Search results for "Cytoprotection"

showing 10 items of 49 documents

Doxycycline reduces nitric oxide production in guinea pig inner ears.

2011

Abstract Objective Gentamicin application is an important therapeutic option to control vertigo spells in Meniere's disease. However, even in the case of low-dose intratympanic application, gentamicin might contribute to a pathological NO-increase leading to cochlear damage and hearing impairment. The study was performed to evaluate the nitric oxide (NO) reducing capacity of doxycycline in the inner ear after NO-induction by gentamicin. Methods In a prospective animal study, a single dose of gentamicin (10 mg/kg body weight) was injected intratympanically into male guinea pigs (n = 48). The auditory brainstem responses (ABRs) were recorded prior to application and 3, 5 and 7 days afterwards…

Malemedicine.medical_specialtyLuminescenceArginineGuinea PigsPharmacologyOrgan cultureNitric OxideNitric oxideGuinea pigchemistry.chemical_compoundOrgan Culture Techniquesotorhinolaryngologic diseasesmedicineEvoked Potentials Auditory Brain StemAnimalsProspective StudiesOrgan of CortiCochleaDoxycyclineomega-N-Methylargininebusiness.industryGeneral MedicineImmunohistochemistrySurgeryUp-Regulationmedicine.anatomical_structureNG-Nitroarginine Methyl EsterOtorhinolaryngologychemistryOrgan of CortiCytoprotectionDoxycyclineEar InnerSurgeryGentamicinGentamicinsbusinessmedicine.drugAuris, nasus, larynx
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modifications peroxysomales associées à l'oxyapoptophagie induite par le 7-cétocholestérol et identification de lipides cytoprotecteurs

2020

Oxidative stress is often increased in several diseases such as age-related diseases (cardiovascular diseases, eye diseases (age-related macular degeneration (AMD) and cataracts), neurodegenerative diseases (Alzheimer's disease, multiple sclerosis), chronic inflammatory diseases (chronic inflammatory bowel disease (IBD)) as well as certain rare genetic diseases (Niemann Pick's disease, X-linked adrenoleukodystrophy (X-ALD)). Oxidative stress can oxidize various molecules, in particular the cholesterol present in lipid membranes, and lead to the formation of oxidized cholesterol derivatives: oxysterols. Some of them, such as 7-ketocholesterol (7KC), are toxic and may be the cause of a type o…

OxysterolOxiapoptophagyStress oxydantOxidative stressCytoprotection[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyOxystérolsPeroxisome[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyPeroxysome7-Ketocholesterol7-CétocholestérolOxyapoptophagie
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Apoptotic Cell Debris and Phosphatidylserine-Containing Lipid Vesicles Induce Apolipoprotein J (Clusterin) Gene Expression in Vital Fibroblasts

2001

The molecular events in cells undergoing programmed cell death (apoptosis) are well studied; however, the response of the surviving neighbor cells to local cell death is largely uncharacterized. Apolipoprotein J (clusterin) is an 80-kDa glycoprotein that has been implied in cytoprotection of the vital cells, presumably by assisting in the clearance of apoptotic vesicles and membrane remnants. Its mRNA is specifically up-regulated in the vital cells of apoptotic tissues. The molecular mechanisms, however, leading to this response are not known. We here show that exposure of vital fibroblasts to apoptotic vesicles, disrupted vital cells, and trypsin-treated membrane remnants induces apoJ mRNA…

Programmed cell deathEndocytic cycleGene ExpressionApoptosisPhosphatidylserinesCell Linechemistry.chemical_compoundCricetinaeAnimalsTrypsinGlycoproteinsClusterinbiologyVesicleCell BiologyPhosphatidylserinePhosphatidic acidFibroblastsLipid MetabolismMolecular biologyCytoprotectionRatsCell biologyClusterinchemistryApoptosisbiology.proteinMolecular ChaperonesExperimental Cell Research
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Prevention of 7-ketocholesterol-induced side effects by natural compounds

2018

Cholesterol oxidation products, also named oxysterols, can be formed either by cholesterol auto-oxidation, enzymatically or both. Among these oxysterols, 7-ketocholesterol (7KC) is mainly formed during radical attacks that take place on the carbon 7 of cholesterol. As increased levels of 7KC have been found in the tissues, plasma and/or cerebrospinal fluid of patients with major diseases, especially age-related diseases (cardiovascular diseases, eye diseases, neurodegenerative diseases), some cancers, and chronic inflammatory diseases, it is suspected that 7KC, could contribute to their development. Since 7KC, provided by the diet or endogenously formed, is not or little efficiently metabol…

Programmed cell deathOxysterol030309 nutrition & dieteticsTocopherolsInflammationPharmacologymedicine.disease_causeAntioxidantsIndustrial and Manufacturing Engineering03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologymedicineHumansNoncommunicable DiseasesKetocholesterolsInflammation0303 health sciencesCholesterolFatty AcidsPolyphenols04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceCytoprotectionOxidative StressMetabolic pathwaychemistryHepatic stellate cellmedicine.symptomOxidation-ReductionOxidative stressFood ScienceCritical Reviews in Food Science and Nutrition
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Lipids Nutrients in Parkinson and Alzheimer’s Diseases: Cell Death and Cytoprotection

2020

Neurodegenerative diseases, particularly Parkinson’s and Alzheimer’s, have common features: protein accumulation, cell death with mitochondrial involvement and oxidative stress. Patients are treated to cure the symptoms, but the treatments do not target the causes; so, the disease is not stopped. It is interesting to look at the side of nutrition which could help prevent the first signs of the disease or slow its progression in addition to existing therapeutic strategies. Lipids, whether in the form of vegetable or animal oils or in the form of fatty acids, could be incorporated into diets with the aim of preventing neurodegenerative diseases. These different lipids can inhibit the cytotoxi…

Programmed cell deathParkinson's diseaseInflammationReviewDiseasePharmacologyMitochondrionmedicine.disease_causelipids nutrientsCatalysislcsh:ChemistryInorganic ChemistrysynucleinFish OilsAlzheimer DiseaseHumansPlant OilsMedicinePhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopybusiness.industryFatty AcidsOrganic ChemistryapoptosisamyloidParkinson DiseaseNutrientsGeneral Medicinemedicine.diseaseLipidsCytoprotectionComputer Science ApplicationsmitochondriaOxidative Stresslcsh:Biology (General)lcsh:QD1-999CytoprotectionParkinson’s diseaseSynucleinTaumedicine.symptombusinessAlzheimer’s diseaseOxidative stressInternational Journal of Molecular Sciences
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Coffee Silverskin and Spent Coffee Suitable as Neuroprotectors against Cell Death by Beauvericin and α-Zearalenol: Evaluating Strategies of Treatment

2021

Coffee silverskin and spent coffee have been evaluated in a neuroblastoma cell line (SH-SY5Y cells) against beauvericin (BEA) and α-zearalenol (α-ZEL)-induced cytotoxicity with different strategies of treatment. First, the direct treatment of mycotoxins and coffee by-products extracts in SH-SY5Y cells was assayed. IC50 values for α-ZEL were 20.8 and 14.0 µM for 48 h and 72 h, respectively and, for BEA only at 72 h, it was 2.5 µM. Afterwards, the pre-treatment with spent coffee obtained by boiling water increased cell viability for α-ZEL at 24 h and 48 h from 10% to 16% and from 25% to 30%, respectively

Programmed cell deathTime Factors030309 nutrition & dieteticsHealth Toxicology and Mutagenesislcsh:MedicineToxicologyCoffeeArticleSH-SY5Y cells03 medical and health scienceschemistry.chemical_compoundInhibitory Concentration 500404 agricultural biotechnologyCell Line TumorDepsipeptidesIc50 valuesHumansViability assayFood scienceCytotoxicityMycotoxinNeuroblastoma cell linespent coffeeNeurons0303 health sciencesCell DeathDose-Response Relationship DrugPlant Extractslcsh:Rbeauvericin04 agricultural and veterinary sciences040401 food scienceCytoprotectionBeauvericinNeuroprotective AgentschemistryCytoprotectionSeedsZeranolα-zearalenolcoffee silverskinToxins
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CD40 ligation protects bronchial epithelium against oxidant-induced caspase-independent cell death.

2006

KEYWORDS CLASSIFICATION: 7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide;Antigens,CD40;Apoptosis;Bronchi;cytology;Caspases;Cell Cycle;Cell Death;Cell Line,Transformed;Cell Survival;Cell Transformation,Viral;Cytoprotection;drug effects;Epithelial Cells;Humans;Italy;mechanisms of carcinogenesis;metabolism;Oxidants;pharmacology;physiology;Research;Simian virus 40;toxicity;Transcription Factor AP-1. CD40 and its ligand regulate pleiotropic biological responses, including cell proliferation, differentiation, and apoptosis. In many inflammatory lung diseases, tissue damage by environmental or endogenous oxidants plays a major role in disease pathogenesis. As the epithelial barrier is a major t…

Pulmonary and Respiratory MedicineNF-BProgrammed cell deathCell SurvivalClinical Biochemistry78-Dihydro-78-dihydroxybenzo(a)pyrene 910-oxideApoptosisBronchiSimian virus 40Inhibitor of apoptosisAntigens CD40CD40HumansCD40 AntigensMolecular BiologyMitosisCaspaseActivator protein–1Cell Line Transformedoxidant stressbiologyCell DeathCell growthCell CycleEpithelial CellsCell BiologyCell cycleCell Transformation ViralOxidantsapoptosiCell biologyTranscription Factor AP-1activator protein–1ApoptosisCytoprotectionCaspasesbiology.proteinNF- BApoptosis-inducing factorOxidant stress
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Cytoprotection assessment against mycotoxins on HepG2 cells by extracts from Allium sativum L

2021

Abstract Cytoprotection effects of Allium sativum L garlic extract from a local garlic ecotype from Ferrara (Italy) on hepatocarcinoma cells, HepG2 cells, is presented in this study. This garlic type is known as Voghiera garlic and has been characterized as PDO (Protected designation of Origin) product. Voghiera garlic extract (VGE) was evaluated against beauvericin (BEA) and two zearalenone (ZEA) metabolites (α-zearalenol (α-ZEL) and β-zearalenol (β-ZEL))-induced cytotoxicity on HepG2 cells by the MTT (3–4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazolium bromide) assay, over 24 h and 48 h. Direct treatment, simultaneous treatment and pre-treatment strategies at the dilution 1:16–1:00 for VG…

Socio-culturaleToxicology03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyPDOHumansVoghieraFood scienceGarlicMycotoxinCytotoxicityHepG2 cellsZearalenoneBeauvericin; Garlic; HepG2 cells; PDO; Voghiera; α-ZEL; β-ZEL; Cytoprotection; Garlic; Hep G2 Cells; Humans; Mycotoxins030304 developmental biology0303 health sciencesHep G2 Cells04 agricultural and veterinary sciencesGeneral Medicineβ-ZELMycotoxinsBeauvericinAllium sativum040401 food scienceCytoprotectionBeauvericinDirect Treatmentchemistryα-ZELCytoprotectionHepg2 cellsFood ScienceFood and Chemical Toxicology
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Side-specific effects by cadmium exposure: Apical and basolateral treatment in a coculture model of the blood–air barrier

2010

Cadmium (Cd{sup 2+}) is a widespread environmental pollutant, which is associated with a wide variety of cytotoxic and metabolic effects. Recent studies showed that intoxication with the heavy metal most importantly targets the integrity of the epithelial barrier. In our study, the lung epithelial cell line, NCI H441, was cultured with the endothelial cell line, ISO-HAS-1, as a bilayer on a 24-well HTS-Transwell (registered) filter plate. This coculture model was exposed to various concentrations of CdCl{sub 2}. The transepithelial electrical resistance decreased on the apical side only after treatment with high Cd{sup 2+} concentrations after 48 h. By contrast, a breakdown of TER to less t…

Time FactorsCell SurvivalToxicologyTight JunctionsProinflammatory cytokineAlveolar cellsCadmium ChlorideCell Line TumorElectric ImpedancemedicineHumansViability assayRespiratory systemFragmentation (cell biology)Cell ShapePharmacologyBlood-Air BarrierDose-Response Relationship DrugChemistryCell PolarityEndothelial CellsEpithelial CellsBlood–air barrierAdherens JunctionsMolecular biologyCoculture TechniquesEndothelial stem cellmedicine.anatomical_structureCytoprotectionImmunologyCytokinesCalciumInflammation MediatorsIntracellularToxicology and Applied Pharmacology
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''Deferoxamine blocks death induced by glutathione depletion in PC 12 cells''

2013

Chouraqui, E. | Leon, A. | Repesse, Y. | Prigent-Tessier, A. | Bouhallab, S. | Bougle, D. | Marie, C. | Duval, D.; International audience; ''The purpose of the present work was to investigate the mechanisms by which glutathione depletion induced by treatment with buthionine sulfoximine (BSO) led within 24-30 h to PC 12 cells apoptosis. Our results showed that treatment by relatively low concentrations (10-30 mu M) of deferoxamine (DFx), a natural iron-specific chelator, almost completely shielded the cells from BSO-induced toxicity and that DFx still remained protective when added up to 9-12 h after BSO treatment. On the other hand, phosphopeptides derived from milk casein and known to carr…

Time FactorsIronApoptosisDeferoxaminePharmacologyIron Chelating AgentsToxicologymedicine.disease_causePC12 Cellschemistry.chemical_compoundOXIDATIVE-STRESSPARKINSONS-DISEASECaseinmedicineAnimalsHomeostasisButhionine sulfoximineButhionine SulfoximineNeuronsCELLULAR IRONDose-Response Relationship DrugbiologyChemistryGeneral NeuroscienceGlutathioneGlutathioneIRON CHELATORRatsDeferoxamineFerritinSYMPATHETIC NEURONSISCHEMIC-STROKEBiochemistryBRAIN IRONCELLULAR IRON''CytoprotectionApoptosisToxicity[ SCCO.NEUR ] Cognitive science/Neurosciencebiology.proteinSERUM DEPRIVATIONHEME OXYGENASE-1NEURODEGENERATIVE DISORDERSOxidative stress''OXIDATIVE-STRESSmedicine.drug
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