Search results for "Dopamine"

showing 10 items of 660 documents

Reduction of Dopamine Level Enhances the Attractiveness of Male Drosophila to Other Males

2009

1932-6203 (Electronic); Dopamine is an important neuromodulator in animals and its roles in mammalian sexual behavior are extensively studied. Drosophila as a useful model system is widely used in many fields of biological studies. It has been reported that dopamine reduction can affect female receptivity in Drosophila and leave male-female courtship behavior unaffected. Here, we used genetic and pharmacological approaches to decrease the dopamine level in dopaminergic cells in Drosophila, and investigated the consequence of this manipulation on male homosexual courtship behavior. We find that reduction of dopamine level can induce Drosophila male-male courtship behavior, and that this beha…

MaleLevodopaanimal structuresDopaminemedia_common.quotation_subjectlcsh:MedicineNeuroscience/Neural HomeostasisCourtshipSexual Behavior Animal03 medical and health sciences0302 clinical medicineDopamineDopaminergic CellmedicineAnimalsHomosexuality Malelcsh:ScienceDrosophila030304 developmental biologymedia_common0303 health sciencesNeuroscience/Behavioral NeuroscienceMultidisciplinaryCourtship displaybiologyNeuroscience/Sensory Systemslcsh:RfungiCourtshipbiology.organism_classificationSex pheromonebehavior and behavior mechanismsDrosophilalcsh:QDrosophila melanogasterNeuroscience030217 neurology & neurosurgeryResearch Articlemedicine.drug
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Partial replication of a DRD4 association in ADHD individuals using a statistically derived quantitative trait for ADHD in a family-based association…

2007

Contains fulltext : 52515.pdf (Publisher’s version ) (Closed access) BACKGROUND: Previous research found an association between single nucleotide polymorphisms (SNPs) in the promoter region of DRD4 and statistically derived phenotypes generated from attention-deficit/hyperactivity disorder (ADHD) symptoms. We sought to replicate this finding by using the same methodology in an independent sample of ADHD individuals. METHODS: Four SNPs were genotyped in and around DRD4 in 2631 individuals in 642 families. We developed a quantitative phenotype at each SNP by weighting nine inattentive and nine hyperactive-impulsive symptoms. The weights were selected to maximize the heritability at each SNP. …

MaleLinkage disequilibriumGenetics and epigenetic pathways of disease [NCMLS 6]Databases FactualMedizinNeuroinformatics [DCN 3]Severity of Illness Index0302 clinical medicinePerception and Action [DCN 1]Determinants in Health and Disease [EBP 1]ChildPromoter Regions GeneticGenetics0303 health sciencesEuropePhenotypeChild PreschoolFemalemedicine.symptomPsychologyFunctional Neurogenomics [DCN 2]medicine.medical_specialtyAdolescentSingle-nucleotide polymorphismQuantitative trait locusImpulsivityMental health [NCEBP 9]Polymorphism Single NucleotideGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesQuantitative Trait HeritableCognitive neurosciences [UMCN 3.2]Genetic modelmental disordersmedicineAttention deficit hyperactivity disorderSNPHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersPsychiatryBiological Psychiatry030304 developmental biologyFamily HealthReceptors Dopamine D4Heritabilitymedicine.diseaseGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivity030217 neurology & neurosurgeryBiological psychiatry
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Increased toxicity of cocaine on human hepatocytes induced by ethanol: role of GSH.

1999

Increased toxicity of cocaine to human hepatocytes is observed when cells are simultaneously incubated with ethanol. Ethanol might exacerbate cocaine hepatocyte toxicity by three different pathways: a) by increasing the oxidative metabolism of cocaine and hence the oxidative damage; b) by the formation of a more toxic metabolite, namely cocaethylene; or c) by decreasing the defence mechanisms of the cell (i.e. GSH). In the present study, experiments were conducted to investigate the feasibility of these hypotheses. In hepatocytes preincubated for 48 hr with ethanol, neither significant changes in cocaine metabolism nor cytotoxicity were found despite differences in hepatocyte p-nitrophenol …

MaleLiver cytologyCell SurvivalPharmacologymedicine.disease_causeBiochemistryLipid peroxidationchemistry.chemical_compoundCocaethyleneCocaineDopamine Uptake InhibitorsmedicineHumansCells CulturedAgedGlutathione TransferasePharmacologyEthanolDrug SynergismGlutathioneCYP2E1Middle AgedOxidative Stressmedicine.anatomical_structurechemistryBiochemistryLiverHepatocyteToxicityFemaleOxidative stressmedicine.drugBiochemical pharmacology
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The high affinity dopamine uptake inhibitor, JHW 007, blocks cocaine-induced reward, locomotor stimulation and sensitization

2009

The discovery and evaluation of high affinity dopamine transport inhibitors with low abuse liability is an important step toward the development of efficacious medications for cocaine addiction. We examined in mice the behavioural effects of (N-(n-butyl)-3Ά-[bis(4Ά-fluorophenyl)methoxy]-tropane) (JHW 007), a benztropine (BZT) analogue that blocks dopamine uptake, and assessed its potential to influence the actions of cocaine in clinically-relevant models of cocaine addiction. In the conditioned place preference (CPP) paradigm, JHW 007 exposure did not produce place conditioning within an ample dose range but effectively blocked the CPP induced by cocaine administration. Similarly, in the CP…

MaleLocomotor activityElevated plus mazemedia_common.quotation_subjectDopamine transportPharmacologyMotor ActivityAnxietyOpen fieldSensitizationMiceDopamine Uptake InhibitorsRewardCocaineDopaminemedicineAnimalsPharmacology (medical)Drug InteractionsMaze LearningBiological PsychiatrySensitizationmedia_commonPharmacologyBenztropineAnalysis of VarianceBehavior AnimalAddictionPlace preferenceBenztropineConditioned place preferencePsychiatry and Mental healthmedicine.anatomical_structureNeurologyConditioning OperantDopamine AntagonistsNeurology (clinical)PsychologyBenztropine analoguesmedicine.drug
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Social defeat-induced increase in the conditioned rewarding effects of cocaine: Role of CX3CL1

2019

Abstract Social stress is associated with higher vulnerability to drug use, as it enhances the reinforcing effects of psychostimulants in rodents. Furthermore, continued or severe stress induces a proinflammatory state of microglial activation and augmented cytokine production. The aim of the present work was to evaluate the role of fractalkine [C-X3-C motif ligand 1 (CX3CL1)], an inflammatory chemokine, in the increased conditioned rewarding effects of cocaine in animals exposed to social defeat stress. In addition, we measured the signaling cascade pathway of CX3CL1 in the hippocampus (HPC) (including p-ERK/ERK, p-p38/p38 MAPK, p-p65/p65 NFκB and p-CREB/CREB ratios). The glutamate recepto…

MaleMAPK/ERK pathwaymedicine.medical_specialtyCREBSocial DefeatSocial defeatMice03 medical and health sciences0302 clinical medicineCocaineDopamine Uptake InhibitorsRewardInternal medicineConditioning PsychologicalCX3CR1AnimalsMedicineCX3CL1Biological PsychiatryMice KnockoutPharmacologySocial stressbiologyChemokine CX3CL1business.industryGlutamate receptorConditioned place preference030227 psychiatryMice Inbred C57BLEndocrinologybiology.proteinbusinessProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Understanding Cannabinoid Psychoactivity with Mouse Genetic Models

2007

Marijuana and its main psychotropic ingredient Δ9-tetrahydrocannabinol (THC) exert a plethora of psychoactive effects through the activation of the neuronal cannabinoid receptor type 1 (CB1), which is expressed by different neuronal subpopulations in the central nervous system. The exact neuroanatomical substrates underlying each effect of THC are, however, not known. We tested locomotor, hypothermic, analgesic, and cataleptic effects of THC in conditional knockout mouse lines, which lack the expression of CB1 in different neuronal subpopulations, including principal brain neurons, GABAergic neurons (those that release γ aminobutyric acid), cortical glutamatergic neurons, and neurons expres…

MaleMESH: Body TemperatureCannabinoid receptormedicine.medical_treatmentGene ExpressionMESH: Receptor Cannabinoid CB1NeocortexMESH: gamma-Aminobutyric AcidMESH: CatalepsyPharmacologyHippocampusMESH: Mice KnockoutMESH: Corpus StriatumBody TemperatureMESH: Autonomic Nervous SystemMESH: NeocortexMice0302 clinical medicineReceptor Cannabinoid CB1MESH: Behavior AnimalCannabinoid receptor type 1MESH: AnimalsMESH: Gene SilencingDronabinolMESH: NociceptorsBiology (General)gamma-Aminobutyric AcidMice Knockout0303 health sciencesBehavior Animalmusculoskeletal neural and ocular physiologyGeneral NeuroscienceMESH: Pain ThresholdNociceptorsMESH: Glutamic AcidMESH: InterneuronsMESH: Motor Activity3. Good healthGABAergicMESH: TetrahydrocannabinolGeneral Agricultural and Biological SciencesResearch Articlemedicine.drugPain ThresholdMESH: Gene ExpressionMESH: Psychotropic DrugsQH301-705.5Glutamic AcidMotor ActivityBiologyAutonomic Nervous SystemGeneral Biochemistry Genetics and Molecular Biologygamma-Aminobutyric acid03 medical and health sciencesGlutamatergicDopamine receptor D1InterneuronsCannabinoid Receptor Modulatorsmental disorders[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsGenetic Predisposition to Disease[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyGene SilencingTetrahydrocannabinolMESH: MiceAnesthesiology and Pain Management030304 developmental biologyPharmacologyCatalepsyPsychotropic DrugsModels GeneticGeneral Immunology and MicrobiologyCannabinoidsIllicit Drugsorganic chemicalsMESH: MaleCorpus StriatumPrimerDisease Models Animalnervous systemCannabinoidNervous System Diseases030217 neurology & neurosurgeryNeurosciencePLoS Biology
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Clozapine: Strong antiaggressive effects with minimal motor impairment

1992

Abstract Clinical studies have shown clozapine to be effective in the treatment of schizophrenia and associated with an extremely low incidence of extrapiramidal side effects. Diverse studies indicate that clozapine is an atypical neuroleptic with a preferential activity on the mesolimbic structures and a lower affinity for striatal D2 receptors than the classical antipsychotics. The purpose of this study was to assess the behavioral properties of clozapine, especially its effects on aggressive and motor behaviors. Individually housed male mice of the OF1 strain were exposed to anosmic “standard opponents” 30 minutes after the last drug administration. One category of animals received a sin…

MaleMale miceExperimental and Cognitive PsychologyAtypical neurolepticMotor ActivityPharmacologyMiceBehavioral NeuroscienceDopamine receptor D2medicineAnimalsClozapineClozapineDose-Response Relationship DrugDrug administrationMotor impairmentmedicine.diseaseAggressionLower affinityMotor SkillsSchizophreniaAnesthesiaArousalPsychologyPsychomotor Performancemedicine.drugPhysiology & Behavior
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Effects of risperidone and SCH 23390 on isolation-induced aggression in male mice.

1998

In this study, the antiaggressive effects of risperidone and SCH 23390 have been explored. Using the paradigm of isolation-induced aggression, 150 albino male mice of the OF1 strain were allocated to control and experimental groups which received three doses of risperidone (0.01, 0.05 and 0.1 mg/kg) or two doses of SCH 23390 (0.05 and 0.1 mg/kg). Only the highest doses of risperidone decreased threat and attack behaviours but all doses significantly impaired motor behaviour. SCH 23390 decreased attack with the two doses used and also produced significant increases in immobility. Although both antipsychotics are antiaggressive, this action seems to be more specific in the case of risperidone…

MaleMale micePharmacologyNeurotransmissionMotor Activitychemistry.chemical_compoundMiceSexual Behavior AnimalDopaminemedicineAnimalsPharmacology (medical)Biological PsychiatryPharmacologySCH-23390RisperidoneAggressionReceptors Dopamine D1BenzazepinesRisperidoneGroomingAggressionPsychiatry and Mental healthDopamine D2 Receptor AntagonistsNeurologychemistryIsolation induced aggressionSocial IsolationDepression ChemicalExploratory BehaviorDopamine AntagonistsFemaleNeurology (clinical)Serotoninmedicine.symptomPsychologymedicine.drugEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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Brain met-enkephalin immunostaining after subacute and subchronic exposure to benzene

1994

Benzene is used in a wide variety of domestic and occupational activities, and due to its lipophilic nature, it accumulates in lipid-rich tissues like the brain. In this sense, neurotoxic action has long been associated with organic solvent exposure and it has been shown that benzene, injected in a single dose or during a prolongued administration, modifies the content of dopamine, noradrenaline, serotonin and its main metabolite 5-hydroxy indolacetic acid, in several brain regions of the rat, then revealing a stimulating action on brain monoamine synthesis and turnover. However, information concerning neurotoxic action of benzene exposure in vivo on peptidergic neuromodulatory systems is s…

MaleMet-enkephalinmedicine.medical_specialtyTime FactorsEnkephalin MethionineHealth Toxicology and MutagenesisCentral nervous systemNeuropeptideBiologyToxicologyRats Sprague-Dawleychemistry.chemical_compoundDopamineInternal medicineImage Processing Computer-AssistedmedicineAnimalsBrain ChemistryStaining and LabelingProteolytic enzymesBrainBenzeneGeneral MedicinePollutionRatsEndocrinologyMonoamine neurotransmittermedicine.anatomical_structurechemistryImmunologic TechniquesSerotoninImmunostainingmedicine.drugBulletin of Environmental Contamination and Toxicology
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NMDA glutamate but not dopamine antagonists blocks drug-induced reinstatement of morphine place preference.

2004

The effects of dopaminergic and glutamatergic antagonists on the drug-induced reinstatement of a previously extinguished morphine conditioned place preference (CPP) in mice were evaluated. Following extinction of a place preference induced by morphine (40 mg/kg), a non-contingent injection of the dopaminergic antagonists SCH 23390 (0.125, 0.5 mg/kg), raclopride (0.3, 1.2 mg/kg), haloperidol (0.1, 0.2 mg/kg) and the dopamine (DA) release inhibitor CGS 10746B (1, 10 mg/kg) or glutamatergic NMDA antagonists memantine (10, 20, 40 mg/kg) and MK-801 (0.1, 0.2, 0.3 mg/kg) alone or with 10 mg/kg morphine was given. Neither the dopaminergic nor the glutamatergic antagonists alone reinstated the plac…

MaleMice Inbred StrainsPharmacologyReceptors N-Methyl-D-AspartateExtinction PsychologicalGlutamatergicMiceDopaminemedicineHaloperidolAnimalsDrug InteractionsRacloprideAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMorphineChemistryGeneral NeuroscienceDopaminergicMemantineConditioned place preferenceAnalgesics OpioidNMDA receptorConditioning OperantDopamine AntagonistsExcitatory Amino Acid Antagonistsmedicine.drugBrain research bulletin
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