Search results for "Cyclazocine"

showing 2 items of 2 documents

On the opioid receptor subtype inhibiting the evoked release of 3H-noradrenaline from guinea-pig atria in vitro

1986

1. Guinea-pig isolated atria were incubated and loaded with 3H-(−)-noradrenaline. The intrinsic nerves were stimulated with trains of 5 or 35 field pulses (4 Hz), and the evoked efflux of 3H-noradrenaline and of total tritium was determined in the presence of atropine, corticosterone, desipramine, and phentolamine by liquid scintillation spectrometry. 2. Ethylketocyclazocine (1.4 nmol/l, IC50), MR 2033 (9.1 nmol/l), dynorphin A (1–13) (25 nmol/l, peptidase inhibitors present), etorphine (71 nmol/l), and [d-Ala2, d-Leu5]-enkephalin (>10 μmol/l, peptidase inhibitors present) inhibited the stimulation-evoked efflux of 3H-noradrenaline in a concentration-dependent manner, but not morphine up to…

Atropinemedicine.medical_specialtyEthylketocyclazocinemedicine.drug_classGuinea PigsPopulationEthylketocyclazocine(+)-NaloxoneIn Vitro TechniquesPharmacologyBinding CompetitiveDynorphinsNorepinephrinechemistry.chemical_compoundOpioid receptorInternal medicinemedicineAnimalsCyclazocineHeart AtriaPhentolamineeducationEndogenous opioidPharmacologyeducation.field_of_studyMorphineNaloxoneMyocardiumReceptors Opioid kappaDesipramineEtorphineDynorphin AGeneral MedicineEnkephalin Leucine-2-AlaninePeptide FragmentsBenzomorphansEndocrinologyEtorphineOpioidchemistryReceptors OpioidSynapsesCorticosteroneEnkephalin Leucinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Failure of opioids to affect excitation and contraction in isolated ventricular heart muscle

1989

The opioid agonists morphine (selective for mu-receptors) and ethylketocyclazocine (selective for kappa-receptors), at concentrations evoking strong effects in neuronal structures, did not significantly affect the configuration of the intracellularly recorded action potential and the force of contraction in ventricular heart muscle isolated from guinea pigs, rabbits and man. These results suggest that any changes of heart functions in vivo in response to opioid-like drugs are probably not mediated postsynaptically at the myocardial cell membrane but rather presynaptically, influencing the release of noradrenaline and/or acetylcholine from the nerve terminals.

medicine.medical_specialtyContraction (grammar)EthylketocyclazocineGuinea PigsAction PotentialsEthylketocyclazocineBiologyGuinea pigNorepinephrineCellular and Molecular NeuroscienceInternal medicineHeart ratemedicineAnimalsCyclazocineHumansOpioid peptideMolecular BiologyPharmacologyMorphineNaloxoneCell BiologyPapillary MusclesMyocardial ContractionAcetylcholineEndocrinologyOpioidSynapsesCirculatory systemMolecular MedicineRabbitsAcetylcholinemedicine.drugExperientia
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