Search results for "Cyclins"

showing 10 items of 31 documents

Prognostic Value of p53, p21/WAF1, Bcl-2, Bax, Bak and Ki-67 Immunoreactivity in pT1 G3 Urothelial Bladder Carcinomas

2001

pT1 G3 bladder carcinomas are heterogeneous with respect to tumor recurrence and progression. Whereas some urologists treat these carcinomas by repeated transurethral resections often followed by intravesical chemotherapy or BCG instillation, others recommend cystectomy after tumor recurrence or early cystectomy after the initial diagnosis. Our goal was to determine the prognostic value of p53, p21/WAF1, Bcl-2, Bax, Bak, and Ki-67 immunoreactivity in these tumors. There were 30 patients with a new histopathological diagnosis of pT1 G3 urothelial carcinoma based on a transurethral resection specimen. Representative sections of these specimens were examined for the above markers. All patients…

Cyclin-Dependent Kinase Inhibitor p21medicine.medical_specialtyPathologyTime FactorsTumor suppressor genemedicine.medical_treatmentBcl 2 baxUrologyDisease-Free SurvivalCystectomyPredictive Value of TestsCyclinsProto-Oncogene ProteinsBiomarkers TumormedicineHumansNeoplasm InvasivenessRetrospective Studiesbcl-2-Associated X ProteinCarcinoma Transitional CellUrinary bladderBladder cancerbiologyMembrane ProteinsGeneral MedicinePrognosismedicine.diseaseImmunohistochemistrySurvival RateKi-67 Antigenbcl-2 Homologous Antagonist-Killer Proteinmedicine.anatomical_structureTransitional cell carcinomaProto-Oncogene Proteins c-bcl-2Urinary Bladder NeoplasmsTumor progressionKi-67biology.proteinTumor Suppressor Protein p53Follow-Up StudiesTumor Biology
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Molecular basis of the functional distinction between Cln1 and Cln2 cyclins

2012

Cln1 and Cln2 are very similar but not identical cyclins. In this work, we tried to describe the molecular basis of the functional distinction between Cln1 and Cln2. We constructed chimeric cyclins containing different fragments of Cln1 and Cln2 and performed several functional analysis that make it possible to distinguish between Cln1 or Cln2. We identified that region between amino acids 225 and 299 of Cln2 is not only necessary but also sufficient to confer Cln2 specific functionality compared with Cln1. We also studied Cln1 and Cln2 subcellular localization identifying additional differences between them. Both cyclins are distributed between the nucleus and the cytoplasm, but Cln1 shows…

CytoplasmSaccharomyces cerevisiae ProteinsTranscription GeneticBlotting WesternGenes FungalGenetic VectorsGreen Fluorescent ProteinsActive Transport Cell NucleusSaccharomyces cerevisiaeKaryopherinsBiologyReportCyclinsGene Expression Regulation FungalmedicineAmino Acid SequenceNuclear export signalMolecular BiologyPeptide sequenceCyclinKaryopherinCell Nucleuschemistry.chemical_classificationCell Cycle CheckpointsCell BiologySubcellular localizationCell nucleusmedicine.anatomical_structureBiochemistrychemistryCytoplasmNuclear transportCDC28 Protein Kinase S cerevisiaePlasmidsDevelopmental BiologyCell Cycle
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Cell Cycle Activation of the Swi6p Transcription Factor Is Linked to Nucleocytoplasmic Shuttling

2003

The control of the subcellular localization of cell cycle regulators has emerged as a crucial mechanism in the regulation of cell division. In the present work, we have characterized the function of the karyopherin Msn5p in the control of the cell cycle of Saccharomyces cerevisiae. Phenotypic analysis of the msn5 mutant revealed an increase in cell size and a functional interaction between Msn5p and the cell cycle transcription factor SBF (composed of the Swi4p and Swi6p proteins), indicating that Msn5p is involved in Start control. In fact, we have shown that the level of Cln2p protein is drastically reduced in an msn5 mutant. The effect on CLN2 expression is mediated at a transcriptional …

CytoplasmSaccharomyces cerevisiae ProteinsTranscription GeneticCell divisionChromosomal Proteins Non-HistoneActive Transport Cell NucleusSaccharomyces cerevisiaeKaryopherinsBiologyDNA-binding proteinCyclinsGene Expression Regulation FungalmedicineCell Growth and DevelopmentMolecular BiologyTranscription factorKaryopherinCell Nucleuschemistry.chemical_classificationCell CycleCell BiologyCell cycleSubcellular localizationCell biologyDNA-Binding ProteinsCell nucleusmedicine.anatomical_structurechemistryCytoplasmMutationCarrier ProteinsTranscription FactorsMolecular and Cellular Biology
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p16INK4A (CDKN2A) gene deletion is a frequent genetic event in synovial sarcomas.

2006

We assessed the frequency of genomic deletion of p16 INK4A (CDKN2A) in synovial sarcomas (SSs) and its possible association with immunoexpression of p16 and cyclin D1 and the Ki-67 proliferation index using dualcolor fluorescence in situ hybridization (FISH) on tissue microarray sections of 41 histologically and molecularly confirmed SSs. A heterozygous p16 INK4A gene deletion was identified in 28 (74%) of 38 cases, with 25 (89%) of them showing abnormal p16 protein expression (20 negative and 5 heterogeneous). Of 25 cases, 19 (76%) exhibiting increased cyclin D1 expression also demonstrated heterozygous p16 INK4A deletion. No significant association was observed between p16 INK4A deletion …

HeterozygoteProliferation indexTumor suppressor geneSoft Tissue NeoplasmsBiologySarcoma SynovialCyclin D1CDKN2ACyclin DCyclinsmedicineBiomarkers TumorHumansCDKN2A Gene DeletionCyclin-Dependent Kinase Inhibitor p16In Situ Hybridization FluorescenceCell Nucleusmedicine.diagnostic_testGeneral Medicinemedicine.diseaseImmunohistochemistrySynovial sarcomaKi-67 AntigenTumor progressionTissue Array AnalysisCancer researchGene DeletionFluorescence in situ hybridizationAmerican journal of clinical pathology
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High-throughput screening with the Eimeria tenella CDC2-related kinase2/cyclin complex EtCRK2/EtCYC3a

2012

The poultry disease coccidiosis, caused by infection with Eimeria spp. apicomplexan parasites, is responsible for enormous economic losses to the global poultry industry. The rapid increase of resistance to therapeutic agents, as well as the expense of vaccination with live attenuated vaccines, requires the development of new effective treatments for coccidiosis. Because of their key regulatory function in the eukaryotic cell cycle, cyclin-dependent kinases (CDKs) are prominent drug targets. The Eimeria tenella CDC2-related kinase 2 (EtCRK2) is a validated drug target that can be activated in vitro by the CDK activator XlRINGO (Xenopus laevis rapid inducer of G2/M progression in oocytes). B…

In silicoPlasmodium falciparumAntiprotozoal AgentsDrug Evaluation PreclinicalProtozoan ProteinsMicrobiologyEimeriaMicrobiology03 medical and health sciences0302 clinical medicineCyclin-dependent kinaseCyclinsparasitic diseasesCDC2 Protein KinaseAnimalsEnzyme Inhibitors030304 developmental biologyCyclin0303 health sciencesCyclin-dependent kinase 1biologyKinaseComputational BiologyPlasmodium falciparumCell cyclebiology.organism_classificationVirologyStandard3. Good healthHigh-Throughput Screening Assays030220 oncology & carcinogenesisCell and Molecular Biology of Microbesbiology.proteinEimeria tenellaMicrobiology
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Inhibition of Delayed-Type Hypersensitivity by Cucurbitacin R through the Curbing of Lymphocyte Proliferation and Cytokine Expression by Means of Nuc…

2009

Cucurbitacin R is known to exhibit an anti-inflammatory effect in different experimental models of inflammation. In this article, we outline the effect of cucurbitacin R on T lymphocyte proliferation, cytokine production, and nuclear factor activation, as well as its influence on various experimental models of delayed-type hypersensitivity (DTH) in mice. Cucurbitacin R reduced the proliferation of phytohemagglutinin A-stimulated human T lymphocytes (IC(50), 18 microM), modifying the cell cycle, as well as the production of cytokines [interleukin (IL)-2, IL-4, IL-10, and especially interferon-gamma] and the induction of the principal cyclins implicated in the cell cycle (A(1), B(1), D(2), an…

Interleukin 2medicine.medical_specialtyT-Lymphocytesmedicine.medical_treatmentAnti-Inflammatory AgentsLymphocyte proliferationBiologyPharmacologyJurkat cellsDrug HypersensitivityJurkat CellsMiceCyclinsInternal medicinemedicineAnimalsHumansHypersensitivity DelayedInterleukin 4Cell ProliferationPharmacologyNFATC Transcription FactorsFootCell CycleIntracellular Signaling Peptides and ProteinsOxazoloneEarNFATCell cycleTriterpenesInterleukin 10EndocrinologyCytokineCytokinesMolecular MedicineDinitrofluorobenzeneFemalemedicine.drugJournal of Pharmacology and Experimental Therapeutics
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Vitamin E deficiency induces liver nuclear factor-κB DNA-binding activity and changes in related genes

2005

The biological functions of vitamin E have been classically attributed to its property as a potent inhibitor of lipid peroxidation in cellular membranes. However, in 1991, Azzi's group first described that alpha-tocopherol inhibits smooth muscle cell proliferation in a protein kinase C (PKC)-dependent way, demonstrating a non-antioxidant cell signalling function for vitamin E. More recently, the capacity of alpha-tocopherol to modulate gene expression with the implication of different transcription factors, beyond its antioxidant properties, has also been established. This study was to determine the effect of vitamin E-deficiency on liver nuclear factor-kappa B (NF-kappaB) DNA-binding activ…

MaleVitaminChromatin ImmunoprecipitationGlutamate-Cysteine Ligasemedicine.medical_treatmentBlotting WesternBiologyBiochemistrychemistry.chemical_compoundCyclin D1CyclinsMalondialdehydemedicineAnimalsVitamin EVitamin E DeficiencyRNA MessengerRats WistarTranscription factorVitamin EBody WeightNF-kappa BPromoterDNAGeneral MedicineCell cycleGlutathioneRatsCell biologyGene Expression RegulationLiverBiochemistrychemistryVitamin E deficiencyChromatin immunoprecipitationFree Radical Research
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Progress in neuropathology of the neuronal ceroid lipofuscinoses.

1999

Abstract Since the last, 6th, International Congress on Neuronal Ceroid-Lipofuscinoses, neuropathological advances in neuronal ceroid lipofuscinoses (NCL) have been made in several areas: (1) In adult NCL (ANCL) lipopigments have now been repeatedly confirmed to contain subunit c of mitochondrial ATP synthase and even sphingolipid activators (saposins). ANCL lipopigments have also been confirmed in extracerebral tissues including skin, skeletal muscle, and spleen, but not yet lymphocytes (2). Among circulating blood cells not only B cells and subclasses of T lymphocytes, i.e., CD4 + , CD8 + , and CD56 cells, but also monocytes have been found to contain NCL lipopigments, indicating that thi…

Pathologymedicine.medical_specialtyEndocrinology Diabetes and MetabolismSpleenNeuropathologyBiologyBiochemistry03 medical and health sciences0302 clinical medicineEndocrinologyNeuronal Ceroid-LipofuscinosesPrecursor cellCyclinsGeneticsmedicineMacrophageHumansVitamin E DeficiencyKufs diseaseMolecular Biology030304 developmental biologySkinNeurons0303 health sciencesMicrogliaBrainmedicine.diseaseSphingolipid3. Good healthProton-Translocating ATPasesmedicine.anatomical_structureSpinal CordMicroglia030217 neurology & neurosurgeryCD8Molecular genetics and metabolism
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Functional distinction between Cln1p and Cln2p cyclins in the control of the Saccharomyces cerevisiae mitotic cycle.

2004

Abstract Cln1p and Cln2p are considered as equivalent cyclins on the basis of sequence homology, regulation, and functional studies. Here we describe a functional distinction between the Cln1p and Cln2p cyclins in the control of the G1/S transition. Inactivation of CLN2, but not of CLN1, leads to a larger-than-normal cell size, whereas overexpression of CLN2, but not of CLN1, results in smaller-than-normal cells. Furthermore, mild ectopic expression of CLN2, but not of CLN1, suppresses the lethality of swi4swi6 and cdc28 mutant strains. In the absence of Cln1p, the kinetics of budding, initiation of DNA replication, and activation of the Start-transcription program are not affected; by cont…

Saccharomyces cerevisiae ProteinsMutantSaccharomyces cerevisiaeBlotting WesternMitosisSaccharomyces cerevisiaeBiologyInvestigationsmedicine.disease_causeS PhaseCyclinsGeneticsmedicineImmunoprecipitationFluorescent Antibody Technique IndirectMitosisCyclinCell SizeGeneticsCyclin-dependent kinase 1MutationDNA replicationbiology.organism_classificationBlotting NorthernBridged Bicyclo Compounds HeterocyclicFlow CytometryMolecular biologyThiazolesMutationThiazolidinesEctopic expressionGenetics
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Karyopherin Msn5 is involved in a novel mechanism controlling the cellular level of cell cycle regulators Cln2 and Swi5

2019

ABSTRACT The yeast β-karyopherin Msn5 controls the SBF cell-cycle transcription factor, responsible for the periodic expression of CLN2 cyclin gene at G1/S, and the nuclear export of Cln2 protein. Here we show that Msn5 regulates Cln2 by an additional mechanism. Inactivation of Msn5 causes a severe reduction in the cellular content of Cln2. This occurs by a post-transcriptional mechanism, since CLN2 mRNA level is not importantly affected in asynchronous cultures. Cln2 stability is not significantly altered in msn5 cells and inactivation of Msn5 causes a reduction in protein level even when Cln2 is stabilized. Therefore, the reduced amount of Cln2 in msn5 cells is mainly due not to a higher …

Swi50301 basic medicineSaccharomyces cerevisiae ProteinsS. cerevisiaeCell Cycle ProteinsSaccharomyces cerevisiaeKaryopherinsCell cycleBiologyProtein degradationCyclin Gene03 medical and health sciences0302 clinical medicineCyclinsGene Expression Regulation FungalPolysomeProtein biosynthesisNuclear export signalMolecular BiologyTranscription factorCyclinMsn5 karyopherinCell BiologyCell cycleActinsCell biologyCln2 cyclin030104 developmental biologyMutagenesisPolyribosomesProtein Biosynthesis030220 oncology & carcinogenesisTranscription FactorsResearch PaperDevelopmental BiologyCell Cycle
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