Search results for "DASE"

showing 10 items of 1891 documents

The Activation Pattern of the Antioxidant Enzymes in the Right Ventricle of Rat in Response to Pressure Overload is of Heart Failure Type

2003

In the left ventricle subjected to pressure overload activity, the antioxidant enzymes increased at the hyperfunctional stage. During the transition to heart failure, these enzymes are down-regulated, oxidative stress increases, and apoptosis progresses. Maladaptative activation of the antioxidant enzymes at an early stage may contribute to the intrinsic vulnerability of right ventricle to pressure overload. The authors studied changes in expression and activity of the enzymes manganese and copper-zinc superoxide dismutases, glutathione peroxidase, and catalase in the right ventricle of rat following induction of pulmonary hypertension by injection of monocrotaline. Increase in the manganes…

medicine.medical_specialtyAntioxidantHeart Ventriclesmedicine.medical_treatmentmedicine.disease_causeAntioxidantsSuperoxide dismutaseInternal medicinePressuremedicineAnimalsRats WistarHeart Failurechemistry.chemical_classificationPressure overloadGlutathione PeroxidaseBase SequenceHypertrophy Right VentricularbiologySequence Analysis RNASuperoxide Dismutasebusiness.industryGlutathione peroxidaseCatalasemedicine.diseasePulmonary hypertensionRatsOxidative Stressmedicine.anatomical_structureEndocrinologychemistryVentricleHeart failureModels Animalbiology.proteinCardiologyReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessOxidative stressHeart Disease
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Janus-faced role of endothelial NO synthase in vascular disease: uncoupling of oxygen reduction from NO synthesis and its pharmacological reversal

2006

Endothelial NO synthase (eNOS) is the predominant enzyme responsible for vascular NO synthesis. A functional eNOS transfers electrons from NADPH to its heme center, where L-arginine is oxidized to L-citrulline and NO. Common conditions predisposing to atherosclerosis, such as hypertension, hypercholesterolemia, diabetes mellitus and smoking, are associated with enhanced production of reactive oxygen species (ROS) and reduced amounts of bioactive NO in the vessel wall. NADPH oxidases represent major sources of ROS in cardiovascular pathophysiology. NADPH oxidase-derived superoxide avidly interacts with eNOS-derived NO to form peroxynitrite (ONOO(-)), which oxidizes the essential NOS cofactor…

medicine.medical_specialtyAntioxidantNitric Oxide Synthase Type IIImedicine.medical_treatmentClinical BiochemistryNitric Oxidemedicine.disease_causeBiochemistrychemistry.chemical_compoundEnosInternal medicinemedicineAnimalsHumansVascular DiseasesEnzyme InhibitorsMolecular BiologyHemeJanus Kinaseschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxidebiology.organism_classificationOxygenEndocrinologychemistrybiology.proteinPeroxynitriteOxidative stressBiological Chemistry
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Effect of a Diet Supplemented with alpha-Tocopherol and beta-Carotene on ATP and Antioxidant Levels after Hepatic Ischemia-Reperfusion.

2008

Ischemia-reperfusion injury associated with liver transplantation remains a serious complication in clinical practice. In the present study the effect of intake of alpha-tocopherol or beta-carotene to limit liver injury by oxidative stress in ischemia and reperfusion was explored. Wistar rats were fed with diets enriched with alpha-tocopherol (20 mg/day) or beta-carotene (3 mg/day) for 21 days. After 21 days, their livers were subjected to 15 and 30 min of ischemia and afterwards were reperfused for 60 min. The recovery of levels of ATP during reperfusion was better in the group of rats whose diets were supplemented with alpha-tocopherol or beta-carotene than in the group control. The suppl…

medicine.medical_specialtyAntioxidantmedicine.medical_treatmentClinical BiochemistryIschemiaMedicine (miscellaneous)medicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundInternal medicineβ-carotenemedicineGSHoxidative stresschemistry.chemical_classificationLiver injuryNutrition and Dieteticsα-tocopherolbiologybusiness.industryGlutathione peroxidaseGlutathionemedicine.diseaseischemia/reperfusionEndocrinologychemistryBiochemistrybiology.proteinOriginal ArticlebusinessReperfusion injuryOxidative stressJournal of clinical biochemistry and nutrition
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Redox modulation of mitochondriogenesis in exercise. Does antioxidant supplementation blunt the benefits of exercise training?

2015

Physical exercise increases the cellular production of reactive oxygen species (ROS) in muscle, liver, and other organs. This is unlikely due to increased mitochondrial production but rather to extramitochondrial sources such as NADPH oxidase or xanthine oxidase. We have reported a xanthine oxidase-mediated increase in ROS production in many experimental models from isolated cells to humans. Originally, ROS were considered as detrimental and thus as a likely cause of cell damage associated with exhaustion. In the past decade, evidence showing that ROS act as signals has been gathered and thus the idea that antioxidant supplementation in exercise is always recommendable has proved incorrect.…

medicine.medical_specialtyAntioxidantmedicine.medical_treatmentPhysical exerciseBiochemistryAntioxidantsSuperoxide dismutasechemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineAnimalsHumansMuscle SkeletalXanthine oxidaseExercisechemistry.chemical_classificationReactive oxygen speciesOrganelle BiogenesisNADPH oxidasebiologyMuscle adaptationGlutathione peroxidaseAdaptation PhysiologicalMitochondria MuscleOxidative StressEndocrinologychemistryDietary Supplementsbiology.proteinOxidation-ReductionFree Radical Biology and Medicine
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Oxidative stress in vascular disease and its pharmacological prevention

2013

Cardiovascular risk factors lead to enhanced production of reactive oxygen species (ROS) generated by NADPH oxidase, xanthine oxidase (XO), the mitochondrial electron-transport chain (ETC), and dysfunctional endothelial nitric oxide synthase (eNOS). When the capacity of antioxidant defense systems [e.g., superoxide dismutase (SOD), catalase, glutathione peroxidase (GPx), heme oxygenase (HO), paraoxonase (PON)] is exceeded, this results in oxidative stress, which can promote atherogenesis. Therefore, pharmacological means to prevent oxidative stress are of major therapeutic interest. Some established drugs and novel therapeutic approaches can prevent oxidative stress and, presumably, vascula…

medicine.medical_specialtyAntioxidantmedicine.medical_treatmentToxicologymedicine.disease_causeAntioxidantsSuperoxide dismutasechemistry.chemical_compoundInternal medicinemedicineAnimalsHumansVascular DiseasesXanthine oxidasePharmacologychemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologyGlutathione peroxidaseHeme oxygenaseOxidative StressEndocrinologychemistrybiology.proteinOxidative stressTrends in Pharmacological Sciences
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Future perspectives of the pharmacological management of diabetic dyslipidemia

2019

Introduction: Diabetic dyslipidemia is frequent among patients with type 2 diabetes mellitus (T2DM) and is characterized by an increase in triglycerides (TGs), low-density lipoprotein cholesterol (LDL-C), and small-dense (atherogenic) particles, and by a decrease in low high-density lipoprotein cholesterol (HDL-C) and apolipoprotein (Apo) A1 that are strongly related to insulin resistance. The increased flux of free fatty acids from adipose tissue to the liver aggravates hepatic insulin resistance and promotes all of aspects of the dyslipidemic state. Areas covered: Statins are the first-line agents for treatment while other lipid-lowering drugs (ezetimibe, fibrate and proprotein convertase…

medicine.medical_specialtyApolipoprotein Bmedicine.drug_classglucagon like peptide-1 receptor agonist (GLP-1RA)Fibrate030226 pharmacology & pharmacystatins03 medical and health sciences0302 clinical medicineInsulin resistanceEzetimibeInternal medicinemedicineHumansHypoglycemic AgentsPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsOmega 3 fatty acidDyslipidemiasHypolipidemic Agentsfibratebiologybusiness.industrydyslipidemianutritional and metabolic diseasesType 2 Diabetes MellitusGeneral MedicineLipidmedicine.diseasesodium/glucose cotransporter 2 inhibitors (SGLT-2is)LipidsEndocrinologyDiabetes Mellitus Type 2Cardiovascular Diseases030220 oncology & carcinogenesisDipeptidyl peptidase-4 inhibitors (DPP-4is)Dietary Supplementsbiology.proteinKexinlipids (amino acids peptides and proteins)Hydroxymethylglutaryl-CoA Reductase InhibitorHydroxymethylglutaryl-CoA Reductase InhibitorsInsulin ResistancebusinessDyslipidemiamedicine.drugezetimibeproprotein convertase subtilisin/kexin type 9 (PCSK9)
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Activities of angiotensin-converting enzymes ACE1 and ACE2 and inhibition by bioactive peptides in porcine ocular tissues.

2009

An active local renin-angiotensin system (RAS) has recently been found in the human eye. The aim of the present study was to compare the activities of central RAS enzymes (ACE1 and 2) in porcine ocular tissues, morphologically and physiologically close to the human eye. In addition, the effects of three ACE-inhibitory tripeptides on these enzymes were evaluated.Enucleated fresh porcine eyes were used. Activities of ACE1 and ACE2 and their inhibition by bioactive tripeptides (Ile-Pro-Pro, Val-Pro-Pro, Leu-Pro-Pro) as well as by a standard ACE-inhibitor captopril were assayed in the vitreous body, the retina and the ciliary body using fluorometric detection methods.Activity of ACE1 as well as…

medicine.medical_specialtyCaptoprilgenetic structuresSwinePeptideAngiotensin-Converting Enzyme InhibitorsTripeptideBiologyIn Vitro TechniquesPeptidyl-Dipeptidase ARetina03 medical and health sciences0302 clinical medicineCiliary bodyInternal medicineRenin–angiotensin systemmedicineAnimalsPharmacology (medical)030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesOligopeptideRetinaCiliary Bodyeye diseases3. Good healthVitreous BodyOphthalmologyEnzymeEndocrinologymedicine.anatomical_structurechemistryAngiotensin-converting enzyme 2030221 ophthalmology & optometrysense organsAngiotensin-Converting Enzyme 2OligopeptidesJournal of ocular pharmacology and therapeutics : the official journal of the Association for Ocular Pharmacology and Therapeutics
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Chelation of synaptic zinc induces overexcitation in the hilar mossy cells of the rat hippocampus.

2004

Complete removal of synaptic zinc by the chelator dietyldithiocarbamate (DEDTC; 500 mg/kg i.p.) in rat was followed by convulsive behaviour including wet dog shakes alternating immobility. Histological analysis 1 day after DEDTC administration detected expression of heat shock protein in the hippocampus restricted to hilar cells. These cells colocalize the marker for neurons and the glutamate receptor GluR2/3 showing that they are excitatory neurons. Additionally, they projected to the contralateral dentate gyrus. Therefore, they correspond to hilar mossy cells. These data show that the synaptic zinc has a role in normal hippocampus avoiding overexcitation, that would impair functionality e…

medicine.medical_specialtyCentral nervous systemPresynaptic TerminalsWheat Germ Agglutinin-Horseradish Peroxidase ConjugateHippocampusAction PotentialsHSP72 Heat-Shock Proteinsc-FosHippocampusSynaptic TransmissionSeizuresInternal medicineNeural PathwaysmedicineAnimalsReceptors AMPAHeat-Shock ProteinsChelating AgentsbiologyGeneral NeuroscienceDentate gyrusGlutamate receptorColocalizationImmunohistochemistryRatsZincEndocrinologymedicine.anatomical_structurenervous systemDentate GyrusMossy Fibers Hippocampalbiology.proteinExcitatory postsynaptic potentialDitiocarbImmediate early geneProto-Oncogene Proteins c-fosNeuroscience letters
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A case of combined Farber and Sandhoff disease

1989

We describe a patient with the biochemically established combination of Farber and Sandhoff disease. A 6-month-old girl of consanguineous Turkish parents presented with hoarseness, stridor, scattered skin nodules, painful swelling of hand joints and ankles, and cherry-red macular spots. Until the age of 2 years her motor and physical condition deteriorated distinctly, however her mental state remained unchanged. A biopsied skin nodule disclosed lysosomal inclusions within storage cells that were typical of Farber disease (curved tubular structures). However, other inclusions (e.g. zebra bodies) were also found. Biochemical findings included ceramide accumulation in skin nodules and cultured…

medicine.medical_specialtyCeramidePathologyBiopsySandhoff diseaseCeramidesLipid Metabolism Inborn Errorschemistry.chemical_compoundInternal medicinemedicineHumansHexosaminidaseSkinSphingolipidsFarber diseasebusiness.industryInfantSandhoff DiseaseCeramidasemedicine.diseaseSkin NoduleEndocrinologyCeramidase activitychemistryPediatrics Perinatology and Child HealthFemalebusinessSphingomyelinEuropean Journal of Pediatrics
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Use of Novel Antidiabetic Agents in Patients with Type 2 Diabetes and COVID-19: A Critical Review

2021

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes coronavirus disease 2019 (COVID-19). The latter is a pandemic that has the potential of developing into a severe illness manifesting as systemic inflammatory response syndrome, acute respiratory distress syndrome, multi-organ involvement and shock. In addition, advanced age and male sex and certain underlying health conditions, like type 2 diabetes mellitus (T2DM), predispose to a higher risk of greater COVID-19 severity and mortality. This calls for an urgent identification of antidiabetic agents associated with more favourable COVID-19 outcomes among patients with T2DM, as well as recognition of their potential underlying…

medicine.medical_specialtyCoronavirus disease 2019 (COVID-19)1 receptor agonists Sodium-glucose co-transporter&nbspEndocrinology Diabetes and MetabolismSevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)COVID-19 Dipeptidyl peptidase&nbspReviewType 2 diabetesSodium-glucose co-transporter 2 inhibitorsType 2 diabetesGlucagon-like peptide 1 receptor agonistsDiabetes mellitusPandemicInternal Medicinemedicine2 diabetesIntensive care medicineAntidiabetic agents4 inhibitors Glucagon-like peptide&nbspbusiness.industryCOVID-19medicine.diseaseSystemic inflammatory response syndromeShock (circulatory)Dipeptidyl peptidase 4 inhibitorsmedicine.symptombusiness2 inhibitors Type&nbspDiabetes Therapy
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