Search results for "Cytotoxic"

showing 10 items of 1673 documents

Interleukin 1 stimulates T lymphocytes to produce granulocyte-monocyte colony-stimulating factor.

1988

T lymphocytes are thought to cooperatively interact with monocytes to produce colony-stimulating factors (CSF). However, little is known about monocyte-mediated signals leading to CSF-secretion by T lymphocytes, although soluble monocyte products have been implicated. We have employed monoclonal antibody anti-T3B covalently coupled to CnBr-activated Sepharose 4B beads, to show that multimeric ligation of T cell antigen receptor leads to T cell receptiveness to interleukin 1 (IL-1), as indicated by T cell production of CSF, which induces growth of myeloid progenitor cells into neutrophil, eosinophil, and monocyte colonies. To investigate the molecular basis of these findings, total RNA was e…

T cellMonocyteCD28General MedicineBiologyMolecular biologyTCIRG1Interleukin 21medicine.anatomical_structuremedicineCytotoxic T cellIL-2 receptorResearch ArticleInterleukin 3Journal of Clinical Investigation
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The mitochondrial protein TCAIM regulates activation of T cells and thereby promotes tolerance induction of allogeneic transplants.

2013

Primary T cell activation and effector cell differentiation is required for rejection of allogeneic grafts in naive recipients. It has become evident, that mitochondria play an important role for T cell activation. Expression of several mitochondrial proteins such as TCAIM (T cell activation inhibitor, mitochondrial) is down-regulated upon T cell receptor triggering. Here we report that TCAIM inhibited spontaneous development of memory and effector T cells. CD4(+) T cells from Tcaim knock-in (KI) mice showed reduced activation, cytokine secretion and proliferation in vitro. Tcaim KI T cells tolerated allogeneic skin grafts upon transfer into Rag-1 KO mice. CD4(+) and CD8(+) T cells from the…

T cellT-LymphocytesBiologyLymphocyte ActivationT-Lymphocytes RegulatoryMitochondrial ProteinsInterleukin 21MicemedicineImmunology and AllergyCytotoxic T cellAnimalsTransplantation HomologousPharmacology (medical)IL-2 receptorAntigen-presenting cellCells CulturedHomeodomain ProteinsMice KnockoutTransplantationMice Inbred BALB CZAP70CD28Cell DifferentiationSkin TransplantationFlow CytometryCell biologyMitochondriaMice Inbred C57BLmedicine.anatomical_structureCytokinesTransplantation ToleranceReactive Oxygen SpeciesImmunologic MemoryCD8American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
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Tonic T cell signalling and T cell tolerance as opposite effects of self-recognition on dendritic cells.

2010

Naive T cells spend most of their time scanning the surface of dendritic cells (DCs), indicating that self-MHC/T cell receptor (TCR) interactions between these immune cells occur routinely in peripheral organs during the steady state. Peripheral self-MHC recognition on DCs drives seemingly opposing effects in the absence of inflammatory stimuli such as deletion of certain self-reactive T cells as well as maintenance of the T cell responsiveness to antigen, both of which shape the T cell repertoire and regulate T cell responses. Here we review recent data on the role of self-MHC recognition on steady-state DCs in the periphery and propose that interactions between T cells and steady-state DC…

T cellT-LymphocytesImmunologyAntigen presentation610 Medicine & healthchemical and pharmacologic phenomenaBiologyLymphocyte ActivationMajor Histocompatibility ComplexmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansIL-2 receptorAntigen-presenting cell2403 ImmunologyAntigen PresentationZAP70CD28Dendritic CellsNatural killer T cellCell biologymedicine.anatomical_structureSelf Tolerance10032 Clinic for Oncology and Hematology2723 Immunology and AllergySignal TransductionCurrent opinion in immunology
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Macrophages are dispensable for superantigen-mediated stimulation and anergy induction of peripheral T cells in vivo.

1994

Bacterial superantigens provoke T lymphocyte activation by cross-linking the variable part of the T cell receptor (TCR) beta-chain with MHC class II molecules on antigen-presenting cells. Although the molecular mechanisms of this interaction are well characterized, the in vivo accessory cell requirements for this stimulation of T lymphocytes by bacterial superantigens remain unknown. In the present study we have addressed the role of splenic macrophages in the activation of V beta 8+ peripheral T cells by staphylococcal enterotoxin B (SEB) in BALB/c mice. SEB-triggered clonal expansion and subsequent induction of unresponsiveness of both CD4+ and CD8+ T cells were investigated in naive anim…

T cellT-LymphocytesImmunologyAntigen-Presenting Cellschemical and pharmacologic phenomenaSpleenCell CommunicationEnterotoxinsMiceSuperantigenmedicineCytotoxic T cellAnimalsAntigen-presenting cellClonal AnergyMHC class IIMice Inbred BALB CSuperantigensbiologyMacrophagesT-cell receptorhemic and immune systemsFlow CytometryMolecular biologymedicine.anatomical_structureImmunologybiology.proteinInterleukin-2CD8Cell DivisionCellular immunology
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With a little help from my old T cell: Memory follicular T helper cells driving autoimmunity?

2014

It has long been known that the B cell repertoire includes cells that are capable of producing autoantibodies and that these cells can be found in humans and also in wild type strains of laboratory mice; however, normally, these B cells do not give rise to plasma cells, and thus do not fulfil their autoimmune potential. In this issue of the European Journal of Immunology, Nusser et al. [Eur. J. Immunol. 2014. 44: 2893–2902] dissect the mechanism by which these B cells are activated and autoantibodies are produced. The authors demonstrate that T cells, most likely antigen-specific, which accumulate with age or as a result of homeostatic proliferation, provide essential help to these autoreac…

T cellZAP70ImmunologyBiologyNatural killer T cellB-1 cellInterleukin 21medicine.anatomical_structureImmunologymedicineImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellEuropean Journal of Immunology
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The Role of the Major Histocompatibility Gene Complex in Murine Cytotoxic T Cell Responses

1980

Publisher Summary An interpretation of the function of major histocompatibility complex (MHC) in cytotoxic T cell responses requires making certain assumptions, which, because of the lack of experimental data, are often based on intuition. This chapter discusses some specific aspects of the role of MHC in cytotoxic T cell responses. It also summarizes the expression of T cell-mediated responses to alloantigens. It also focuses on H-2-restricted cytotoxic T lymphocytes (CTL) responses and discusses the influence of the MHC on cytotoxic T cell specificity and CTL responsiveness against foreign antigens. T cell responses to alloantigens mirror all the functional activities seen in H-2-restrict…

T cellchemical and pharmacologic phenomenaBiologyMHC restrictionMajor histocompatibility complexmedicine.anatomical_structureAntigenImmunologyMHC class Imedicinebiology.proteinCytotoxic T cellAntigen-presenting cellCD8
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Intrahepatic myeloid-cell aggregates enable local proliferation of CD8+T cells and successful immunotherapy against chronic viral liver infection

2013

Chronic infection is difficult to overcome because of exhaustion or depletion of cytotoxic effector CD8(+) T cells (cytotoxic T lymphoytes (CTLs)). Here we report that signaling via Toll-like receptors (TLRs) induced intrahepatic aggregates of myeloid cells that enabled the population expansion of CTLs (iMATEs: 'intrahepatic myeloid-cell aggregates for T cell population expansion') without causing immunopathology. In the liver, CTL proliferation was restricted to iMATEs that were composed of inflammatory monocyte-derived CD11b(+) cells. Signaling via tumor-necrosis factor (TNF) caused iMATE formation that facilitated costimulation dependent on the receptor OX40 for expansion of the CTL popu…

T cellmedicine.medical_treatmentImmunologyPopulationGreen Fluorescent ProteinsMice TransgenicBiologyCD8-Positive T-LymphocytesLymphocytic ChoriomeningitisMicemedicineImmunology and AllergyCytotoxic T cellAnimalsLymphocytic choriomeningitis virusMyeloid CellseducationCell ProliferationMice Knockouteducation.field_of_studyLiver infectionCD11b AntigenMicroscopy ConfocalLiver DiseasesImmunotherapyReceptors OX40Flow CytometryMice Inbred C57BLCTL*Chronic infectionmedicine.anatomical_structureAnimals NewbornLiverToll-Like Receptor 9ImmunologyChronic DiseaseHost-Pathogen InteractionsImmunotherapyCD8Signal TransductionT-Lymphocytes CytotoxicNature Immunology
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An innate cell-mediated, murine ulcerative colitis-like syndrome in the absence of nuclear factor of activated T cells.

2004

Abstract Background & Aims: Nuclear factor of activated T cells transcription factors plays a central role in immunity by regulating the expression of multiple cytokines and other regulatory molecules, many of which have been heavily implicated in the pathogenesis of inflammatory bowel disease. However, few studies have directly investigated the nuclear factor of activated T cells proteins in inflammatory bowel disease. We describe here a specific role for nuclear factor of activated T cells c2 in the pathogenesis of murine inflammatory bowel disease. Methods: Mice deficient for nuclear factor of activated T cells c2, recombinase activating gene-2, or both and transgenic or nontransgenic fo…

T-LymphocytesBiologyInterleukin 21MicemedicineImmune ToleranceCytotoxic T cellAnimalsIL-2 receptorB-LymphocytesImmunity CellularMice Inbred BALB CHepatologyNFATC Transcription FactorsZAP70Innate lymphoid cellGastroenterologyNuclear ProteinsT helper cellRectal ProlapseNatural killer T cellAcquired immune systemMice Mutant StrainsDNA-Binding ProteinsMice Inbred C57BLmedicine.anatomical_structureImmunologyCancer researchColitis UlcerativeTranscription FactorsGastroenterology
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Induction of apoptosis in a leukemia cell line by triterpene saponins from Albizia adianthifolia

2004

Abstract Triterpenoid saponins, which are present in plants and some marine animals, exert various important pharmacological effects. The present study examines the effects of adianthifoliosides A, B, and D (AdA, AdB, and AdD) together with two prosapogenins (Pro1 and Pro2) obtained from Albizia adianthifolia (Mimosaceae) on human leukemia T-cells (Jurkat cells) and on splenocytes. AdA, AdB, and AdD were found to exhibit a cytotoxic effect on Jurkat cells, whereas the prosapogenins were found to exert a lymphoproliferative effect on this cell type. Furthermore, all tested compounds were found to exert a synergistic lymphoproliferative activity with concanavalin A (ConA) on splenocytes. The …

T-LymphocytesClinical BiochemistryPharmaceutical ScienceAlbizziaApoptosisHemolysisBiochemistryJurkat cellsCell LineJurkat Cellschemistry.chemical_compoundDrug DiscoveryTumor Cells CulturedHumansCytotoxic T cellPropidium iodideOleanolic AcidCytotoxicityMolecular BiologyPlants MedicinalDose-Response Relationship DrugbiologyChemistryOrganic ChemistryBiological activitySaponinsbiology.organism_classificationAntineoplastic Agents PhytogenicTriterpenesBiochemistryConcanavalin ACell culturebiology.proteinMolecular MedicineAlbizia adianthifoliaBioorganic & Medicinal Chemistry
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Synergistic activation of dendritic cells by combined Toll-like receptor ligation induces superior CTL responses in vivo.

2006

Toll-like receptors (TLRs) are able to interact with pathogen-derived products and their signals induce the coordinated activation of innate and adaptive immune mechanisms. Dendritic cells (DCs) play a central role in these events. As the different TLRs are able to trigger MyD88/TRIF-dependent and -independent signaling pathways, we wondered if the simultaneous activation of these signaling cascades would synergize with respect to DC activation and induce superior cytotoxic T-lymphocyte (CTL) activity in vivo. We observed that indeed the combined activation of MyD88-dependent and -independent signaling induced by TLR7 and TLR3 ligands provoked a more rapid and more sustained bone marrow–der…

T-LymphocytesImmunologyBone Marrow CellsBiologyLigandsBiochemistryT-Lymphocytes RegulatoryMiceCytotoxic T cellAnimalsAntigen-presenting cellAdaptor Proteins Signal TransducingCD86Toll-like receptorCD40Membrane GlycoproteinsToll-Like ReceptorsImmunityhemic and immune systemsCell BiologyHematologyDendritic cellDendritic CellsAcquired immune systemCell biologyToll-Like Receptor 3Mice Inbred C57BLCTL*Toll-Like Receptor 7ImmunologyMyeloid Differentiation Factor 88biology.proteinSignal TransductionT-Lymphocytes CytotoxicBlood
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