Search results for "DOPAMINERGIC"

showing 10 items of 200 documents

Synthesis and evaluation in rats of the dopamine D2/3 receptor agonist 18F-AMC20 as a potential radioligand for PET

2015

Dopamine D2/3 receptor (D2/3R) agonist PET tracers are better suited for the imaging of synaptic dopaminergic neurotransmission than D2/3R antagonists and may also offer the opportunity to study in vivo the high-affinity state of D2/3R (D2/3RHigh). With the aim to develop 18F-labeled D2/3R agonists suitable for widespread clinical application, we report here on the synthesis and in vitro and in vivo evaluation of a D2/3R agonist ligand from the aminomethyl chromane (AMC) class-(R)-2-[(4- 18Fluorobenzylamino)methyl]chroman-7-ol (18F-AMC20). Methods: In vitro affinities of AMC20 toward dopaminergic receptor subtypes were measured in membrane homogenates prepared from HEK293 cells expressing h…

MaleBenzylaminesChemistry Techniques SyntheticPharmacologyLigandsRats Sprague-DawleyCricetinaeBINDINGRadioligandIN-VIVORacloprideRadiochemistryChemistryDopaminergicBrainStereoisomerismLigand (biochemistry)SERIESADDICTIONDopamine receptorDopamine AgonistsHIGH-AFFINITY STATEmedicine.drugAgonistmedicine.drug_classDRUG-ABUSECHO CellsPOSITRON-EMISSION-TOMOGRAPHYCricetulusFIn vivoDopamine receptor D2medicineAnimalsHumansRadiology Nuclear Medicine and imagingBenzopyransChromansReceptors Dopamine D2F-18Receptors Dopamine D3Biological TransportAgonist tracerRatsKineticsHEK293 CellsPETDopamine receptorRaclopridePositron-Emission TomographyRADIOTRACERSYSTEMJournal of Nuclear Medicine
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Mice lacking α-synuclein display functional deficits in the nigrostriatal dopamine system

2000

alpha-Synuclein (alpha-Syn) is a 14 kDa protein of unknown function that has been implicated in the pathophysiology of Parkinson's disease (PD). Here, we show that alpha-Syn-/- mice are viable and fertile, exhibit intact brain architecture, and possess a normal complement of dopaminergic cell bodies, fibers, and synapses. Nigrostriatal terminals of alpha-Syn-/- mice display a standard pattern of dopamine (DA) discharge and reuptake in response to simple electrical stimulation. However, they exhibit an increased release with paired stimuli that can be mimicked by elevated Ca2+. Concurrent with the altered DA release, alpha-Syn-/- mice display a reduction in striatal DA and an attenuation of …

MaleCalbindinsNeuroscience(all)DopamineDopamine AgentsLong-Term PotentiationPresynaptic TerminalsSynucleinsGene ExpressionGlutamic AcidSubstantia nigraNerve Tissue ProteinsNeurotransmissionMotor ActivityHippocampusSynaptic TransmissionReuptakechemistry.chemical_compoundMiceS100 Calcium Binding Protein GDopamineDopaminergic CellmedicineAnimalsAutoreceptorsAlpha-synucleinMice KnockoutNeuronsGeneral NeuroscienceRab3A GTP-Binding ProteinCorpus Striatumrab3A GTP-Binding Proteinnervous system diseasesMice Inbred C57BLSubstantia NigraAmphetaminechemistrynervous systemalpha-SynucleinCalciumFemaleBeta-synucleinNeuroscienceLocomotionmedicine.drug
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Interaction of morphine and haloperidol on agonistic and motor behaviors of male mice.

1997

To further clarify the interaction between opioid and dopaminergic systems, the effects of simultaneous administration of morphine hydrochloride (1.25 or 2.5 mg/kg) and haloperidol (0.1 mg/kg) on aggressive behavior of male mice were explored. Isolated male mice (experimental animals) were confronted in a neutral area with anosmic, group-housed consepecifics (standard opponents) 30 min after injection of both compounds, and aggression was evaluated by estimation of times allocated to 11 different behavioral categories. In the first experiment (which functioned as a pilot study), the two doses of morphine were explored. In the second one, incorporating a more complete experimental design, on…

MaleClinical BiochemistryMice Inbred StrainsPharmacologyMotor ActivityToxicologyBiochemistryBehavioral NeuroscienceMicemedicineHaloperidolAgonistic behaviourAnimalsDrug InteractionsSocial BehaviorBiological PsychiatryPharmacologyMorphineAggressionDopaminergicAntagonistDrug interactionGroomingAnalgesics OpioidOpioidMorphineExploratory BehaviorDopamine AntagonistsHaloperidolmedicine.symptomPsychologyAgonistic Behaviormedicine.drugPharmacology, biochemistry, and behavior
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Gender differences in escape-avoidance behavior of mice after haloperidol administration.

1993

Gender differences in the disruptive effects of haloperidol on some reinforced behaviors have been observed in different species. However, the inhibitory action of haloperidol on the acquisition and performance of escape-avoidance behavior has only been investigated in male subjects. The present experiment was designed to investigate possible gender differences in the effects of haloperidol on the initial phase of an escape-avoidance learning task. Male and female mice of the OF1 strain were given a single training session in a shuttle-box. Thirty minutes prior to the behavioral test, mice were injected IP with haloperidol (0.25 mg/kg) or physiological saline (10 ml/kg). Latencies of escape…

MaleClinical BiochemistryPhysiologyMice Inbred StrainsMotor ActivityToxicologyInhibitory postsynaptic potentialBiochemistryDevelopmental psychologyBehavioral NeuroscienceMiceEscape ReactionmedicineHaloperidolAvoidance LearningAnimalsBiological PsychiatryPharmacologySex CharacteristicsDopaminergicDopamine antagonistControl subjectsInitial phaseToxicityHaloperidolFemalePsychologyHormonemedicine.drugPharmacology, biochemistry, and behavior
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Subthalamic deep brain stimulation improves time perception in Parkinson's disease.

2004

Alterations in temporal estimation have been observed in Parkinson's disease (PD) and have been associated with dopaminergic dysfunction. To investigate whether deep brain stimulation might reverse these abnormalities in PD, patients treated with electrode implantation for subthalamic deep brain stimulation were required to reproduce time intervals in different experimental conditions (off deep brain stimulation/off therapy, on deep brain stimulation/off therapy, on therapy/off deep brain stimulation). Patients treated with deep brain stimulation in off deep brain stimulation/off therapy displayed the anomalous pattern of responses typically observed in PD. When subthalamic deep brain stimu…

MaleDeep brain stimulationParkinson's diseaseDeep brain stimulation; Memory; Parkinsons disease; Time perception;Parkinson's diseasemedicine.medical_treatmentCentral nervous systemElectric Stimulation TherapyNOmemoryParkinsons diseaseBasal gangliamedicineHumansAnalysis of Variance; Parkinson Disease; Humans; Electric Stimulation Therapy; Time Perception; Aged; Middle Aged; Subthalamus; Male; FemaleDeep transcranial magnetic stimulationPrefrontal cortexAgedAnalysis of VarianceSettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicabusiness.industryGeneral NeuroscienceDopaminergicParkinson DiseaseTime perceptionMiddle Agedmedicine.diseasedeep brain stimulationmedicine.anatomical_structureSubthalamusTime PerceptionSettore MED/26 - NeurologiaFemalebusinessNeuroscienceNeuroreport
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Hampered long-term depression and thin spine loss in the nucleus accumbens of ethanol-dependent rats.

2014

Alcoholism involves long-term cognitive deficits, including memory impairment, resulting in substantial cost to society. Neuronal refinement and stabilization are hypothesized to confer resilience to poor decision making and addictive-like behaviors, such as excessive ethanol drinking and dependence. Accordingly, structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing the use of alcohol after chronic ingestion. Here we show that ethanol-dependent rats display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, accompanied by a reduction of tyrosine hydroxylase immunostaining and postsynaptic density 95…

MaleDendritic spineDendritic SpinesGlutamic AcidNucleus accumbensNeurotransmissionMedium spiny neuronSynaptic TransmissionNucleus AccumbensOrgan Culture TechniquesAnimalsRats WistarLong-term depressionLong-Term Synaptic Depressiondopamine synaptic plasticity Golgi glutamateMultidisciplinaryNeuronal PlasticityEthanolDopaminergic NeuronsLong-Term Synaptic DepressionCentral Nervous System DepressantsRatsAlcoholismPNAS PlusSynaptic plasticitySettore BIO/14 - FarmacologiaPsychologyNeurosciencePostsynaptic densityProceedings of the National Academy of Sciences of the United States of America
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[18F]PR04.MZ PET/CT Imaging for Evaluation of Nigrostriatal Neuron Integrity in Patients With Parkinson Disease.

2020

Introduction Degeneration of dopaminergic, nigrostriatal neurons is the hallmark of Parkinson disease (PD), and PET quantification of dopamine transporters is a widely accepted method for differential diagnosis between idiopathic PD and essential tremor. [18F]PR04.MZ is a new PET tracer with excellent imaging properties allowing for precise quantification of striatal and extrastriatal dopamine transporter. Here we describe our initial experience with [18F]PR04.MZ PET/CT in a larger cohort of healthy controls and PD patients as a proof-of-concept study for this tracer. Methods Eighteen healthy subjects, 19 early PD patients (Hoehn-Yahr I–II), and 13 moderate-advanced PD patients (Hoehn-Yahr …

MaleDopamineCaudate nucleusPET imagingSubstantia nigra[18F]PR04.MZ030218 nuclear medicine & medical imagingCohort Studies03 medical and health sciences0302 clinical medicineDopaminePositron Emission Tomography Computed TomographyMedicineHumansRadiology Nuclear Medicine and imagingdopamine transporterDopamine transporterAgedNeuronsDopamine Plasma Membrane Transport ProteinsbiologyEssential tremorbusiness.industryPars compactaPutamenDopaminergicParkinson DiseaseGeneral MedicineOriginal ArticlesMiddle Agedmedicine.diseaseSubstantia Nigranervous system030220 oncology & carcinogenesisbiology.proteinmovement disordersFemalebusinessNuclear medicinemedicine.drugClinical nuclear medicine
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Dual motor responses elicited by ethanol in the posterior VTA: Consequences of the blockade of μ-opioid receptors

2015

A recent hypothesis, based on electrophysiological and behavioural findings, suggests that ethanol simultaneously exerts opposed effects on the activity of dopamine (DA) neurons in the ventral tegmental area (VTA) through two parallel mechanisms, one promoting and the other reducing the GABA release onto VTA DA neurons. In this sense, the activating effects are mediated by salsolinol, a metabolite of ethanol, acting on the μ-opioid receptors (MORs) located in VTA GABA neurons. The inhibitory effects are, however, triggered by the non-metabolized fraction of ethanol which would cause the GABAA receptors-mediated inhibition of VTA DA neurons. Since both trends tend to offset each other, only…

MaleDopamineReceptors Opioid muPoison controlMotor ActivityPharmacologyInhibitory postsynaptic potentialDopaminemental disordersmedicineAnimalsPharmacology (medical)Rats WistarReceptorPharmacologyEthanolChemistryGABAA receptorDopaminergic NeuronsVentral Tegmental AreaReceptors GABA-ARatsVentral tegmental areaPsychiatry and Mental healthElectrophysiologymedicine.anatomical_structurenervous systemOpioidmedicine.drugJournal of Psychopharmacology
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γ1- and γ2-melanocyte stimulating hormones induce central anxiogenic effects and potentiate ethanol withdrawal responses in the elevated plus-maze te…

2008

Little is known about the endogenous functions of gamma1- and gamma2-melanocyte stimulating hormones (gamma1- and gamma2-MSH). Although gamma-MSHs bind to melanocortin receptor subtypes 3 and 4, we have previously shown that these peptides also influence non-melanocortinergic processes, such as dopaminergic and GABAergic. The aim of this study was to determine the effects of gamma1- and gamma2-MSH (at doses 0.3, 1 and 2 nmol/mouse/5 microl) on the anxiety levels in mice in elevated plus maze. Three experimental paradigms were performed to assess the effects of peptides on: a) ethanol withdrawal; b) acute ethanol-induced anxiolytic action; c) peptides per se. We used ethanol as the model sub…

MaleElevated plus mazemedicine.medical_specialtyMelanocyte-stimulating hormonemedicine.drug_classClinical BiochemistryAnxietyToxicologyBiochemistryAnxiolyticMiceBehavioral NeuroscienceMelanocortin receptorInternal medicinemedicineAnimalsMelanocyte-Stimulating HormonesMaze LearningBiological PsychiatryPharmacologyMice Inbred ICRDose-Response Relationship DrugEthanolDopaminergicSubstance Withdrawal SyndromeEndocrinologyAnxiogenicGABAergicPsychologyHormonePharmacology Biochemistry and Behavior
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Cocaine enhances the conditioned rewarding effects of MDMA in adolescent mice.

2015

Although the consumption of cocaine is frequent in young users of MDMA (3,4-methylenedioxymethamphetamine), the influence of exposure to cocaine on the rewarding effects of MDMA in adolescents has not been studied. The purpose of the present work was to evaluate the effect of co-administration of cocaine (1 and 10 mg/kg) and a sub-threshold dose of MDMA (1.25 mg/kg) on the acquisition of conditioned place preference (CPP) (experiment 1). In addition, the effect of pre-treatment with cocaine on MDMA-induced CPP was evaluated (experiment 2). Levels of monoamines in striatum, hippocampus and cortex were measured in both experiments. Our hypotheses were that cocaine co-administration or pre-tre…

MaleGeneral NeuroscienceN-Methyl-34-methylenedioxyamphetamineDopaminergicHippocampusMDMADrug SynergismStriatumPharmacologyConditioned place preferenceDrug synergismMiceMonoamine neurotransmitterCocaineRewardmental disordersConditioning PsychologicalmedicineConditioningAnimalsBiogenic MonoaminesPsychologypsychological phenomena and processesmedicine.drugBrain research bulletin
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