Search results for "Endogeny"

showing 10 items of 147 documents

Impulse interval-dependent effect of sympathetic nerve stimulation on evoked acetylcholine release from the rabbit perfused atria preparation.

1995

The aim of the present study was to explore possible prejunctional effects mediated by impulse activity of sympathetic terminals on evoked acetylcholine release in an organ innervated by the autonomic ground plexus. Rabbit atria were isolated with the extrinsic right vagus and sympathetic nerves intact and perfused with Tyrode solution. Acetylcholine overflow was determined after labelling of the transmitter stores with [14C]choline and fractionation of the radioactivity on cation exchange columns. The overflow of endogenous noradrenaline was measured by HPLC and electrochemical detection. The vagus nerve was stimulated at 2 Hz for 3 min four times at intervals of 10 min. During the second …

Malemedicine.medical_specialtySympathetic Nervous SystemSympathetic nerveStimulationEndogenyElectrochemical detectionImpulse (physics)In Vitro TechniquesReceptors PresynapticCholinechemistry.chemical_compoundInternal medicinemedicineElectrochemistryCholineAnimalsChromatography High Pressure LiquidPharmacologyPlexusGanglia SympatheticChemistryMyocardiumHeartVagus NerveGeneral MedicineMyocardial ContractionAcetylcholineElectric StimulationPerfusionEndocrinologycardiovascular systemFemaleRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Bromocriptine and Prostatic Carcinoma: Plasma Kinetics, production and Tissue Uptake of3H-Testosterone in Vivo

1978

The influence of the anti-prolactin bromocriptine on plasma kinetics, production rate and tissue uptake of testosterone was investigated in 15 patients with newly diagnosed stages C and D prostatic carcinoma. Bromocriptine was given for 5 days in a daily dose of 15 mg. orally. The studies were performed with the single injection technique using the 2-compartment model. Plasma testosterone, serum prolactin, and luteinizing and follicle-stimulating hormones were determined initially. Blood samples were drawn up to 5 hours after the injection of 3H-testosterone. For tissue studies a transrectal needle biopsy was done 3 hours post-injection. Bromocriptine suppressed prolactin and the endogenous…

Malemedicine.medical_specialtyUrologyEndogenyIn vivoInternal medicinemedicineCarcinomaHumansTestosteroneBromocriptineAgedbusiness.industryBiopsy NeedleProstatic NeoplasmsLuteinizing Hormonemedicine.diseaseBromocriptineProlactinProlactinKineticsPlasma kineticsEndocrinologyNeedle biopsyFollicle Stimulating HormonebusinessHormonemedicine.drugJournal of Urology
researchProduct

Endogenous oxytocin is essential for the buffering effects of pair housing against the increase in cocaine reward induced by social stress.

2019

Social factors have a dual influence on addictive disorders. While social defeat stress in rodents increases the response to drug reward, positive social conditions, such as pair housing, increase stress resilience. The objective of the present study was to confirm whether oxytocin (OT) mediates this social buffering. To this end, male mice were housed in pairs and administered the OT receptor antagonist atosiban prior to each stress episode or for ten days after the stress protocol. The response to cocaine was assessed using a conditioned place preference paradigm. Our results confirmed that OT activity mediates the protective effect of pair housing and highlights its therapeutic potential.

Malemedicine.medical_specialtymedicine.drug_classmedia_common.quotation_subjectExperimental and Cognitive PsychologyEndogenyOxytocinSocial defeat03 medical and health sciencesBehavioral NeuroscienceMice0302 clinical medicineCocaineRewardInternal medicinemedicineAnimals0501 psychology and cognitive sciences050102 behavioral science & comparative psychologySocial Behaviormedia_commonSocial stressbusiness.industryAddiction05 social sciencesAtosibanReceptor antagonistConditioned place preferenceEndocrinologyOxytocinHousingbusiness030217 neurology & neurosurgeryStress Psychologicalmedicine.drugPhysiologybehavior
researchProduct

Nitric Oxide Modulates the Acute Increase of Gastrointestinal Transit Induced by Endotoxin in Rats: a Possible Role for Tachykinins

1997

Abstract Because of the evidence that endogenous nitric oxide (NO) plays an essential role in the physiological regulation of gastrointestinal motility we have investigated, by use of the NO synthase inhibitor, NG-nitro-l-arginine methyl ester (l-NAME), the role of endogenous NO in the acute endotoxin-induced changes of gastrointestinal transit. Pre-treatment with E. coli endotoxin (100 μg kg−, i.v.) induced a significant increase in the gastrointestinal transit of a charcoal suspension in anaesthetized rats. Previous administration of the NO synthase inhibitor, l-NAME (10 mg kg−, i.v.) significantly prevented the effects of endotoxin. l-arginine (200 mg kg−, i.v.) and the substance P antag…

Malemedicine.medical_specialtymedicine.medical_treatmentAnti-Inflammatory AgentsPharmaceutical ScienceMotilityEndogenySubstance PBiologyNitric OxideDexamethasoneNitric oxidePathogenesischemistry.chemical_compoundTachykininsInternal medicineNeuromodulationmedicineAnimalsEnzyme InhibitorsRats WistarGastrointestinal TransitDexamethasonePharmacologyRatsEndotoxinsNG-Nitroarginine Methyl EsterEndocrinologyCytokinemedicine.anatomical_structurechemistryToxicityNitric Oxide Synthasemedicine.drugJournal of Pharmacy and Pharmacology
researchProduct

A non-redundant role for OX40 in the competitive fitness of Treg in response to IL-2.

2010

OX40 stimulation is known to enhance activation of effector T cells and to inhibit induction and suppressive function of Treg. Here we uncovered a novel role of OX40 in sustaining Treg competitive fitness in vivo, during repopulation of lymphopenic hosts and reconstitution of BM chimeras. Defective expansion of OX40-null Treg diminished their ability to suppress inflammation in a model of lymphopenia-driven colitis. OX40-mediated promotion of Treg fitness spanned beyond lymphopenic environments, as endogenous Treg in OX40-null mice showed decreased accumulation during thymic development, enhanced susceptibility to antibody-mediated depletion and defective turnover following thymectomy. In v…

Malemedicine.medical_treatmentImmunologyBlotting Westernchemical and pharmacologic phenomenaEndogenyInflammationSuppressor of Cytokine Signaling ProteinsT-Lymphocytes RegulatoryMiceSuppressor of Cytokine Signaling 1 ProteinLymphopeniaOX40; Treg; IL-2.medicineSTAT5 Transcription FactorImmunology and AllergyAnimalsOX40PhosphorylationReceptorSTAT5Cell ProliferationMice KnockoutbiologyEffectorCell growthSuppressor of cytokine signaling 1hemic and immune systemsReceptors OX40IL-2.ColitisFlow Cytometrycytokinescompetitive fitnessSpecific Pathogen-Free OrganismsThymectomyMice Inbred C57BLTregRadiation ChimeraImmunologybiology.proteinInterleukin-2costimulatory moleculesmedicine.symptomcompetitive fitness; costimulatory molecules; cytokines; treg
researchProduct

Tumornekrosefaktor und Interferon als prognostische Marker der HIV-Infektion

1991

Peripheral blood cells were obtained from patients at different stages of their human immunodeficiency virus (HIV) infection. It was found that the capacity to generate interferon alpha was reduced already at Walter Reed stage 2 (WR) while the interferon gamma capacity remained largely unaffected until WR stage 4. Endogenous tumor necrosis factor (TNF) alpha production increased as the HIV disease progressed. The data obtained add to our knowledge on destruction of the immune system by the HIV. Moreover TNF and acid labile interferon alpha might contribute to HIV replication and disease progression. Nevertheless the tests performed are too time-consuming to be introduced into routine analys…

Microbiology (medical)Human immunodeficiency virus (HIV)Alpha (ethology)Alpha interferonEndogenyGeneral MedicineBiologymedicine.disease_causeVirologyInfectious DiseasesImmune systemInterferonImmunologymedicineTumor necrosis factor alphaInterferon gammamedicine.drugInfection
researchProduct

Reduction of nevirapine-driven HIV mutations by carbamazepine is modulated by CYP3A activity

2014

Item does not contain fulltext OBJECTIVES: The reduction in mother-to-child transmission of HIV-1 by single-dose nevirapine given at birth onset is achieved at the expense of de novo HIV-1 resistance mutations. In the VITA1 study, single-dose carbamazepine accelerated nevirapine elimination, but the accompanying trend towards fewer de novo HIV-1 mutations was statistically non-significant. METHODS: We investigated if the effect of carbamazepine was confounded by the individual variability in nevirapine metabolism and transport. RESULTS: Nine of 34 (26%) single-dose nevirapine-treated women had one or more nevirapine-associated resistance mutations, compared with 3 of 34 (9%) in the single-d…

Microbiology (medical)NevirapineCYP3AAnti-HIV AgentsHuman immunodeficiency virus (HIV)Mutation MissenseEndogenyHIV InfectionsPharmacologyBiologymedicine.disease_causeChemopreventionPregnancyDrug Resistance ViralmedicineClinical endpointCytochrome P-450 CYP3AHumansPharmacology (medical)NevirapinePharmacologyMutationCYP3A4Cytochrome P-450 CYP3A InducersCarbamazepinelnfectious Diseases and Global Health Radboud Institute for Health Sciences [Radboudumc 4]Infectious DiseasesCarbamazepineTreatment OutcomeHIV-1Femalemedicine.drug
researchProduct

Electrophysiological Analysis of Effects of Exogenous and Endogenous Adenosine in Hippocampal Slices

1988

Little doubt remains that adenosine serves a modulatory role in the nervous system (Sattin and Rail, 1970; Shimizu and Daly, 1970; Fredholm and Hedqvist, 1980; Phillis and Wu, 1981; Stone, 1981; Daly et al., 1984; Dunwiddie, 1985). While electrophysiologically active concentrations of adenosine have been found in the extracellular fluid, the source and control of the levels of this active adenosine are unclear. There are indications that adenosine is released (or coreleased) from synaptic endings, fulfilling a typical transmitter role (Burnstock, 1975, 1981; Nagy et al., 1986). A synaptic hyperpolarization in autonomic neurons has been identified as an adenosine-mediated potential (Akasu et…

Nervous systemElectrophysiologymedicine.anatomical_structureChemistrymedicineEndogenyPopulation spikeHippocampal formationHyperpolarization (biology)Inhibitory postsynaptic potentialAdenosineNeurosciencemedicine.drug
researchProduct

Endogenous and exogenous nitric oxide inhibits norepinephrine release from rat heart sympathetic nerves.

1995

Abstract This study was designed to elucidate whether nitric oxide (NO) controls norepinephrine (NE) release from sympathetic nerves of the rat heart. Hearts were perfused in the Langendorff mode with Tyrode’s solution. The right sympathetic nerve was stimulated with trains of 1 or 3 Hz and NE release was measured. The NO synthase (NOS) inhibitor N G -nitro- l -arginine (L-NNA) enhanced the evoked NE release in a concentration-dependent manner. This facilitation was independent of the increase in perfusion pressure and was stereospecifically reversed by l -arginine but not d -arginine. Another NOS inhibitor, N G -methyl- l -arginine, produced a similar increase in NE release. The NO-donor …

Nervous systemMalemedicine.medical_specialtySympathetic Nervous SystemPhysiologyEndogenyNeurotransmissionIn Vitro TechniquesArginineNitric OxideNitric oxideNorepinephrine (medication)Rats Sprague-Dawleychemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsEnzyme Inhibitorsomega-N-MethylargininebiologyChemistryHeartImmunohistochemistryRatsNitric oxide synthasemedicine.anatomical_structureEndocrinologyCatecholaminebiology.proteinLiberationFemaleNitric Oxide SynthaseCardiology and Cardiovascular Medicinemedicine.drugCirculation research
researchProduct

Treatment of skeletal impairment in patients with endogenous hypercortisolism: when and how?

2014

Guidelines for the management of osteoporosis induced by endogenous hypercortisolism are not available. Both the American College of Rheumatology and the International Osteoporosis Foundation recommend to modulate the treatment of exogenous glucocorticoid-induced osteoporosis (GIO) based on the individual fracture risk profile (calculated by FRAX) and dose of glucocorticoid used, but it is difficult to translate corticosteroid dosages to different degrees of endogenous hypercortisolism, and there are no data on validation of FRAX stratification method in patients with endogenous hypercortisolism. Consequently, it is unclear whether such recommendations may be adapted to patients with endoge…

Oncologymedicine.medical_specialtyFRAXEndocrinology Diabetes and MetabolismOsteoporosisHypercortisolismAdrenal incidentaloma; Cushing's disease; Glucocorticoids; Osteoporosis; Bone Density Conservation Agents; Cushing Syndrome; Glucocorticoids; Humans; Osteoporosis; Osteoporotic Fractures; Risk FactorsEndogenyDiseaseadrenal incidentalomaBone remodelingRisk FactorsInternal medicinemedicineHumansCushing SyndromeBone Density Conservation Agentsglucocorticoidsbusiness.industrycushing's diseaseCushing's diseasemedicine.diseaseosteoporosisRheumatologyglucocorticoids; osteoporosis; cushing's disease; adrenal incidentalomaEndocrinologyAdrenal incidentaloma; CUSHING'S DISEASE; Glucocorticoids; OsteoporosisbusinessGlucocorticoidOsteoporotic Fracturesmedicine.drug
researchProduct