Search results for "DEA"

showing 10 items of 4491 documents

Promises and Pitfalls in the Use of PD-1/PD-L1 Inhibitors in Multiple Myeloma

2018

In the biology of multiple myeloma (MM), immune dysregulation has emerged as a critical component for novel therapeutic strategies. This dysfunction is due to a reduced antigen presentation, a reduced effector cell ability and a loss of reactive T cells against myeloma, together with a bone marrow microenvironment that favors immune escape. The Programmed Death-1 (PD-1) pathway is associated with the regulation of T cell activation and with the apoptotic pathways of effector memory T cells. Specifically, the binding with PD-1 ligand (PD-L1) on the surface of tumor plasma cells down-regulates T cell-proliferation, thus contributing to the immune escape of tumor cells. In relapsed and/or refr…

0301 basic medicinePD-L1lcsh:Immunologic diseases. AllergyDurvalumabMini ReviewT-LymphocytesT cellProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationT cellsPembrolizumabmedicine.disease_causeB7-H1 Antigen03 medical and health sciences0302 clinical medicineBone Marrowimmune dysregulationPD-L1PD-1Tumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyImmune dysregulation; Multiple myeloma; PD-1; PD-L1; T cells; Animals; B7-H1 Antigen; Bone Marrow; Humans; Multiple Myeloma; Programmed Cell Death 1 Receptor; T-Lymphocytes; Tumor MicroenvironmentMultiple myelomabiologybusiness.industryImmune dysregulationmedicine.diseasemultiple myeloma030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinCancer researchNivolumabbusinesslcsh:RC581-607Frontiers in Immunology
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Circulating programmed death ligand-1 (cPD-L1) in non-smallcell lung cancer (NSCLC)

2018

// Silvia Vecchiarelli 1, * , Francesco Passiglia 2, * , Armida D’Incecco 3, * , Marianna Gallo 4 , Antonella De Luca 4 , Elisa Rossi 5 , Federica D’Inca 1 , Gabriele Minuti 1 , Lorenza Landi 1 , Chiara Bennati 1 , Michela Spreafico 1 , Manolo D’Arcangelo 1 , Valentina Mazza 1 , Nicola Normanno 4 and Federico Cappuzzo 1 1 Department of Oncology and Hematology, AUSL della Romagna, Ravenna, Italy 2 Department of Surgical, Oncological and Stomatological Disciplines, University of Palermo, Palermo, Italy 3 Medical Oncology and Immunotherapy, University Hospital of Siena, Siena, Italy 4 Cell Biology and Biotherapy Unit, Istituto Nazionale Tumori "Fondazione G Pascale"-IRCCS, Naples, Italy 5 Fond…

0301 basic medicinePD-L1medicine.medical_specialtymedicine.medical_treatmentnon-small cell lung cancer (NSCLC)Gastroenterology03 medical and health sciences0302 clinical medicineInternal medicinemedicineLung cancerSurvival analysisChemotherapyHematologybusiness.industrybiomarkersBiomarkermedicine.disease030104 developmental biologyOncology030220 oncology & carcinogenesisCohortMann–Whitney U testImmunotherapybusinessNon-small-cell lung cancerResearch PaperProgrammed death
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Contribution of cholesterol and oxysterols to the pathophysiology of Parkinson's disease

2016

International audience; Neurodegenerative diseases are a major public health issue worldwide. Some countries, including France, have engaged in research into the causes of Parkinson's disease, Alzheimer's disease, and multiple sclerosis and the management of these patients. It should lead to a better understanding of the mechanisms leading to these diseases including the possible involvement of lipids in their pathogenesis. Parkinson's disease is a progressive neurodegenerative disorder characterized by the loss of dopaminergic neurons in the substantia nigra and the accumulation of α-synuclein (Lewy bodies). Several in vivo studies have shown a relationship between the lipid profile [chole…

0301 basic medicinePathologymedicine.medical_specialtyParkinson's diseaseOxysterolParkinson's diseasePresynaptic TerminalsSubstantia nigraDiseaseBiologyBioinformaticsBiochemistryPathogenesisProtein Aggregates03 medical and health scienceschemistry.chemical_compoundOxysterol0302 clinical medicinePhysiology (medical)medicineHumans[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyAlpha-synucleinCell Deathmedicine.diagnostic_testDopaminergic NeuronsMultiple sclerosisParkinson DiseaseOxysterols[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismmedicine.diseaseSubstantia NigraCholesterol030104 developmental biologychemistryalpha-Synucleinlipids (amino acids peptides and proteins)Lipid profileOxidation-Reduction030217 neurology & neurosurgeryFree Radical Biology and Medicine
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Prospective risk of stillbirth and neonatal complications in twin pregnancies: systematic review and meta-analysis

2016

Twin pregnancies are at increased risk of stillbirth. Uncomplicated twin pregnancies are commonly delivered earlier to prevent stillbirth; however, there is a risk of neonatal complications associated with being born prior to 39 weeks’ gestation. The optimal gestational age for delivery in twin pregnancies is unknown and likely varies by chorionicity. The present study aimed to determine the prospective risk of stillbirth in women with uncomplicated monochorionic and dichorionic twin pregnancies, and neonatal mortality risks, when delivered beyond 34 weeks of gestation. Data on twin pregnancies that reported rates of stillbirth were obtained from MEDLINE, EMBASE, and Cochrane Library. Separ…

0301 basic medicinePediatricsNeonatal intensive care unitPerinatal DeathCochrane LibraryInfant Newborn Diseases0302 clinical medicineObstetrics and gynaecologyPregnancyRisk FactorsTwins DizygoticMedicineProspective StudiesProspective cohort studyreproductive and urinary physiologyTwin Pregnancyhealth care economics and organizations030219 obstetrics & reproductive medicineObstetricsIncidence (epidemiology)Absolute risk reductionObstetrics and GynecologyGestational ageProspective riskGeneral MedicineStillbirthfemale genital diseases and pregnancy complicationsMeta-analysisGestationFemalemedicine.medical_specialtyeducationGestational AgeTwin-to-twin transfusion syndrome03 medical and health sciences030225 pediatricsJournal ArticleHumansGynecologyPregnancybusiness.industryResearchInfant NewbornTwins Monozygoticmedicine.diseaseConfidence interval030104 developmental biologyIntensive Care NeonatalPregnancy TwinbusinessBMJ (Clinical research ed.)
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Global, regional, and national levels of maternal mortality, 1990–2015: a systematic analysis for the Global Burden of Disease Study 2015

2016

BACKGROUND: In transitioning from the Millennium Development Goal to the Sustainable Development Goal era, it is imperative to comprehensively assess progress toward reducing maternal mortality to identify areas of success, remaining challenges, and frame policy discussions. We aimed to quantify maternal mortality throughout the world by underlying cause and age from 1990 to 2015.METHODS: We estimated maternal mortality at the global, regional, and national levels from 1990 to 2015 for ages 10-54 years by systematically compiling and processing all available data sources from 186 of 195 countries and territories, 11 of which were analysed at the subnational level. We quantified eight underl…

0301 basic medicinePediatricsNutrition and DiseaseMILLENNIUM DEVELOPMENT GOALSSUSTAINABLE DEVELOPMENT GOALSANTENATAL CAREGlobal Health0302 clinical medicineVoeding en Ziekte11. SustainabilityGlobal healthHQHealthcare FinancingEMERGENCY OBSTETRIC CARE030212 general & internal medicineCooperative Behavior10. No inequalityReproductive healthMedicine(all)education.field_of_study030219 obstetrics & reproductive medicineMedicine (all)1. No povertyObstetrics and GynecologyPublic Health Global Health Social Medicine and EpidemiologyPrenatal CareGeneral Medicine11 Medical And Health SciencesLOW-RESOURCE SETTINGS3142 Public health care science environmental and occupational healthFamily Planning Service3. Good healthGBD 2015 Maternal Mortality CollaboratorsGovernment ProgramsMaternal MortalityReproductive HealthFamily Planning ServicesMaternal deathHEALTHLife Sciences & BiomedicineHumanCOUNTRIESmedicine.medical_specialtyPopulation610Prenatal careArticle03 medical and health sciencesMedicine General & InternalSDG 3 - Good Health and Well-beingCASH TRANSFER PROGRAMEnvironmental healthGeneral & Internal Medicineparasitic diseasesmedicineLife ScienceQUALITYHumansGlobal Burden of Disease StudyeducationVLAGScience & TechnologyMedical Assistancebusiness.industryKlinisk medicinParturitionObstetric transitionmedicine.diseaseQPInfant mortalityFolkhälsovetenskap global hälsa socialmedicin och epidemiologiStandardized mortality ratio030104 developmental biologyRISK-FACTORSRGClinical MedicinebusinessRA
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EAST/SeSAME syndrome: Review of the literature and introduction of four new Latvian patients.

2018

EAST (Epilepsy, Ataxia, Sensorineural deafness, Tubulopathy) or SeSAME (Seizures, Sensorineural deafness, Ataxia, Mental retardation, and Electrolyte imbalance) syndrome is a rare autosomal recessive syndrome first described in 2009 independently by Bockenhauer and Scholl. It is caused by mutations in KCNJ10, which encodes Kir4.1, an inwardly rectifying K+ channel found in the brain, inner ear, kidney and eye. To date, 16 mutations and at least 28 patients have been reported. In this paper, we review mutations causing EAST/SeSAME syndrome, clinical manifestations in detail, and efficacy of treatment in previously reported patients. We also report a new Latvian kindred with 4 patients. In co…

0301 basic medicinePediatricsmedicine.medical_specialtyAtaxiaHearing Loss SensorineuralKCNJ10030105 genetics & hereditySensorineural deafnessKidney03 medical and health sciencesEpilepsyTubulopathySeizuresIntellectual DisabilityIntellectual disabilityGeneticsmedicineEAST syndromeHumansEye AbnormalitiesPotassium Channels Inwardly RectifyingGenetics (clinical)SeSAME syndromebiologybusiness.industryBrainmedicine.diseaseLatvia030104 developmental biologyPhenotypeEar InnerMutationbiology.proteinmedicine.symptombusinessClinical genetics
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Antioxidant effects of resveratrol in the cardiovascular system

2016

The antioxidant effects of resveratrol (3,5,4'-trihydroxy-trans-stilbene) contribute substantially to the health benefits of this compound. Resveratrol has been shown to be a scavenger of a number of free radicals. However, the direct scavenging activities of resveratrol are relatively poor. The antioxidant properties of resveratrol in vivo are more likely to be attributable to its effect as a gene regulator. Resveratrol inhibits NADPH oxidase-mediated production of ROS by down-regulating the expression and activity of the oxidase. This polyphenolic compound reduces mitochondrial superoxide generation by stimulating mitochondria biogenesis. Resveratrol prevents superoxide production from un…

0301 basic medicinePharmacologyOxidase testAntioxidantendocrine system diseasesbiologySuperoxideSirtuin 1organic chemicalsmedicine.medical_treatmentfood and beveragesMitochondrionResveratrolmedicine.disease_cause03 medical and health scienceschemistry.chemical_compound030104 developmental biologychemistryBiochemistrymedicinebiology.proteinProtein deacetylaseOxidative stressBritish Journal of Pharmacology
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N-Desmethyldauricine Induces Autophagic Cell Death in Apoptosis-Defective Cells via Ca2+ Mobilization

2017

Resistance of cancer cells to chemotherapy remains a significant problem in oncology. Mechanisms regulating programmed cell death, including apoptosis, autophagy or necrosis, in the treatment of cancers have been extensively investigated over the last few decades. Autophagy is now emerging as an important pathway in regulating cell death or survival in cancer therapy. Recent studies demonstrated variety of natural small-molecules could induce autophagic cell death in apoptosis-resistant cancer cells, therefore, discovery of novel autophagic enhancers from natural products could be a promising strategy for treatment of chemotherapy-resistant cancer. By computational virtual docking analysis,…

0301 basic medicinePharmacologyProgrammed cell deathautophagyKinaseDrug discoveryAutophagylcsh:RM1-950BiologyCell biology03 medical and health sciences030104 developmental biologylcsh:Therapeutics. PharmacologyApoptosisautophagic cell deathN-desmethyldauricineSERCACancer cellCytotoxic T cellPharmacology (medical)apoptosis-resistantProtein kinase AOriginal ResearchFrontiers in Pharmacology
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Tetrandrine, an Activator of Autophagy, Induces Autophagic Cell Death via PKC-α Inhibition and mTOR-Dependent Mechanisms

2017

Emerging evidence suggests the therapeutic role of autophagic modulators in cancer therapy. This study aims to identify novel traditional Chinese medicinal herbs as potential anti-tumor agents through autophagic induction, which finally lead to autophagy mediated-cell death in apoptosis-resistant cancer cells. Using bioactivity-guided purification, we identified tetrandrine (Tet) from herbal plant, Radix stephaniae tetrandrae, as an inducer of autophagy. Across a number of cancer cell lines, we found that breast cancer cells treated with tetrandrine show an increase autophagic flux and formation of autophagosomes. In addition, tetrandrine induces cell death in a panel of apoptosis-resistant…

0301 basic medicinePharmacologyProgrammed cell deathautophagylcsh:RM1-950AutophagyCaspase 3BiologytetrandrineCaspase 7Cell biologyTetrandrine03 medical and health scienceschemistry.chemical_compoundlcsh:Therapeutics. Pharmacology030104 developmental biologychemistryCancer cellmTORPharmacology (medical)apoptosis-resistantPKC-αProtein kinase API3K/AKT/mTOR pathwayOriginal ResearchFrontiers in Pharmacology
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Coarse-grained models of double-stranded DNA based on experimentally determined knotting probabilities

2018

Abstract To accurately model double-stranded DNA in a manner that is computationally efficient, coarse-grained models of DNA are introduced, where model parameters are selected by fitting the spectrum of observable DNA knots: We develop a general method to fit free parameters of coarse-grained chain models by comparing experimentally obtained knotting probabilities of short DNA chains to knotting probabilities that are computed in Monte Carlo simulations, resulting in coarse-grained DNA models which are tailored to reflect DNA topology in the best possible way. The method is exemplified by fitting ideal chain models as well as a bead-spring model with excluded volume interactions, to model …

0301 basic medicinePhysicsPersistence lengthQuantitative Biology::BiomoleculesPolymers and PlasticsGeneral Chemical EngineeringMonte Carlo methodfood and beveragesObservableGeneral ChemistryBiochemistry03 medical and health sciencesMolecular dynamics030104 developmental biologyMaterials ChemistryEnvironmental ChemistryStatistical physicsIdeal chainTopology (chemistry)AnsatzFree parameterReactive and Functional Polymers
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