Search results for "Core"

showing 10 items of 1999 documents

Interaction of wild-type and naturally occurring deleted variants of hepatitis B virus core polypeptides leads to formation of mosaic particles

2000

AbstractThe simultaneous presence of hepatitis B virus (HBV) genomes carrying wild-type (wt) and in-frame deleted variants of the HBV core gene has been identified as a typical feature of HBV-infected renal transplant patients with severe liver disease. To investigate possible interactions of wt and deleted core polypeptides a two-vector Escherichia coli expression system ensuring their concomitant synthesis has been developed. Co-expression of wt and a mutant core lacking 17 amino acid residues (77–93) within the immunodominant region led to the formation of mosaic particles, whereas the mutant alone was incapable of self-assembly.

Hepatitis B virusBlotting WesternMutantBiophysicsBiologymedicine.disease_causeBiochemistryGenomeHepatitis B virus PRE betaLiver diseaseStructural BiologyEscherichia coliGeneticsmedicineProtein Structure QuaternaryMolecular BiologyEscherichia coliSequence DeletionHepatitis B virusImmunodominant EpitopesHepatitis B virus coreViral Core ProteinsVirus AssemblyWild typeGenetic VariationCell Biologymedicine.diseaseDimer formationHepatitis B Core AntigensPrecipitin TestsVirologyMolecular biologyRecombinant ProteinsMosaic particleMicroscopy ElectronPeptidesDimerizationC gene deletionProtein BindingFEBS Letters
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Hepatitis B core particles as a universal display model: a structure-function basis for development

1999

AbstractBecause it exhibits a remarkable capability to accept mutational intervention and undergo correct folding and self-assembly in all viable prokaryotic and eukaryotic expression systems, hepatitis B core (HBc) protein has been favored over other proposed particulate carriers. Structurally, the unusual α-helical organization of HBc dimeric units allows introduction of foreign peptide sequences into several areas of HBc shells, including their most protruding spikes. Progress toward full resolution of the spatial structure as well as accumulation of chimeric HBc-based structures has brought closer the knowledge-based design of future vaccines, gene therapy tools and other artificial par…

Hepatitis B virusGenes ViralCryo-electron microscopyMacromolecular SubstancesProtein ConformationBiophysicsComputational biologyBiologyBiochemistryMolecular displayEpitopesProtein structureStructural BiologyGeneticsProkaryotic expressionAnimalsHumansMolecular BiologyDrug CarriersBinding SitesSpatial structureViral Core ProteinsStructure functionHepatitis B core proteinvirus diseasesCell BiologyBasis (universal algebra)Self-assemblyAntigenicityVirologyBiological EvolutionHepatitis B Core Antigensdigestive system diseasesFolding (chemistry)Protein structureElectron cryomicroscopyDimerizationHepatitis b coreFEBS Letters
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Molecular Epidemiology and Immunology of Hepatitis B Virus Infection – An Update

2003

Hepatitis B virus (HBV) continues to be one of the most important viral pathogens in humans. This review provides an update on the molecular epidemiology and immunology of HBV infection. DNA sequencing has allowed replacement of the initial serotypic classification of HBV strains by a more systematic genotype system that currently consists of 7 members (genotypes A–G). More recently, sequence analysis of virus isolates from many individual patients has revealed the occurrence of certain mutational hot spots in the genome, some of which appear to correlate with the patient’s immunological and/or disease status; however, cause and effect are not always easily discernible. This holds particula…

Hepatitis B virusGenotypeMolecular Sequence DataPopulationBiologymedicine.disease_causeVirusVirologyGenotypemedicineHumansAmino Acid SequencePromoter Regions GeneticeducationHepatitis B viruseducation.field_of_studyMutationMolecular epidemiologyViral Core ProteinsVirionHepatitis BVirologyReverse transcriptaseVaccinationInfectious DiseasesMutationImmunologyIntervirology
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Stop codon insertion restores the particle formation ability of hepatitis B virus core-hantavirus nucleocapsid protein fusions.

2003

In recent years, epitopes of various origin have been inserted into the core protein of hepatitis B virus (HBc), allowing the formation of chimeric HBc particles. Although the C-terminus of a C-terminally truncated HBc (HBcΔ) tolerates the insertion of extended foreign sequences, the insertion capacity is still a limiting factor for the construction of multivalent vaccines. Previously, we described a new system to generate HBcΔ mosaic particles based on a read-through mechanism in an <i>Escherichia coli</i> suppressor strain [J Gen Virol 1997;78:2049–2053]. Those mosaic particles allowed the insertion of a 114-amino acid (aa)-long segment of a Puumala hantavirus (PUUV) nucleocap…

Hepatitis B virusHepatitis B virus DNA polymerasevirusesRecombinant Fusion ProteinsMolecular Sequence Datamedicine.disease_causeEpitopeHepatitis B virus PRE betaMiceVirologyparasitic diseasesmedicineAnimalsNucleocapsidHantavirusHepatitis B virusMice Inbred BALB CBase SequenceChemistryHepatitis B virus coreVirionvirus diseasesNucleocapsid ProteinsVirologyMolecular biologyHepatitis B Core Antigensdigestive system diseasesStop codonNS2-3 proteaseInfectious DiseasesCodon TerminatorImmunizationIntervirology
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Priming of cytotoxic T cell responses to exogenous hepatitis B virus core antigen is B cell dependent

2003

The hepatitis B virus (HBV) core antigen (HBcAg) has a unique ability to bind a high frequency of naive human and murine B cells. The role of HBcAg-binding naive B cells in the immunogenicity of HBcAg is not clear. The HBcAg-binding properties of naive B cells were characterized using HBcAg particles with mutated spike region (residues 76-85) sequences. Deletion of residues 76-85 (HBcDelta76-85) destroyed naive B cell binding, whereas deletion of residues 79-85 did not. HBcAg particles with an Ile instead of the natural Ala at position 80 did not bind naive B cells, whereas reversion of Ile80--Ala restored B cell binding. Destroying the B cell-binding ability of HBcAg had a marginal effect …

Hepatitis B virusMolecular Sequence DataNaive B cellPriming (immunology)Biologymedicine.disease_causeMiceAntigenVirologymedicineAnimalsCytotoxic T cellHepatitis B VaccinesAmino Acid SequenceHepatitis B AntibodiesB cellHepatitis B virusB-LymphocytesVaccines SyntheticBinding SitesImmunogenicityVirionvirus diseasesHepatitis BHepatitis B Core AntigensVirologyRecombinant Proteinsdigestive system diseasesMice Inbred C57BLHBcAgmedicine.anatomical_structureImmunizationT-Lymphocytes Cytotoxic
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Pre-core mutants of hepatitis B virus in patients receiving immunosuppressive treatment after orthotopic liver transplantation.

1996

Orthotopic liver transplantation (OLT) is a possible treatment for acute or chronic liver failure due to hepatitis B virus (HBV) infection, but reinfection of the graft can be a serious complication. The aim of this study was to monitor HBV markers, to analyse pre-core-/core-mutations as well as to identify the viral population causing reinfection after OLT, and to investigate the emergence or disappearance of these mutants in patients receiving immunosuppressive treatment. Fifty-four pre-and posttransplant serum samples of 17 patients were analysed. All patients underwent OLT for HBV-related liver disease and had HBV-DNA before and after OLT. Total DNA was extracted from all sera and a 240…

Hepatitis B virusTime Factorsmedicine.medical_treatmentPopulationLiver transplantationInterferon alpha-2medicine.disease_causeAntiviral AgentsLiver diseaseVirologyMedicineHumansHepatitis B e AntigensProtein PrecursorseducationHepatitis B viruseducation.field_of_studyHepatitis B Surface Antigensbiologybusiness.industryInterferon-alphaImmunosuppressionSequence Analysis DNAHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis BVirologyHepatitis B Core AntigensRecombinant ProteinsLiver TransplantationTransplantationsurgical procedures operativeInfectious DiseasesHepadnaviridaeDNA ViralbusinessImmunosuppressive AgentsFollow-Up StudiesJournal of medical virology
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Mosaic particles formed by wild-type hepatitis B virus core protein and its deletion variants consist of both homo- and heterodimers.

2003

AbstractCo-expression in Escherichia coli of wild-type (wt) hepatitis B virus core protein (HBc) and its naturally occurring variants with deletions at amino acid positions 77–93 or 86–93 leads to formation of mosaic particles, which consist of three dimer subunit compositions. These compositions are wt/variant HBc heterodimers and two types of homodimers, formed by wt HBc or the variant HBc themselves. Mosaic particles were found also when both HBc deletion variants 77–93 and 86–93 were co-expressed in E. coli. These findings are discussed in terms of their significance for hepatitis B virus pathogenesis and prospective use of mosaic particles in vaccine development.

Hepatitis B virusvirusesProtein subunitDimerBiophysicsExpressionPlasma protein bindingBiologymedicine.disease_causeMosaic particlesBiochemistrychemistry.chemical_compoundHepatitis B virus core proteinProtein structureStructural Biologyparasitic diseasesGeneticsmedicineHepatitis B VaccinesCloning MolecularProtein Structure QuaternaryMolecular BiologyEscherichia coliSequence Deletionchemistry.chemical_classificationHepatitis B virusViral Core ProteinsWild typevirus diseasesGenetic VariationCell BiologyHepatitis BDimer formationVirologyMolecular biologydigestive system diseasesAmino acidProtein SubunitschemistryDimerizationProtein BindingFEBS letters
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e System and intrahepatocelullar HBcAG and HBsAG in HBsAG positive patients with liver diseases and healthy carriers.

1977

Patients with hepatitis-B surface antigen positive liver diseases and healthy carriers were studied for the presence of e-antigen and anti-e as well as for intrahepatocellular HBsAG and hepatitis-B core antigen. The e-antigen was demonstrated in 9 out of 12 patients with chronic perisitent hepatitis, in 15 out of 39 patients with chronic active hepatitis, in 3 out of 40 patients with acute type B hepatitis, and in 2 out of 9 patients with a protracted course of type B hepatitis. No e-antigen was found in healthy HBsAG carriers nor in patients with complete recovery from type B hepatitis one year after onset of the disease. Anti-e was detected in 24 out of 61 healthy HBsAG carriers with a no…

HepatitisHBsAgChronic Activebusiness.industryLiver cellLiver DiseasesGastroenterologyChronic persistent hepatitisDiseasemedicine.diseaseHepatitis BHepatitis B Core AntigensHepatitisHepatitis B AntigensHBcAgAntigenLiverImmunologyAcute DiseaseCarrier StateChronic DiseasemedicineHumansAntigensbusinessScandinavian journal of gastroenterology
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"Table 22" of "Test of CP invariance in vector-boson fusion production of the Higgs boson in the H → ττ channel in proton–proton collisions at s=13Te…

2020

Post-fit BDT distributions in the $Z\to \ell\ell$ CR for the $\tau_{\mathrm{lep}}\tau_{\mathrm{lep}}$ SF analysis channel. The size of the combined statistical, experimental, and theoretical uncertainties is given. The exact value of the $p_{\mathrm{T}}$ cut on the leptons depends on the trigger.

Higgs Production13000.0Proton-Proton ScatteringBDT ScoreHigh Energy Physics::PhenomenologyTau-Tau DecayHigh Energy Physics::ExperimentP P -> H + X
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"Table 20" of "Test of CP invariance in vector-boson fusion production of the Higgs boson in the H → ττ channel in proton–proton collisions at s=13Te…

2020

Post-fit BDT distributions in the top-quark CR for the $\tau_{\mathrm{lep}}\tau_{\mathrm{lep}}$ SF channel. The size of the combined statistical, experimental, and theoretical uncertainties is given. The exact value of the $p_{\mathrm{T}}$ cut on the leptons depends on the trigger.

Higgs Production13000.0Proton-Proton ScatteringBDT ScoreHigh Energy Physics::PhenomenologyTau-Tau DecayHigh Energy Physics::ExperimentP P -> H + X
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