Search results for "Development Biology"

showing 10 items of 80 documents

Molecular and patho-physiological basis of syndromes with developmental anomalies and intellectual disability

2013

Intellectual disability (ID) corresponds to abnormal intellectual performances and adaptive functions, beginning in childhood. It is estimated that 2-3% of individuals develop a ID, which represents a significant medical challenge since people with ID are frequently in situations of social dependence. Overall, a critical involvement of genetic factors in this disease is suspected. To date, several hundreds of genes are known to be responsible for ID. The ID is particularly characterized by extreme clinical and genetic heterogeneity, that made it resistant to conventional genetic studies. However, it is classicaly separated between syndromic ID, which may be clinically recognizable due to as…

Exome sequencingMendelian disorders[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyShprintzen-Goldberg syndromeIntellectual disabilitySyndromes microdélétionnels[SDV.GEN.GH] Life Sciences [q-bio]/Genetics/Human geneticsAnomalies du développementDéficience intellectuelleSéquençage d’exomeMicrodeletionnal syndromesSyndrome de Shprintzen-Goldberg[SDV.BDD] Life Sciences [q-bio]/Development BiologyMultiple congenital anomalies[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyMaladies mendéliennes
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Age-Related Changes in the Gut Microbiota Modify Brain Lipid Composition

2020

PMCID: PMC6970973; International audience; Understanding the molecular mechanisms underlying the changes observed during aging is a prerequisite to design strategies to prevent age-related diseases. Aging is associated with metabolic changes, including alteration in the brain lipid metabolism. These alterations may contribute to the development of pathophysiological conditions. Modifications in the gut microbiota composition are also observed during aging. As communication axes exist between the gut microbiota and the brain and knowing that microbiota influences the host metabolism, we speculated on whether age-associated modifications in the gut microbiota could be involved in the lipid ch…

Fatty Acid DesaturasesMale0301 basic medicinelcsh:QR1-502Gene ExpressionGut floralcsh:MicrobiologyFatty Acids MonounsaturatedMiceCellular and Infection MicrobiologyAging brain[SDV.BDD]Life Sciences [q-bio]/Development BiologyOriginal Researchchemistry.chemical_classificationFatty AcidsAge FactorsBrainLipidscortexInfectious DiseasesFatty Acids Unsaturated[SDV.IMM]Life Sciences [q-bio]/Immunologylipids (amino acids peptides and proteins)SphingomyelinStearoyl-CoA DesaturasePolyunsaturated fatty acidMicrobiology (medical)medicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyFatty Acid ElongasesFADS1FADS2030106 microbiologyImmunologyBiologyliverdigestive systemMicrobiology03 medical and health scienceslipidInternal medicine[SDV.BDD] Life Sciences [q-bio]/Development BiologymedicinemicrobiotaAnimalsGerm-Free LifephospholipidagingFatty acidcholesterolLipid Metabolismbiology.organism_classificationGastrointestinal MicrobiomeTransplantation[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologyEndocrinologychemistryfatty acid[SDV.AEN]Life Sciences [q-bio]/Food and NutritionFrontiers in Cellular and Infection Microbiology
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Membrane potential dye imaging of ventromedial hypothalamus neurons from adult mice to study glucose sensing

2013

Studies of neuronal activity are often performed using neurons from rodents less than 2 months of age due to the technical difficulties associated with increasing connective tissue and decreased neuronal viability that occur with age. Here, we describe a methodology for the dissociation of healthy hypothalamic neurons from adult-aged mice. The ability to study neurons from adult-aged mice allows the use of disease models that manifest at a later age and might be more developmentally accurate for certain studies. Fluorescence imaging of dissociated neurons can be used to study the activity of a population of neurons, as opposed to using electrophysiology to study a single neuron. This is par…

General Chemical Engineeringneurons/cytology/metabolism/ physiologystaining and labeling/ methodsventromedial hypothalamic[ SDV.BA ] Life Sciences [q-bio]/Animal biologyMembrane Potentials0302 clinical medicinePremovement neuronal activity[SDV.BDD]Life Sciences [q-bio]/Development BiologyNeuronsMembrane potential0303 health scienceseducation.field_of_studyGeneral Neuroscience[SDV.BA]Life Sciences [q-bio]/Animal biologynucleus/cytology/metabolism/ physiologyanimalsmedicine.anatomical_structureHypothalamus[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]fluorescent dyes/ chemistryinbred c57blmicePopulationConnective tissuefluorescence/ methodsBiologyGeneral Biochemistry Genetics and Molecular Biologyspectrometry03 medical and health sciencesmaleExtracellularmedicine[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyeducationFluorescent Dyes030304 developmental biologyStaining and LabelingGeneral Immunology and Microbiologymembrane potentials/physiologyMice Inbred C57BLElectrophysiologyGlucoseSpectrometry Fluorescencenervous systemVentromedial Hypothalamic Nucleus[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]NeuronNeuroscience030217 neurology & neurosurgeryglucose/ metabolismNeuroscience
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SILAC labeling coupled to shotgun proteomics analysis of membrane proteins of liver stem/hepatocyte allows to candidate the inhibition of TGF-beta pa…

2014

Background: Despite extensive research on hepatic cells precursors and their differentiated states, much remains to be learned about the mechanism underlying the self-renewal and differentiation.Results: We apply the SILAC (stable isotope labeling by amino acids in cell culture) approach to quantitatively compare the membrane proteome of the resident liver stem cells (RLSCs) and their progeny spontaneously differentiated into epithelial/hepatocyte (RLSCdH). By means of nanoLC-MALDI-TOF/TOF approach, we identified and quantified 248 membrane proteins and 57 of them were found modulated during hepatocyte differentiation. Functional clustering of differentially expressed proteins by Ingenuity …

Hepatocyte differentiationProteomicsStem cellChemistryResearchLiver Stem CellProteomicProteomicsBioinformaticsBiochemistrySILACCell biologyMembrane proteinStable isotope labeling by amino acids in cell cultureTGF beta signaling pathwayHepatocyte; Proteomics; SILAC; Stem cell; Biochemistry; Molecular Biology[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyHepatocyteStem cellShotgun proteomics[SDV.BDD]Life Sciences [q-bio]/Development BiologyMolecular BiologyProteome Science
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A functional analysis of ACP-20, an adult-specific cuticular protein gene from the beetle Tenebrio. Role of an intronic sequence in transcriptional a…

2004

0962-1075 (Print) Comparative Study Journal Article Research Support, Non-U.S. Gov't; A gene encoding the adult cuticular protein ACP-20 was isolated in Tenebrio. It consists of three exons interspersed by two introns, intron 1 interrupting the signal peptide. To understand the regulatory mechanisms of ACP-20 expression, ACP-20 promoter-luciferase reporter gene constructs were transfected into cultured pharate adult wing epidermis. Transfection assays needed the presence of 20-hydroxyecdysone, confirming that ACP-20 is up-regulated by ecdysteroids. Analysis of 5' deletion constructs revealed that three regions are necessary for high levels of transcription. Interaction experiments between i…

MESH : Molecular Sequence Data[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Genes Reporter/physiologyMESH : Transcriptional Activation/geneticsMESH : Introns/geneticsPromoter Regions (Genetics)/drug effects/physiologyExon0302 clinical medicineGenes ReporterTranscriptional regulationTrans-Activation (Genetics)/genetics/*physiologyMESH : Tenebrio/geneticsLuciferasesPromoter Regions GeneticTenebrioPeptide sequenceMESH : Metamorphosis Biological/geneticsComputingMilieux_MISCELLANEOUS0303 health sciencesMESH : Amino Acid SequenceMetamorphosis BiologicalMESH : Luciferases/metabolismEcdysone/metabolism/pharmacology3. Good healthInsect ProteinsMESH : TransfectionSequence AnalysisSignal peptideTranscriptional ActivationEcdysoneanimal structuresSequence analysisMolecular Sequence DataMESH : Transcriptional Activation/physiologyReporter/physiologyBiological/genetics/*physiologyMESH : Insect Proteins/physiologyBiologyLuciferases/metabolismTransfectionTenebrio/*genetics/physiologyMESH : Ecdysone/pharmacology03 medical and health sciencesGeneticsAnimalsAmino Acid Sequence[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyMolecular BiologyGeneMESH : Introns/physiology030304 developmental biologyGene LibraryMESH : Metamorphosis Biological/physiologyReporter gene[SDV.GEN.GPO]Life Sciences [q-bio]/Genetics/Populations and Evolution [q-bio.PE]Base SequenceMetamorphosisIntronIntrons/genetics/physiologyMESH : Ecdysone/metabolismSequence Analysis DNADNAMESH : Gene LibraryMolecular biologyIntronsGenesMESH : Tenebrio/physiologyEpidermis/metabolism Gene LibraryInsect ScienceMESH : Insect Proteins/geneticsMESH : Epidermis/metabolismMESH : Base SequenceMESH : AnimalsEpidermisMESH : Promoter Regions Genetic/drug effects[SDE.BE]Environmental Sciences/Biodiversity and EcologyInsect Proteins/*genetics/*physiology030217 neurology & neurosurgeryEpidermis/metabolismMESH : Promoter Regions Genetic/physiologyMESH : Sequence Analysis DNA
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The sperm of aging male bustards retards their offspring's development.

2015

Understanding whether the sperm of older males has a diminished capacity to produce successful offspring is a key challenge in evolutionary biology. We investigate this issue using 10 years of reproductive data on captive long-lived houbara bustards (Chlamydotis undulata), where the use of artificial insemination techniques means parents can only influence offspring quality via their gametes. Here we show that paternal aging reduces both the likelihood that eggs hatch and the rate at which chicks grow, with older males producing the lightest offspring after the first month. Surprisingly, this cost of paternal aging on offspring development is of a similar scale to that associated with mater…

Male0106 biological sciencesAvian clutch sizeAgingZygoteOffspringmedicine.medical_treatmentmedia_common.quotation_subjectGeneral Physics and AstronomyZoologySemen analysisBiologyInsemination010603 evolutionary biology01 natural sciencesArticleGeneral Biochemistry Genetics and Molecular BiologyBirds03 medical and health sciencesmedicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalsBody Size[ SDV.BDD ] Life Sciences [q-bio]/Development Biology[SDV.BDD]Life Sciences [q-bio]/Development BiologyInsemination Artificial030304 developmental biologymedia_commonGenetics0303 health sciencesMultidisciplinaryZygotemedicine.diagnostic_testReproductionArtificial inseminationAge FactorsGeneral ChemistryClutch SizeSpermatozoaSpermSemen AnalysisFemaleGenetic FitnessReproduction[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Sex-pairing pheromones and reproductive isolation in three sympatric Cornitermes species (Isoptera, Termitidae, Syntermitinae)

2011

International audience; The species-specificity of pairing has been studied in three sympatric Neotropical termites: Cornitermes bequaerti, Cornitermes cumulans and Cornitermes silvestrii (Termitidae, Syntermitinae). Bioassays showed that sex attraction was highly species-specific between C. bequaerti and C cumulans but not between C. cumulans and C. silvestrii. The sex-pairing pheromone of the three species is secreted by the tergal glands of female alates. It consists of a common compound (3Z,6Z,8E)-dodeca-3,6,8-trien-1-ol. In C. bequaerti, this polyunsaturated alcohol is the only compound of the sex-pairing pheromone, whereas it is associated with the oxygenated sesquiterpene (E)-nerolid…

Male0106 biological sciencesPHEROMONAL BLENDPhysiology(3Z6Z8Z)-DODECA-368-TRIEN-1-OLIsopteraAlate(E)-NEROLIDOL010603 evolutionary biology01 natural sciences(Z)-DODEC-3-en-1-OLSexual Behavior AnimalSpecies SpecificityBotanyAnimalsSex AttractantsSPECIES-SPECIFIC PHEROMONE[SDV.BDD]Life Sciences [q-bio]/Development BiologybiologySexual attractionReproduction[SDV.BA]Life Sciences [q-bio]/Animal biology[SDV.BDLR]Life Sciences [q-bio]/Reproductive BiologyReproductive isolationbiology.organism_classificationAttractionSPECIES RECOGNITION010602 entomologyTermitidaeSympatric speciationInsect ScienceSex pheromonePheromoneFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]
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New insights into the pathogenesis of Beckwith-Wiedemann and Silver-Russell syndromes: contribution of small copy number variations to 11p15 imprinti…

2011

International audience; The imprinted 11p15 region is organized in two domains, each of them under the control of its own imprinting control region (ICR1 for the IGF2/H19 domain and ICR2 for the KCNQ1OT1/CDKN1C domain). Disruption of 11p15 imprinting results in two fetal growth disorders with opposite phenotypes: the Beckwith-Wiedemann (BWS) and the Silver-Russell (SRS) syndromes. Various 11p15 genetic and epigenetic defects have been demonstrated in BWS and SRS. Among them, isolated DNA methylation defects account for approximately 60% of patients. To investigate whether cryptic copy number variations (CNVs) involving only part of one of the two imprinted domains account for 11p15 isolated…

MaleBeckwith–Wiedemann syndrome[SDV.GEN] Life Sciences [q-bio]/GeneticsMESH: Base SequenceMESH: DNA MethylationCopy-number variationImprinting (psychology)[SDV.BDD]Life Sciences [q-bio]/Development BiologyGenetics (clinical)GeneticsComparative Genomic Hybridization0303 health sciencesKCNQ1OT1MESH: Polymorphism Single Nucleotide030305 genetics & hereditycopy number variation11p15 regionPedigreegenomic imprintingMESH: Silver-Russell SyndromeDNA methylationBeckwith-Wiedemann syndromeFemaleMESH: DNA Copy Number VariationsMESH: Beckwith-Wiedemann SyndromeAdultDNA Copy Number VariationsMESH: PedigreeBiologyPolymorphism Single Nucleotide03 medical and health sciences[SDV.BDD] Life Sciences [q-bio]/Development BiologyGeneticsmedicineHumansEpigenetics030304 developmental biology[SDV.GEN]Life Sciences [q-bio]/GeneticsMESH: HumansBase SequenceChromosomes Human Pair 11MESH: AdultDNA Methylationmedicine.diseaseMESH: MaleMESH: Genomic ImprintingMESH: Comparative Genomic HybridizationUniparental IsodisomySilver-Russell syndromeMESH: Chromosomes Human Pair 11Genomic imprintingMESH: Femalefetal growthfetal growth.
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Olfaction scaffolds the developing human from neonate to adolescent and beyond

2020

The impact of the olfactory sense is regularly apparent across development. The fetus is bathed in amniotic fluid (AF) that conveys the mother's chemical ecology. Transnatal olfactory continuity between the odours of AF and milk assists in the transition to nursing. At the same time, odours emanating from the mammary areas provoke appetitive responses in newborns. Odours experienced from the mother's diet during breastfeeding, and from practices such as pre-mastication, may assist in the dietary transition at weaning. In parallel, infants are attracted to and recognize their mother's odours; later, children are able to recognize other kin and peers based on their odours. Familiar odours, su…

MaleBreastfeedingAdaptation BiologicalDevelopmental psychology0302 clinical medicineParent-Child RelationsChild[SDV.BDD]Life Sciences [q-bio]/Development BiologyReciprocity (cultural anthropology)media_commonAged 80 and over0303 health sciencesFamily unitcommunicationC100food and beveragesC500ArticlesMiddle AgedSmellMate choiceChild Preschoolbehavior and behavior mechanismsmaternal effectsFemaleGeneral Agricultural and Biological SciencesPsychologypsychological phenomena and processesolfactionAdultAdolescentOffspringmedia_common.quotation_subjectemotionOlfactionsocial cognitionGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesYoung AdultSocial cognitionPerceptionparasitic diseasesHumansNonverbal Communicationattachment030304 developmental biologyAgedfungiInfant NewbornInfantC400Olfactory PerceptionOdorants030217 neurology & neurosurgery
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NOTCH, a new signaling pathway implicated in holoprosencephaly.

2011

International audience; Genetics of Holoprosencephaly (HPE), a congenital malformation of the developing human forebrain, is due to multiple genetic defects. Most genes that have been implicated in HPE belong to the sonic hedgehog signaling pathway. Here we describe a new candidate gene isolated from array comparative genomic hybridization redundant 6qter deletions, DELTA Like 1 (DLL1), which is a ligand of NOTCH. We show that DLL1 is co-expressed in the developing chick forebrain with Fgf8. By treating chick embryos with a pharmacological inhibitor, we demonstrate that DLL1 interacts with FGF signaling pathway. Moreover, a mutation analysis of DLL1 in HPE patients revealed a three-nucleoti…

MaleMESH: Signal TransductionCandidate gene[SDV.GEN] Life Sciences [q-bio]/GeneticsChick EmbryoMESH: Amino Acid SequenceMESH: Base SequenceHoloprosencephalyMESH: Animals[SDV.BDD]Life Sciences [q-bio]/Development BiologyGenetics (clinical)Sequence DeletionGenetics0303 health sciencesReceptors NotchMESH: Androstenediols030305 genetics & heredityMESH: Infant NewbornIntracellular Signaling Peptides and ProteinsGeneral MedicineMESH: Sequence DeletionMESH: Chick EmbryoCell biologyembryonic structuresFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]MESH: Membrane ProteinsSignal transductionMESH: HoloprosencephalySignal TransductionAdultmusculoskeletal diseasesCell signalingcongenital hereditary and neonatal diseases and abnormalitiesanimal structuresMolecular Sequence DataNotch signaling pathwayMESH: Sequence AlignmentBiologyArticle03 medical and health sciencesFGF8[SDV.BDD] Life Sciences [q-bio]/Development BiologyHoloprosencephalyAndrostenediolsGeneticsmedicineAnimalsHumans[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Amino Acid SequenceMolecular Biology030304 developmental biology[SDV.GEN]Life Sciences [q-bio]/GeneticsMESH: Molecular Sequence DataMESH: HumansBase SequenceInfant NewbornMembrane ProteinsMESH: Adultmedicine.diseaseMESH: MaleForebrainMutation testingMESH: Receptors NotchSequence AlignmentMESH: Female
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