Search results for "DEFICIT"

showing 10 items of 470 documents

Association between DNA methylation and ADHD symptoms from birth to school age: a prospective meta-analysis

2020

Funder: Gouvernement du Canada | Canadian Institutes of Health Research (Instituts de Recherche en Santé du Canada); doi: https://doi.org/10.13039/501100000024

0301 basic medicineOncology3124 Neurology and psychiatryEpigenesis GeneticACTIVATION0302 clinical medicine3123 Gynaecology and paediatricsPregnancyProspective StudiesProspective cohort studyChildRISK0303 health sciencesSchoolsATTENTION-DEFICIT/HYPERACTIVITY DISORDERMethylation3. Good healthPsychiatry and Mental healthMeta-analysisChild PreschoolDNA methylation/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingBiomarker (medicine)FemalePACKAGEmedicine.medical_specialtyAdolescent515 PsychologyArticlelcsh:RC321-571Cellular and Molecular Neuroscience03 medical and health sciencesSDG 3 - Good Health and Well-beingInternal medicinemedicineGeneticsAttention deficit hyperactivity disorderHumansEpigeneticslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological Psychiatry030304 developmental biologyGenetic associationPregnancybusiness.industryInfant NewbornPERFORMANCEDNA Methylationmedicine.diseaseGENE030104 developmental biologyAttention Deficit Disorder with HyperactivityCOHORT PROFILEEtiologybusinessPsychiatric disorders030217 neurology & neurosurgery
researchProduct

Parkinson's disease: towards better preclinical models and personalized treatments.

2016

Non peer reviewed

0301 basic medicineParkinson's diseaseeducationMEDLINEBioinformatics03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDEFICITSMedicineAnimalsHumansMolecular BiologyPharmacologybusiness.industryParkinson DiseaseCell Biologymedicine.diseaseMolecular medicine3. Good healthMICE030104 developmental biologyNeuroprotective AgentsCell Biology; Molecular Biology; Molecular Medicine; Pharmacology; Cellular and Molecular NeuroscienceMolecular Medicine3111 Biomedicinebusiness030217 neurology & neurosurgeryCellular and molecular life sciences : CMLS
researchProduct

Violent offending in borderline personality disorder and attention deficit/hyperactivity disorder.

2018

Abstract The prevalence of borderline personality disorder (BPD) and attention deficit/hyperactivity disorder (ADHD) is significantly higher among offenders compared to the prevalence found in the general population. Both disorders share important diagnostic characteristics and thus it has been suggested that they might follow a common developmental pathway. In this narrative review, we first discuss the potential links of disorder inherent symptoms such as impulsivity and emotion regulation difficulties and how they might elevate the risk of violent delinquency. We continue with highlighting that comorbidities particularly from the antisocial spectrum as well as comorbid substance use diso…

0301 basic medicinePopulationPoison controlViolenceImpulsivitybehavioral disciplines and activities03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineBorderline Personality Disordermental disordersmedicineJuvenile delinquencyAttention deficit hyperactivity disorderHumanseducationBorderline personality disorderPharmacologyeducation.field_of_studyAntisocial personality disorderCriminalsmedicine.diseaseEmotional RegulationSubstance abuse030104 developmental biologyAttention Deficit Disorder with HyperactivityImpulsive Behaviormedicine.symptomPsychology030217 neurology & neurosurgeryClinical psychologyNeuropharmacology
researchProduct

Narrowing the Genetic Causes of Language Dysfunction in the 1q21.1 Microduplication Syndrome

2018

The chromosome 1q21.1 duplication syndrome (OMIM# 612475) is characterized by head anomalies, mild facial dysmorphisms, and cognitive problems, including autistic features, mental retardation, developmental delay, and learning disabilities. Speech and language development are sometimes impaired, but no detailed characterization of language problems in this condition has been provided to date. We report in detail on the cognitive and language phenotype of a child who presents with a duplication in 1q21.1 (arr[hg19] 1q21.1q21.2(145,764,455-147,824,207) × 3), and who exhibits cognitive delay and behavioral disturbances. Language is significantly perturbed, being the expressive domain the most …

0301 basic medicineProbandmedicine.medical_specialtycognitive delayAudiologychromosome 1q21.1 duplication syndrome03 medical and health sciences0302 clinical medicineMotor speechmedicinebusiness.industrylanguage deficitslcsh:RJ1-570DyslexiaCDH1LROBO1lcsh:PediatricsCognitionFOXP2Pragmaticsmedicine.diseaseComprehensionLanguage development030104 developmental biologyPediatrics Perinatology and Child Healthspeech problemsbusiness030217 neurology & neurosurgery
researchProduct

Identification of microRNAS differentially regulated by water deficit in relation to mycorrhizal treatment in wheat.

2019

Arbuscular mycorrhizal fungi (AMF) are soil microrganisms that establish symbiosis with plants positively influencing their resistance to abiotic stresses. The aim of this work was to identify wheat miRNAs differentially regulated by water deficit conditions in presence or absence of AMF treatment. Small RNA libraries were constructed for both leaf and root tissues considering four conditions: control (irrigated) or water deficit in presence/absence of mycorrhizal (AMF) treatment. A total of 12 miRNAs were significantly regulated by water deficit in leaves: five in absence and seven in presence of AMF treatment. In roots, three miRNAs were water deficit-modulated in absence of mycorrhizal t…

0301 basic medicineSmall RNABiologyPlant Roots03 medical and health sciences0302 clinical medicineSymbiosisTranscription (biology)Gene Expression Regulation PlantStress PhysiologicalMycorrhizaeBotanymicroRNAGeneticsProtein biosynthesisTranscriptional regulationGene Regulatory NetworksMolecular BiologyDurum wheatWater deficitTriticummiRNAPlant ProteinsAbiotic componentGene Expression ProfilingfungiGene Expression Regulation DevelopmentalGeneral MedicineCell redox homeostasisDroughtsPlant LeavesMicroRNAs030104 developmental biologyRootRNA Plant030220 oncology & carcinogenesisWheatMolecular biology reports
researchProduct

Surge of Peripheral Arginine Vasopressin in a Rat Model of Birth Asphyxia

2018

Mammalian birth is accompanied by a period of obligatory asphyxia, which consists of hypoxia (drop in blood O2 levels) and hypercapnia (elevation of blood CO2 levels). Prolonged, complicated birth can extend the asphyxic period, leading to a pathophysiological situation, and in humans, to the diagnosis of clinical birth asphyxia, the main cause of hypoxic-ischemic encephalopathy (HIE). The neuroendocrine component of birth asphyxia, in particular the increase in circulating levels of arginine vasopressin (AVP), has been extensively studied in humans. Here we show for the first time that normal rat birth is also accompanied by an AVP surge, and that the fetal AVP surge is further enhanced in…

0301 basic medicineVasopressinmedicine.medical_specialtySTRESSArgininehypothalamic-pituitary axis (HPA axis)blood gasesHYPOXIAbirth asphyxia3124 Neurology and psychiatrylcsh:RC321-571neonatal03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineCopeptinInternal medicineMedicineBRAINlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryPRECURSORNEURONSperinatalOriginal ResearchRELEASEAsphyxiaFetusPARAVENTRICULAR NUCLEUSbusiness.industry3112 NeurosciencescopeptinENCEPHALOPATHYarginine vasopressin (AVP)Hypoxia (medical)base deficit030104 developmental biologyEndocrinologyHypothalamusHYPOTHALAMUSmedicine.symptombusinessHypercapnia030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
researchProduct

A new “sudden fright paradigm” to explore the role of (epi)genetic modulations of the DAT gene in fear-induced avoidance behavior

2020

Alterations in dopamine (DA) reuptake are involved in several psychiatric disorders whose symptoms can be investigated in knock out rats for the DA transporter (DAT-KO). Recent studies evidenced the role of epigenetic DAT modulation in depressive-like behavior. Accordingly, we used heterozygous (HET) rats born from both HET parents (termed MIX-HET), compared to HET rats born from WT-mother and KO-father (MAT-HET), implementing the role of maternal care on DAT modulation. We developed a "sudden fright" paradigm (based on dark-light test) to study reaction to fearful inputs in the DAT-KO, MAT-HET, MIX-HET, and WT groups. Rats could freely explore the whole 3-chambers apparatus; then, they wer…

0301 basic medicineanimal structuresEmotionsStimulus (physiology)Epigenesis GeneticReuptakechoice behavior03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineDopamineDAT-KO ratAvoidance LearningGeneticsmedicineAnimalsFear conditioningEpigeneticsprefrontal cortex.Prefrontal cortexdopamine transporterDopamine transporterDopamine Plasma Membrane Transport ProteinsBehavior AnimalbiologyFearfear conditioningRatsDisease Models Animal030104 developmental biologyNeurologyAttention Deficit Disorder with Hyperactivitybiology.proteinSettore BIO/14 - Farmacologiaconditioned preferenceHistone deacetylaseNeuroscience030217 neurology & neurosurgerymedicine.drug
researchProduct

The still under-investigated role of cognitive deficits in PML diagnosis

2017

Background: Despite cognitive deficits frequently represent the first clinical manifestations of Progressive Multifocal Leukoencephalopathy (PML) in Natalizumab-treated MS patients, the importance of cognitive deficits in PML diagnosis is still under-investigated. The aim of the current study is to investigate the cognitive deficits at PML diagnosis in a group of Italian patients with PML. Methods: Thirty-four PML patients were included in the study. The demographic and clinical data, the lesion load and localization, and the longitudinal clinical course was compared between patients with (n = 13) and without (n = 15) cognitive deficit upon PML suspicion (the remaining six patients were asy…

0301 basic medicinecognitionmedicine.medical_specialtyPediatricscognition; italian database; natalizumab; neuropsychological impairment; progressive multifocal leukoencephalopathy; neurology (clinical); neurology; immunology; immunology and allergy; natalizumab; cognition; neuropsychological impairment; italian databaseNeurologySettore MED/17 - Malattie InfettiveAsymptomaticApraxiaprogressive multifocal leukoencephalopathyimmunology03 medical and health sciences0302 clinical medicinenatalizumabitalian databasemedicineDementiaimmunology and allergyPsychiatryCognitive deficitneurology (clinical)Progressive multifocal leukoencephalopathyneurologyNeuropsychologyCognitionProgressive multifocal leukoencephalopathy Natalizumab Cognition Neuropsychological impairment Italian databasemedicine.disease030104 developmental biologyCognition; Italian database; Natalizumab; Neuropsychological impairment; Progressive multifocal leukoencephalopathyCognition Italian database Natalizumab Neuropsychological impairment Progressive multifocal leukoencephalopathyCognition; Italian database; Natalizumab; Neuropsychological impairment; Progressive multifocal leukoencephalopathy; Immunology and Allergy; Immunology; Neurology; Neurology (clinical)Settore MED/26 - Neurologiamedicine.symptomPsychologyneuropsychological impairment030217 neurology & neurosurgery
researchProduct

Natural visibility graphs for diagnosing attention deficit hyperactivity disorder (ADHD)

2016

“NOTICE: this is the author’s version of a work that was accepted for publication in Electronic Notes in Discrete Mathematics. Changes resulting from the publishing process, such as peer review, editing, corrections, structural formatting, and other quality control mechanisms may not be reflected in this document. Changes may have been made to this work since it was submitted for publication. A definitive version was subsequently published in Electronic Notes in Discrete Mathematics, [Volume 54, October 2016, Pages 337-342] DOI 10.1016/j.endm.2016.09.058 ¨

0301 basic medicinemedia_common.quotation_subjectAttention deficit hyperactivity disorder03 medical and health sciencesDisk formatting0302 clinical medicinemedicineDiscrete Mathematics and CombinatoricsAttention deficit hyperactivity disorderNatural (music)Quality (business)Mathematicsmedia_commonNoticebusiness.industryApplied MathematicsVisibility (geometry)medicine.disease030104 developmental biologyPublishingKolmogorov-Smirnov testbusinessMATEMATICA APLICADANatural visibility graph030217 neurology & neurosurgeryCognitive psychology
researchProduct

Transient hypothyroidism during lactation alters the development of the corpus callosum in rats. An in vivo magnetic resonance image and electron mic…

2020

Magnetic resonance imaging (MRI) data of children with late diagnosed congenital hypothyroidism and cognitive alterations such as abnormal verbal memory processing suggest altered telencephalic commissural connections. The corpus callosum (CC) is the major inter-hemispheric commissure that contra-laterally connects neocortical areas. However, in late diagnosed neonates with congenital hypothyroidism, the possible effect of early transient and chronic postnatal hypothyroidism still remains unknown. We have studied the development of the anterior, middle and posterior CC, using in vivo MRI and electron microscopy in hypothyroid and control male rats. Four groups of methimazole (MMI) treated r…

0301 basic medicineneocortical developmentmedicine.medical_specialtyNeuroscience (miscellaneous)autismattention deficit/hyperactivity disorderCorpus callosumNerve conduction velocitylcsh:RC321-571lcsh:QM1-695law.invention03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineIn vivolawInternal medicineLactationmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchthyroid hormonesiodine dietmedicine.diagnostic_testbusiness.industrycongenital hypothyroidismpsychiatric diseasesMagnetic resonance imaginglcsh:Human anatomyCommissuremedicine.diseaseCongenital hypothyroidismNeuroanatomy030104 developmental biologyEndocrinologymedicine.anatomical_structureAnatomyElectron microscopebusiness030217 neurology & neurosurgery
researchProduct