Search results for "Enterocyte"

showing 8 items of 28 documents

Chitosan-coated mesoporous MIL-100(Fe) nanoparticles as improved bio-compatible oral nanocarriers

2017

Nanometric biocompatible Metal-Organic Frameworks (nanoMOFs) are promising candidates for drug delivery. Up to now, most studies have targeted the intravenous route, related to pain and severe complications; whereas nanoMOFs for oral administration, a commonly used non-invasive and simpler route, remains however unexplored. We propose here the biofriendly preparation of a suitable oral nanocarrier based on the benchmarked biocompatible mesoporous iron(III) trimesate nanoparticles coated with the bioadhesive polysaccharide chitosan (CS). This method does not hamper the textural/structural properties and the sorption/release abilities of the nanoMOFs upon surface engineering. The interaction …

Materials scienceBiocompatibilityBioadhesiveQuímica organometàl·licaNanoparticleAdministration OralNanotechnology02 engineering and technologySurface engineering010402 general chemistry01 natural sciencesFerric CompoundsArticleChitosanchemistry.chemical_compoundHumansChitosanMultidisciplinaryNanotecnologia021001 nanoscience & nanotechnology3. Good health0104 chemical sciencesDrug LiberationKineticsLysergic Acid DiethylamideEnterocyteschemistryDrug deliveryNanoparticlesNanocarriersCaco-2 Cells0210 nano-technologyMesoporous material
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7keto-stigmasterol and 7keto-cholesterol induce differential proteome changes to intestinal epitelial (Caco-2) cells

2015

Abstract Recent studies have expanded the appreciation of the roles of oxysterols triggering inflammatory, immune cytotoxic and apoptotic processes, but have not been considered for proteome analysis. A comparative proteomic study in intestinal epithelial cell cultures incubated (60 μM/24 h) with 7keto-cholesterol or 7keto-stigmasterol was performed. The influence of both compounds was studied following the nLC-TripleTOF analysis. Findings were compared to results for control cultures. In the principal component analysis (PCA) of proteome patterns, two components were extracted accounting for 99.8% of the variance in the protein expression. PCA analysis clearly discriminated between the per…

ProteomeStigmasterolInflammationBiologyToxicologyPeptide MappingImmune systemmedicineHumansRNA MessengerKetocholesterolsTranscription factorPrincipal Component AnalysisCell growthGene Expression ProfilingGeneral MedicineOxidantsCell biologyEnterocytesGene Expression RegulationBiochemistryCell cultureApoptosisProteomeMacrophage migration inhibitory factorCaco-2 Cellsmedicine.symptomFood ScienceFood and Chemical Toxicology
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Iron bioavailability in iron-fortified cereal foods: The contribution of in vitro studies

2015

Iron deficiency anemia is the most common nutritional deficiency in humans. Not all dietary ingested iron, heme or nonheme, will be available to absorption and negative imbalance between iron requirements and absorption leads to iron deficiency and/or anemia. The recommended iron values usually are based on the genetic and on diet iron-bioavailability, which can be considered as the principal factor that change among the cultures and influences the distinct levels of recommendation among countries. Dietary changes present practical limitations due to be difficult to change food habits. The iron food fortification is considered more cost effective and economically more attractive than iron s…

Risk0301 basic medicineIron OverloadAnemiaFlourWheat flourGlobal HealthIndustrial and Manufacturing EngineeringFerrousFood group03 medical and health sciencesmedicineAnimalsHumansCookingFood science030109 nutrition & dieteticsAnemia Iron-DeficiencyChemistryFood fortificationfood and beveragesBreadGeneral MedicineIron deficiencymedicine.diseaseBioavailabilityEnterocytesFood StorageIntestinal AbsorptionIron-deficiency anemiaFood FortifiedCaco-2 CellsEdible GrainNutritive ValueIron DietaryFood ScienceCritical Reviews in Food Science and Nutrition
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Searching for wheat plants with low toxicity in celiac disease: Between direct toxicity and immunologic activation.

2009

Abstract Background Natural or induced variations in the noxiousness of gluten proteins for celiac disease (CD) patients are currently being investigated for their potential in breeding wheat crops with reduced toxicity. Aims We evaluated the bread wheat line C173 for its effects on the in vitro -grown duodenal mucosa of CD patients. Methods In vitro -grown duodenal mucosa biopsies of 19 CD patients on a gluten-free diet were exposed to peptic/tryptic-digested prolamins from bread wheat line C173 lacking gliadin–glutenin subunits, analyzed for morphology, cytokine and anti-tTG antibody production, and compared with mucosa biopsies exposed to prolamins from wild-type cv. San Pastore. Results…

Settore MED/09 - Medicina InternaEnterocytemedicine.medical_treatmentAntibodiesTissue Culture TechniquesImmunologic activationInterferon-gammamedicineHumansIntestinal MucosaProlaminCommon wheatTriticumHepatologybiologyGastroenterologyfood and beveragestoxicityimmunologic activation.Interleukin-10Cytokinemedicine.anatomical_structureAnti-transglutaminase antibodiesImmunologyToxicitybiology.proteinInterleukin-2AntibodyGene Deletionwheat plantceliac diseaseProlamins
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Role of tir and intimin in the virulence of rabbit enteropathogenic Escherichia coli serotype O103:H2.

2000

ABSTRACT Attaching and effacing (A/E) rabbit enteropathogenic Escherichia coli (REPEC) strains belonging to serogroup O103 are an important cause of diarrhea in weaned rabbits. Like human EPEC strains, they possess the locus of enterocyte effacement clustering the genes involved in the formation of the A/E lesions. In addition, pathogenic REPEC O103 strains produce an Esp-dependent but Eae (intimin)-independent alteration of the host cell cytoskeleton characterized by the formation of focal adhesion complexes and the reorganization of the actin cytoskeleton into bundles of stress fibers. To investigate the role of intimin and its translocated coreceptor (Tir) in the pathogenicity of REPEC, …

Time Factors[SDV]Life Sciences [q-bio]MutantAdministration OralPATHOGENICITEmedicine.disease_causeBacterial AdhesionMICROSCOPIE ELECTRONIQUE A TRANSMISSIONFecesCytoskeleton0303 health sciencesVirulenceEscherichia coli ProteinsEnterobacteriaceae3. Good health[SDV] Life Sciences [q-bio]IntestinesInfectious DiseasesMolecular and Cellular PathogenesisRabbitsLocus of enterocyte effacementBacterial Outer Membrane ProteinsImmunologyMolecular Sequence DataVirulenceReceptors Cell SurfaceBiologyMicrobiologydigestive systemMicrobiologyCell Line03 medical and health sciencesBacterial ProteinsIleummedicineEscherichia coliAnimalsHumansEnteropathogenic Escherichia coliAdhesins BacterialEscherichia coli030304 developmental biologyIntiminModels Genetic030306 microbiologyGenetic Complementation TestEpithelial Cellsbiochemical phenomena metabolism and nutritionbiology.organism_classificationActin cytoskeleton[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyActinsKineticsMicroscopy ElectronMicroscopy FluorescenceMutagenesisParasitologyCarrier ProteinsHeLa CellsInfection and immunity
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Fermentation Products of Commensal Bacteria Alter Enterocyte Lipid Metabolism

2020

eferred to byJia Wen, John F. RawlsFeeling the Burn: Intestinal Epithelial Cells Modify Their Lipid Metabolism in Response to Bacterial Fermentation ProductsCell Host & Microbe, Volume 27, Issue 3, 11 March 2020, Pages 314-316; International audience; Despite the recognized capacity of the gut microbiota to regulate intestinal lipid metabolism, the role of specific commensal species remains undefined. Here, we aimed to understand the bacterial effectors and molecular mechanisms by which Lactobacillus paracasei and Escherichia coli regulate lipid metabolism in enterocytes. We show that L-lactate produced by L. paracasei inhibits chylomicron secretion from enterocytes and promotes lipid stora…

[SDV.IMM] Life Sciences [q-bio]/ImmunologyEnterocyteBiologyGut floraMicrobiologyCell Linelipids03 medical and health sciences0302 clinical medicineLipid oxidationVirologyChylomicronsmedicineEscherichia coliAnimalsSecretionSymbiosis030304 developmental biology0303 health sciencescommensal bacteriaAMPKLipid metabolismMetabolismLacticaseibacillus paracaseiL-lactatebiology.organism_classificationLipid MetabolismCell biologyIntestinesMice Inbred C57BLmedicine.anatomical_structureEnterocytesFermentation[SDV.IMM]Life Sciences [q-bio]/ImmunologyParasitologyFemalelipids (amino acids peptides and proteins)acetatesmall intestine030217 neurology & neurosurgeryChylomicron
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Enteropathogenic Escherichia coli O103 from rabbit elicits actin stress fibers and focal adhesions in HeLa epithelial cells, cytopathic effects that …

1997

Escherichia coli O103, a major agent of weaned-rabbit diarrhea in Western Europe, was previously shown to produce diarrhea and attaching-and-effacing intestinal lesions in experimentally infected rabbits and to possess a homolog of the eaeA gene of enteropathogenic E. coli (EPEC). In the present study, we have shown that although negative in the fluorescent-actin staining test on HeLa cells, prototype rabbit E. coli O103 strain B10 was able to induce an original cytopathic effect (CPE) in the same interaction model. This CPE was characterized by a generalized reorganization of the actin cytoskeleton and the formation of focal adhesions on the entire surface of the target cells. These effect…

[SDV]Life Sciences [q-bio]ImmunologyMutantVirulencemedicine.disease_causeMicrobiologyMicrobiology03 medical and health sciencesCytopathogenic Effect ViralmedicineCell AdhesionEscherichia coliAnimalsHumansEnteropathogenic Escherichia coliEscherichia coliEscherichia coli InfectionsComputingMilieux_MISCELLANEOUS030304 developmental biologyCytopathic effect0303 health sciencesbiologyCell DeathVirulence030306 microbiologyEpithelial Cellsbiochemical phenomena metabolism and nutritionbiology.organism_classificationActin cytoskeletonEnterobacteriaceaeActins3. Good healthIntestines[SDV] Life Sciences [q-bio]Infectious DiseasesMutagenesisParasitologyRabbitsLocus of enterocyte effacementResearch ArticleHeLa Cells
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Intestinal conversion of linoleic acid to arachidonic acid in the rat

1993

Abstract The arachidonic acid (C20:4, n-6) appearing in intestinal lymph during linoleic acid (C18:2, n-6) absorption may originate from enterocyte synthesis or from the liver either after secretion in biliary phospholipids at the same time dietary linoleic acid absorption occurs or via plasma. The radioactivity measured in the total bile collected during the 6 hours of linoleic acid absorption is too small to explain hepatic origin of the C20:4 detected by high performance liquid chromatography analysis of labeled fatty acids recovered in the lymph, in the intestinal mucosa, and the intestinal wall at the peak of linoleic acid intestinal absorption. This study confirms the probability that…

chemistry.chemical_classificationNutrition and DieteticsEnterocyteEndocrinology Diabetes and MetabolismLinoleic acidClinical BiochemistryBiologyBiochemistryIntestinal absorptionchemistry.chemical_compoundmedicine.anatomical_structurechemistryIntestinal mucosaBiochemistrymedicineArachidonic acidLymphDigestionMolecular BiologyPolyunsaturated fatty acidThe Journal of Nutritional Biochemistry
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