Search results for "Corticosterone"

showing 10 items of 72 documents

N6 -Methyladenosine Modification in Chronic Stress Response Due to Social Hierarchy Positioning of Mice

2021

Appropriately responding to stressful events is essential for maintaining health and well-being of any organism. Concerning social stress, the response is not always as straightforward as reacting to physical stressors, e.g., extreme heat, and thus has to be balanced subtly. Particularly, regulatory mechanisms contributing to gaining resilience in the face of mild social stress are not fully deciphered yet. We employed an intrinsic social hierarchy stress paradigm in mice of both sexes to identify critical factors for potential coping strategies. While global transcriptomic changes could not be observed in male mice, several genes previously reported to be involved in synaptic plasticity, l…

Social stressMethyltransferase complexbehaviorQH301-705.5sex differenceStressorCell BiologyBiologydominancechemistry.chemical_compoundtranscriptomicschemistryCorticosteroneepigenetic modificationSynaptic plasticityChronic stressmethyltransferaseMRNA methylationN6-MethyladenosineBiology (General)NeuroscienceDevelopmental BiologyFrontiers in Cell and Developmental Biology
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Effects of chronic treatment with testosterone propionate on aggression and hormonal levels in intact male mice.

1998

Effects of testosterone propionate, an anabolic-androgenic steroid (AAS), on aggression in gonadally intact male mice were examined. Animals were given weekly injections of 3.75, 7.5, 15, and 30 mg/kg of drug or sesame oil for 10 weeks. During the last 3 weeks, behavioral tests were conducted and at the end of the experiment, body, liver and testes weight and hormonal data were collected. The treatment had minimal behavioral and endocrine effects. It resulted in shorter latencies of 'threat' only in the last agonistic encounter, increases in testosterone levels and decreases in testes weight in a non-linear dose-dependant way. The action of treatment was different on threat and attack, the …

Testosterone propionateMalemedicine.medical_specialtymedicine.drug_classEndocrinology Diabetes and MetabolismIndividualityTesticlechemistry.chemical_compoundMiceEndocrinologyAnabolic AgentsCorticosteroneInternal medicinemedicineAgonistic behaviourAnimalsTestosteroneSocial BehaviorBiological PsychiatryTestosteroneDose-Response Relationship DrugEndocrine and Autonomic SystemsAggressionAndrogenAggressionPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologychemistrymedicine.symptomPsychologyArousalCorticosteroneAgonistic BehaviorHormonePsychoneuroendocrinology
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Individual vulnerability to stress is associated with increased demand for intravenous heroin self-administration in rats

2019

AbstractOpioid use is a widespread epidemic, and traumatic stress exposure is a critical risk factor in opioid use and relapse. There is a significant gap in our understanding of how stress contributes to heroin use, and there are limited studies investigating individual differences underlying stress reactivity and subsequent stress-induced heroin self-administration. We hypothesized that greater individual vulnerability to stress would predict higher demand for heroin self-administration in a within-subjects rodent model of stress and heroin use comorbidity. Male rats were exposed to inescapable intermittent swim stress and individual biological (corticosterone) or behavioral (open field, …

economic demandheroin demandCognitive NeuroscienceOpen fieldlcsh:RC321-571Heroin03 medical and health sciencesBehavioral Neurosciencestresschemistry.chemical_compound0302 clinical medicineCorticosteronemental disorderscue reinstatementmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Research030304 developmental biology0303 health sciencesbusiness.industrystress reinstatementswim-stressStressorTraumatic stressExtinction (psychology)medicine.diseaseComorbidityNeuropsychology and Physiological Psychologychemistrypost-traumatic stress disorderheroinSelf-administrationbusiness030217 neurology & neurosurgeryNeurosciencemedicine.drugClinical psychology
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Zeitlicher Ablauf der ACTH-induzierten Stimulierung der Corticosteroidbiosynthese

1969

The time course of ACTH action was studied during incubation of quartered rat adrenals by estimation of corticosterone production and secretion rates. The results suggest a two step action of ACTH during the acute stimulation of corticosteroid biosynthesis: A modest initial stimulation was observed within the first 30 min of exposure to ACTH which was overlapped by a marked secondary stimulation between 30 and 60 min.

endocrine systemmedicine.medical_specialtymedicine.drug_classChemistryTwo stepStimulationRat Adrenalschemistry.chemical_compoundEndocrinologyCorticosteroneInternal medicineTime coursemedicineCorticosteroidSecretionIncubationhormones hormone substitutes and hormone antagonists
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Context dependent variation in corticosterone and phenotypic divergence of Rana arvalis populations along an acidification gradient

2022

Background Physiological processes, as immediate responses to the environment, are important mechanisms of phenotypic plasticity and can influence evolution at ecological time scales. In stressful environments, physiological stress responses of individuals are initiated and integrated via the release of hormones, such as corticosterone (CORT). In vertebrates, CORT influences energy metabolism and resource allocation to multiple fitness traits (e.g. growth and morphology) and can be an important mediator of rapid adaptation to environmental stress, such as acidification. The moor frog, Rana arvalis, shows adaptive divergence in larval life-histories and predator defense traits along an acidi…

endocrine systemviitasammakkoRanidaeEvolutionAcidification; Adaptive divergence; Amphibians; Corticosterone; Environmental stress; Evolutionary physiology; Phenotypic plasticityEnvironmental stressPhenotypic plasticitykortikosteroniEvolutionsbiologiAcidificationAmphibiansAdaptive divergencehappamoituminenQH359-425polycyclic compoundsAnimalsHumanshormonaaliset vaikutuksetQH540-549.5sopeutuminenEkologiEvolutionary BiologyEcologysammakkoeläimetHydrogen-Ion ConcentrationAdaptation PhysiologicalLarvaEvolutionary physiologyfenotyyppiAnuraCorticosteroneAcidsympäristönmuutoksethormones hormone substitutes and hormone antagonistsfysiologiset vaikutuksetResearch ArticleBMC Ecology and Evolution
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Aldosterone biosynthesis induced by ACTH and angiotensin II in newborn rat adrenocortical cells transfected by c-EJ-Ha-ras oncogene

1991

Abstract Adrenocortical cells were obtained by fractionated trypsination of newborn rat adrenal glands and transfected with a plasmid containing the EJ T24 -Ha-ras oncogene. Isolation of adhesive cells led to a proliferative cell line with an overexpression of 21 kDa ras protein. These cells incubated with corticosterone or deoxycorticosterone as the precursor produced a high level of 18-hydroxycorticosterone and aldosterone as identified by gas chromatography- mass spectrometry. ACTH and angiotensin II increased the basal production of aldosterone nineteen-fold and six-fold respectively. Under ACTH stimulation the ratio between aldosterone and 18-hydroxycorticosterone production was 1:3. T…

medicine.medical_specialtyBiophysicsBiologyPeptide hormoneTransfectionBiochemistryMass SpectrometryProto-Oncogene Proteins p21(ras)chemistry.chemical_compoundAdrenocorticotropic HormoneCorticosteroneInternal medicineAdrenal GlandsmedicineAnimals18-HydroxycorticosteroneAldosteroneMolecular BiologyChromatography High Pressure LiquidAldosteroneOncogeneAdrenal cortexCell growthAngiotensin IICell BiologyAngiotensin IIRatsEndocrinologymedicine.anatomical_structurechemistryCell cultureBiochemical and Biophysical Research Communications
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18-hydroxylation in the Y-1 adrenal cell line: response to ACTH and to culture conditions.

1992

The 18-hydroxylation of deoxycorticosterone in the Y-1 adrenal cell line was studied under various incubation and cell culture conditions and compared to 11 beta-hydroxylation. Repeated incubation of the substrate increased both 18- and 11 beta-hydroxylation in the Y-1 cells. Furthermore, both 18- and 11 beta-hydroxylation were increased with increased serum concentration and prolonged incubation time. While the increase in 11 beta-hydroxylation seemed to be independent of the type of serum, 18-hydroxylation was much more important in cells cultured in fetal or newborn calf serum supplemented medium than in those cultured in horse serum supplemented medium. As expected, ACTH treatment incre…

medicine.medical_specialtyCytochromeEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryHydroxylationBiochemistryHydroxylationchemistry.chemical_compoundMiceEndocrinologyAdrenocorticotropic HormoneCytochrome P-450 Enzyme SystemInternal medicineAdrenal GlandsmedicineAnimalsCytochrome P-450 CYP11B2DesoxycorticosteroneMolecular BiologyIncubationCells CulturedFetusbiologyDose-Response Relationship DrugSubstrate (chemistry)Cell BiologyFetal BloodIn vitroCulture MediaSteroid hormoneEndocrinologyBloodchemistryCell cultureSteroid Hydroxylasesbiology.proteinMolecular MedicineSteroid 11-beta-HydroxylaseThe Journal of steroid biochemistry and molecular biology
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Postnatal Overfeeding in Rodents by Litter Size Reduction Induces Major Short- and Long-Term Pathophysiological Consequences

2013

Numerous studies have demonstrated that the early postnatal environment can influence body weight and energy homeostasis into adulthood. Rodents raised in small litters have been shown to be a useful experimental model to study the short- and long-term consequences of early overnutrition, which can lead to modifications not only in body weight but also of several metabolic features. Postnatal overfeeding (PNOF) induces early malprogramming of the hypothalamic system, inducing acquired persisting central leptin and insulin resistance and an increase in orexigenic signals. Visceral white adipose tissue, lipogenic activity, and inflammatory status are increased in PNOF rodents, while brown adi…

medicine.medical_specialtyLitter Sizemedia_common.quotation_subjectHypothalamusAppetiteMedicine (miscellaneous)White adipose tissueHyperphagiaBiologyEnergy homeostasisToxicology03 medical and health scienceschemistry.chemical_compoundOvernutrition0302 clinical medicineInsulin resistanceCorticosteroneInternal medicineBrown adipose tissuemedicineAnimalsHumansHormone metabolismObesityInfant Nutritional Physiological Phenomena030304 developmental biologymedia_common2. Zero hunger0303 health sciencesNutrition and DieteticsLeptinInfantAppetitemedicine.diseaseHormonesEndocrinologymedicine.anatomical_structureAdipose TissuechemistryCardiovascular DiseasesInsulin ResistanceEnergy Intake030217 neurology & neurosurgeryThe Journal of Nutrition
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Stress hormones promote growth of B16-F10 melanoma metastases: an interleukin 6- and glutathione-dependent mechanism

2013

[EN] Background: Interleukin (IL)-6 (mainly of tumor origin) activates glutathione (GSH) release from hepatocytes and its interorgan transport to B16-F10 melanoma metastatic foci. We studied if this capacity to overproduce IL-6 is regulated by cancer cell-independent mechanisms. Methods: Murine B16-F10 melanoma cells were cultured, transfected with red fluorescent protein, injected i.v. into syngenic C57BL/6J mice to generate lung and liver metastases, and isolated from metastatic foci using high-performance cell sorting. Stress hormones and IL-6 levels were measured by ELISA, and CRH expression in the brain by in situ hybridization. DNA binding activity of NF-kappa B, CREB, AP-1, and NF-IL…

medicine.medical_specialtyTranscription GeneticMelanoma ExperimentalInterleukin 6ApoptosisEnzyme-Linked Immunosorbent AssayIn situ hybridizationBiologyMetastasesCREBReal-Time Polymerase Chain ReactionGeneral Biochemistry Genetics and Molecular BiologyFlow cytometryMiceNorepinephrineAdrenocorticotropic HormoneInternal medicineCell Line TumormedicineAnimalsNeoplasm MetastasisIn Situ HybridizationMedicine(all)medicine.diagnostic_testBase SequenceBiochemistry Genetics and Molecular Biology(all)Interleukin-6ResearchStress hormonesInterleukinGeneral MedicineTransfectionCell sortingMolecular biologyGlutathionehumanitiesEndocrinologyElectroporationApoptosisbiology.proteinCorticosteroneDNA ProbesHormoneTranscription FactorsJournal of Translational Medicine
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Imbalance between the neuromodulatory actions of corticotropin-releasing hormone and corticosterone as a pathophysiologic mechanism in panic disorder

1990

medicine.medical_specialtybusiness.industryMechanism (biology)Panic disordermedicine.diseasePathophysiologyPsychiatry and Mental healthchemistry.chemical_compoundCorticotropin-releasing hormoneEndocrinologychemistryCorticosteroneInternal medicineMedicinebusinessBiological PsychiatryJournal of Psychiatric Research
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