Search results for "Cytokines"

showing 10 items of 845 documents

Circulating mir-21 and mir-146a are associated with increased cytokines and CD36 in Algerian obese male participants

2020

The microRNAs have come up as crucial mediators of energy balance and metabolic control. CD36 is potential biomarker of obesity and metabolic syndrome. This study investigates the concentration of miR-146a and miR-21 and CD 36 in blood samples of obese and healthy young participants. We assessed the association of mir-146a and mir-21 with inflammatory states in Algerian young participants.Our study included male obese, without co-morbidities (miR-146a and miR-21 concentrations were significantly decreased; however, CD36 expression was increased in obese subjects. Interestingly, miR-146a and miR-21 concentrations were negatively correlated to IL-6, TNF-α, and CD36 in obese participants.We de…

Malemedicine.medical_specialtyPhysiologyCD36Adipokine030209 endocrinology & metabolism03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicinemicroRNAHumansMedicineObesitybiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryGeneral Medicinemedicine.diseaseObesityMicroRNAsEndocrinologyAlgeria030220 oncology & carcinogenesisPotential biomarkersMetabolic control analysisbiology.proteinCytokinesMetabolic syndromebusinessBiomarkersArchives of Physiology and Biochemistry
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Molecular and functional interactions between tumor necrosis factor-alpha receptors and the glutamatergic system in the mouse hippocampus: Implicatio…

2009

Tumor necrosis factor (TNF)-alpha is a proinflammatory cytokine acting on two distinct receptor subtypes, namely p55 and p75 receptors. TNF-alpha p55 and p75 receptor knockout mice were previously shown to display a decreased or enhanced susceptibility to seizures, respectively, suggesting intrinsic modifications in neuronal excitability. We investigated whether alterations in glutamate system function occur in these naive knockout mice with perturbed cytokine signaling that could explain their different propensity to develop seizures. Using Western blot analysis of hippocampal homogenates, we found that p55(-/-) mice have decreased levels of membrane GluR3 and NR1 glutamate receptor subuni…

Malemedicine.medical_specialtyReceptors Kainic acidMicrodialysisAction PotentialsGlutamic AcidKainate receptorAMPA receptorIn Vitro TechniquesBiologyHippocampusReceptors N-Methyl-D-Aspartateelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundGlutamatergicReceptors Kainic AcidSeizuresInternal medicinemedicineAnimalsReceptors Tumor Necrosis Factor Type IIReceptors AMPAMice KnockoutNeuronsInflammationTumor Necrosis Factor-alphaGeneral NeuroscienceGlutamate receptorProtein SubunitsEndocrinologymedicine.anatomical_structureReceptors Glutamatenervous systemchemistryReceptors Tumor Necrosis Factor Type IMetabotropic glutamate receptorAstrocytesCytokinesNMDA receptorNBQXDisease SusceptibilityAstrocyte
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Visceral obesity and metabolic syndrome: two faces of the same medal?

2009

In this review, we have analyzed the role of visceral obesity in the occurrence of metabolic syndrome (MetS). MetS is a common metabolic disorder that has been related recently to the increasing prevalence of obesity. The disorder is defined in various ways, but in the near future a new definition(s) should be applicable worldwide. The pathophysiology has been largely attributed, in the past years, to insulin resistance, although several epidemiological and pathophysiological data now indicate visceral obesity as a main factor in the occurrence of all the components of MetS. In view of this, relationships among visceral obesity, free fatty acids, dyslipidemia and insulin resistance have bee…

Malemedicine.medical_specialtySettore MED/09 - Medicina InternaAdipokineFatty Acids NonesterifiedBioinformaticsInsulin resistanceAdipokinesInternal medicineEpidemiologyInternal MedicineMedicineHumansDyslipidemiasMetabolic SyndromeAdiponectinbusiness.industryMetabolic disordermedicine.diseaseObesityEndocrinologyObesity AbdominalEmergency MedicineFemaleAdiponectinMetabolic syndromeInsulin ResistancebusinessDyslipidemiaMetabolic syndrome - Visceral obesity - Adipocytokines - AdiponectinInternal and emergency medicine
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Immune-inflammatory markers and arterial stiffness indexes in subjects with acute ischemic stroke.

2010

Abstract No study has yet evaluated the relationship between arterial stiffness indexes and immuno-inflammatory pathway in patients with an acute cardiovascular or cerebrovascular event. The aim of our study was to evaluate in patients with acute ischemic stroke the relationship between immune-inflammatory markers and arterial stiffness indexes. Methods 107 subjects with acute ischemic stroke and 107 controls without stroke. We evaluated plasma levels of C-reactive protein (CRP), interleukin-1beta (IL-1β), tumor necrosis factor-alpha (TNF-α), interleukin-6 (IL-6) and interleukin-10 (IL-10), E-selectin, P-selectin, intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1…

Malemedicine.medical_specialtySettore MED/09 - Medicina InternaEmbolismTissue plasminogen activatorVon Willebrand factorIschemiaInternal medicinevon Willebrand FactormedicineHumanscardiovascular diseasesStrokePulse wave velocityAgedAged 80 and overInflammationbiologybusiness.industryCerebral infarctionMiddle Agedmedicine.diseaseAtherosclerosisischemic stroke arterial stiffnessSurgeryStrokeCarotid ArteriesImmune SystemAcute Diseasecardiovascular systemArterial stiffnessCardiologybiology.proteinCytokinesAortic stiffnessFemaleCardiology and Cardiovascular MedicinebusinessPlasminogen activatorcirculatory and respiratory physiologymedicine.drugAtherosclerosis
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Changes in natriuretic peptide and cytokine plasma levels in patients with heart failure, after treatment with high dose of furosemide plus hypertoni…

2011

BACKGROUND AND AIMS: Neurohormonal activation and inflammation characterizes heart failure, relates to outcome, and is a therapeutic target. The aim of this study was to evaluate the effects of high-dose furosemide plus small-volume hypertonic saline solutions (HSS) on natriuretic peptides and immuno-inflammatory marker levels and to analyze, after treatment, the response to acute saline loading. METHODS AND RESULTS: 120 patients with heart failure treated with high-dose furosemide+HSS (Furosemide/HSS group) were matched with: 30 subjects with heart failure treated with high-dose furosemide (furosemide group), 30 controls with asymptomatic left-ventricular dysfunction (ALVD) (asymptomatic g…

Malemedicine.medical_specialtySettore MED/09 - Medicina Internamedicine.drug_classEndocrinology Diabetes and Metabolismmedicine.medical_treatmentSodiumMedicine (miscellaneous)chemistry.chemical_elementSodium ChlorideHeart failurenatriuretic petide cytokine furosemideAsymptomaticGastroenterologyFurosemideInternal medicineNatriuretic Peptide BrainmedicineNatriuretic peptideHumansSalineAgedAged 80 and overHeart FailureSaline Solution HypertonicNutrition and Dieteticsbusiness.industryFurosemideMiddle Agedmedicine.diseaseHypertonic salineCytokineEndocrinologychemistryCase-Control StudiesHeart failureCytokinesFemalemedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugNutrition, Metabolism and Cardiovascular Diseases
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Influence of HMB Supplementation and Resistance Training on Cytokine Response to Resistance Exercise

2014

The purpose of this study was to determine the effects of a multinutritional supplement including amino acids, β-hydroxy-β-methylbutyrate (HMB), and carbohydrates on cytokine responses to resistance exercise and training.Seventeen healthy, college-aged men were randomly assigned to a Muscle Armor™ (MA; Abbott Nutrition, Columbus, OH) or placebo supplement group and 12 weeks of resistance training. An acute resistance exercise protocol was administered at 0, 6, and 12 weeks of training. Venous blood samples at pre-, immediately post-, and 30-minutes postexercise were analyzed via bead multiplex immunoassay for 17 cytokines.After 12 weeks of training, the MA group exhibited decreased interfer…

Malemedicine.medical_specialtyStrength trainingmedicine.medical_treatmentInterleukin-1betaMedicine (miscellaneous)Immune functionGranulocytePlaceboBody Mass IndexInterferon-gammaYoung AdultDouble-Blind MethodMuscle damageInternal medicineMyokineMyokinemedicineDietary CarbohydratesValeratesHumansMicronutrientsAmino Acidsta315Chemokine CCL4Immune function; Muscle damage; Myokine; Nutrition; Strength trainingChemokine CCL2NutritionNutrition and DieteticsInterleukin-13business.industryInterleukin-6MonocyteBody WeightInterleukin-8InterleukinResistance TrainingVenous bloodHealthy VolunteersInterleukin-10Cytokinemedicine.anatomical_structureEndocrinologyNutrition AssessmentImmunologyDietary SupplementsCytokinesStrength trainingbusiness
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Assessment of heart rate variability (HRV) in subjects with type 2 diabetes mellitus with and without diabetic foot: correlations with endothelial dy…

2021

Abstract Background Some studies have suggested that patients with diabetes and foot complications have worse cardiovascular and cerebrovascular risk profiles, higher degrees of endothelial dysfunction and arterial stiffness and a higher inflammatory background than patients with diabetes without diabetic foot complications. Patients with diabetes mellitus have an alteration in the sympathovagal balance as assessed by means of heart rate variability (HRV) analysis, which is also related to the presence of endothelial dysfunction. Other studies suggest a possible role of inflammation coexisting with the alteration in the sympathovagal balance in favor of the atherosclerotic process in a mixe…

Malemedicine.medical_specialtySympathetic Nervous SystemSettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismPopulationHyperemia030209 endocrinology & metabolism030204 cardiovascular system & hematologyGPI-Linked Proteins03 medical and health sciences0302 clinical medicineHeart RateLectinsHRV diabetic foot diabetesInternal medicineDiabetes mellitusmedicineDiseases of the circulatory (Cardiovascular) systemHumansOutpatient clinicHeart rate variabilityeducationReactive hyperemiaSerpinsAgedOriginal Investigationeducation.field_of_studybusiness.industryHeartVagus NerveMiddle Agedmedicine.diseaseDiabetic footDiabetic FootCross-Sectional StudiesDiabetes Mellitus Type 2RC666-701Case-Control StudiesArterial stiffnessCardiologyCytokinesFemaleEndothelium VascularSample collectionInflammation MediatorsCardiology and Cardiovascular MedicinebusinessBiomarkersCardiovascular Diabetology
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Characterization of a T-cell-derived mast cell costimulatory activity (MCA) that acts synergistically with interleukin 3 and interleukin 4 on the gro…

1990

The proliferation of mucosal mast cells (MMC) depends on the presence of interleukin 3 (IL 3) and can be further enhanced by interleukin 4 (IL 4). The supernatant of a TH2 cell clone (ST2/K.9) stimulated by concanavalin A was found to contain a factor, provisionally termed mast cell costimulatory activity (MCA), that substantially enhances the proliferation of MMC promoted by a combination of IL 3 and IL 4. In comparison to other lymphokines MCA is rather resistant to tryptic digestion but is very sensitive to pH values lower than 6.0 and to organic solvents. Chromatographic fractionation of MCA revealed that activity is associated with protein(s) or glycoprotein(s) of 35 to 40 kDa. Partial…

Malemedicine.medical_specialtyT-LymphocytesImmunologyBone Marrow CellsBiologyBiochemistryMast cell proliferationCell LineBiological FactorsMiceEpidermal growth factorInternal medicinemedicineImmunology and AllergyAnimalsMast CellsMolecular BiologyInterleukin 4Interleukin 3LymphokinesLymphokineDrug SynergismHematologyMast cellHematopoietic Stem CellsMolecular biologyClone Cellsmedicine.anatomical_structureEndocrinologyMice Inbred DBAChromatography GelCytokinesTumor necrosis factor alphaFemaleInterleukin-3Interleukin-4Leukemia inhibitory factorCell DivisionCytokine
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Insulin resistance as common molecular denominator linking obesity to Alzheimer’s disease

2015

Alzheimer’s disease (AD) is an aging-related multi-factorial disorder to which metabolic factors contribute at what has canonically been considered a centrally mediated process. Although the exact underlying mechanisms are still unknown, obesity is recognized as a risk factor for AD and the condition of insulin resistance seems to be the link between the two pathologies. Using mice with high fat diet (HFD) obesity we dissected the molecular mechanisms shared by the two disorders. Brains of HFD fed mice showed elevated levels of APP and Aβ 40 /Aβ 42 together with BACE, GSK3β and Tau proteins involved in APP processing and Aβ accumulation. Immunofluorescence, Thioflavin T staining experiments…

Malemedicine.medical_specialtyTime FactorsAdipokineAmyloidogenic ProteinsInflammationBiologyDiet High-Fatmedicine.disease_causeAdipokines Alzheimer’s disease gene expression inflammation insulin resistance mitochondrial dysfunction obesity.Settore BIO/09 - FisiologiaGlycogen Synthase Kinase 3MiceInsulin resistanceAlzheimer DiseaseInternal medicinemedicineAnimalsInsulinObesityReceptorGSK3BGlycogen Synthase Kinase 3 betaSettore BIO/16 - Anatomia UmanaNeurodegenerationBrainmedicine.diseaseReceptor InsulinMice Inbred C57BLDisease Models AnimalOxidative StressInsulin receptorEndocrinologyGene Expression RegulationNeurologyCase-Control Studiesbiology.proteinCytokinesNeurology (clinical)Amyloid Precursor Protein SecretasesInsulin Resistancemedicine.symptomOxidative stressSignal Transduction
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Spatiotemporal distribution of gp130 cytokines and their receptors after status epilepticus: comparison with neuronal degeneration and microglial act…

2003

Although numerous studies have demonstrated the neurotrophic capacity of gp130 cytokines, it remains unclear whether endogenously expressed cytokines actually function in a direct neuromodulatory manner. Therefore, using the lithium-pilocarpine status epilepticus model, we performed a detailed in situ hybridization time-course study of five gp130 cytokines (interleukin [IL]-6, leukemia inhibitory factor [LIF], IL-11, oncostatin-m [OSM], and ciliary neurotrophic factor), gp130, and the receptors of the cytokines we found to be induced (IL-6 receptor [IL-6R], LIF receptor [LIF-R], and IL-11 receptor [IL-11R]). Additionally, to further understand the regulation of these cytokines, we compared …

Malemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentBiologyHippocampal formationHippocampusRats Sprague-DawleyStatus EpilepticusInternal medicinemedicineAnimalsReceptors CytokineReceptorMicrogliaGeneral NeuroscienceDentate gyrusGlycoprotein 130Granule cellRatsmedicine.anatomical_structureEndocrinologyCytokineNerve DegenerationCytokinesMicrogliaLeukemia inhibitory factorSignal TransductionNeuroscience
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