Search results for "Development"

showing 10 items of 26949 documents

The metabolism and availability of essential fatty acids in animal and human tissues.

1994

Essential fatty acids (EFA), which are not synthesized in animal and human tissues, belong to the n-6 and n-3 families of polyunsaturated fatty acids (PUFA), derived from linoleic acid (LA, 18:2n-6) and alpha-linolenic acid (LNA, 18:3n-3). Optimal requirements are 3-6% of ingested energy for LA and 0.5-1% for LNA in adults. Requirements in LNA are higher in development. Dietary sources of LA and LNA are principally plants, while arachidonic acid (AA, 20:4n-6) is found in products from terrestrian animals, and eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) are found in products from marine animals. EFA are principally present in dietary triacylglycerols, which should be hydrolyze…

medicine.medical_specialtyAgingLinolenic acidLinoleic acidBiological AvailabilityBiologyIntestinal absorptionchemistry.chemical_compoundInternal medicine[SDV.BDD] Life Sciences [q-bio]/Development BiologymedicineAnimalsHumansNutritional Physiological Phenomena[SDV.BDLR] Life Sciences [q-bio]/Reproductive Biologychemistry.chemical_classificationFatty Acids EssentialNutritional Requirementsfood and beveragesMetabolismEicosapentaenoic acidDietary FatsHormones[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologyBiochemistrychemistryIntestinal AbsorptionDocosahexaenoic acidOrgan Specificitylipids (amino acids peptides and proteins)Arachidonic acidDigestionPolyunsaturated fatty acidReproduction, nutrition, development
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Age-dependent changes of nuclear envelope protein phosphokinase and protein phosphatase activities. Significance for altered nucleo-cytoplasmic mRNA …

1984

Nuclear envelopes are associated with a protein phosphokinase and a phosphoprotein phosphatase, whose activities are modulated by poly(A) in an opposite manner. The activities of these enzymes were determined in nuclear ghosts from liver and oviduct of quails of different age and of different hormone status. Under optimal conditions, kinase activity was found to increase in immature animals 8-fold in response to diethylstilbestrol; co-administration of progesterone had no marked effect on enzyme activity. After the initial burst, the activity of the enzyme increased only slightly during ageing. Two proteins present in nuclear ghosts of Mr 64 000 and of Mr 106 000 are phosphorylated during t…

medicine.medical_specialtyAgingNuclear Envelopemedicine.medical_treatmentPhosphataseOviductsQuailInternal medicinemedicinePhosphoprotein PhosphatasesAnimalsProtein phosphorylationRNA MessengerKinase activityPhosphorylationProtein kinase ADiethylstilbestrolProgesteronebiologyKinaseBiological TransportEnzyme assayMolecular WeightSteroid hormoneEndocrinologyLiverbiology.proteinPhosphorylationFemaleProtein KinasesDevelopmental BiologyMechanisms of ageing and development
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Age-related changes in the endocannabinoid system in the mouse hippocampus.

2015

Previous studies have demonstrated that the endocannabinoid system significantly influences the progression of brain ageing, and the hippocampus is one of the brain regions most vulnerable to ageing and neurodegeneration. We have further examined age-related changes in the hippocampal endocannabinoid system by measuring the levels of anandamide (AEA) and 2-arachidonoylglycerol (2-AG) in young and old mice from two different mouse strains. We found a decrease in 2-AG but not AEA levels in aged mice. In order to identify the cause for 2-AG level changes, we investigated the levels of several enzymes that contribute to synthesis and degradation of 2-AG in the hippocampus. We found a selective …

medicine.medical_specialtyAgingPolyunsaturated Alkamides2-ArachidonoylglycerolHippocampusmetabolism [Hippocampus]Arachidonic AcidsHippocampal formationBiologyHippocampusGlycerideschemistry.chemical_compoundMicepathology [Aging]Internal medicinemetabolism [Arachidonic Acids]medicineanandamideAnimalsglyceryl 2-arachidonateddc:610metabolism [Aging]NeurodegenerationAnandamidemedicine.diseasemetabolism [Endocannabinoids]Endocannabinoid systemMonoacylglycerol lipaseLipoprotein Lipasepathology [Hippocampus]metabolism [Polyunsaturated Alkamides]EndocrinologychemistryAgeingphysiopathology [Hippocampus]lipids (amino acids peptides and proteins)metabolism [Lipoprotein Lipase]metabolism [Glycerides]Developmental BiologyEndocannabinoidsMechanisms of ageing and development
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Glucose 6-P dehydrogenase delays the onset of frailty by protecting against muscle damage.

2021

Background: Frailty is a major age-associated syndrome leading to disability. Oxidative damage plays a significant role in the promotion of frailty. The cellular antioxidant system relies on reduced nicotinamide adenine dinucleotide phosphate (NADPH) that is highly dependent on glucose 6-P dehydrogenase (G6PD). The G6PD-overexpressing mouse (G6PD-Tg) is protected against metabolic stresses. Our aim was to examine whether this protection delays frailty. Methods: Old wild-type (WT) and G6PD-Tg mice were evaluated longitudinally in terms of frailty. Indirect calorimetry, transcriptomic profile, and different skeletal muscle quality markers and muscle regenerative capacity were also investigate…

medicine.medical_specialtyAging[SDV]Life Sciences [q-bio]Respiratory chainOxidative phosphorylationDiseases of the musculoskeletal systemGlucosephosphate DehydrogenaseMitocondrisLipid peroxidation03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineEnvellimentPhysiology (medical)Internal medicineAdipocytemedicineNADPHAnimalsOrthopedics and Sports MedicineRespiratory exchange ratio030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesDisabilityFrailtybusiness.industryMusclesQM1-695Skeletal muscleGlucose 1-DehydrogenaseGlutathioneOriginal Articles3. Good healthMitochondriamedicine.anatomical_structureEndocrinologyGlucosechemistryRC925-935Human anatomyHealthspanOriginal ArticleAntioxidantbusinessReactive oxygen species030217 neurology & neurosurgeryJournal of cachexia, sarcopenia and muscle
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Oral findings in Rett syndrome : a systematic review of the dental literature

2010

Rett syndrome (RS) is a chromosome X-linked genetic neurological disorder characterized by developmental regression, particularly in relation to expressive language and use of the hands, together with profound mental retardation, that almost exclusively affects females. The present review describes the 35 cases of RS Publisher in the indexed literature (Medline) ? the first corresponding to 1985 and the last to the year 2007. Certain oral manifestations of the disease are derived from the drug treatment prescribed to control the disease, while others are common to other clinical conditions characterized by convulsion activity, difficulties for correct oral hygiene, walking problems and/or a…

medicine.medical_specialtyAlternative medicineMEDLINERett syndromeNeurological disorderDiseaseOral hygieneConvulsionRett SyndromemedicineHumansChildPsychiatryGeneral Dentistrybusiness.industrymedicine.disease:CIENCIAS MÉDICAS [UNESCO]OtorhinolaryngologyUNESCO::CIENCIAS MÉDICASBruxismFemaleSurgerymedicine.symptomMouth DiseasesbusinessDevelopmental regression
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Estradiol or genistein prevent Alzheimer's disease-associated inflammation correlating with an increase PPAR gamma expression in cultured astrocytes.

2009

Inflammation has been implicated in neurodegenerative disorders such as Alzheimer's disease (AD). The main inflammatory players in AD are the glial cells which initiate the inflammatory response. One of the earliest neuropathological changes in AD is the accumulation of astrocytes at sites of A beta deposition. It is desirable to find methods of tipping the balance towards anti-inflammatory state. Estrogenic compounds have shown anti-inflammatory and also antioxidant activity. Astrocytes were pretreated with 17-beta estradiol or with genistein, and 48 h later treated with 5 microM amyloid beta (A beta) for 24 h. We found that A beta induces inflammatory mediators, such as cyclooxygenase 2 (…

medicine.medical_specialtyAmyloid betaInterleukin-1betaGenisteinPeroxisome proliferator-activated receptorNitric Oxide Synthase Type IIInflammationEnzyme-Linked Immunosorbent Assaychemistry.chemical_compoundInternal medicinemedicineAnimalsDrug InteractionsMolecular BiologyProtein Kinase InhibitorsCells Culturedchemistry.chemical_classificationCerebral CortexAmyloid beta-PeptidesbiologyDose-Response Relationship DrugEstradiolTumor Necrosis Factor-alphaGeneral NeuroscienceInterleukinEstrogensGenisteinPeptide FragmentsRatsPPAR gammaEndocrinologymedicine.anatomical_structurechemistryGene Expression RegulationCyclooxygenase 2Astrocytesbiology.proteinNeurogliaTumor necrosis factor alphaNeurology (clinical)medicine.symptomDevelopmental BiologyAstrocyteBrain research
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Impact of Sex Differences and Diabetes on Coronary Atherosclerosis and Ischemic Heart Disease

2018

Cardiovascular diseases (CVD) including coronary artery disease (CAD) and ischemic heart disease (IHD) are the main cause of mortality in industrialized countries. Although it is well known that there is a difference in the risk of these diseases in women and men, current therapy does not consider the sexual dimorphism; i.e., differences in anatomical structures and metabolism of tissues. Here, we discuss how genetic, epigenetic, hormonal, cellular or molecular factors may explain the different CVD risk, especially in high-risk groups such as women with diabetes. We analyze whether sex may modify the effects of diabetes at risk of CAD. Finally, we discuss current diagnostic techniques in th…

medicine.medical_specialtyAnatomical structureslcsh:MedicineReviewDisease030204 cardiovascular system & hematologyCoronary artery disease03 medical and health sciences0302 clinical medicineInternal medicineDiabetes mellitusgendercell signalingsexSettore MED/05 - Patologia ClinicaMedicinecardiovascular diseasesCoronary atherosclerosis030304 developmental biology0303 health sciencesdiabetesbusiness.industrylcsh:Rbiomarkers; cell signaling; coronary artery disease; diabetes; gender; sex; womenbiomarkersGeneral Medicinemedicine.diseaseSexual dimorphismdiabeteCardiologybiomarkerwomenbusinessIschemic heartDeveloped countrycoronary artery diseaseJournal of Clinical Medicine
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Inhibition of Delta-like 4 mediated signaling induces abortion in mice due to deregulation of decidual angiogenesis.

2013

Objective: To explore whether the Dll4/Notch1 pathway plays a key role in regulating the vascular endothelial growth factor (VEGF)/VEGF receptor 2 (VEGFR2) driven decidual angiogenesis and related pregnancy through induction of a tip/stalk phenotype. Methods: Progesterone-replaced ovariectomized pregnant mice received a single injection of YW152F (Dll4 blocking antibody, BAb) or placebo at embryonic day (E) 4.5. Animals were sacrificed at different time points; blood and uterus were collected for further analysis. Number of embryos and implantation site, uteri weight, and serum progesterone levels were assessed. Alterations in the tip/stalk phenotype were determined by quantitative immunofl…

medicine.medical_specialtyAngiogenesisNotch pathwayNotch signaling pathwayUterusEmbryonic DevelopmentNeovascularization PhysiologicApoptosisGestational AgeDll4BiologyPregnancy disruptionAndrologychemistry.chemical_compoundMicePregnancyInternal medicinemedicineDeciduaAnimalsAntibodies BlockingAdaptor Proteins Signal TransducingCell ProliferationCell growthDeciduaCalcium-Binding ProteinsIntracellular Signaling Peptides and ProteinsObstetrics and GynecologyMembrane ProteinsEmbryoVEGFVascular Endothelial Growth Factor Receptor-2Vascular endothelial growth factorDisease Models Animalmedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryApoptosiscardiovascular systemEmbryo LossFemaleAngiogenesisDevelopmental BiologySignal TransductionPlacenta
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Diabetes, oxidative stress and therapeutic strategies.

2014

Abstract Background Diabetes has emerged as a major threat to health worldwide. Scope of Review The exact mechanisms underlying the disease are unknown; however, there is growing evidence that excess generation of reactive oxygen species (ROS), largely due to hyperglycemia, causes oxidative stress in a variety of tissues. Oxidative stress results from either an increase in free radical production, or a decrease in endogenous antioxidant defenses, or both. ROS and reactive nitrogen species (RNS) are products of cellular metabolism and are well recognized for their dual role as both deleterious and beneficial species. In type 2 diabetic patients, oxidative stress is closely associated with ch…

medicine.medical_specialtyAntioxidantEndogenous Factorsmedicine.medical_treatmentBiophysicsInflammationEndogeny030204 cardiovascular system & hematologyBiologyPharmacologymedicine.disease_causeBiochemistryAntioxidants03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineDiabetes mellitusmedicineDiabetes MellitusHumansMolecular BiologyReactive nitrogen speciesComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesmedicine.disease3. Good health[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemOxidative StressEndocrinologychemistrymedicine.symptomOxidoreductasesReactive Oxygen SpeciesOxidative stressBiochimica et biophysica acta
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Prevention of apoptosis by deferoxamine during 4 hours of cold cardioplegia and reperfusion: in vitro study of isolated working rat heart model.

2002

INTRODUCTION: Heart transplantation is often accompanied by multiple functional alterations, especially in reperfusion period. These are probably related to the reactive oxygen species (ROS) formation catalyzed by transition metals such as iron and copper, and thus the preservation time of the donor hearts is limited. Metabolic protection of the heart grafts is a permanent objective of numerous experiments. Recently, an iron chelator deferoxamine (DFX) was proposed as antioxidant agent for storage solutions in heart grafts. Oxidative stress is also known to mediate the apoptotic cell death in different tissues during ischemia-reperfusion. METHODS: The aim of this study was to evaluate a pos…

medicine.medical_specialtyAntioxidantmedicine.medical_treatment030204 cardiovascular system & hematologyPharmacologymedicine.disease_causePathology and Forensic Medicine03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicineHeart ratemedicine030304 developmental biologychemistry.chemical_classificationHeart transplantation0303 health sciencesReactive oxygen speciesbusiness.industry3. Good healthDeferoxaminemedicine.anatomical_structurechemistryVentricleApoptosisCardiologybusinessOxidative stressmedicine.drugPathophysiology : the official journal of the International Society for Pathophysiology
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