Search results for "Disposition"

showing 10 items of 832 documents

Serotonin transporter gene polymorphism in eating disorders: Data from a new biobank and META-analysis of previous studies.

2016

Objectives Growing interest focuses on the association between 5-HTTLPR polymorphism and eating disorders (ED), but published findings have been conflicting. Methods The Italian BIO.VE.D.A. biobank provided 976 samples (735 ED patients and 241 controls) for genotyping. We conducted a literature search of studies published up to 1 April 2015, including studies reporting on 5HTTLPR genotype and allele frequencies in obesity and/or ED. We ran a meta-analysis, including data from BIO.VE.D.A. – comparing low and high-functioning genotype and allele frequencies in ED vs. controls. Results Data from 21 studies, plus BIO.VE.D.A., were extracted providing information from 3,736 patients and 2,707 co…

medicine.medical_specialtyAnorexia Nervosa5-HTTLPR; anorexia nervosa; binge eating; bulimia nervosa; Eating disorders; Biological Psychiatry; Psychiatry and Mental Health5-HTTLPR03 medical and health sciences0302 clinical medicinebinge eatingGene FrequencymedicineHumansGenetic Predisposition to DiseaseObesityPsychiatryBulimia NervosaBiological PsychiatrySerotonin transporterBiological Specimen BanksSerotonin Plasma Membrane Transport ProteinsPolymorphism GeneticbiologyBinge eatingBulimia nervosaEating disordermedicine.diseaseBiobank030227 psychiatryPsychiatry and Mental healthEating disordersMeta-analysis5-HTTLPREating disordersbiology.proteinGene polymorphismmedicine.symptomPsychology030217 neurology & neurosurgeryThe world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry
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Environmental risk factors and cardiovascular diseases: a comprehensive expert review.

2021

Abstract Non-communicable diseases (NCDs) are fatal for more than 38 million people each year and are thus the main contributors to the global burden of disease accounting for 70% of mortality. The majority of these deaths are caused by cardiovascular disease (CVD). The risk of NCDs is strongly associated with exposure to environmental stressors such as pollutants in the air, noise exposure, artificial light at night, and climate change, including heat extremes, desert storms, and wildfires. In addition to the traditional risk factors for CVD such as diabetes, arterial hypertension, smoking, hypercholesterolaemia, and genetic predisposition, there is a growing body of evidence showing that …

medicine.medical_specialtyArtificial lightPhysiologybusiness.industryStressorDiseasemedicine.diseaseCardiovascular SystemEnvironmental riskCardiovascular DiseasesRisk FactorsPhysiology (medical)Environmental healthUrbanizationDiabetes mellitusEpidemiologyGenetic predispositionDiabetes MellitusMedicineHumansCardiology and Cardiovascular MedicinebusinessNoncommunicable DiseasesCardiovascular research
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Effects of l-Carnitine in Patients with Autism Spectrum Disorders: Review of Clinical Studies

2019

Carnitine is an amino acid derivative, which plays several important roles in human physiology, in the central nervous system, and for mitochondrial metabolism, in particular. Altered carnitine metabolic routes have been associated with a subgroup of patients with autism spectrum disorders (ASD) and could add to the pathophysiology associated with these disorders. We review the current evidence about the clinical effects of carnitine administration in ASD in both non-syndromic forms and ASD associated with genetic disorders. Two randomized clinical trials and one open-label prospective trial suggest that carnitine administration could be useful for treating symptoms in non-syndromic ASD. Th…

medicine.medical_specialtyAutism Spectrum DisorderCentral nervous systemPharmaceutical ScienceReviewComorbidityAnalytical Chemistrylaw.inventionlcsh:QD241-44103 medical and health sciences0302 clinical medicineNeurodevelopmental disorderlcsh:Organic chemistryRandomized controlled triallawCarnitineInternal medicinemental disordersDrug DiscoveryIntellectual disabilitymedicineHumansgenetic disordersGenetic Predisposition to DiseaseCarnitinePhysical and Theoretical ChemistryRandomized Controlled Trials as Topic030304 developmental biology0303 health sciencesDose-Response Relationship Drugbusiness.industryOrganic Chemistryvitaminmedicine.diseaseneurodevelopmental disorderPathophysiologyClinical trialTreatment Outcomemedicine.anatomical_structureChemistry (miscellaneous)Molecular MedicineAutismnutritional supplementationbusinessmetabolism030217 neurology & neurosurgerymedicine.drugMolecules
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Heredity of interocular similarities in components of refraction: a population-based twin study among 66- to 79-year-old female twins.

2019

Purpose: To examine genetic influences on interocular similarities in ocular refraction and components of refraction among elderly female twins. Methods: Refraction was assessed in 94 monozygotic (MZ) and 74 dizygotic (DZ) female twins aged 66–78 years. Absolute values of interocular differences (Aniso variables) in spherical refraction (SR), refractive astigmatism (AST), spherical equivalent (SE), corneal refractive power (CR), corneal astigmatism (CAST), anterior chamber depth (ACD) and axial length (AL) were calculated. The differences between sisters in each of the Aniso variables were calculated and graded into two categories, best differentiating the groups, here isometropic and aniso…

medicine.medical_specialtyBiometryOLDER POPULATIONASTIGMATISMEmmetropiaBiologymedicine.disease_causeRefraction OcularCornea03 medical and health sciencesansiometropia0302 clinical medicineOphthalmologyHereditymedicineDiseases in TwinsTwins DizygoticHumansGenetic Predisposition to Disease3125 Otorhinolaryngology ophthalmology10. No inequalityAnisometropiaAgedspherical equivalentHERITABILITYisometropiaGeneral MedicineTwins MonozygoticHeritabilitymedicine.diseaseRefractive ErrorsTwin studyRefractionZygosityANISOMETROPIAPREVALENCEcorneal refractionOphthalmologyExact testastigmatism030221 ophthalmology & optometryRISK-FACTORSFemaleemmetropia030217 neurology & neurosurgeryActa ophthalmologica
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Relation of PvuII site polymorphism in the COL1A2 gene to the risk of fractures in prepubertal Finnish girls.

2003

Genetic susceptibility to fractures may be detectable in early childhood. We evaluated the associations between the polymorphic PvuII site of the COL1A2 gene and bone properties assessed by different modalities (dual-energy X-ray absorptiometry; peripheral quantitative computed tomography; gel coupling scanning quantitative ultrasonometry; ultrasound bone sonometry), bone turnover markers, and the occurrence of fractures in 244 prepubertal Finnish girls. Tanner stage and physical characteristics did not differ significantly among girls with different COL1A2 genotypes. The polymorphism was not significantly associated with different bone properties or any of the bone turnover markers when gi…

medicine.medical_specialtyBone densityPhysiologyOsteoporosisBiologyPolymorphism Single NucleotideCollagen Type IBone remodelingFractures BoneBone DensityRisk FactorsInternal medicineGenotypeGeneticsmedicineHumansGenetic Predisposition to DiseaseTibiaQuantitative computed tomographyChildDeoxyribonucleases Type II Site-SpecificFinlandRetrospective StudiesBone mineralBinding SitesPolymorphism Geneticmedicine.diagnostic_testPubertyAnthropometrymedicine.diseaseEndocrinologyFemaleBone RemodelingCollagenPhysiological genomics
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Genetic bases of urinary albumin excretion and related traits in hypertension

2010

Epidemiological as well as animal studies have recognized the potential role of genetic factors in the development of microalbuminuria and related traits (renal insufficiency, end-stage renal disease and nephroangiosclerosis) in hypertension. To unravel genetic variants of susceptibility, candidate gene, linkage and genome wide scan analysis has been used. In spite of the great efforts that have been made in the field, sound knowledge about the major genetic variants causing the susceptibility to develop renal damage in hypertension is scarce, since many associations were not replicated or only showed association in a certain subgroup of patients. Looking initially at genes of the most impo…

medicine.medical_specialtyCandidate geneGenetic LinkagePhysiologyGenome-wide association studyDiseaseBioinformaticsGenomeRenin-Angiotensin SystemGenetic linkageInternal medicineInternal MedicinemedicineAlbuminuriaAnimalsHumansGenetic Predisposition to DiseaseNatriuretic PeptidesGenebusiness.industrymedicine.diseaseReceptors AdrenergicOxidative StressEndocrinologyHypertensionAlbuminuriaKallikreinsMicroalbuminuriamedicine.symptomCardiology and Cardiovascular MedicinebusinessGenome-Wide Association StudyJournal of Hypertension
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Dual specificity phosphatase 1 knockout mice show enhanced susceptibility to anaphylaxis but are sensitive to glucocorticoids.

2007

Dual specificity phosphatase DUSP1 (otherwise known as mitogen-activated phosphatase 1 or MKP-1) dephosphorylates MAPKs, particularly p38, and negatively regulates innate immunity. Recent studies have shown that the DUSP1 gene is transcriptionally up-regulated by glucocorticoids (GCs) and that the antiinflammatory action of GCs is impaired in DUSP1-/- mice. Here we show that GC-mediated dephosphorylation of ERK-1 and ERK-2 activated by IgE receptor cross-linking is unimpaired in bone marrow-derived mast cells (BMMCs) of DUSP1-/- mice. Dephosphorylation of phospho-p38 MAPK is impaired but only at early times of GC treatment. Proinflammatory cytokine and chemokine gene expression (CCL2, IL-6,…

medicine.medical_specialtyChemokinePhosphataseImmunoglobulin Ep38 Mitogen-Activated Protein KinasesProinflammatory cytokineDephosphorylationMiceEndocrinologyInternal medicineSepsisDual-specificity phosphatasemedicineAnimalsGenetic Predisposition to DiseaseMolecular BiologyAnaphylaxisGlucocorticoidsMice KnockoutMitogen-Activated Protein Kinase 1Mice Inbred C3HMitogen-Activated Protein Kinase 3biologyInterleukin-6Tumor Necrosis Factor-alphaDegranulationDual Specificity Phosphatase 1General MedicineMice Inbred C57BLEndocrinologyGene Expression RegulationMice Inbred DBAbiology.proteinCytokinesTumor necrosis factor alphaMolecular endocrinology (Baltimore, Md.)
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HLA-DQ: Celiac diseasevsinflammatory bowel disease

2018

Aim To determine the genetic predisposition to celiac disease (CeD) in inflammatory bowel disease (IBD) patients by quantifying the frequency of CeD-related human leucocyte antigen (HLA) (HLA-CeD: HLA-DQ2 and -DQ8) in IBD patients globally, by type of IBD and gender, and by calculating the protective/risk contribution of these haplotypes in the development of the IBD disease. Methods We conducted a prospective study with IBD patients from our Unit. Clinical information was gathered and blood was tested for HLA-CeD. The control group was made up of unrelated Valencian organ donors. Results 1034 subjects were analyzed: 457 IBD [207 ulcerative coliti (UC) and 250 Crohn's disease (CD)] patients…

medicine.medical_specialtyCrohn's diseaseHLA-DQB1business.industryGastroenterologynutritional and metabolic diseasesGeneral MedicineHuman leukocyte antigenmedicine.diseaseGastroenterologyInflammatory bowel diseaseUlcerative colitis03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisInternal medicineHLA-DQGenetic predispositionMedicine030211 gastroenterology & hepatologybusinessProspective cohort studyWorld Journal of Gastroenterology
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Evaluation of cytokine polymorphisms (TNFalpha, IFNgamma and IL-10) in Down patients with coeliac disease.

2005

Abstract Background In Down syndrome there is an increased prevalence of coeliac disease, but the reasons for this association are yet unknown. Aims To evaluate a possible correlation between TNFα, IFNγ and IL-10 genotype polymorphisms with the susceptibility to coeliac disease in Down syndrome patients. Methods Single nucleotide polymorphisms of TNFα (−308G → A promoter region), IFNγ (+874T → A promoter region) and IL-10 (−1082G → A promoter region) have been studied in 10 Down patients with coeliac disease, in 40 Down patients without coeliac disease and in 220 healthy controls. Clinical features were also studied in coeliac disease–Down syndrome patients. Results The 10 coeliac disease–D…

medicine.medical_specialtyDown syndromeAdolescentSingle-nucleotide polymorphismGastroenterologyPolymorphism Single NucleotideCoeliac diseaseSerologyInterferon-gammaInternal medicineGenotypeBiopsymedicineHumansGenetic Predisposition to DiseaseChildPolymorphism GeneticHepatologymedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantmedicine.diseasesyndromedigestive system diseasesSurgeryInterleukin-10Interleukin 10Celiac DiseaseChild PreschoolCytokinesDownDown SyndromebusinessTrisomyDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
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No paradox, no progress: inverse cancer comorbidity in people with other complex diseases.

2011

Salvador Martínez [et al.]. 5 p., 2 tables and references.

medicine.medical_specialtyDown syndromeAnorexia NervosaNeuregulin-1DiseaseComorbidity03 medical and health sciences0302 clinical medicineAlzheimer DiseaseNeoplasmsEpidemiologymedicineHumansGenetic Predisposition to DiseasePsychiatryComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesbusiness.industryCancerParkinson Diseasemedicine.disease[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]Comorbidity3. Good healthOncologySchizophreniaAnorexia nervosa (differential diagnoses)SchizophreniaAlzheimer's diseaseDown Syndromebusiness030217 neurology & neurosurgeryChromosomes Human Pair 8The Lancet. Oncology
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