Search results for "Disposition"

showing 10 items of 832 documents

Reducing Body Myopathy with Cytoplasmic Bodies and Rigid Spine Syndrome: A Mixed Congenital Myopathy

2001

At the age of five years a male child started to develop a progressive rigid spine, torsion scoliosis, and flexion contractures of his elbows, knees, hips, and ankles owing to severe proximal and distal muscle weakness. He had three muscle biopsies from three different muscles at ages 7, 11, and 14 years, respectively. Myopathologically, these muscle tissues contained numerous inclusions which, at the ultrastructural level, turned out to be reducing bodies and cytoplasmic bodies, often in close spatial proximity. Similar histological inclusions, although not further identified by histochemistry and electron microscopy, were seen in his maternal grandmother's biopsied muscle tissue who had d…

AdultMaleMuscle tissuePathologymedicine.medical_specialtyWeaknessScoliosisSpinal Muscular Atrophies of ChildhoodSarcomereMyositis Inclusion BodymedicineHumansGenetic Predisposition to DiseaseMuscle SkeletalMyopathyMyositisAgedInclusion Bodiesbusiness.industrySyndromeGeneral MedicineAnatomymedicine.diseasePenetrancePedigreemedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthDisease ProgressionLordosisFemaleDesminNeurology (clinical)medicine.symptombusinessMyopathies Structural CongenitalNeuropediatrics
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TNFalpha, IFNgamma and IL-10 gene polymorphisms in a sample of Sicilian patients with coeliac disease.

2005

Coeliac disease is associated with DQ2 and DQ8 alleles, but other genes also confer an additional genetic risk.Defining whether the genetic profiles of interleukin-10, tumour necrosis factor alpha and interferon gamma are associated with an increased coeliac disease risk.The functionally gene polymorphisms of tumour necrosis factor alpha (-308G/A), interferon gamma (+874T/A) and interleukin-10 (-1082G/A) were typed using sequence specific primer-polymerase chain reaction in 110 Sicilian coeliac disease patients and in 220 Sicilian healthy controls.No differences in genotype frequencies of interleukin-10 polymorphisms were found between coeliac disease patients and healthy controls. A signif…

AdultMaleNecrosisAdolescentGenotypeCoeliac diseaseInterferon-gammaGene FrequencymedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneSicilyPolymorphism GeneticHepatologybusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantGluten intoleranceMiddle Agedmedicine.diseaseGenotype frequencyInterleukin-10Interleukin 10Celiac DiseaseCase-Control StudiesChild PreschoolImmunologyTumor necrosis factor alphaFemalemedicine.symptombusinessDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
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Association of a variant in the muscarinic acetylcholine receptor 2 gene (CHRM2 ) with nicotine addiction

2009

Genetic factors contribute to the overall risk of developing nicotine addiction, which is the major cause of preventable deaths in western countries. However, knowledge regarding specific polymorphisms influencing smoking phenotypes remains scarce. In the present study we provide evidence that a common single nucleotide polymorphism (SNP) in the 5′ untranslated region of CHRM2, the gene coding for the muscarinic acetylcholine receptor 2 is associated with nicotine addiction. CHRM2 was defined as a candidate gene for nicotine addiction based on previous evidence that linked variations in CHRM2 to alcohol and drug dependence. A total of more than 5,500 subjects representative of the German po…

AdultMaleNicotineCandidate geneAdolescentmedia_common.quotation_subjectSingle-nucleotide polymorphismBiologyBioinformaticsNicotineCellular and Molecular NeuroscienceMuscarinic acetylcholine receptormedicineHumansSNPGenetic Predisposition to DiseaseAlleleAllelesGenetics (clinical)Agedmedia_commonAged 80 and overGeneticsReceptor Muscarinic M2AddictionSmokingGenetic VariationTobacco Use DisorderOdds ratioMiddle AgedPsychiatry and Mental healthFemalemedicine.drugAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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Association Study of Nonsynonymous Single Nucleotide Polymorphisms in Schizophrenia

2010

Genome-wide association studies using several hundred thousand anonymous markers present limited statistical power. Alternatively, association studies restricted to common nonsynonymous single nucleotide polymorphisms (nsSNPs) have the advantage of strongly reducing the multiple testing problem, while increasing the probability of testing functional single nucleotide polymorphisms (SNPs).We performed a case-control association study of common nsSNPs in Galician (northwest Spain) samples using the Affymetrix GeneChip Human 20k cSNP Kit, followed by a replication study of the more promising results. After quality control procedures, the discovery sample consisted of 5100 nsSNPs at minor allel…

AdultMaleNonsynonymous substitutionGenotype"psychosis"methods [Genetic Association Studies]"mental brain homeostasis"Single-nucleotide polymorphismBiologygenetics [Cation Transport Proteins]Polymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineHumansGenetic Predisposition to Diseasegenetics [Schizophrenia]ddc:610statistics & numerical data [Genetic Association Studies]genetics [Genetic Predisposition to Disease]Cation Transport ProteinsGenetic Association StudiesBiological PsychiatryAged030304 developmental biology"ZIP8"Aged 80 and overGenetics0303 health sciences"mental brain homeostasis"; "psychosis"; "metal ion transporters"; "ZIP8"; "whole-genome assosiation"; "SLC39A3"Zip8 protein humanMiddle Aged3. Good health"whole-genome assosiation"SpainCase-Control StudiesSchizophreniaFemale"SLC39A3""metal ion transporters"030217 neurology & neurosurgeryBiological Psychiatry
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High prevalence of BRCA1 deletions in BRCAPRO-positive patients with high carrier probability.

2007

Mutation screening of the BRCA1 and BRCA2 genes in probands with familial breast/ovarian cancer has been greatly improved by the multiplex ligation-dependent probe amplification (MLPA) assay able to evidence gene rearrangements not detectable by standard screening methods. However, no criteria for selection of cases to be submitted to the MLPA test have been reported yet. We used the BRCAPro software for the selection of familial breast/ovarian cancer probands investigated with the MLPA approach after negative BRCA1/2 conventional mutation screening. One hundred and seventy-seven probands were investigated for germline BRCA1/2 mutations after assessment of genetic risk using BRCAPro. Proban…

AdultMaleOncologyProbandcongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyendocrine system diseasesBreast NeoplasmsGermlineBreast Neoplasms MaleGermline mutationBreast cancerRisk FactorsInternal medicinePrevalenceHumansMedicineGenetic Predisposition to DiseaseMultiplexMultiplex ligation-dependent probe amplificationskin and connective tissue diseasesAgedSequence DeletionOvarian NeoplasmsGeneticsBRCA1 Proteinbusiness.industryGenetic Carrier ScreeningProstatic NeoplasmsHematologyMiddle Agedmedicine.diseaseBRCA1 BRCA2 BRCAPro breast cancer MLPA ovarian cancerPedigreeOncologyMutation (genetic algorithm)FemalebusinessOvarian cancerSoftware
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Interaction of genetic vulnerability to schizophrenia and Communication Deviance of adoptive parents associated with MMPI schizophrenia vulnerability…

2008

The aim of this study was to establish possible genotype-environment interaction in high-risk and low-risk adoptees' vulnerability to schizophrenia. The study population consisted of a subgroup of 41 adoptive families with a high genetic risk adoptee and 58 families with a low genetic risk adoptee from the Finnish Adoptive Family Study of Schizophrenia. Communication style was assessed based on the Communication Deviance (CD) of the adoptive parents, and the adoptees' vulnerability indicators were measured with the Minnesota Multiphasic Personality Inventory (MMPI). Taken separately, only the genetic liability to schizophrenia, but not the communication style of the adoptive parents, was si…

AdultMaleParentsmedicine.medical_specialtyAdolescentGenotypeSchizophrenia (object-oriented programming)VulnerabilityHostilityEnvironmentSeverity of Illness IndexDevelopmental psychologyMinnesota Multiphasic Personality InventoryCommunication devianceMMPIAdoptionmedicineHumansGenetic Predisposition to DiseaseGenetic riskPsychiatryGenetic vulnerabilityCommunicationPsychiatry and Mental healthSchizophreniaPopulation studyFemalemedicine.symptomPsychologyNordic journal of psychiatry
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Gene-environment interaction as a predictor of early adjustment in first episode psychosis.

2017

Abstract Background This study aims to explore the gene-environment interaction hypothesis applied to pre-symptomatic neurodevelopmental phenotypes of first episode psychosis (FEP), that is, genetic factors might increase vulnerability to the effects of environmental adverse conditions occurring at later stages of development. Methods We constructed a schematic ‘two-hit’ model, with Val/Val homozygosity for the catechol- O -methyltransferase ( COMT ) Val158Met polymorphism as the ‘first hit’ and history of obstetric complications and parental socioeconomic status as ‘second hits’. Early adjustment, measured using the Premorbid Adjustment Scale, was considered the main outcome. The study pop…

AdultMaleParentsmedicine.medical_specialtyGenotypePremorbid Adjustment ScaleCatechol O-MethyltransferasePolymorphism Single NucleotideStatistics Nonparametric03 medical and health sciencesYoung Adult0302 clinical medicineMethioninePolymorphism (computer science)First episode psychosismedicineHumansGenetic Predisposition to DiseaseGene–environment interactionPsychiatrySocioeconomic statusBiological PsychiatryRetrospective StudiesAdverse conditionsValinemedicine.disease030227 psychiatryPsychiatry and Mental healthPsychotic DisordersSchizophreniaPopulation studyFemaleGene-Environment InteractionPsychology030217 neurology & neurosurgeryDemographySchizophrenia research
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Missense PANK2 mutation without "eye of the tiger" sign: MR findings in a large group of patients with pantothenate kinase-associated neurodegenerati…

2011

Purpose: To present some unusual MR findings in a group of patients from the south-west of the Dominican Republic suffering from Pantothenate Kinase Associated Neurodegeneration (PKAN). Materials and Methods: Twenty patients and one preclinical case homozygous for the PANK2 mutation, 13 heterozygous gene carriers and 14 healthy volunteers were scanned prospectively using a 3 Tesla system. Results: All patients showed the typical signal reduction within the globus pallidus and the substantia nigra. A surprising finding was the absence of the bright spot (“tiger's eye”) in the medial part of the pallidum in 6 patients, but not in the preclinical case. Both fractional anisotropy (FA) and mean …

AdultMalePathologymedicine.medical_specialtyHeterozygoteInternal capsuleAdolescentMutation MissenseSubstantia nigraSensitivity and SpecificityPantothenate kinase-associated neurodegenerationWhite matterYoung AdultFractional anisotropymedicineMissense mutationHumansRadiology Nuclear Medicine and imagingGenetic Predisposition to DiseaseChildAgedPantothenate Kinase-Associated Neurodegenerationbusiness.industryBrainReproducibility of ResultsMiddle AgedPANK2medicine.diseaseMagnetic Resonance ImagingPhosphotransferases (Alcohol Group Acceptor)medicine.anatomical_structureGlobus pallidusnervous systemFemalebusinessJournal of magnetic resonance imaging : JMRI
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Malignant paraganglioma caused by a novel germline mutation of the succinate dehydrogenase D-gene--a case report.

2008

Background Paragangliomas of the head and neck are rare, mostly benign tumors. Approximately 10% to 15% of paragangliomas are caused by mutations in the succinate dehydrogenase (SDH) genes B, C, or D. These are often multifocal as part of paraganglioma syndromes and hormone secreting, and malignant particularly associated with mutations in SDHB. Methods and Results A 29-year-old man was seen with recurrent paraganglioma. The patient's father reportedly suffered from bilateral carotid body tumors. Imaging studies showed metastases in both lungs and the liver. There was no increased hormone production by the tumor. Sequence analysis of the SDH genes revealed a novel C to T nonsense mutation i…

AdultMalePathologymedicine.medical_specialtySDHBNonsense mutationmedicine.disease_causeRisk AssessmentSkull Base NeoplasmsPheochromocytomaParagangliomaTreatment RefusalGermline mutationParagangliomamedicineMalignant ParagangliomaHumansGenetic Predisposition to DiseaseGerm-Line MutationNeoplasm StagingMutationbusiness.industryBiopsy Needlemedicine.diseaseImmunohistochemistryPedigreeSuccinate DehydrogenaseOtorhinolaryngologyPositron-Emission TomographySDHDbusinessHeadneck
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Pro-inflammatory gene variants in myocardial infarction and longevity: implications for pharmacogenomics.

2008

Inflammation and genetics play an important role in the pathogenesis of coronary heart disease (CHD). However, despite the increasing appreciation of the role of genetics in CHD and myocardial infarction (MI) pathogenesis, pharmacogenomic approaches to uncover drug target have not been extensively explored. Cyclo-oxygenases (COXs) and 5-lipoxygenase (5-LO) are the key enzymes in the conversion of arachidonic acid to prostaglandins (PG) and leukotrienes (LT) and are implicated in a wide variety of inflammatory disorders, including atherosclerosis. In fact, PGE2 activates Matrix Metallo-proteinases whereas LTB4 is a chemoactractant for monocytes and activates gene expression in inflammatory c…

AdultMalePathologymedicine.medical_specialtymedia_common.quotation_subjectLongevityMyocardial InfarctionIMMUNOGENETICSINFARCTIONINFLAMMATIONLONGEVITYPHARMACOGENOMICSInflammationDiseaseBioinformaticsPathogenesisYoung AdultDrug Delivery SystemsRisk FactorsDrug DiscoverymedicineHumansGenetic Predisposition to DiseaseMyocardial infarctionAlleleAllelesmedia_commonAged 80 and overInflammationPharmacologyArachidonate 5-Lipoxygenasebusiness.industryAge FactorsLongevityMiddle Agedmedicine.diseasePhenotypeCyclooxygenase 2PharmacogeneticsPharmacogenomicsFemalemedicine.symptombusinessPharmacogenetics
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