Search results for "Digestive System"

showing 10 items of 1747 documents

HSP110 promotes colorectal cancer growth through STAT3 activation.

2017

IF 7.932; International audience; Heat shock protein 110 (HSP110) is induced by different stresses and, through its anti-apoptotic and chaperoning properties, helps cells survive these adverse situations. In colon cancers, HSP110 is abnormally abundant. We have recently shown that colorectal cancer patients with microsatellite instability (MSI) had an improved response to chemotherapy because they harbor an HSP110-inactivating mutation (HSP110DE9). In this work, we used patient biopsies, human colorectal cancer cells grown in vitro and in vivo (xenografts), and intestinal crypts to demonstrate that HSP110 is also involved in colon cancer growth. We showed that HSP110 induces colon cancer ce…

STAT3 Transcription Factor0301 basic medicineCancer ResearchColorectal cancerBiopsyMice Nudecolorectal cancer[SDV.CAN]Life Sciences [q-bio]/CancerMouse model of colorectal and intestinal cancerBiologymedicine.disease_causeMolecular oncology[ SDV.CAN ] Life Sciences [q-bio]/CancerSTAT3Mice03 medical and health sciences0302 clinical medicineGrowth factor receptorCell Line TumorGeneticsmedicineAnimalsHumansHSP110 Heat-Shock ProteinsIntestinal MucosaPhosphorylationSTAT3Molecular BiologyCell ProliferationMicrosatellite instabilityCell cyclemedicine.diseaseMolecular biologydigestive system diseases3. Good health030104 developmental biology030220 oncology & carcinogenesisCancer researchbiology.proteinFemaleColorectal NeoplasmsCarcinogenesisNeoplasm TransplantationHSP110Protein Binding
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Signaling molecules: the pathogenic role of the IL-6/STAT-3 trans signaling pathway in intestinal inflammation and in colonic cancer.

2008

Although the precise etiology of inflammatory bowel diseases (IBD) still remains unclear, considerable progress has been made in the identification of novel signal transduction pathways that elucidate the immunopathogenesis involved in the perpetuation of the inflammatory process. As both ulcerative colitis and Crohn's disease are associated with an increased risk for developing colorectal cancer (CRC) and precancerous dysplastic epithelial changes, further studies have concentrated on finding a common signaling pathway that could serve as a mechanistic link between inflammation and associated colonic cancer in IBD. This review presents the current data concerning the pathogenic role of the…

STAT3 Transcription FactorCell signalingColorectal cancerClinical BiochemistryAnti-Inflammatory AgentsInflammationAntineoplastic AgentsDiseaseSuppressor of cytokine signallingDrug DiscoverymedicineAnimalsHumansAutocrine signallingPharmacologybusiness.industryInterleukin-6medicine.diseaseInflammatory Bowel DiseasesUlcerative colitisdigestive system diseasesCell Transformation NeoplasticColonic NeoplasmsCancer researchMolecular Medicinemedicine.symptomSignal transductionbusinessSignal TransductionCurrent drug targets
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Activation of Intestinal Epithelial Stat3 Orchestrates Tissue Defense during Gastrointestinal Infection

2015

Gastrointestinal infections with EHEC and EPEC are responsible for outbreaks of diarrheal diseases and represent a global health problem. Innate first-line-defense mechanisms such as production of mucus and antimicrobial peptides by intestinal epithelial cells are of utmost importance for host control of gastrointestinal infections. For the first time, we directly demonstrate a critical role for Stat3 activation in intestinal epithelial cells upon infection of mice with Citrobacter rodentium - a murine pathogen that mimics human infections with attaching and effacing Escherichia coli. C. rodentium induced transcription of IL-6 and IL-22 in gut samples of mice and was associated with activat…

STAT3 Transcription FactorColonAntimicrobial peptideslcsh:MedicineInflammation-digestive systemMicrobiologyMiceMedizinische FakultätmedicineCitrobacter rodentiumAnimalsHumansddc:610Intestinal Mucosalcsh:ScienceSTAT3PathogenMice KnockoutGastrointestinal tractMultidisciplinarybiologylcsh:REnterobacteriaceae InfectionsEpithelial CellsColitisMucusEpitheliumIntestinesMice Inbred C57BLmedicine.anatomical_structureImmunologybiology.proteinCitrobacter rodentiumlcsh:Qmedicine.symptomResearch ArticlePLoS ONE
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Mouse models of inflammatory bowel disease.

2007

Animal models of intestinal inflammation are indispensable for our understanding of the pathogenesis of Crohn disease and Ulcerative colitis, the idiopathic forms of inflammatory bowel disease in humans. The clinical appearance of human IBD is heterogeneous, a fact that is also reflected by the steadily increasing number of mouse strains displaying IBD like intestinal alterations. The analysis of these models together with genetic studies in humans greatly enhanced our insights into immunoregulatory processes in the gut and led to the generally accepted hypothesis that a deregulated immune response against components of the intestinal microbiota is critically involved in IBD pathophysiology…

STAT3 Transcription FactorPharmaceutical ScienceMice Transgenicdigestive systemInflammatory bowel diseasePathogenesisMiceImmune systemImmunityMedicineAnimalsHumansCrohn's diseasebusiness.industryCrohn diseaseNF-kappa BSTAT4 Transcription Factormedicine.diseaseCadherinsInflammatory Bowel DiseasesUlcerative colitisdigestive system diseasesPathophysiologyImmunity InnateInterleukin-10Disease Models AnimalImmunologybusinessAdvanced drug delivery reviews
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Inhibition of VEGF expression through blockade of Hif1a and STAT3 signalling mediates the anti-angiogenic effect of melatonin in HepG2 liver cancer c…

2013

Background: Hepatocellular carcinoma (HCC) growth relies on angiogenesis via vascular endothelial growth factor (VEGF) release. Hypoxia within tumour environment leads to intracellular stabilisation of hypoxia inducible factor 1 alpha (Hif1α) and signal transducer and activator of transcription (STAT3). Melatonin induces apoptosis in HCC, and shows anti-angiogenic features in several tumours. In this study, we used human HepG2 liver cancer cells as an in vitro model to investigate the anti-angiogenic effects of melatonin. Methods: HepG2 cells were treated with melatonin under normoxic or CoCl2-induced hypoxia. Gene expression was analysed by RT–qPCR and western blot. Melatonin-induced anti-…

STAT3 Transcription FactorTranscriptional ActivationVascular Endothelial Growth Factor ACancer ResearchCarcinoma HepatocellularTranscription GeneticAngiogenesisAngiogenesis InhibitorsApoptosismelatoninP300-CBP Transcription FactorsHif1αBiologyMelatoninSTAT3chemistry.chemical_compoundHypoxia-Inducible Factor 1-AlphamedicineHuman Umbilical Vein Endothelial CellsHumansp300-CBP Transcription FactorsSTAT3Promoter Regions GeneticTube formationNeovascularization PathologicLiver NeoplasmsCobaltHep G2 Cellshepatocellular carcinomaHypoxia-Inducible Factor 1 alpha SubunitVEGFCell Hypoxiadigestive system diseasesCyclic S-OxidesVascular endothelial growth factorGene Expression Regulation NeoplasticVascular endothelial growth factor AOncologychemistryCancer researchbiology.proteinTranslational Therapeuticsmedicine.drugSignal Transduction
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Effectiveness of salivary stimulation using xylitol-malic acid tablets as coadjuvant treatment in patients with gastro-oesophageal reflux disease : e…

2020

[Background] Besides dental erosion syndrome, other oral syndromes could benefit from the stimulation of sali-vary secretion, in patients with gastro-oesophageal reflux disease (GORD). Our aims is evaluate the improvement of oral extra-oesophageal manifestations in patients with GORD using xylitol–malic acid tablets to stimulate salivary secretion.

Salivamedicine.medical_specialtyErythemaMalatesGastroenterology03 medical and health sciences0302 clinical medicineGastro-Internal medicinemedicineotorhinolaryngologic diseasesHumansSalivaGeneral DentistryXylitolOral Medicine and Pathologybusiness.industryResearchdigestive oral and skin physiologyRefluxHeartburnTooth wearfood and beverages030206 dentistryDry mouth:CIENCIAS MÉDICAS [UNESCO]digestive system diseasesClinical trialstomatognathic diseasesOtorhinolaryngologyGastroesophageal refluxErosionRegurgitation (digestion)UNESCO::CIENCIAS MÉDICASGastroesophageal RefluxQuality of LifeSurgerymedicine.symptombusinessTablets
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Oral immunization with HCV-NS3-transformed Salmonella: induction of HCV-specific CTL in a transgenic mouse model.

2001

Abstract Background & Aims: The ability to induce cytotoxic T cells is considered an important feature of a candidate hepatitis C virus (HCV) vaccine. We used an oral immunization strategy with attenuated HCV-NS3–transformed Salmonella typhimurium to deliver DNA directly to the gut-associated lymphoid tissue. Methods: HLA-A2.1 transgenic mice were immunized once with transformed attenuated Salmonella . HCV-specific CD8 + T cells were analyzed in vitro as well as in vivo by challenge of mice with recombinant HCV-NS3 vaccinia virus. Results: Salmonella (10 8 colony-forming units; 20 μg plasmid DNA) induced cytotoxic and IFN-γ–producing CD8 + T cells specific for the immunodominant epitope NS3…

Salmonella typhimuriumViral Hepatitis VaccinesSalmonellavirusesAdministration OralMice TransgenicHepacivirusViral Nonstructural Proteinsmedicine.disease_causeMajor histocompatibility complexEpitopeVirusMicrobiologychemistry.chemical_compoundInterferon-gammaMiceInterferonHLA-A2 AntigenmedicineVaccines DNACytotoxic T cellAnimalsVaccines SyntheticHepatologybiologyGastroenterologyvirus diseasesbiochemical phenomena metabolism and nutritionVirologydigestive system diseaseschemistrybiology.proteinImmunizationVacciniaCD8medicine.drugT-Lymphocytes CytotoxicGastroenterology
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Acidic Monosaccharides become Incorporated into Calcite Single Crystals*.

2020

Streptococcus suis is an encapsulated bacterium and one of the most important swine pathogens and a zoonotic agent for which no effective vaccine exists. Bacterial capsular polysaccharides (CPSs) are poorly immunogenic, but anti-CPS antibodies are essential to the host defense against encapsulated bacteria. In addition to the previously known serotypes 2 and 14, that are non-immunogenic, we have recently purified and described the CPS structures for serotypes 1, 1/2, 3, 7, 8, and 9. Here, we aimed to elucidate how these new structurally diverse CPSs interact with the immune system to generate anti-CPS antibody responses. CPS-stimulated dendritic cells produced significant levels of C–C moti…

SerotypeChemokineSecondary infectionStreptococcus suisCrystal structure010402 general chemistry01 natural sciencesCatalysisMicrobiologyCalcium Carbonatechemistry.chemical_compound03 medical and health sciencesImmune systemMonosaccharideTitermax[SDV.IB.BIO]Life Sciences [q-bio]/Bioengineering/Biomaterials030304 developmental biologychemistry.chemical_classificationCalcite0303 health sciencesMineralsbiology010405 organic chemistryChemistryBiomoleculeOrganic Chemistrycrystal growthMonosaccharidesGeneral ChemistryPolymerbiology.organism_classificationdigestive system diseases3. Good health0104 chemical sciencesbioinspired synthesiX-ray diffractionCalcium carbonateChemical engineeringcarbohydratebiology.proteinAntibodyCrystallizationAcidsBacteriaBiomineralizationMacromoleculeChemistry (Weinheim an der Bergstrasse, Germany)
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Immunity and Nutrition: The Right Balance in Inflammatory Bowel Disease

2022

Inflammatory bowel disease (IBD) is an increasingly urgent medical problem that strongly impairs quality of life for patients. A global rise in incidence has been observed over the last few decades, with the highest incidence rates recorded in North America and Europe. Still, an increased incidence has been reported in the last ten years in newly industrialized countries in Asia, including China and India, both with more than one billion inhabitants. These data underline that IBD is an urgent global health problem. In addition, it is estimated that between 20% and 30% of IBD patients will develop colorectal cancer (CRC) within their lifetime and CRC mortality is approximately 50% amongst IB…

Settore MED/04 - Patologia GeneraleQH301-705.5General MedicineInflammatory Bowel Diseasesdigestive system diseasesGastrointestinal MicrobiomeIntestinesQuality of LifeHumansCytokinesDysbiosisImmune bowel diseasePolyunsaturated fatty acidsBiology (General)
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Hepatocellular carcinoma treatment over sorafenib: epigenetics, microRNAs and microenvironment. Is there a light at the end of the tunnel?

2015

Introduction: Sorafenib is currently the only approved therapy in hepatocellular carcinoma (HCC). Alternative first- and second-line treatments are a significant unmet medical need, and several biologic agents have been tested in recent years, with poor results. Therefore, angiogenic pathways and the cytokine cascade remain possible targets in HCC. Recent studies suggest a role of epigenetic processes, associated with the initiation and development of HCC. In this field, DNA methylation, micro-RNAs (miRNAs) and tumor microenvironment cells became a possible new target for HCC treatment. Areas covered: This review explains the possible role of DNA methylation and histone deacetylase inhibito…

Settore MED/06 - Oncologia MedicaClinical BiochemistrytivantinibEpigenesis GeneticAntineoplastic Agentchemistry.chemical_compoundHistone Deacetylase InhibitorDrug DiscoveryTumor MicroenvironmentMolecular Targeted TherapyplateletmicroRNALiver Neoplasmshepatocellular carcinomaSorafenibVEGFLiver NeoplasmHepatocellular carcinomaDNA methylationMolecular MedicineepigeneticHumanmedicine.drugPhenylurea CompoundSorafenibNiacinamideCarcinoma HepatocellularAntineoplastic AgentsBiologymicroRNAmedicineAnimalsHumansEpigeneticsTivantinibPharmacologyTumor microenvironmentAnimalDrug Discovery3003 Pharmaceutical SciencePhenylurea CompoundsDNA Methylationmedicine.diseasedigestive system diseasesHistone Deacetylase InhibitorsMicroRNAschemistryDrug DesignImmunologyCancer researchHistone deacetylaseExpert opinion on therapeutic targets
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