Search results for "DEA"

showing 10 items of 4491 documents

Coexposure to sulfamethoxazole and cadmium impairs development and attenuates transcriptional response in sea urchin embryo

2017

Abstract Among sulfonamides, sulfamethoxazole represents one of the most widely employed. A considerable amount of sulfamethoxazole is introduced into the marine environment after utilization in aquaculture. The cytotoxicity of sulfamethoxazole relies mainly on arylhydroxylamine metabolites and it is associated with the production of reactive oxygen species. Cadmium represents a metal largely employed in several anthropic activities and it is toxic for all living organisms even at low concentrations. Since it is not degraded, cadmium irreversibly accumulates into cells. In order to understand the mechanisms of response to changes in the chemical environment, we investigated by light microsc…

0301 basic medicineEmbryo NonmammalianSulfamethoxazoleHealth Toxicology and Mutagenesis; Defense mechanisms;Gene ExpressionAquaculture010501 environmental sciencesmedicine.disease_cause01 natural sciencesCoexposureToxicologychemistry.chemical_compoundCadmium ChlorideGene expression profile; Stress responsechemistry.chemical_classificationCadmiumeducation.field_of_studyEchinodermSulfamethoxazoleChemistry (all)General MedicinePollutionCadmiumDefense mechanismEchinodermsmedicine.drugProgrammed cell deathEnvironmental EngineeringPopulationchemistry.chemical_elementSettore BIO/11 - Biologia MolecolareCadmium chlorideBiologyMicrobiologyCoexposure; Defense mechanisms; Echinoderms; Gene expression profile; Stress response; Chemistry (all); Environmental Chemistry03 medical and health sciencesDetoxificationmedicineAnimalsEnvironmental Chemistryeducation0105 earth and related environmental sciencesReactive oxygen speciesStress responsePublic Health Environmental and Occupational HealthGeneral ChemistryGene expression profile030104 developmental biologychemistrySea UrchinsWater Pollutants ChemicalOxidative stress
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Survey on the presence of A. simplex s.s. and A. pegreffii hybrid forms in Central-Western Mediterranean Sea

2016

Abstract In this study, 1029 fish and cephalopod samples came from Central-Western Mediterranean (FAO 37.1.1 and FAO 37.1.3) were analysed for Anisakidae larvae research with the aim to identify possible hybridisations between Anisakis pegreffii and Anisakis simplex s.s. species. A total of 1765 larvae were detected, with prevalence values between 8.1% and 100%. The morphologic analysis revealed characters attributable to morphotype I of Anisakis in 98.5% of the examined larvae, while 1.5% belonged to the morphotype II. PCR-based Restriction Fragment Length Polymorphism (PCR–RFLP) analysis of the entire ITS region (ITS1, 5.8S and ITS2) of nuclear ribosomal DNA (rDNA) was performed with Hinf…

0301 basic medicineFish DiseaseSympatric areaAnisakidaeZoologyAnisakisPerciformesPCR-RFLPFish Diseases03 medical and health sciencesAscaridoideaDNA Ribosomal SpacerGenotypeMediterranean SeaAnimalsPerciformeSequence analysiRibosomal DNAGeneticsBase SequencebiologyAnimalChimeraAnisakis simplexSouth Mediterranean Sea030108 mycology & parasitologybiology.organism_classificationHorse mackerelPerciformesAnisakidae030104 developmental biologyInfectious DiseasesCephalopodaHybrid genotypeLarvaParasitologyRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthParasitology International
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The Gemmellaro Collection: first record of an anomuran from the Tithonian of Sicily, Italy

2017

A recent field trip to Sicily and an examination of decapod crustacean collections at the Museo Geologico G.G. Gemmellaro in the centre of Palermo, Sicily (Italy), has demonstrated that most of the anomuran and brachyuran material described by Gemmellaro (Gemmellaro GG. 1869. Studi paleontologici sulla fauna del Calcare àTerebratula janitordel nord di Sicilia. Palermo: Lao, vol. 1, pp. 11–18) from the Tithonian of that island is still present. Interestingly, a single specimen in this lot was never mentioned, described or illustrated by that author. The species to which this particular individual is here shown to belong,Gastrosacus tuberosus, was first described and named 26 years later, in …

0301 basic medicineGalathea010506 paleontologyFaunaMediterranean01 natural sciences03 medical and health sciencesSingle specimenUpper Jurassic0105 earth and related environmental sciencesbiologylcsh:QE1-996.5HolotypeGeologySettore GEO/01 - Paleontologia E Paleoecologiabiology.organism_classificationArchaeologyGalatheoideaAnomura / Galatheoidea / Upper Jurassic / Mediterranean / palaeobiogeographylanguage.human_languagelcsh:Geologypalaeobiogeographylanguage030101 anatomy & morphologyAnomuraTerebratulaSicilianGeology
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ZNF518B gene up-regulation promotes dissemination of tumour cells and is governed by epigenetic mechanisms in colorectal cancer

2019

AbstractMost of colorectal cancer CRC-related death is due to metastasis and the finding of markers for prognosis of invasiveness, constitutes an appealing challenge. Here, after analysing cDNA array containing 43 tumour and 5 normal mucosa samples, we report that the expression of the ZNF518B gene as a whole and that of its two major splicing isoforms are significantly increased in tumours. The canonical isoform was also up-regulated in a patients’ cohort containing 70 tumour and 69 adjacent tissue samples. The effects of silencing ZNF518B on the phenotype of CRC cell lines were then studied. The gene does not affect cell proliferation, but plays a significant role in cell migration and in…

0301 basic medicineGene isoformEpithelial-Mesenchymal TransitionCelllcsh:MedicineBiologyArticleHistone DeacetylasesEpigenesis GeneticHistones03 medical and health sciences0302 clinical medicineCell MovementCell Line TumorGene expressionmedicineGene silencingHumansProtein IsoformsEpigeneticsNeoplasm Metastasislcsh:ScienceGeneCell ProliferationNeoplasm StagingMultidisciplinaryGene Expression Profilinglcsh:RPrognosisColorectal cancer3. Good healthDNA-Binding ProteinsGene Expression Regulation Neoplastic030104 developmental biologymedicine.anatomical_structureHistoneGene Knockdown TechniquesCancer researchbiology.proteinH3K4me3lcsh:QEpigeneticsColorectal Neoplasms030217 neurology & neurosurgeryScientific Reports
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Apoptosis induced by a HIPK2 full-length-specific siRNA is due to off-target effects rather than prevalence of HIPK2-Δe8 isoform

2017

Small interfering RNAs (siRNAs) are widely used to study gene function and extensively exploited for their potential therapeutic applications. HIPK2 is an evolutionary conserved kinase that binds and phosphorylates several proteins directly or indirectly related to apoptosis. Recently, an alternatively spliced isoform skipping 81 nucleotides of exon 8 (Hipk2-Δe8) has been described. Selective depletion of Hipk2 full-length (Hipk2-FL) with a specific siRNA that spares the Hipk2-Δe8 isoform has been shown to strongly induce apoptosis, suggesting an unpredicted dominant-negative effect of Hipk2-FL over the Δe8 isoform. From this observation, we sought to take advantage and assessed the therape…

0301 basic medicineGene isoformMaleProgrammed cell deathSmall interfering RNACell SurvivalBlotting WesternMice Nudecolorectal cancerApoptosisHIPK2BiologyProtein Serine-Threonine KinasesGene Expression Regulation Enzymologic03 medical and health sciencesExonRNA interferenceCell Line TumorAnimalsHumansViability assayoff-target effectCell Line TransformedSettore MED/04 - Patologia GeneraleKinaseReverse Transcriptase Polymerase Chain ReactionAlternative splicingalternative splicing isoformoff-target effectsExonsHCT116 CellsMolecular biologyXenograft Model Antitumor AssaysCell biologyGene Expression Regulation NeoplasticIsoenzymesAlternative Splicing030104 developmental biologyRNAi TherapeuticsOncologyalternative splicing isoformsNeoplastic Stem CellsRNA InterferenceHIPK2; alternative splicing isoforms; colorectal cancer; off-target effects; siRNA therapeutic applicationsiRNA therapeutic applicationCarrier ProteinsColorectal NeoplasmsGene DeletionResearch Paper
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Histone Deacetylase Inhibitors from Marine Invertebrates

2020

Simple Summary Histone deacetylases (HDACs) are enzymes that control gene expression and are involved in the onset of serious human pathologies, including cancer; hence, their inhibitors (HDACis) have received increased attention in recent years. It is known that marine invertebrates produce significant amounts of molecules showing active pharmacological properties and an extensive spectrum of biomedical applications. This review is focused on the description of the molecular, biochemical, and, where available, physiological aspects of marine invertebrate-derived compounds that possess HDACi properties, taking into consideration their possible utilization as treatment agents against differe…

0301 basic medicineGene isoformbiomedical applicationsmarine invertebratesSettore BIO/05 - ZoologiaComputational biologyReviewhistone deacetylase inhibitorsGeneral Biochemistry Genetics and Molecular BiologyChromatin remodelinganticancer compound03 medical and health sciencesCnidaria0302 clinical medicineNon-histone proteinmarine invertebrateGene expressionEpigeneticsSettore BIO/06 - Anatomia Comparata E Citologiahistone deacetylase inhibitorlcsh:QH301-705.5General Immunology and MicrobiologybiologyMarine invertebratesanticancer compoundsPorifera030104 developmental biologyHistonelcsh:Biology (General)030220 oncology & carcinogenesisbiology.proteinbiomedical applicationHistone deacetylaseGeneral Agricultural and Biological SciencesEchinodermataBiology
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Production Conditions Affect the In Vitro Anti-Tumoral Effects of a High Concentration Multi-Strain Probiotic Preparation.

2016

A careful selection of the probiotic agent, standardization of the dose and detailed characterization of the beneficial effects are essential when considering use of a probiotic for the dietary management of serious diseases. However, changes in the manufacturing processes, equipment or facilities can result in differences in the product itself due to the live nature of probiotics. The need to reconfirm safety and/or efficacy for any probiotic product made at a different factory is therefore mandatory. Recently, under the brand VSL#3®, a formulation produced by a manufacturer different from the previous one, has been commercialized in some European countries (the UK and Holland). VSL#3 is a…

0301 basic medicineGenetics and Molecular Biology (all)Cell LinesCancer Treatmentlcsh:MedicineApoptosisMedicine (all); Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Biochemistrylaw.inventionProbiotic0302 clinical medicinelawMedicine and Health SciencesMedicineCell Cycle and Cell DivisionEnzyme-Linked Immunoassayslcsh:ScienceStainingMultidisciplinaryCell DeathMedicine (all)Inflammatory Bowel DiseasesCell StainingApoptotic deathProbiotic agentOncologyCell Processes030211 gastroenterology & hepatologyBiological CulturesResearch ArticleTumor cellsAffect (psychology)Research and Analysis MethodsMicrobiology03 medical and health sciencesImmunoassaysBeneficial effectsBacteriabusiness.industryProbioticslcsh:ROrganismsBiology and Life SciencesCell BiologyIn vitroBiotechnology030104 developmental biologyAgricultural and Biological Sciences (all)Specimen Preparation and TreatmentImmunologyImmunologic Techniqueslcsh:QCaco-2 CellsbusinessPLoS ONE
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Identification and Successful Negotiation of a Metabolic Checkpoint in Direct Neuronal Reprogramming

2016

Despite the widespread interest in direct neuronal reprogramming, the mechanisms underpinning fate conversion remain largely unknown. Our study revealed a critical time point after which cells either successfully convert into neurons or succumb to cell death. Co-transduction with Bcl-2 greatly improved negotiation of this critical point by faster neuronal differentiation. Surprisingly, mutants with reduced or no affinity for Bax demonstrated that Bcl-2 exerts this effect by an apoptosis-independent mechanism. Consistent with a caspase-independent role, ferroptosis inhibitors potently increased neuronal reprogramming by inhibiting lipid peroxidation occurring during fate conversion. Genome-w…

0301 basic medicineGeneticsProgrammed cell deathCell typeCellular Reprogramming TechniquesMutantCell BiologyBiologyIn vitroCell biology03 medical and health sciencesTransduction (genetics)030104 developmental biologyIn vivoGeneticsMolecular MedicineReprogrammingCell Stem Cell
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A dual role of caspase-8 in triggering and sensing proliferation-associated DNA damage, a key determinant of liver cancer development.

2017

Summary Concomitant hepatocyte apoptosis and regeneration is a hallmark of chronic liver diseases (CLDs) predisposing to hepatocellular carcinoma (HCC). Here, we mechanistically link caspase-8-dependent apoptosis to HCC development via proliferation- and replication-associated DNA damage. Proliferation-associated replication stress, DNA damage, and genetic instability are detectable in CLDs before any neoplastic changes occur. Accumulated levels of hepatocyte apoptosis determine and predict subsequent hepatocarcinogenesis. Proliferation-associated DNA damage is sensed by a complex comprising caspase-8, FADD, c-FLIP, and a kinase-dependent function of RIPK1. This platform requires a non-apop…

0301 basic medicineGenome instabilityMaleliver; Hepatocellular carcinoma; DNA damage response; replication stress; apoptosisCancer ResearchDNA RepairCarcinogenesisFas-Associated Death Domain ProteinApoptosisurologic and male genital diseasesDNA damage responseDna Damage Response ; Apoptosis ; Hepatocellular Carcinoma ; Liver ; Replication StressHistonesMice0302 clinical medicineRisk FactorsFADDPhosphorylationCellular SenescenceCaspase 8biologyLiver Neoplasmshepatocellular carcinomaLiver regeneration3. Good healthHistoneOncologyReceptors Tumor Necrosis Factor Type I030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesFemalebiological phenomena cell phenomena and immunityCell agingCarcinoma HepatocellularDNA damageDNA repairreplication stressCaspase 8liverArticleGenomic Instability03 medical and health sciencesAnimalsHepatectomyHumansCrosses GeneticCell ProliferationJNK Mitogen-Activated Protein KinasesCell BiologyLiver Regeneration030104 developmental biologyImmunologyChronic Diseasebiology.proteinCancer researchHepatocytesMyeloid Cell Leukemia Sequence 1 ProteinDNA Damage
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Evolutionary impact of copy number variation rates.

2017

[Objective]: Copy number variation is now recognized as one of the major sources of genetic variation among individuals in natural populations of any species. However, the relevance of these unexpected observations goes beyond diagnosing high diversity. [Results]: Here, it is argued that the molecular rates of copy number variation, mainly the deletion rate upon variation, determine the evolutionary road of the genome regarding size. Genetic drift will govern this process only if the efective population size is lower than the inverse of the deletion rate. Otherwise, natural selection will do.

0301 basic medicineGenome sizeDNA Copy Number VariationsGene duplicationPopulation geneticsPopulation geneticslcsh:MedicineBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesEffective population sizeGenetic driftGenetic variationAnimalsHumansCopy-number variationlcsh:Science (General)Genome sizelcsh:QH301-705.5GeneticsNatural selectionlcsh:RGenetic DriftBirth–death processGeneral MedicineBiological EvolutionResearch Note030104 developmental biologyGenetics Populationlcsh:Biology (General)Evolutionary biologyNeutral theory of molecular evolutionNeutral evolutionlcsh:Q1-390BMC research notes
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