Search results for "Edema"

showing 10 items of 625 documents

Treatment of macular oedema (excluding age-related macular degeneration (AMD))

2021

Macular oedema corresponds to an increase in macular thickness most often associated with a rupture of the hematoretinal barriers. Diagnosis is based on the notion of reduced vision, but above all on multimodal imaging in which optical coherence tomography (OCT) plays an essential role both for the initial diagnosis and for follow-up. The mechanisms involved in the occurrence of macular oedema are primarily vascular endothelial growth factor (VEGF) and cytokines regardless of the aetiology of the oedema. Treatment often involves a combination of local treatment - primarily intravitreal injections of anti-VEGF or corticosteroids - and, in certain situations, such as diabetes or uveitis, gene…

Inflammation[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyOCT[SDV.MHEP.OS] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansDiabetesŒdème maculaire OCT Diabète Occlusion veineuse Inflammation VEGF CorticoïdesCorticoidsMacular oedemaVEGFVenous occlusion
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Cellular targets in diabetic retinopathy therapy

2021

Despite the existence of treatment for diabetes, inadequate metabolic control triggers the appearance of chronic complications such as diabetic retinopathy. Diabetic retinopathy is considered a multifactorial disease of complex etiology in which oxidative stress and low chronic inflammation play essential roles. Chronic exposure to hyperglycemia triggers a loss of redox balance that is critical for the appearance of neuronal and vascular damage during the development and progression of the disease. Current therapies for the treatment of diabetic retinopathy are used in advanced stages of the disease and are unable to reverse the retinal damage induced by hyperglycemia. The lack of effective…

Inflammationbusiness.industryEndocrinology Diabetes and MetabolismInflammationDiseaseDiabetic retinopathyReviewmedicine.diseaseBioinformaticsmedicine.disease_causePathophysiologyCellular targetDiabetic retinopathyOxidative stressDiabetic macular edemaMetabolic control analysisDiabetes mellitusInternal MedicinemedicineEtiologymedicine.symptombusinessOxidative stressWorld Journal of Diabetes
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Experimental Hypertensive Intracerebral Mass Hemorrhage in Cats

1986

About two–thirds of all patients with spontaneous intracerebral hemorrhage suffer from systemic hypertension. Additionally systemic hypertension is the main factor indicating a poor prognosis of intracerebral hemorrhage. This has been shown in our statistical evaluation of 117 cases of conservatively treated spontaneous intracerebral hemorrhage. More than 48% of the hypertensive patients died, but only 30% of the normotensive patients (Fig. 1). In an experimental study we investigated the influence of systemic hypertension and normotension on epidural pressure and on the formation of brain edema during the first 12 h after artificial intracerebral hemorrhage.

Intracerebral hemorrhagePoor prognosisVasogenic edemaCATSbusiness.industryBrain edemaAnesthesiamedicineSpontaneous intracerebral hemorrhagemedicine.diseasebusinessHypertensive group
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The Influence of Systemic Hypertension on Intracranial Pressure and Edema Formation in Experimental Intracerebral Hemorrhage

1986

Systemic hypertension is not only the most frequent cause of intracerebral hemorrhage but also the most important complication of this disease. In our clinical investigation of 117 cases almost 70% of the patients suffered from systemic hypertension. Despite advances in diagnosis (Schiirmann, Dei Anang, 1980) mortality of hypertensive patients was 48%. Normotensive hemorrhages showed a better prognosis: only 31% of the patients died and 33% (17% in hypertensive patients) obtained good neurological recovery (Wallenfang et al., 1985).

Intracerebral hemorrhagebusiness.industryEdema fluidmedicine.diseaseWhite matterchemistry.chemical_compoundmedicine.anatomical_structurechemistryClinical investigationAnesthesiamedicineEdema formationComplicationbusinessIntracranial pressureEvans Blue
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Prevention and treatment of intracranial hypertension

2007

Intracranial pressure (ICP) is the pressure exerted by cranial contents on the dural envelope. It comprises the partial pressures of brain, blood and cerebrospinal fluid (CSF). Normal intracranial pressure is somewhere below 10 mmHg; it may increase as a result of traumatic brain injury, stroke, neoplasm, Reye's syndrome, hepatic coma, or other pathologies. When ICP increases above 20 mmHg it may damage neurons and jeopardize cerebral perfusion. If such a condition persists, treatment is indicated. Control of ICP requires measurement, which can only be performed invasively. Standard techniques include direct ventricular manometry or measurement in the parenchyma with electronic or fiberopti…

Intracranial PressureTraumatic brain injurymedicine.medical_treatmentBrain EdemaCerebral autoregulationNeurosurgical ProceduresmedicineHumansCerebral perfusion pressureIntracranial pressurePostoperative Carebusiness.industrymedicine.diseaseRespiration ArtificialHydrocephalusHypertonic salineAnesthesiology and Pain MedicineCerebral blood flowBrain InjuriesAnesthesiaPneumocephalusDrainageDecompressive craniectomyIntracranial HypertensionbusinessHydrocephalusBest Practice & Research Clinical Anaesthesiology
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Intravitreal Aflibercept Injection for Macular Edema Resulting from Central Retinal Vein Occlusion

2014

PURPOSE: To evaluate the efficacy and safety of intravitreal aflibercept injections for treatment of macular edema secondary to central retinal vein occlusion (CRVO). DESIGN: A randomized, multicenter, double-masked phase 3 study. PARTICIPANTS: A total of 177 treatment-naive patients with macular edema secondary to CRVO were randomized in a 3:2 ratio. METHODS: Patients received either 2-mg intravitreal aflibercept or sham injections every 4 weeks for 20 weeks. From week 24 to 48, the aflibercept group received aflibercept as needed (pro re nata [PRN]), and the sham group continued receiving sham injections. MAIN OUTCOME MEASURES: The primary efficacy end point was the proportion of patients…

Intraocular pressuremedicine.medical_specialtyVisual acuitygenetic structuresbusiness.industryPhases of clinical researchmedicine.diseaseeye diseases3. Good healthlaw.inventionOphthalmologyCentral retinal vein occlusionRandomized controlled trialPro re natalawOphthalmologymedicinemedicine.symptombusinessMacular edemaAfliberceptmedicine.drugOphthalmology
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The Spectrum of Ocular Manifestations in Patients with Waldenström's Macroglobulinemia.

2021

Purpose: To investigate the ocular manifestations in 91 Waldenstrom's macroglobulinemia (WM) patients.Methods: Retrospective, cross-sectional, observational analysis.Results: Ocular impairments, detected in 19 patients, included flame-shaped hemorrhages, venous sausaging, papilledema, macular detachments, or central retinal vein occlusion in 16 patients; paraproteinemic keratopathy in 2; and a CANOMAD syndrome in 1. Best-corrected visual acuity was ≥0.5 logMAR units in 11 of 38 eyes. Intraocular pressure was increased in seven eyes. Genetic analysis in seven patients showed a mutation in the MYD88 gene in six patients and a nonsense mutation in the CXCR4 gene in five patients. Plasmapheresi…

Intraocular pressuremedicine.medical_specialtyVisual acuitygenetic structuresmedicine.medical_treatment03 medical and health sciences0302 clinical medicineCentral retinal vein occlusionOphthalmologymedicineImmunology and AllergyPapilledemabusiness.industryMacroglobulinemiamedicine.diseaseeye diseases3. Good healthOphthalmology030220 oncology & carcinogenesis030221 ophthalmology & optometryRituximabPlasmapheresismedicine.symptombusinessmedicine.drugRetinopathyOcular immunology and inflammation
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Acute endothelial failure after cosmetic iris implants (NewIris®)

2011

We report a case of an acute endothelial failure after the implantation of a new cosmetic, colored, artificial iris diaphragm implant called NewIris(®). A 21-year-old woman came to us complaining of progressive loss of vision and pain after NewIris lenses had been implanted. Decreased visual acuity, corneal edema, and increased intraocular pressure in both eyes appeared only 3 weeks after the surgery. The lenses were removed as soon as possible but had already severely affected the endothelial cell count. NewIris implants are an alternative to cosmetic contact lenses, but they are not as safe as other phakic anterior chamber intraocular lenses, nor are they a good option for the patient.

Intraocular pressuremedicine.medical_specialtygenetic structuresbusiness.industryCase Reportendothelial failureNewIrisEndothelial cell counteye diseasescosmetic iris implantsSurgeryOphthalmologymedicine.anatomical_structureCorneal edemaIntraocular lensesOphthalmologyDecreased Visual AcuitymedicineImplantsense organsIris (anatomy)businessClinical Ophthalmology (Auckland, N.Z.)
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Structural considerations on the iridoids as anti-inflammatory agents.

1994

Abstract Twelve iridoid glycosides have been evaluated for their anti-inflammatory activity on two models: the carrageenan-induced mouse paw edema and the TPA-induced mouse ear edema. Loganic acid was the most active (44.4% edema inhibition) on the former test, whereas the catalpol derivative mixture isolated from Scrophularia, aucubin, verbenalin, and loganin, showed the highest activity (from 72.0 to 80.0% edema inhibition) on the latter. The results allowed us to establish the relationship between the structure and anti-inflammatory activity on the basis of the different patterns of substitution, particularly hydroxylation, unsaturation, and acylation.

Iridoid GlycosidesIridoidmedicine.drug_classAnti-Inflammatory AgentsPharmaceutical ScienceIridoid GlucosidesAnalytical Chemistrychemistry.chemical_compoundMiceStructure-Activity RelationshipEdemaDrug DiscoverymedicineAnimalsGlycosidesAucubinPharmacologyChemistryLoganinTerpenesOrganic ChemistryCatalpolVerbenalinComplementary and alternative medicineBiochemistryMolecular MedicineFemalemedicine.symptomPlanta medica
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Swelling of glial cells in lactacidosis and by glutamate: significance of Cl(-)-transport.

1993

Swelling of glial and nerve cells is characteristic of brain damage in cerebral ischemia or trauma. The therapeutical efficiency of inhibition of Cl(-)-transport by a novel antagonist, the diuretic torasemide, on cytotoxic swelling of glial cells from lactacidosis, or glutamate was analyzed. Lactacidosis and the interstitial accumulation of glutamate are hallmarks of the pathophysiological alterations in ischemic or traumatic brain tissue. C6 glioma cells harvested from culture and suspended in a physiological medium were either exposed to pH 6.2, or 5.0 by lactic acid, or exposed to 1 mM glutamate at normal pH. Cell swelling and viability were quantified by flow cytometry. Lactacidosis of …

IschemiaGlutamic AcidBrain EdemaPharmacologyIon Channelschemistry.chemical_compoundChloridesGlutamatesChloride ChannelsmedicineTumor Cells CulturedAnimalsDiureticsMolecular BiologyAcidosisCell SizeSulfonamidesIon TransportChemistryGeneral NeuroscienceGlutamate receptorMembrane ProteinsGlutamic acidmedicine.diseaseTorsemideLactic acidRatsmedicine.anatomical_structureBiochemistryCell cultureNeurogliaAcidosis LacticNeurology (clinical)Swellingmedicine.symptomNeurogliaDevelopmental BiologyBrain research
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