Search results for "Cyclic compound"

showing 10 items of 819 documents

Dietary cholic acid lowers plasma levels of mouse and human apolipoprotein A-I primarily via a transcriptional mechanism

2000

To induce dietary atherosclerosis in mice, high-fat/high-cholesterol (HF) diets are frequently supplemented with cholic acid (CA). This diet produces low plasma levels of high-density lipoprotein (HDL) and high levels of low-density lipoprotein (LDL). However, HF diets without any added CA, which more closely resemble human diets, increase levels of both HDL and LDL, suggesting that CA may be responsible for the lowering of HDL. Our aim was to examine the potential mechanism responsible for the lowering of HDL. Nontransgenic (NTg) C57BL mice and apoA-I-transgenic (apoAI-Tg) mice, with greatly increased basal apoA-I and HDL levels, were used. Mice were fed the following four diets: control (…

medicine.medical_specialtyBile acidmedicine.drug_classCholesterolResponse elementCholic acidnutritional and metabolic diseasesBiologyBiochemistrychemistry.chemical_compoundEndocrinologyHigh-density lipoproteinchemistryInternal medicineLow-density lipoproteinpolycyclic compoundsmedicinelipids (amino acids peptides and proteins)Hepatic lipaseLipoproteinEuropean Journal of Biochemistry
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Liver collagen proportionate area predicts decompensation in patients with recurrent hepatitis C virus cirrhosis after liver transplantation

2012

Background and Aims:  Current histological scoring systems do not subclassify cirrhosis. Computer-assisted digital image analysis (DIA) of Sirius Red-stained sections measures fibrosis morphologically producing a fibrosis ratio (collagen proportionate area [CPA]). CPA could have prognostic value within a disease stage, such as cirrhosis. The aim of the present study was to evaluate CPA in patients with recurrent hepatitis C virus (HCV) allograft cirrhosis and assess its relationship with hepatic venous pressure gradient (HVPG). Methods:  In 121 consecutively-transplanted HCV patients with HVPG, measured contemporaneously with transjugular liver biopsies, 65 had Ishak stage 5 or 6 disease (4…

medicine.medical_specialtyCirrhosisHepatologybusiness.industryPortal venous pressuremedicine.medical_treatmentGastroenterologyHepatitis CLiver transplantationmedicine.diseaseGastroenterologychemistry.chemical_compoundchemistryInterquartile rangeInternal medicinecardiovascular systemmedicinePortal hypertensionheterocyclic compoundsDecompensationbusinessSirius RedJournal of Gastroenterology and Hepatology
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Free and sulfoconjugated dehydroepiandrosterone, cyclic adenosine-3′,5′-monophosphate, and free estriol in maternal and cord blood

1976

When free DHEA, its sulfatide, and sulfate were assayed in maternal plasma as well as in umbilical cord arterial and venous plasma, rather high concentrations were found in either fraction from cord arterial plasma, reflecting the fetal contribution not only of free DHEA and DHEA sulfate, but also of the lipophile steroid sulfatide. Since high DHEA levels were associated with elevated c-AMP concentrations, a certain interrelationship of both parameters is indicated. In the course of delivery, a rapid decrease of free estriol in maternal plasma was observed. Higher concentration of free estriol in umbilical venous plasma pointed at its placental biosynthesis from fetal precursors.

medicine.medical_specialtyCordmedicine.medical_treatmentDehydroepiandrosteroneUmbilical cordSteroidCellular and Molecular NeurosciencePregnancyInternal medicineCyclic AMPpolycyclic compoundsmedicineHumansMolecular BiologyPharmacologyFetusLabor ObstetricSulfoglycosphingolipidsEstriolSulfatesChemistryVenous PlasmaEstriolDehydroepiandrosteroneCell BiologyFetal BloodEndocrinologymedicine.anatomical_structureCord bloodMolecular MedicineFemalehormones hormone substitutes and hormone antagonistsExperientia
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DNA-dependent DNA polymerase pattern in noninfected and herpesvirus infected rabbit kidney cells.

1973

In this paper we report on a DNA-dependent DNA polymerase produced in herpesvirus infected cells which is not present in virions. It differs from the polymerases of noninfected cells by its molecular weight as well as by its insensitivity to cytosine arabinoside triphosphate (ara-CTP).

medicine.medical_specialtyDNA polymerasevirusesDeoxyribonucleotidesKidneyTritiumchemistry.chemical_compoundMedical microbiologyAdenosine TriphosphateVirologyRabbit kidneymedicineAnimalsSimplexvirusheterocyclic compoundsPolymeraseCells CulturedbiologyCell-Free SystemCytarabineGeneral Medicinebiochemical phenomena metabolism and nutritionVirologyMolecular biologyMolecular WeightchemistryDNA Nucleotidyltransferasesbiology.proteinChromatography GelRabbitsArabinofuranosylcytosine triphosphateDNAArchiv fur die gesamte Virusforschung
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Muscle adenylate kinase in Duchenne muscular dystrophy

1986

Abstract On the basis of electrophoretic and enzyme inhibition studies it was postulated that an aberrant adenylate kinase occurs in muscle and serum of patients with Duchenne muscular dystrophy (Schirmer, R.H. and Thuma, E. (1972) Biochim. Biophys. Acta 268, 92–97; Hamada, M. et al. (1981) Biochim. Biophys. Acta 660, 227–237; Hamada et al. (1985) J. Biol. Chem. 260, 11595–11602. On the basis of the following results we conclude that Duchenne muscular dystrophy patients do not possess an unusual adenylate kinase isoenzyme. (1) In muscle biopsies from five Duchenne patients, the electrophoretic mobility of adenylate kinase and the inhibition of the enzyme by P 1 , P 5 -di(adenosine-5′)pentap…

medicine.medical_specialtyDTNBDuchenne muscular dystrophyBiophysicsAdenylate kinaseDithionitrobenzoic AcidBiochemistryIsozymeMuscular Dystrophieschemistry.chemical_compoundNormal muscleInternal medicinemedicineHumansheterocyclic compoundsSulfhydryl CompoundsMolecular Biologychemistry.chemical_classificationAdenine NucleotidesMusclesAdenylate KinasePhosphotransferasesElectrophoresis Cellulose Acetatemedicine.diseaseMOPSIsoenzymesEndocrinologyEnzymechemistryPMSFDinucleoside PhosphatesBiochimica et Biophysica Acta (BBA) - General Subjects
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Digoxin-related leukocytoclastic vasculitis in a very elderly woman: A case report

2016

International audience; Even though digoxin causes many side effects, few cases of skin involvement are recorded in the French Pharmacovigilance Database. We report a case of leukocytoclastic vasculitis (LV) very probably due to digoxin. A 91-year-old woman, hospitalized following a fall, presented cardiac decompensation in a context of rapid atrial fibrillation requiring treatment with digoxin. Eight days later, a rash appeared on her back and trunk. It was neither itchy, nor painful and persisted despite local treatment. There were no other clinical anomalies. After a few days, the rash spread with appearance of bullous lesions, ulcerations and a necrosis on lymphedema of the two legs. Am…

medicine.medical_specialtyDigoxinDigoxinContext (language use)Vascularite leucocytoclasique030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicinePharmacovigilanceBiopsypolycyclic compoundsmedicinecardiovascular diseasesAdverse side effects030203 arthritis & rheumatologymedicine.diagnostic_testbusiness.industryAtrial fibrillationLeukocytoclastic vasculitismedicine.diseaseEffets indésirablesDermatologyRashLymphedemaSkin biopsyDigoxinemedicine.symptomCardiology and Cardiovascular Medicinebusiness[ SDV.MHEP.DERM ] Life Sciences [q-bio]/Human health and pathology/Dermatologymedicine.drug
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3H-cyproterone acetate: binding characteristics to human uterine progestagen receptors

1985

The availability of tritium labeled cyproterone acetate (CPA) facilitated the systematic investigation of the binding characteristics of this compound for human uterine progesterone receptors (PgR). The binding parameters of 3H-CPA are compared to those of 3H-R5020 and 3H-progesterone. The rate constants of association (k1M-1sec-1) to PgR were 7.8 X 10(3) for 3H-R5020, 4.5 X 10(4) for 3H-progesterone and 4.0 X 10(4) for 3H-CPA. The rate constants of dissociation (k-1, sec-1) were 3.6 X 10(-5) for 3H-R5020, 21.3 X 10(-5) for 3H-progesterone and 17.8 X 10(-5) for 3H-CPA. The Kd-values (M), as obtained by titration analysis and subsequent Scatchard plot analysis were 1.2 X 10(-9) for 3H-R5020,…

medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentStatistics as TopicTritiumBinding CompetitivePromegestoneSteroidchemistry.chemical_compoundEndocrinologyInternal medicineCentrifugation Density GradientmedicineHumansPotencyheterocyclic compoundsCyproteroneBinding siteCyproterone AcetateReceptorProgesteroneUterusCyproterone acetateKineticsEndocrinologychemistryDihydrotestosteronecardiovascular systemCyproteroneFemaleTritiumReceptors Progesteronehormones hormone substitutes and hormone antagonistsmedicine.drugJournal of Endocrinological Investigation
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Histamine-sensitive adenylate cyclase of human gastric mucosa: a model for H2-receptor excitation.

1978

Recent studies revealed that human gastric mucosa contains a histamine-sensitive adenylate cyclase which is almost completely localized within the acid-secreting area of the stomach. In an attempt to further characterize the effector system of histamine’s action, we compared the effects of H 1 — and H 2-receptor agonists upon the adenylate cyclase in human fundic gastric mucosa.

medicine.medical_specialtyNucleotidase activityAdenylate kinaseIn Vitro TechniquesCyclasechemistry.chemical_compoundHistamine H2 receptorInternal medicinemedicineGastric mucosaEthylaminesHumansheterocyclic compoundsPharmacology (medical)Receptors Histamine H2Receptors Histamine H1Pharmacologybusiness.industryEffectorStomachMethylhistaminesdigestive system diseasesStimulation Chemicalmedicine.anatomical_structureEndocrinologychemistryGastric MucosaReceptors HistaminebusinessHistamineResearch ArticleAdenylyl CyclasesHistamine
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Chronic heart damage following doxorubicin treatment is alleviated by lovastatin.

2014

The anticancer efficacy of anthracyclines is limited by cumulative dose-dependent early and delayed cardiotoxicity resulting in congestive heart failure. Mechanisms responsible for anthracycline-induced heart damage are controversially discussed and effective preventive measures are preferable. Here, we analyzed the influence of the lipid lowering drug lovastatin on anthracycline-induced late cardiotoxicity three month after treatment of C57BL/6 mice with five low doses of doxorubicin (5×3mg/kg BW; i.p.). Doxorubicin increased the cardiac mRNA levels of BNP, IL-6 and CTGF, while the expression of ANP remained unchanged. Lovastatin counteracted these persisting cardiac stress responses evoke…

medicine.medical_specialtyStatinCardiotonic AgentsAnthracyclinemedicine.drug_classBiologymedicine.disease_causeDNA MitochondrialInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsDoxorubicinHSP70 Heat-Shock ProteinsLovastatinPharmacologyCardiotoxicityEjection fractionAntibiotics AntineoplasticInterleukin-6Gene Expression ProfilingMyocardiumConnective Tissue Growth Factormedicine.diseaseFibrosisCardiotoxicityMice Inbred C57BLEndocrinologyDoxorubicinHeart failureFemaleLovastatinOxidative stressmedicine.drugDNA DamagePharmacological research
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MECHANISMS OF TENSION DEVELOPMENT INDUCED BY MONENSIN IN GUINEA-PIG AORTA : EFFECTS OF VERAPAMIL, OUABAIN, PRAZOSIN AND GLIBENCLAMIDE

1993

The influence of monensin (10 µmol/L) alone and in the presence of verapamil (3 µmol/L) or ouabain (1 mmol) and prazosin (1 µmol/L) or glibenclamide (10 µmol/L) were studied on the muscle tension of guinea pig aorta. Changes in tissue sodium, potassium and calcium ion contents of the aortic muscle produced by monensin in the presence of prazosin were evaluated. Monensin in normal Tyrode's solution containing prazosin caused an increase in the resting tension followed by a decrease and returned to normal values. Verapamil reduced the muscle tension induced by monensin. After ouabain, monensin induced sustained increase in the resting tension. Glibenclamide partially reversed the relaxant pha…

medicine.medical_specialtyanimal structuresVoltage-dependent calcium channelChemistryMonensinchemistry.chemical_elementCalciumOuabaincarbohydrates (lipids)Glibenclamidechemistry.chemical_compoundEndocrinologyMuscle tensionInternal medicinecardiovascular systemPrazosinmedicineVerapamilheterocyclic compoundsmedicine.drugZagazig Journal of Pharmaceutical Sciences
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