Search results for "Dama"

showing 10 items of 1437 documents

Odpowiedzialność Skarbu Państwa wobec akcjonariuszy (udziałowców) upadłego za szkodę wyrządzoną przez Sąd Upadłościowy poprzez niesłuszne orzeczenie …

2021

Niniejsze opracowanie odnosi się do zagadnień związanych z odpowiedzialnością odszkodowawczą Skarbu Państwa w stosunku do udziałowców dłużnika z tytułu błędnego orzeczenia o opcji upadłości likwidacyjnej zamiast o opcji restrukturyzacji. Postępowanie wszczyna się na wniosek uprawnionego. O otwarciu postępowania decyduje sąd. W praktyce może dojść do sytuacji, w której będą złożone konkurencyjne względem siebie wnioski: albo zmierzające do likwidacji majątku dłużnika, albo mające na celu jego restrukturyzację. Opracowanie odnosi się do dwóch stanów prawnych: do stanu prawnego obowiązującego w latach 2003–2015 i do stanu prawnego zującego od 2016 r. W artykule poddano analizie ustawowe kryter…

restructuring optionlikwidacjarestrukturyzacjathe State Treasury’s liability for damageupadłość likwidacyjnaodpowiedzialność odszkodowawcza Skarb Państwabankruptcy optionliquidationActa Iuridica Resoviensia
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Late Activation of Stress-activated Protein Kinases/c-Jun N-terminal Kinases Triggered by Cisplatin-induced DNA Damage in Repair-defective Cells

2011

Although stress-activated protein kinases/c-Jun N-terminal kinases (SAPK/JNK) are rapidly activated by genotoxins, the role of DNA damage in this response is not well defined. Here we show that the SEK1/MKK4-mediated dual phosphorylation of SAPK/JNK (Thr-183/Tyr-185) correlates with the level of cisplatin-DNA adducts at late times (16–24 h) after drug treatment in both human and mouse cells. Transfection of platinated plasmid DNA also caused SAPK/JNK activation. A defect in transcription-coupled nucleotide excision repair resting on a mutation in Cockayne syndrome group B protein promoted the late SAPK/JNK activation following cisplatin exposure. Signaling to SAPK/JNK was accompanied by act…

rho GTP-Binding ProteinsDNA RepairMAP Kinase Kinase 4DNA repairDNA damageDNA damage response; DNA repair; cisplatin-DNA adducts; SAPK/JNKp38 mitogen-activated protein kinasesAntineoplastic AgentsCell Cycle ProteinsAtaxia Telangiectasia Mutated ProteinsProtein Serine-Threonine KinasesDNA and ChromosomesBiologyBiochemistryAtaxia Telangiectasia Mutated ProteinsDNA AdductsMiceRadiation IonizingAnimalsHumansDNA Breaks Double-StrandedMolecular BiologyReplication protein ACells CulturedMice KnockoutKinaseTumor Suppressor ProteinsJNK Mitogen-Activated Protein KinasesCell BiologyMolecular biologyDNA-Binding ProteinsEnzyme Activationc-Jun N-terminal kinasesbiology.proteinCisplatinSignal TransductionNucleotide excision repairJournal of Biological Chemistry
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Lesiones de hemisferio derecho. Materiales y análisis pragmático (Volumen VII del corpus PerLA).

2011

El corpus PerLA (“Percepción, Afasia y Lenguaje”), surge en el área de Lingüística General de la Universitat de València como respuesta a la necesidad de integrar el estudio de las patologías lingüísticas en las tendencias actuales de la pragmática y la lingüística de corpus. En esta ocasión se presentan seis transcripciones en las que participa un hablante con lesión cerebral de hemisferio derecho, una situación que ha sido poco investigada desde planteamientos lingüístico-logopédicos, y para la que la bibliografía suele proponer síntomas de escaso alcance verbal, como la disprosodia o la agnosia visual. No obstante, el análisis detenido de los datos ecológicos permite comprobar la alterac…

right brain damage:LINGÜÍSTICA [UNESCO]lesiones de hemisferio derecholingüística de corpuscorpus linguisticslingüística clínicaUNESCO::LINGÜÍSTICAclinical linguistics
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La resistibile ascesa del risarcimento punitivo nell’ordinamento italiano (a proposito dell’ordinanza n. 9978/2016 della Corte di Cassazione)

2017

risarcimento - punitive damages - tutela compensativa - tutela restitutoria - tutela sanzionatoria - ordine pubblicoSettore IUS/01 - Diritto Privato
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Alain Damasio, Les furtifs

2019

Clamart, La Volte, 2019, 704 p.; International audience

roman et histoireextrême-gauchealtermondialisme[SHS.HIST] Humanities and Social Sciences/Historyanticipation[SHS.HIST]Humanities and Social Sciences/HistoryAlain Damasio
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Defying Stereotypes with Nanodiamonds: Stable Primary Diamondoid Phosphines

2016

International audience; Direct unequal C-H bond difunctionalization of phosphorylated diamantane was achieved in high yield from the corresponding phosphonates. Reduction of the functionalized phosphonates provides access to novel primary and secondary alkyl/aryl diamantane phosphines. The prepared primary diamantyl phosphines are quite air stable compared to their adamantyl and especially alkyl or aryl analogues. This finding is corroborated by comparing the singly occupied molecular orbital energy levels of the corresponding phosphine radical cations obtained by density functional theory computations.

room-temperaturemolecular tripoddeactivated aryl chlorideshomogeneous catalysts010402 general chemistryDiamondoidselective preparationchemistry01 natural sciencesMedicinal chemistryChemical reaction[ CHIM ] Chemical Scienceschemistry.chemical_compoundOrganic chemistry[CHIM]Chemical SciencesarylationAlkylNanodiamonds ; Diamondoid Phosphines ; diamantane ; adamantane ; adamantylphosphinechemistry.chemical_classification010405 organic chemistryChemistryligandsArylOrganic Chemistrypalladiumphosphorylated adamantanes3. Good health0104 chemical sciencesChemical bondDensity functional theoryDiamantanePhosphine
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Blood Flow Restriction Exercise: Considerations of Methodology, Application, and Safety

2020

safetymedicine.medical_specialtyBFRPhysiologyIschemiaocclusionischemiaMuscle damageBlood flow restrictionlcsh:PhysiologyMuscle hypertrophyInternal medicinemuscle fiber degenerationPhysiology (medical)Occlusionmedicinemuscle hypertrophylcsh:QP1-981business.industryGeneral CommentaryhypoxiaBlood flowHypoxia (medical)medicine.diseaseCardiologyfatiguemedicine.symptombusinessMuscle fiber degenerationFrontiers in Physiology
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Invasive Northern red oaks benefit temnothorax crassispinus (Hymenoptera: Formicidae) ant colonies

2018

Non-native plant species can modify their environment, and their influence on food chains is well recognized. How ever, the phenomenon of non-nutrient dependent interaction between non-native plants and native animals has received little attention to date. The Northern Red Oak (Quercus rubra) is a non-native, invasive tree species in Europe, which strongly negatively influences co-occurring plants. However, a part of the native fauna is able to utilize the resources offered by this species in its non-native range. We studied a common species of wood ant, Temnothorax crassispinus, in forests under canopies of non-native Northern Red Oak and native oaks, Pedunculate Oak (Q. robur), and Sessil…

seed damageInvasive plant speciesfungieducationacornsfood and beveragescavity-nesting ant
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Bcl-xL as a Modulator of Senescence and Aging

2021

Many features of aging result from the incapacity of cells to adapt to stress conditions. When cells are overwhelmed by stress, they can undergo senescence to avoid unrestricted growth of damaged cells. Recent findings have proven that cellular senescence is more than that. A specific grade of senescence promotes embryo development, tissue remodeling and wound healing. However, constant stresses and a weakening immune system can lead to senescence chronicity with aging. The accumulation of senescent cells is directly related to tissue dysfunction and age-related pathologies. Centenarians, the most aged individuals, should accumulate senescent cells and suffer from their deleterious effects,…

senescenceReviewmedicine.disease_causelcsh:Chemistry0302 clinical medicineImmunologic Surveillancelcsh:QH301-705.5SpectroscopyCellular Senescenceimmunosenescence0303 health sciencesapoptosisGeneral MedicineImmunosenescenceComputer Science ApplicationsCell biologyOrgan Specificity030220 oncology & carcinogenesisDisease SusceptibilitycentenariansProtein BindingSignal TransductionSenescencebcl-X ProteinBcl-xLBiologyCatalysisInorganic Chemistry03 medical and health sciencesImmune systemStress PhysiologicalmedicineAnimalsHumansPhysical and Theoretical ChemistrySenolyticMolecular Biology030304 developmental biologyBcl-xLOrganic ChemistryIntrinsic apoptosisagingGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999senolyticsbiology.proteinWound healingOxidative stressBiomarkersDNA DamageInternational Journal of Molecular Sciences
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Silibin hepatoprotection occur via NFKB signaling inhibition in murine nonalcholic fatty liver disease

2009

silibin liver disease mitochondrial damageSettore BIO/16 - Anatomia Umana
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