Search results for "Determinism"

showing 10 items of 88 documents

Soliloquia de humani arbitrii libertate ar potestate / [Constantius Applanus

1496

Sign.: a12, b-z4, &4, t/4, R/4, A-C4, D-K8, L12. - Data completa, 4 octubre, 1496 L. rom. - 2 mides. - 28 lín. - Min. p. inic. - Postil·les marginals. - Filigr.: balança encerclada. - Errors de fol.

Lliure albir i determinisme Obres anteriors a 1800DIG-BH
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Soliloquia de humani arbitrii libertate ar potestate

1496

Sign.: a12, b-z4, &4, t/4, R/4, A-C4, D-K8, L12. - Data completa, 4 octubre, 1496 L. rom. - 2 mides. - 28 lín. - Min. p. inic. - Postil·les marginals. - Filigr.: balança encerclada. - Errors de fol

Lliure albir i determinisme Obres anteriors al 1800
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Rumination for innovation? : analyzing the longitudinal effects of work-related rumination on creativity at work and off-job recovery

2017

ABSTRACTWork-related rumination is not a single construct, but consists of a dimension associated with negative emotions or affect (affective rumination), and a dimension associated with reflective thinking and applying strategies to solve problems (problem-solving pondering). In this three-wave longitudinal study across two years (N = 630) we investigated whether the relationships between work-related rumination, off-job recovery, and creativity at work varied along the two dimensions of work-related rumination. In addition, we tested whether the relationships followed normal, reversed, or reciprocal causation. The results showed, first, that in a one-year perspective affective rumination,…

Longitudinal studymedia_common.quotation_subject050109 social psychologyponderingAffect (psychology)Work relatedDevelopmental psychologyrecoverystress0502 economics and businessmedicine0501 psychology and cognitive sciencesMeasurement invarianceApplied Psychologyta515media_commonPsykologia - Psychology05 social sciencesPerspective (graphical)ruminationReciprocal determinismCreativityinnovationRuminationmedicine.symptomPsychology050203 business & management
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A follow-up study of a genome-wide association scan identifies a susceptibility locus for venous thrombosis on chromosome 6p24.1.

2010

International audience; To identify genetic susceptibility factors conferring increased risk of venous thrombosis (VT), we conducted a multistage study, following results of a previously published GWAS that failed to detect loci for developing VT. Using a collection of 5862 cases with VT and 7112 healthy controls, we identified the HIVEP1 locus on chromosome 6p24.1 as a susceptibility locus for VT. Indeed, the HIVEP1 rs169713C allele was associated with an increased risk for VT, with an odds ratio of 1.20 (95% confidence interval 1.13-1.27, p = 2.86 x 10(-9)). HIVEP1 codes for a protein that participates in the transcriptional regulation of inflammatory target genes by binding specific DNA …

MESH : Transcription Factors[SDV]Life Sciences [q-bio]Genome-wide association study030204 cardiovascular system & hematologyMESH : Chromosomes Human Pair 60302 clinical medicineGenetics(clinical)Genetics (clinical)GeneticsVenous Thrombosis0303 health sciencesMESH: Polymorphism Single NucleotideMESH : Polymorphism Single NucleotideMESH: Genetic Predisposition to DiseaseMESH: Follow-Up StudiesMESH: Transcription FactorsMESH : Venous ThrombosisMESH: Case-Control StudiesDNA-Binding ProteinsChromosomes Human Pair 6MESH : DNA-Binding ProteinsErratumMESH : Genome-Wide Association StudyMESH : Case-Control StudiesMESH: Chromosomes Human Pair 6Locus (genetics)BiologyPolymorphism Single NucleotideGenetic determinism03 medical and health sciencesReportGenetic predispositionGeneticsHumansGenetic Predisposition to DiseaseAlleleGene030304 developmental biologyMESH: Humans[ SDV ] Life Sciences [q-bio]MESH : Humanslinking inflammation protein atherothrombosis sequence riskCase-control studyChromosomeMESH : Follow-Up StudiesCase-Control StudiesMESH: Genome-Wide Association StudyMESH: Venous ThrombosisMESH : Genetic Predisposition to Disease030217 neurology & neurosurgeryMESH: DNA-Binding ProteinsFollow-Up StudiesGenome-Wide Association StudyTranscription Factors
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Genome-wide association scan of quantitative traits for attention deficit hyperactivity disorder identifies novel associations and confirms candidate…

2008

Contains fulltext : 70192.pdf (Publisher’s version ) (Closed access) Attention deficit hyperactivity disorder (ADHD) is a complex condition with environmental and genetic etiologies. Up to this point, research has identified genetic associations with candidate genes from known biological pathways. In order to identify novel ADHD susceptibility genes, 600,000 SNPs were genotyped in 958 ADHD proband-parent trios. After applying data cleaning procedures we examined 429,981 autosomal SNPs in 909 family trios. We generated six quantitative phenotypes from 18 ADHD symptoms to be used in genome-wide association analyses. With the PBAT screening algorithm, we identified 2 SNPs, rs6565113 and rs5526…

MaleCandidate geneGenetics and epigenetic pathways of disease [NCMLS 6]2804 Cellular and Molecular NeuroscienceMedizinGenome-wide association studyNeuroinformatics [DCN 3]Genome2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]Cluster AnalysisGenetics(clinical)Genetics (clinical)Genetics0303 health sciences10058 Department of Child and Adolescent PsychiatryCadherinsPedigreePsychiatry and Mental healthFemaleFunctional Neurogenomics [DCN 2]CDH13AlgorithmsGenetic Markers2716 Genetics (clinical)GenotypeQuantitative Trait Loci610 Medicine & healthSingle-nucleotide polymorphismBiologyQuantitative trait locusPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]medicineADHDAttention deficit hyperactivity disorderHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersGene030304 developmental biologyProbabilityModels GeneticGenome Humancandidate genefamily-based associationmedicine.diseaseIntronsHaplotypesGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivitygenome-wide association030217 neurology & neurosurgeryGenome-Wide Association StudyAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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A high-density SNP linkage scan with 142 combined subtype ADHD sib pairs identifies linkage regions on chromosomes 9 and 16

2008

As part of the International Multi-centre ADHD Gene (IMAGE) project we have completed an affected sibling pair study of 142 narrowly defined DSM-IV combined type ADHD proband-sibling pairs. We found suggestive linkage on chromosomes 9 and 16 with non-parametric multipoint peak LOD scores of 2.13 and 3.1 respectively. There have been several previous ADHD linkage scans. The UCLA study (Fisher et al. 2002; Ogdie et al. 2004; Ogdie et al. 2003), the Dutch study (Bakker et al. 2003), the German study (Hebebrand et al. 2006) and the MGH Study (Faraone et al., submitted) applied the affected sib pair (ASP) strategy; the Columbian study used extended pedigrees ascertained from a population isolate…

MaleGenetics and epigenetic pathways of disease [NCMLS 6]GENOMEWIDE SCANMedizin2804 Cellular and Molecular NeuroscienceCHILDRENComorbidityNeuroinformatics [DCN 3]Severity of Illness IndexDevelopmental psychology2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]HETEROGENEITYIsraelChildGeneticsObserver Variation0303 health sciencesATTENTION-DEFICIT/HYPERACTIVITY DISORDERPSYCHIATRIC-DISORDERSDOPAMINE TRANSPORTER GENEASSOCIATION10058 Department of Child and Adolescent PsychiatryEuropePsychiatry and Mental healthFemalePsychologyChromosomes Human Pair 9linkageFunctional Neurogenomics [DCN 2]GenotypeDEFICIT HYPERACTIVITY DISORDER610 Medicine & healthPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismWhite PeopleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]Genetic linkage1312 Molecular BiologymedicineSNPAttention deficit hyperactivity disorderHumansADHDSiblingMolecular Biology030304 developmental biologyLinkage (software)SiblingsChromosomemedicine.diseaseSib pairsUnited Statesaffected sib pairsGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with HyperactivityCONDUCT DISORDERLod ScoreDISEQUILIBRIUM030217 neurology & neurosurgeryChromosomes Human Pair 16
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Acromesomelic dysplasia Maroteaux type maps to human chromosome 9.

1998

SummaryAcromesomelic dysplasias are skeletal disorders that disproportionately affect the middle and distal segments of the appendicular skeleton. We report genetic mapping studies in four families with acromesomelic dysplasia Maroteaux type (AMDM), an autosomal recessive osteochondrodysplasia. A peak LOD score of 5.1 at recombination fraction 0 was obtained with fully informative markers on human chromosome 9. In three of the four families, the affected offspring are products of consanguineous marriages; if it is assumed that these affected offspring are homozygous by descent for the region containing the AMDM locus, a 6.9-cM AMDM candidate interval can be defined by markers D9S1853 and D9…

MaleGenotypeGenetic LinkageLocus (genetics)Chromosome 9ConsanguinityBiologyOsteochondrodysplasiasGenetic determinismBone and BonesConsanguinityGene mappingmedicineGeneticsHumansGenetics(clinical)OsteochondrodysplasiaGenetics (clinical)GeneticsChromosome 9Chromosome Mappingmedicine.diseaseOsteochondrodysplasiaPedigreeRadiographyMappingAcromesomelic dysplasia Maroteaux typeFemaleChromosome 20Lod ScoreChromosomes Human Pair 9Acromesomelic dysplasiaResearch ArticleMicrosatellite Repeats
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Depressive Symptoms and Alcohol Use are Genetically and Environmentally Correlated Across Adolescence

2010

Depressive symptoms and alcohol use are frequently positively associated during adolescence. This study aimed to assess the heritability of each phenotype across adolescence; to assess potential shared liabilities; to examine changes in the nature of shared liabilities across adolescence; and to investigate potential causal relationships between depressive symptoms and alcohol use. We studied a longitudinally assessed sample of adolescent Finnish twins (N = 1,282) to test hypotheses about genetic and environmental influences on these phenotypes within and across ages, using data from assessments at ages 12, 14, and 17.5 years. The heritability of depressive symptoms is consistent across ado…

MaleHeterozygotemedicine.medical_specialtyAdolescentAlcohol DrinkingnuoruusikäGenetics BehavioralEnvironmentArticleStructural equation modelingDevelopmental psychologyDiseases in TwinsGeneticsmedicineHumansLongitudinal StudiesChildFinlandGenetics (clinical)Ecology Evolution Behavior and SystematicsDepression (differential diagnoses)masentuneisuusCausal modelModels GeneticDepressionPublic healthReciprocal determinismHeritabilityTwin studyAlcoholismHealth psychologyalkoholin käyttöPhenotypeFemalePsychologyDemographyBehavior Genetics
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Phenotype severity in the bladder exstrophy-epispadias complex: analysis of genetic and nongenetic contributing factors in 441 families from North Am…

2011

Objective To identify genetic and nongenetic risk factors that contribute to the severity of the bladder exstrophy-epispadias complex (BEEC). Study design Patients with BEEC from North America (n = 167) and Europe (n = 274) were included. The following data were collected: associated anomalies, parental age at conception, mode of conception, periconceptional folic acid supplementation, maternal risk factors during pregnancy, and environmental risk factors. The patients were divided into 3 subgroups according to phenotype severity: (i) mild, epispadias (n = 43); (ii) intermediate, classic bladder exstrophy (n = 366); and (iii) severe, cloacal exstrophy (n = 31). These subgroups then were com…

MalePediatricsmedicine.medical_treatmentSeverity of Illness IndexIntracytoplasmic sperm injectionPregnancyRisk FactorsSurveys and QuestionnairesSmokingPrenatal CareMiddle AgedCleft PalateEuropePhenotypeVitamin B ComplexFemaleAntacidsMaternal AgeAdultmedicine.medical_specialtyEpispadiasCleft Lip610 Medicine & healthmacromolecular substancesEpispadiasPrenatal careFertilization in VitroGenetic determinismPaternal AgeArticleFolic AcidSeverity of illnessmedicineHumans10220 Clinic for Surgery2735 Pediatrics Perinatology and Child HealthSperm Injections IntracytoplasmicSex DistributionPregnancybusiness.industryBladder Exstrophymedicine.diseaseCloacal exstrophySurgeryBladder exstrophyRadiographyPregnancy Trimester FirstPediatrics Perinatology and Child HealthNorth AmericabusinessThe Journal of pediatrics
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Mutations in the PDS Gene in German Families with Pendred’s Syndrome: V138F Is a Founder Mutation

2003

Pendred's syndrome, an autosomal-recessive condition characterized by congenital sensorineural hearing loss and goiter, is caused by mutations in the PDS gene. Located on chromosome 7q22-q31, it encodes a chloride-iodide transporter expressed in the thyroid, inner ear, and kidney. We investigated the PDS gene of six affected individuals from four unrelated families with Pendred's syndrome by direct sequencing. PDS mutations were identified in homozygous or compound heterozygous state in all six cases. A homozygous missense mutation leading to the amino acid substitution S133T was detected in a family of Turkish origin. The mutations found in the other affected individuals, who originate fro…

MaleThreoninemedicine.medical_specialtyAdolescentTurkeyHearing Loss SensorineuralEndocrinology Diabetes and MetabolismClinical BiochemistryMutation MissenseBiologyCompound heterozygositymedicine.disease_causeBiochemistryGenetic determinismEndocrinologyHypothyroidismGermanyInternal medicineSerinemedicineHumansMissense mutationAlleleChildPendred syndromeGeneticsMutationBase SequenceBiochemistry (medical)HaplotypeInfant NewbornMembrane Transport Proteinsfood and beveragesSyndromemedicine.diseaseFounder EffectPedigreeEndocrinologyAmino Acid SubstitutionHaplotypesSulfate TransportersChild PreschoolMicrosatelliteFemaleCarrier ProteinsThe Journal of Clinical Endocrinology & Metabolism
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