Search results for "Disorders"

showing 10 items of 4560 documents

Anti-inflammatory and cognitive effects of interferon-β1a (IFNβ1a) in a rat model of Alzheimer’s disease

2018

Background: Aβ 1-42 peptide abnormal production is associated with the development and maintenance of neuroinflammation and oxidative stress in brains from Alzheimer disease (AD) patients. Suppression of neuroinflammation may then represent a suitable therapeutic target in AD. We evaluated the efficacy of IFNβ1a in attenuating cognitive impairment and inflammation in an animal model of AD. Methods: A rat model of AD was obtained by intra-hippocampal injection of Aβ 1-42 peptide (23 μg/2 μl). After 6 days, 3.6 μg of IFNβ1a was given subcutaneously (s.c.) for 12 days. Using the novel object recognition (NOR) test, we evaluated changes in cognitive function. Measurement of pro-inflammatory or …

0301 basic medicineTime Factorsmedicine.medical_treatmentHippocampusCell CountPharmacologymedicine.disease_causeHippocampuslcsh:RC346-429Superoxide Dismutase-10302 clinical medicineNeuroinflammationNF-kBMicrogliaGeneral NeuroscienceMicrofilament ProteinsROSPro-inflammatory cytokineIFNβ1amedicine.anatomical_structureCytokineNeurologyIL-10CytokinesFemalemedicine.symptomAlzheimer's diseaseInterferon beta-1aPro-inflammatory cytokinesImmunologyAβ 1-42InflammationProinflammatory cytokine03 medical and health sciencesCellular and Molecular NeuroscienceHippocampuAlzheimer DiseaseGlial Fibrillary Acidic ProteinmedicineAnimalsAβ1-42Rats WistarSODMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationInflammationAmyloid beta-PeptidesNeuroscience (all)Superoxide Dismutasebusiness.industryResearchCalcium-Binding ProteinsRecognition Psychologymedicine.diseasePeptide FragmentsRatsDisease Models Animal030104 developmental biologyLipid PeroxidationCognition DisordersReactive Oxygen Speciesbusiness030217 neurology & neurosurgeryOxidative stressJournal of Neuroinflammation
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Relationship between adipic acid concentration and the core symptoms of autism spectrum disorders.

2016

Dicarboxylic acids are an important source of information about metabolism and potential physiopathological alterations in children with autism spectrum disorders (ASDs). We measured the concentration between dicarboxylic adipic and suberic acids in children with an ASD and typically-developing (TD) children and analyzed any relationships between the severity of the core symptoms of ASDs and other clinical features (drugs, supplements, drugs, or diet). The core symptoms of autism were evaluated using the DSM-IV criteria, and adipic acid and suberic acid were measured in urine samples. Overall, no increase in the concentration of adipic acid in children with ASDs compared to TD children, how…

0301 basic medicineVitamin bMalePediatricsmedicine.medical_specialtyAdolescentAutism Spectrum DisorderAdipatesUrinebehavioral disciplines and activitiesSeverity of Illness Index03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemental disordersmedicineHumansDicarboxylic AcidsChildBiological PsychiatryAdipic acidbusiness.industrySignificant differenceMetabolismmedicine.diseaseDiagnostic and Statistical Manual of Mental DisordersPsychiatry and Mental health030104 developmental biologyEndocrinologychemistryCase-Control StudiesChild PreschoolDietary SupplementsVitamin B ComplexAutismFemaleCore symptomsCaprylatesbusinessSuberic acid030217 neurology & neurosurgeryPsychiatry research
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Clinical Correlates of Functional Motor Disorders: An Italian Multicenter Study

2020

Background\ud Functional motor disorders (FMDs) are abnormal movements that are significantly altered by distractive maneuvers and are incongruent with movement disorders seen in typical neurological diseases.\ud \ud Objective\ud The objectives of this article are to (1) describe the clinical manifestations of FMDs, including nonmotor symptoms and occurrence of other functional neurological disorders (FND); and (2) to report the frequency of isolated and combined FMDs and their relationship with demographic and clinical variables.\ud \ud Methods\ud For this multicenter, observational study, we enrolled consecutive outpatients with a definite diagnosis of FMDs attending 25 tertiary movement …

0301 basic medicineWeaknessPediatricsmedicine.medical_specialtyMovement disordersfunctional neurological disordersdiagnosisPopulationfunctional weakneDisease030105 genetics & heredityfunctional weakness03 medical and health sciences0302 clinical medicinefunctional neurological disordermedicineeducationResearch Articleseducation.field_of_studyfunctional neurological disorders; functional dystonia; functional tremor; functional weakness; diagnosisbusiness.industryfunctional neurological disorders functional dystonia functional tremor functional weakness diagnosisFunctional weaknessfunctional dystoniatremorNeurologyMulticenter studyAnxietyfunctional tremorSettore MED/26 - NeurologiaObservational studydystoniaNeurology (clinical)medicine.symptombusinessfunctional neurological disorders functional dystonia functional tremor functional weakness diagnosis.030217 neurology & neurosurgery
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New insights into the clinical and molecular spectrum of the novel CYFIP2-related neurodevelopmental disorder and impairment of the WRC-mediated acti…

2021

International audience; Purpose: A few de novo missense variants in the cytoplasmic FMRP-interacting protein 2 (CYFIP2) gene have recently been described as a novel cause of severe intellectual disability, seizures, and hypotonia in 18 individuals, with p.Arg87 substitutions in the majority.Methods: We assembled data from 19 newly identified and all 18 previously published individuals with CYFIP2 variants. By structural modeling and investigation of WAVE-regulatory complex (WRC)-mediated actin polymerization in six patient fibroblast lines we assessed the impact of CYFIP2 variants on the WRC.Results: Sixteen of 19 individuals harbor two previously described and 11 novel (likely) disease-ass…

0301 basic medicine[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyWAVEregulatory complex (WRC)030105 genetics & heredityBiologyArticleIntellectual disability; Epilepsy; CYFIP2; WAVE-regulatory complex (WRC); WASF03 medical and health sciencesNeurodevelopmental disorderSeizuresWAVE-regulatory complex (WRC)medicineCYFIP2Missense mutationHumansGenetics(clinical)WASFGeneGenetics (clinical)ActinAdaptor Proteins Signal TransducingGenetics/dk/atira/pure/subjectarea/asjc/2700/2716medicine.diseaseActin cytoskeletonPhenotypeHypotoniaActins3. Good healthddc:030104 developmental biology[SDV.BDD.EO]Life Sciences [q-bio]/Development Biology/Embryology and OrganogenesisNeurodevelopmental Disordersintellectual disabilityCYFIP2epilepsymedicine.symptom
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Evaluation of DNA Methylation Episignatures for Diagnosis and Phenotype Correlations in 42 Mendelian Neurodevelopmental Disorders

2020

Contains fulltext : 218274.pdf (Publisher’s version ) (Closed access) Genetic syndromes frequently present with overlapping clinical features and inconclusive or ambiguous genetic findings which can confound accurate diagnosis and clinical management. An expanding number of genetic syndromes have been shown to have unique genomic DNA methylation patterns (called "episignatures"). Peripheral blood episignatures can be used for diagnostic testing as well as for the interpretation of ambiguous genetic test results. We present here an approach to episignature mapping in 42 genetic syndromes, which has allowed the identification of 34 robust disease-specific episignatures. We examine emerging pa…

0301 basic medicine[SDV]Life Sciences [q-bio]Computational biology030105 genetics & heredityBiologyPediatricsArticleCohort Studiesmolecular diagnostics03 medical and health sciencessymbols.namesakeGenetic HeterogeneityGene duplicationGeneticsHumansHunter-McAlpine syndromeGenetics (clinical)Mass screening030304 developmental biologyEpiSignGenetics0303 health sciencesNeurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]DNA methylationGenetic heterogeneity030305 genetics & heredityCorrectionSyndromeDNA MethylationMolecular diagnosticsPhenotypePenetranceHuman genetics3. Good healthepisignaturegenomic DNA030104 developmental biologyPhenotypeNeurodevelopmental DisordersDNA methylationuncertain clinical casesMendelian inheritancesymbolsIdentification (biology)VUS classification
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Physiological Functions of the β-Site Amyloid Precursor Protein Cleaving Enzyme 1 and 2

2017

BACE1 was discovered as the β-secretase for initiating the cleavage of amyloid precursor protein (APP) at the β-secretase site, while its close homology BACE2 cleaves APP within the β-amyloid (Aβ) domain region and shows distinct cleavage preferences in vivo. Inhibition of BACE1 proteolytic activity has been confirmed to decrease Aβ generation and amyloid deposition, and thus specific inhibition of BACE1 by small molecules is a current focus for Alzheimer’s disease therapy. While BACE1 inhibitors are being tested in advanced clinical trials, knowledge regarding the properties and physiological functions of BACE is highly important and this review summarizes advancements in BACE1 research ov…

0301 basic medicineamyloid plaquessecretaseReviewamyloid precursor proteinBiology03 medical and health sciencesCellular and Molecular Neurosciencemental disordersAmyloid precursor proteinaspartic proteaseBACE substratesGlucose homeostasisMolecular Biologychemistry.chemical_classificationNeurogenesisBACE2P3 peptideBACE1Biochemistry of Alzheimer's disease030104 developmental biologyEnzymechemistryBiochemistrySynaptic plasticitybiology.proteinAmyloid precursor protein secretaseAlzheimer’s diseaseNeuroscienceFrontiers in Molecular Neuroscience
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Hypocellularity in the murine model for Down Syndrome Ts65Dn is not affected by adult neurogenesis

2016

Down syndrome (DS) is caused by the presence of an extra copy of the chromosome 21 and it is the most common aneuploidy producing intellectual disability. Neural mechanisms underlying this alteration may include defects in the formation of neuronal networks, information processing and brain plasticity. The murine model for DS, Ts65Dn, presents reduced adult neurogenesis. This reduction has been suggested to underlie the hypocellularity of the hippocampus as well as the deficit in olfactory learning in the Ts65Dn mice. Similar alterations have also been observed in individuals with DS. To determine whether the impairment in adult neurogenesis is, in fact, responsible for the hypocellularity …

0301 basic medicineanimal diseasesHippocampusSubventricular zoneBiotecnologiaHippocampusSubgranular zonelcsh:RC321-57103 medical and health sciences0302 clinical medicinedoublecortinNeuroplasticitymental disordersmedicineBrdUlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchbiologyGeneral NeuroscienceNeurogenesisOlfactory BulbOlfactory bulbDoublecortinCell biologyadult neurogenesisTs65Dn mice030104 developmental biologymedicine.anatomical_structureHypocellularityPsicobiologianervous systembiology.proteinDown SyndromeKi67Neuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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Resveratrol in Autism Spectrum Disorders: Behavioral and Molecular Effects

2020

Resveratrol (RSV) is a polyphenolic stillbenoid with significant anti-oxidative and anti-inflammatory properties recently tested in animal models of several neurological diseases. Altered immune alteration and oxidative stress have also been found in patients with autism spectrum disorders (ASD), and these alterations could add to the pathophysiology associated with ASD. We reviewed the current evidence about the effects of RSV administration in animal models and in patients with ASD. RSV administration improves the core-symptoms (social impairment and stereotyped activity) in animal models and it also displays beneficial effects in other behavioral abnormalities such as hyperactivity, anxi…

0301 basic medicineantioxidantPhysiologyClinical BiochemistryReviewResveratrolBioinformaticsmedicine.disease_causeBiochemistrylaw.inventionnatural compound03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemRandomized controlled triallawMedicinedevelopmental disordersMolecular BiologyRisperidonebusiness.industryanimal modellcsh:RM1-950Cell Biologymedicine.diseasePathophysiologylcsh:Therapeutics. Pharmacology030104 developmental biologychemistryAutismAnxietynutraceuticalmedicine.symptombusiness030217 neurology & neurosurgeryOxidative stressmedicine.drugAntioxidants
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Melatonin in Early Nutrition: Long-Term Effects on Cardiovascular System

2021

Breastfeeding protects against adverse cardiovascular outcomes in the long term. Melatonin is an active molecule that is present in the breast milk produced at night beginning in the first stages of lactation. This indoleamine appears to be a relevant contributor to the benefits of breast milk because it can affect infant health in several ways. The melatonin concentration in breast milk varies in a circadian pattern, making breast milk a chrononutrient. The consumption of melatonin can induce the first circadian stimulation in the infant’s body at an age when his/her own circadian machinery is not functioning yet. This molecule is also a powerful antioxidant with the ability to act on infa…

0301 basic medicineantioxidantcardiovascular disordersBreastfeedingPhysiologymelatoninStimulationReviewGut floraCardiovascular System0302 clinical medicinenewbornLactationNutritional Physiological PhenomenaBiology (General)Infant Nutritional Physiological PhenomenaSpectroscopybiologycardiovascular programmingfood and beveragesGeneral MedicineCircadian RhythmComputer Science ApplicationsChemistryBreast Feedingmedicine.anatomical_structureearly life nutritionbreast milkDisease Susceptibilitymedicine.symptomMetabolic Networks and Pathwaysmedicine.drugQH301-705.5InflammationBreast milkCatalysisInorganic ChemistryMelatonin03 medical and health sciencesmedicineAnimalsHumansLactationCircadian rhythmPhysical and Theoretical ChemistryQD1-999Molecular Biologygut microbiotabusiness.industryOrganic ChemistryInfant Newbornbiology.organism_classificationGastrointestinal MicrobiomeOxidative Stress030104 developmental biologycircadian rhythmsbusiness030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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MicroRNAs Dysregulation and Metabolism in Multiple System Atrophy.

2019

Multiple system atrophy (MSA) is an adult onset, fatal disease, characterized by an accumulation of alpha-synuclein (α-syn) in oligodendroglial cells. MicroRNAs (miRNAs) are small non-coding RNAs involved in post-translational regulation and several biological processes. Disruption of miRNA-related pathways in the central nervous system (CNS) plays an important role in the pathogenesis of neurodegenerative diseases, including MSA. While the exact mechanisms underlying miRNAs in the pathogenesis of MSA remain unclear, it is known that miRNAs can repress the translation of messenger RNAs (mRNAs) that regulate the following pathogenesis associated with MSA: autophagy, neuroinflammation, α-syn …

0301 basic medicineautophagyalpha-synucleinCentral nervous systemmultiple system atrophyReviewBiologylcsh:RC321-571neuroinflammationPathogenesis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAtrophystomatognathic systemmicroRNAmental disordersmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeuroinflammationAlpha-synucleinmicroRNAGeneral NeuroscienceAutophagyTranslation (biology)medicine.diseaseCell biologynervous system diseases030104 developmental biologymedicine.anatomical_structurechemistrynervous system030217 neurology & neurosurgeryNeuroscienceFrontiers in neuroscience
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