Search results for "Dot"

showing 10 items of 5147 documents

Uncoupling of eNOS in Cardiovascular Disease

2017

Abstract Under physiological conditions, nitric oxide (NO) produced by the endothelial NO synthase (eNOS) represents a key vasoprotective factor. Under conditions of cardiovascular diseases, such as hypertension, diabetes, and atherosclerosis, eNOS may become uncoupled. Uncoupled eNOS generates superoxide at the expense of NO and contributes significantly to endothelial dysfunction and atherogenesis. Major mechanisms of eNOS uncoupling include depletion of tetrahydrobiopterin, an essential cofactor for the eNOS enzyme, and deficiency of l -arginine, the eNOS substrate, and/or eNOS S-glutathionylation. Reversal of eNOS uncoupling may represent a novel therapeutic strategy for the prevention …

0301 basic medicinemedicine.medical_specialtyArgininebiologySuperoxidebusiness.industryNitric Oxide Synthase Type IIITetrahydrobiopterin030204 cardiovascular system & hematologybiology.organism_classificationmedicine.diseaseVasoprotectiveNitric oxide03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicineEndocrinologychemistryEnosInternal medicinemedicineEndothelial dysfunctionbusinessmedicine.drug
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Effects of resveratrol on eNOS in the endothelium and the perivascular adipose tissue

2017

Under physiological conditions, nitric oxide (NO) is produced in the vasculature mainly by the endothelial NO synthase (eNOS). Experiments using gene-disrupted mice have demonstrated that eNOS has antihypertensive, antithrombotic, and antiatherosclerotic effects. Recent studies show that eNOS is expressed not only in the endothelium but also in the perivascular adipose tissue (PVAT). Resveratrol prevents eNOS uncoupling and upregulates eNOS expression and activity. These effects of resveratrol are well established for the eNOS enzyme in the endothelium. Interestingly, resveratrol also improves PVAT function. However, a causal role for eNOS in the effects of resveratrol on PVAT function has …

0301 basic medicinemedicine.medical_specialtyEndotheliumAdipose tissue030204 cardiovascular system & hematologyResveratrolGeneral Biochemistry Genetics and Molecular BiologyNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineHistory and Philosophy of ScienceEnosInternal medicineAntithromboticmedicinebiologyChemistryGeneral NeuroscienceNitric Oxide Synthase Type IIIbiology.organism_classificationNitric oxide synthase030104 developmental biologyEndocrinologymedicine.anatomical_structurebiology.proteinAnnals of the New York Academy of Sciences
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Crucial role for Nox2 and sleep deprivation in aircraft noise-induced vascular and cerebral oxidative stress, inflammation, and gene regulation

2018

Abstract Aims Aircraft noise causes endothelial dysfunction, oxidative stress, and inflammation. Transportation noise increases the incidence of coronary artery disease, hypertension, and stroke. The underlying mechanisms are not well understood. Herein, we investigated effects of phagocyte-type NADPH oxidase (Nox2) knockout and different noise protocols (around-the-clock, sleep/awake phase noise) on vascular and cerebral complications in mice. Methods and results C57BL/6j and Nox2 −/− (gp91phox −/−) mice were exposed to aircraft noise (maximum sound level of 85 dB(A), average sound pressure level of 72 dB(A)) around-the-clock or during sleep/awake phases for 1, 2, and 4 days. Adverse effec…

0301 basic medicinemedicine.medical_specialtyEndotheliumAircraft10208 Institute of NeuropathologyInflammation610 Medicine & health030204 cardiovascular system & hematologySystemic inflammationmedicine.disease_cause2705 Cardiology and Cardiovascular Medicine03 medical and health sciences0302 clinical medicineBasic ScienceVascular BiologyInternal medicineeNOS uncouplingmedicineHumansEndothelial dysfunctionEndothelial dysfunctionInflammationSystemic inflammationbusiness.industryEnvironmental stressorCerebral redox balancemedicine.diseaseEnvironmental stressorSleep deprivationNoiseSleep deprivationOxidative Stress030104 developmental biologymedicine.anatomical_structureEndocrinologyNADPH oxidase-derived oxidative stress570 Life sciences; biologymedicine.symptombusinessNoiseCardiology and Cardiovascular MedicineOxidative stressNoise exposure
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Dexamethasone Preconditioning in Cardiac Procedures Reduces Decreased Antithrombin Activity and Is Associated to Beneficial Outcomes: Role of Endothe…

2018

13 páginas, 6 figuras, 2 tablas.

0301 basic medicinemedicine.medical_specialtyEndotheliumAngiogenesisAntithrombinEndotelio.Urology030204 cardiovascular system & hematologyDexamethasonelaw.invention03 medical and health sciences0302 clinical medicineAortic valve replacementlawmedicine.arteryCardiopulmonary bypassMedicinePharmacology (medical)Dexametasona - Uso terapéutico.DexamethasoneOriginal ResearchPharmacologyAortabusiness.industryCardiopulmonary bypasslcsh:RM1-950AntithrombinEndothelium.Antitrombinas.Endothelial functionCardiac surgerymedicine.diseaseAntithrombins.Cardiac surgerylcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structureHeart - Surgery.Dexamethasone - Therapeutic use.Corazón - Cirugía.Angiogenesisbusinessmedicine.drug
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Relationship between PMN-endothelium interactions, ROS production and Beclin-1 in type 2 diabetes.

2020

Type 2 diabetes is closely related to oxidative stress and cardiovascular diseases. In this study, we hypothesized that polymorphonuclear leukocytes (PMN)-endothelium interactions and autophagy are associated. We evaluated PMN-endothelial interactions, ROS production and autophagy parameters in 47 type 2 diabetic patients and 57 control subjects. PMNs from type 2 diabetic patients exhibited slower rolling velocity (p < 0.001), higher rolling flux (p < 0.001) and adhesion (p < 0.001) in parallel to higher levels of total (p < 0.05) and mitochondrial ROS (p < 0.05). When the protein expression of autophagy markers was analysed, an increase of Beclin-1 (p < 0.05), LC3I (p < 0.05), LC3II (p < 0…

0301 basic medicinemedicine.medical_specialtyEndotheliumNeutrophilsClinical BiochemistryType 2 diabetesMitochondrionmedicine.disease_causeBiochemistry03 medical and health sciences0302 clinical medicineInternal medicineDiabetes mellitusmedicineAutophagyCell AdhesionHumansEndotheliumlcsh:QH301-705.5lcsh:R5-920ChemistryOrganic ChemistryAutophagyRolling velocityType 2 diabetesROSmedicine.diseaseControl subjectsMitochondria030104 developmental biologymedicine.anatomical_structureEndocrinologylcsh:Biology (General)Diabetes Mellitus Type 2Oxidative stressCase-Control StudiesPMN-Endothelium interactionsBeclin-1lcsh:Medicine (General)Reactive Oxygen Species030217 neurology & neurosurgeryOxidative stressResearch PaperRedox biology
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Mechanisms underlying the influence of oestrogen on cardiovascular physiology in women.

2019

Women show a lower incidence of cardiovascular diseases than age-matched men, but this benefit disappears after menopause. Oestrogen-mediated vascular actions are mainly attributed to oestradiol and exerted by oestrogen receptors (ERα, ERβ and G protein-coupled oestrogen receptor), through rapid and/or genomic mechanisms, but these effects depend on ageing and inflammation. A cardiovascular approach in women's health has arisen due to controversy regarding oestrogen's beneficial impact as reported in experimental and observational studies and large randomized trials. These can be explained, in part, by two mutually non-exclusive hypotheses. On the one hand, the timing hypothesis, which stat…

0301 basic medicinemedicine.medical_specialtyEndotheliumPhysiologyMedicinaEstrogen receptorInflammationProstacyclin03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansEndothelial dysfunctionReceptorskin and connective tissue diseasesMolecular Structurebusiness.industryEstrogen Receptor alphaEstrogensmedicine.diseaseCardiovascular physiologyPostmenopause030104 developmental biologymedicine.anatomical_structureEndocrinologyAgeingFemaleEndothelium Vascularmedicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Journal of physiologyReferences
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Oral Unsaturated Fat Load Impairs Postprandial Systemic Inflammation in Primary Hypercholesterolemia Patients.

2021

Context: Primary hypercholesterolemia (PH) is a lipid disorder characterized by elevated levels of cholesterol and low-density lipoprotein (LDL). Low-grade systemic inflammation is associated with PH, which might explain the higher incidence of cardiovascular diseases in this setting.Objective: To evaluate the effect of an oral unsaturated fat load (OUFL) on different immune parameters and functional consequences in patients with PH in postprandial state.Design: A commercial liquid preparation of long-chain triglycerides (Supracal®; ω6/ω3 ratio &amp;gt;20/1, OUFL) was administered to 20 patients and 10 age-matched controls. Whole blood was collected before (fasting state) and 4 h after admi…

0301 basic medicinemedicine.medical_specialtyEndotheliumleukocytesinflammatory mediatorsRM1-950030204 cardiovascular system & hematologySystemic inflammationendothelial dysfunction03 medical and health scienceschemistry.chemical_compound0302 clinical medicineoral unsaturated fat loadInternal medicineprimary hypercholesterolemiamedicineplatelet activationPharmacology (medical)Platelet activationEndothelial dysfunctionOriginal ResearchPharmacologysystemic inflammationCholesterolbusiness.industryUnsaturated fatmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurePostprandialchemistryTherapeutics. Pharmacologymedicine.symptombusinessLipoproteinFrontiers in pharmacology
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Effect of Roux-en-Y Bariatric Bypass Surgery on Subclinical Atherosclerosis and Oxidative Stress Markers in Leukocytes of Obese Patients: A One-Year …

2020

Little is known about the mechanisms underlying the cardioprotective effect of Roux en-Y gastric bypass (RYGB) surgery. Therefore, the aim of the present study was to investigate whether weight loss associated with RYGB improves the oxidative status of leukocytes and ameliorates subclinical atherosclerotic markers. This is an interventional study of 57 obese subjects who underwent RYGB surgery. We determined biochemical parameters and qualitative analysis of cholesterol, leukocyte and systemic oxidative stress markers &mdash

0301 basic medicinemedicine.medical_specialtyGPX1obesityPhysiologybariatric surgeryClinical Biochemistry030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryleukocyte-endothelium interactionsArticleSuperoxide dismutase03 medical and health scienceschemistry.chemical_compound0302 clinical medicineWeight lossInternal medicinemedicineoxidative stressMolecular BiologySubclinical infectionbiologySuperoxideCholesterolbusiness.industrylcsh:RM1-950nutritional and metabolic diseasesCell Biology030104 developmental biologyEndocrinologylcsh:Therapeutics. PharmacologychemistryMyeloperoxidasebiology.proteinmedicine.symptomatherosclerosisbusinessOxidative stress
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Roles of Vascular Oxidative Stress and Nitric Oxide in the Pathogenesis of Atherosclerosis.

2017

Major reactive oxygen species (ROS)–producing systems in vascular wall include NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase, xanthine oxidase, the mitochondrial electron transport chain, and uncoupled endothelial nitric oxide (NO) synthase. ROS at moderate concentrations have important signaling roles under physiological conditions. Excessive or sustained ROS production, however, when exceeding the available antioxidant defense systems, leads to oxidative stress. Animal studies have provided compelling evidence demonstrating the roles of vascular oxidative stress and NO in atherosclerosis. All established cardiovascular risk factors such as hypercholesterolemi…

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyOxidative phosphorylationBiologymedicine.disease_causeNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compoundSpecies SpecificityInternal medicinemedicineAnimalsHumansXanthine oxidasechemistry.chemical_classificationReactive oxygen speciesNitric Oxide Synthase Type IIIAtherosclerosisEndothelial stem cellOxidative Stress030104 developmental biologyEndocrinologychemistryEndothelium VascularCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidative stressNicotinamide adenine dinucleotide phosphateCirculation research
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The Adverse Effects of Environmental Noise Exposure on Oxidative Stress and Cardiovascular Risk

2018

Abstract Epidemiological studies have provided evidence that traffic noise exposure is linked to cardiovascular diseases such as arterial hypertension, myocardial infarction, and stroke. Noise is a nonspecific stressor that activates the autonomous nervous system and endocrine signaling. According to the noise reaction model introduced by Babisch and colleagues, chronic low levels of noise can cause so-called nonauditory effects, such as disturbances of activity, sleep, and communication, which can trigger a number of emotional responses, including annoyance and subsequent stress. Chronic stress in turn is associated with cardiovascular risk factors, comprising increased blood pressure and …

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical Biochemistrytraffic noise exposure030204 cardiovascular system & hematologyenvironmental risk factorsmedicine.disease_causeBiochemistryendothelial dysfunctionCoronary artery disease03 medical and health sciences0302 clinical medicinestress hormonesComprehensive Invited ReviewRisk FactorsInternal medicineDiabetes mellitusmedicineHumansChronic stressMyocardial infarctionEndothelial dysfunctionMolecular BiologyStrokeGeneral Environmental Sciencebusiness.industryaircraft noise exposureEnvironmental ExposureCell Biologymedicine.diseaseStrokeOxidative Stress030104 developmental biologyBlood pressureCardiovascular DiseasesHypertensionCardiologyGeneral Earth and Planetary SciencesEnvironmental PollutantsNoiseReactive Oxygen SpeciesbusinessOxidative stressAntioxidants &amp; Redox Signaling
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