Search results for "DOPAMINE"

showing 10 items of 660 documents

A NEW DOPAMINE-AMINOACID CONJUGATE: SYNTHESIS AND DETERMINATION OF PHYSICO-CHEMICAL PROPERTIES USEFUL TO CROSS THE BBB

2012

Aminoacidic prodrugs dopamine PAMPA-BBB Caco-2 modelSettore CHIM/09 - Farmaceutico Tecnologico Applicativo
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112 Workshop Summary: DOPAMINERGIC MECHANISMS OF PAIN CONTROL

2007

Anesthesiology and Pain MedicinePain controlbusiness.industryMedicineDopaminergic mechanismsbusinessNeuroscienceEuropean Journal of Pain
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Molecular bases of anorexia nervosa, bulimia nervosa and binge eating disorder: shedding light on the darkness

2017

International audience; Eating-disorders (EDs) consequences to human health are devastating, involving social, mental, emotional, physical and life-threatening aspects, concluding on impairment and death in cases of extreme anorexia nervosa. It also implies that people suffering an ED need to find psychiatric and psychological help as soon as possible to achieve a fully physical and emotional recovery. Unfortunately, to date, there is a crucial lack of efficient clinical treatment to these disorders. In this review, we present an overview concerning the actual pharmacological and psychological treatments, the knowledge of cells, circuits, neuropeptides, neuromodulators and hormones in the h…

Anorexia NervosaPsychotherapistcognitive-behavioral therapy[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionneuromodulatorsAnorexia nervosa/bulimia03 medical and health sciencesCellular and Molecular NeuroscienceHuman health0302 clinical medicineplacebo-controlled trialBinge-eating disordermesolimbic dopamine systemGeneticsmedicineHumansGenetic Predisposition to DiseaseBulimia NervosaClinical treatmentregulate feeding-behaviornucleus-accumbens shellborderline personality-disordergene-environment interactionsmedicine.disease030227 psychiatryEating disorderssubstance use disordersAnorexia nervosa (differential diagnoses)genetic approachesrandomized controlled-trialEating disordersgenome-wide associationpharmacologyPsychology[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBinge-Eating Disorder030217 neurology & neurosurgeryClinical psychologyJournal of Neurogenetics
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Novel imine antioxidants at low nanomolar concentrations protect dopaminergic cells from oxidative neurotoxicity.

2009

Strong evidence indicates that oxidative stress may be causally involved in the pathogenesis of Parkinson's disease. We have employed human dopaminergic neuroblastoma cells and rat primary mesencephalic neurons to assess the protective potential of three novel bisarylimine antioxidants on dopaminergic cell death induced by complex I inhibition or glutathione depletion. We have found that exceptionally low concentrations (EC(50) values approximately 20 nM) of these compounds (iminostilbene, phenothiazine, and phenoxazine) exhibited strong protective effects against the toxicities of MPP(+), rotenone, and l-buthionine sulfoximine. Investigating intracellular glutathione levels, it was found t…

Antioxidantmedicine.medical_treatmentDopamineGlutathione reductaseNeurotoxinsBiologymedicine.disease_causeProtein oxidationBiochemistryAntioxidantsLipid peroxidationRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundCell Line TumormedicineAnimalsHumansCells CulturedMembrane Potential MitochondrialCell DeathDose-Response Relationship DrugNeurotoxicityParkinson DiseaseRotenoneGlutathionemedicine.diseaseGlutathioneMitochondriaRatsSubstantia NigraOxidative StressNeuroprotective AgentschemistryBiochemistryElectron Transport Chain Complex ProteinsCytoprotectionNerve DegenerationIminesOxidation-ReductionOxidative stressJournal of neurochemistry
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Ursolic acid ameliorates stress and reactive oxygen species in C. elegans knockout mutants by the dopamine Dop1 and Dop3 receptors.

2020

Abstract Background Depression and stress-related disorders are leading causes of death worldwide. Standard treatments elevating serotonin or noradrenaline levels are not sufficiently effective and cause adverse side effects. A connection between dopamine pathways and stress-related disorders has been suggested. Compounds derived from herbal medicine could be a promising alternative. We examined the neuroprotective effects of ursolic acid (UA) by focusing on dopamine signalling. Methods Trolox equivalent capacity assay was used to determine the antioxidant activities of UA in vitro. C. elegans N2 wildtype and dopamine receptor-knockout mutants (dop-1-deficient RB665 and dop-3-deficient LX70…

Antioxidantmedicine.medical_treatmentDopamineLongevityPharmaceutical SciencePharmacologyNeuroprotectionAntioxidants03 medical and health scienceschemistry.chemical_compoundGene Knockout Techniques0302 clinical medicineDopamineStress PhysiologicalDrug DiscoverymedicineAnimalsHumansReceptorCaenorhabditis elegansCaenorhabditis elegans Proteins030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesChemistryReceptors Dopamine D2Receptors Dopamine D1Receptors Dopamine D3TriterpenesMolecular Docking SimulationComplementary and alternative medicineDopamine receptor030220 oncology & carcinogenesisMutationMolecular MedicineSerotoninTroloxReactive Oxygen Speciesmedicine.drugSignal TransductionPhytomedicine : international journal of phytotherapy and phytopharmacology
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Electrochemistry of copper complexes with macrocyclic polyamines containing pyrazole units.

2006

The voltammetric behaviour of bi- and mono-nuclear complexes formed in solution by Cu(2+) with three polyazacyclophanes containing pyrazole units in aqueous solution is described. Cyclic and square wave voltammetric responses at glassy carbon electrodes indicate that the reduction of copper-macrocycle complexes in solution takes place in two successive one-electron per Cu transfer processes coupled with preorganization and protonation reactions that mimic the behaviour of superoxide dismutase. The electrochemistry of ternary Cu(2+)-receptor-dopamine complexes exhibits significant differences with respect to the protection of the neurotransmitter from post-electron transfer cyclization react…

Aqueous solutionMacrocyclic CompoundsSuperoxide DismutaseDopamineInorganic chemistryMolecular Mimicrychemistry.chemical_elementProtonationSquare wavePyrazoleGlassy carbonElectrochemistryCopperInorganic Chemistrychemistry.chemical_compoundchemistryPolymer chemistryElectrochemistryOrganometallic CompoundsPolyaminesPyrazolesTernary operationCopperDalton transactions (Cambridge, England : 2003)
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A Methanol Extract ofBrugmansia arboreaAffects the Reinforcing and Motor Effects of Morphine and Cocaine in Mice

2013

Previous reports have shown that several of the effects of morphine, including the development of tolerance and physical withdrawal symptoms, are reduced by extracts ofBrugmansia arborea(L.) Lagerheim (Solanaceae) (B. arborea). In the present study we evaluate the action of the methanol extract ofB. arborea(7.5–60 mg/kg) on the motor and reinforcing effects of morphine (20 and 40 mg/kg) and cocaine (25 mg/kg) using the conditioned place preference (CPP) procedure. At the doses employed,B. arboreadid not affect motor activity or induce any effect on CPP. The extract partially counteracted morphine-induced motor activity and completely blocked the CPP induced by 20 mg/kg morphine. On the othe…

Article Subjectbiologybusiness.industryDopaminergiclcsh:Other systems of medicinePharmacologylcsh:RZ201-999biology.organism_classificationConditioned place preferenceComplementary and alternative medicineMechanism of actionBrugmansiaMorphinemedicineCholinergicMotor activitymedicine.symptombusinessCocaine abuseResearch Articlemedicine.drugEvidence-Based Complementary and Alternative Medicine
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Storage and release of false transmitters after infusion of (+)- and (?)-?-methyldopamine

1971

Rabbits were given an infusion of 10 mg/kg (−)- or 30 mg/kg (+)-α-methyldopamine and killed after 135 min. The noradrenaline content of the heart was decreased to 26±5 and 34±2%, respectively, of the control value. After infusion of the (+)-isomer the missing noradrenaline was replaced by (−)-α-methylnoradrenaline. Electrical stimulation of the sympathetic nerves or infusion of acetylcholine plus atropine caused an output of noradrenaline and (−)-α-methylnoradrenaline from the isolated heart. The two amines were released in the same proportion as they were stored in the heart and the total output of both amines equalled the output of noradrenaline from control hearts. Nerve stimulation caus…

AtropineMalemedicine.medical_specialtySympathetic Nervous SystemDopamineMetaboliteAdrenergicBlood PressureStimulationSynaptic TransmissionMethyldopamineMethylaminesNorepinephrinechemistry.chemical_compoundIsomerismInternal medicinemedicineAnimalsPharmacologyCardiac cycleMyocardiumSignificant differenceHeartGeneral MedicineAcetylcholineElectric StimulationAtropineEndocrinologychemistryFemaleRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedebergs Archiv f�r Pharmakologie
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Effects of dopamine infusion on plasma catecholamines in preterm and term newborn infants.

1991

Newborn infants (21 preterm and 13 term) received dopamine infusions at a low (2.5-3.4 micrograms/kg per min) and/or high (5-10 micrograms/kg per min) infusion rate and changes in plasma catecholamines were monitored. The mean baseline values for dopamine, noradrenaline and adrenaline were between 240 and 560, 125 and 144 and 62 and 82 pg/ml, respectively. During low-rate infusion of dopamine, there was a significant increase in plasma dopamine (20-100 fold), noradrenaline (three- to five-fold) and adrenaline (threefold). Administration of dopamine at the high rate resulted in an even larger increase in the plasma catecholamines (dopamine, 100-300 fold; noradrenaline, seven- to eightfold; a…

Baseline valuesHigh ratemedicine.medical_specialtyDose-Response Relationship DrugEpinephrinebusiness.industryDopamineInfant NewbornNorepinephrine (medication)NorepinephrineEpinephrineEndocrinologyDopamineInternal medicinePediatrics Perinatology and Child HealthPlasma concentrationmedicineCatecholamineHumansbusinessPerfusionInfant Prematuremedicine.drugEuropean journal of pediatrics
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Preparation of dopaminergic N-alkyl-benzyltetrahydroisoquinolines using a 'one-pot' procedure in acid medium.

2000

The preparation of N-methyl-BTHIQ (4) from N-phenylethyl-phenacetamide (1) by cyclization, reduction and N-alkylation in acid medium has been achieved in good yield in a 'one-pot' procedure. Acylation of imine (2) intermediate afforded the Z and E stereoselectivity in the enamide formation. 6-Hydroxy-BTHIQ (7) shows selectivity for D2 dopamine receptors, while its N-methylated homologue (8) displays higher affinities for both D1 and D2 receptor types, with an unexpected increase in D1 dopamine receptor affinity.

Bicyclic moleculeStereochemistryReceptors Dopamine D2Receptors Dopamine D1Spectrum AnalysisOrganic ChemistryClinical BiochemistryDopaminergicImineDopamine AgentsPharmaceutical ScienceIsoquinolinesBiochemistryChemical synthesisAcylationchemistry.chemical_compoundchemistryDopamine receptor D2Drug DiscoveryMolecular MedicineStereoselectivitySelectivityMolecular Biology
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