease (CD) and Ulcerative Colitis (UC). CD Shows a transmural granulomatous inflammation that can involve any segment of the intestine affecting all layers of the intestinal wall, while UC is limited to the mucosa and superficial submucosa of the colon. In physiological conditions the gut is costantly exposed to various antigens, commensal microflora and pathogens and the inflammatory response is finely balanced. Anyhow i some individuals with geneti…

Settore MED/18 - Chirurgia GeneraleSettore MED/09 - Medicina InternaIBD Ulcerative Colitis Crohn Disease mesenchymal stem cells murin experimental studySettore BIO/11 - Biologia Molecolare
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AMYLOIDOSIS AND INFLAMMATORY BOWEL DISEASE: FACT OR MITH?

2017

Inflammatory Bowel Disease (IBD), which includes both Crohn's Disease (CD) and Ulcerative Colitis (UC), is a chronic idiopathic inflammatory disorder affecting the gastrointestinal tract. Extraintestinal manifestations (EIM) are common in patients with IBD, and occur in 6-47% of patients with CD or UC. EIM can involve organs other than the gastrointestinal tract such as skin, eyes, joints, biliary tract and kidneis. Renal and urinary involvement particularly occurs in 4-23% of patients with IBD. Among the renal complications of IBD, seconfary amyloidosis (AA-type, AAA) is a rare but serious complication. renal amyloidosis has been proven to be the most common lethal manifestation of IBD-ass…

Settore MED/18 - Chirurgia GeneraleSettore MED/12 - GastroenterologiaCrohn' disease Ulcerative Colitis Amyloidisis IBD
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CROHN'S DISEASE AND EXTRAINTESTINAL GRANULOMATOUS LESIONS

2018

Crohn's disease (CD) is an inflammatory bowel disease with a multifactorial etiology. Clinical features include mucosal erosion, diarrhea, weight loss and other complications such as formation of granuloma. in CD, granuloma is a non-neoplastic epithelioid lesio, formed by a compact aggregate of histiocytes with the absence of a central necrosis, however, the correlation amomg CD and the formation of granulomas is unknown. Many casesof granulomas in the extracellular site, related to Cd, have been reported in the literature. These granulomas, at times, rapresented the only visible manifestation of the pathology. extra intestinal granuloma have been found on ovaries, lungs, male genitalia, fe…

Settore MED/18 - Chirurgia GeneraleSettore MED/12 - GastroenterologiaCrohn's disease granulomatous lesions extra-intestinal manifestations colitis IBD
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An innate cell-mediated, murine ulcerative colitis-like syndrome in the absence of nuclear factor of activated T cells.

2004

Abstract Background & Aims: Nuclear factor of activated T cells transcription factors plays a central role in immunity by regulating the expression of multiple cytokines and other regulatory molecules, many of which have been heavily implicated in the pathogenesis of inflammatory bowel disease. However, few studies have directly investigated the nuclear factor of activated T cells proteins in inflammatory bowel disease. We describe here a specific role for nuclear factor of activated T cells c2 in the pathogenesis of murine inflammatory bowel disease. Methods: Mice deficient for nuclear factor of activated T cells c2, recombinase activating gene-2, or both and transgenic or nontransgenic fo…

T-LymphocytesBiologyInterleukin 21MicemedicineImmune ToleranceCytotoxic T cellAnimalsIL-2 receptorB-LymphocytesImmunity CellularMice Inbred BALB CHepatologyNFATC Transcription FactorsZAP70Innate lymphoid cellGastroenterologyNuclear ProteinsT helper cellRectal ProlapseNatural killer T cellAcquired immune systemMice Mutant StrainsDNA-Binding ProteinsMice Inbred C57BLmedicine.anatomical_structureImmunologyCancer researchColitis UlcerativeTranscription FactorsGastroenterology
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Regulation of T-cell apoptosis in inflammatory bowel disease: to die or not to die, that is the mucosal question.

2001

T-cell resistance against apoptosis contributes to inappropriate T-cell accumulation and the perpetuation of chronic mucosal inflammation in inflammatory bowel diseases (IBDs). Anti-interleukin-12 (IL-12) and anti-IL-6 receptor antibodies suppress colitis activity by the induction of T-cell apoptosis. These findings have important implications for the design of effective treatment regimens in IBD.

T-LymphocytesImmunologyApoptosisInflammatory bowel diseaseImmunology and AllergyMedicineEffective treatmentAnimalsHumansColitisIntestinal MucosaReceptorImmunity MucosalT-cell apoptosisbiologyCell Deathbusiness.industryInflammatory Bowel Diseasesmedicine.diseaseInflammatory Bowel Diseasesdigestive system diseasesApoptosisImmunologybiology.proteinAntibodybusinessTrends in immunology
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Oleuropein protects against dextran sodium sulfate-induced chronic colitis in mice.

2013

The anti-inflammatory effect of oleuropein (1), the major phenolic secoiridoid in Olea europaea, was evaluated in an experimental model of chronic colitis in mice. Animals were exposed to four repeated cycles of dextran sodium sulfate in drinking water followed by a 7-day rest period. Animals receiving a standard diet supplemented with 0.25% of 1 (equivalent to 500 mg/kg/day) for 56 days exhibited a decrease of inflammatory symptoms, as reflected by improvement of disease activity index and histopathological changes. It was found that 1 decreased inflammatory cell recruitment and the release of inflammatory cytokines interleukin (IL)-1β and IL-6 with increased IL-10 levels in colon tissue. …

T-LymphocytesInterleukin-1betaIridoid GlucosidesAnti-Inflammatory AgentsPharmaceutical ScienceNitric Oxide Synthase Type IIPharmacologyp38 Mitogen-Activated Protein KinasesAnalytical Chemistrychemistry.chemical_compoundMiceOleuropeinOleaDrug DiscoveryAnimalsIridoidsIntestinal MucosaChronic colitisPyransPharmacologybiologyMolecular StructureExperimental modelInterleukin-6Organic ChemistryDextran Sulfatebiology.organism_classificationColitisInterleukin-10Mice Inbred C57BLComplementary and alternative medicinechemistryOleaCyclooxygenase 2Molecular MedicineDextran sodium sulfateJournal of natural products
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Cutting Edge: IL-23 Cross-Regulates IL-12 Production in T Cell-Dependent Experimental Colitis

2006

Abstract Although IL-12 and IL-23 share the common p40 subunit, IL-23, rather than IL-12, seems to drive the pathogenesis of experimental autoimmune encephalomyelitis and arthritis, because IL-23/p19 knockout mice are protected from disease. In contrast, we describe in this study that newly created LacZ knockin mice deficient for IL-23 p19 were highly susceptible for the development of experimental T cell-mediated TNBS colitis and showed even more severe colitis than wild-type mice by endoscopic and histologic criteria. Subsequent studies revealed that dendritic cells from p19-deficient mice produce elevated levels of IL-12, and that IL-23 down-regulates IL-12 expression upon TLR ligation. …

T-LymphocytesTransgeneT cellImmunologyDown-RegulationMice TransgenicInterleukin-23PathogenesisMiceInterleukin 23AnimalsImmunology and AllergyMedicineColitisCells Culturedbusiness.industryInterleukinsExperimental autoimmune encephalomyelitisColitismedicine.diseaseInterleukin-12Survival RateDisease Models AnimalProtein Subunitsmedicine.anatomical_structureImmunologyKnockout mouseInterleukin-23 Subunit p19Interleukin 12Disease SusceptibilitybusinessThe Journal of Immunology
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Adaptation of TECCU App Based on Patients´ Perceptions for the Telemonitoring of Inflammatory Bowel Disease: A Qualitative Study Using Focus Groups

2020

Background: Despite the continuous adaptation of eHealth systems for patients with inflammatory bowel disease (IBD), a significant disconnection persists between users and developers. Since non-adherence remains high, it is necessary to better understand the patients&rsquo

Telemedicine020205 medical informaticsHealth Toxicology and Mutagenesiseducationlcsh:Medicine02 engineering and technologyArticle03 medical and health sciences0302 clinical medicineNursingCrohn Diseaseinflammatory bowel disease0202 electrical engineering electronic engineering information engineeringeHealthMedicineHumansAdaptation (computer science)E-healthbusiness.industrylcsh:RPublic Health Environmental and Occupational HealthUsabilityInflammatory Bowel DiseasesFocus groupMobile Applicationsdigestive system diseasesClinical trialPatient Compliancefocus groupse-health030211 gastroenterology & hepatologyColitis UlcerativetelemedicineThematic analysisbusinessqualitative researchQualitative researchInternational Journal of Environmental Research and Public Health
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Efecte protector dels compostos fenòlics àcid el·làgic, apocinina i rutina en el tractament de la colitis ulcerosa induïda per dextranosulfat sòdic

2014

L’ús de productes naturals entre aquells pacients que pateixen malalties cròniques s’inclou dins de les opcions més emprades dels tractaments complementaris i/o alternatius a la medicina convencional. Entre els constituents naturals d'origen vegetal amb interès terapèutic destaquen els compostos fenòlics, alguns dels quals han demostrat ser efectius en el tractament de la colitis ulcerosa (CU). Aquesta malaltia, d’etiologia desconeguda, es caracteritza per una inflamació del còlon que afecta la mucosa des del recte fins a la seua totalitat. El present treball ha abordat l’avaluació de les propietats protectores de tres compostos amb estructura fenòlica -àcid el·làgic, apocinina i rutina-, i…

UNESCO::CIENCIAS MÉDICAS:CIENCIAS MÉDICAS [UNESCO]medicamentos naturalescolitis ulcerosa
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