Search results for "Epilepsy"

showing 10 items of 420 documents

Early Seizures After Acute Stroke: A Study of 1,640 Cases

1994

We evaluated prospectively the occurrence of seizures within 15 days of a first stroke or transient ischemic episode in 1,640 patients to study relation between seizures and type of stroke. Seizures occurred in 90 patients (5.4%), including 36 (4.4%) of 814 with infarct owing to atheroma, 21 (16.6%) of 126 with infarct owing to cardiogenic embolus, 3 (1%) of 273 owing to lacunar infarct, 5 (1.9%) of 259 owing to transient ischemic attack (TIA), 21 (16.2%) of 129 owing to supratentorial hematoma, and 4 (16.6%) of 24 owing to subarachnoid hemorrhage. Thirteen (14.6%) of 89 subcortical infarcts were associated with seizures. Seizures were the initial sign of stroke in 80 (89%) of 90 cases and …

MaleSubarachnoid hemorrhageComorbidityCentral nervous system diseaseHematomaEmbolusmedicineHumansProspective StudiesRegistriescardiovascular diseasesProspective cohort studyStrokeAgedAged 80 and overEpilepsybusiness.industryVascular diseaseIncidenceCerebral InfarctionIntracranial Embolism and ThrombosisMiddle AgedSubarachnoid HemorrhageIntracranial Arteriosclerosismedicine.diseaseCerebrovascular DisordersNeurologyEmbolismIschemic Attack TransientAnesthesiacardiovascular systemRegression AnalysisFemaleFranceNeurology (clinical)businessEpilepsia
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Symbolic function explored in children with epilepsy and headache.

2007

Introduzione. L’attacco alla capacità di pensare è uno dei possibili effetti che possiamo osservare quando il corpo si ammala. Lo scopo di questo studio è esplorare la funzione simbolica in bambini con epilessia e cefalea. Metodi. Sono stati reclutati 75 piccoli pazienti della Clinica Neuropsichiatrica infantile dell’Università di Palermo - 48 maschi (61%) e 27 femmine (39%). Tutti soffrivano di epilessia (53 - 71%) e cefalea primaria (22 - 29%). La loro età variava dai 7 agli 11 anni (media: 9.4±1.2). È stato reclutato anche un gruppo di controllo composto da soggetti che non avevano mai sofferto di malattie croniche o neurologiche. Tale gruppo era pareggiato con il gruppo di pazienti per …

MaleSymbolismEpilepsyepilepsy headache defense mechanisms symbolic function simbolisation mentalizationHeadacheHumansFemaleNeuropsychological TestsChildCognition DisordersSeverity of Illness IndexMinerva pediatrica
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Non-convulsive status epilepticus associated with tiagabine in a pediatric patient

2003

We report a 4-year-old patient who developed non-convulsive status epilepticus (NCSE) following tiagabine (TGB) as add-on treatment for refractory partial seizures. NCSE occurred while the patient received TGB 0.83mg/kg/day. In our case, the TGB reduction led to a significant improvement of electroclinical features. The mechanisms of this abnormal effect are not clear. GABA-ergic hyperfunction and/or multiplicity of interlinked brain GABA systems associated with individual specific sensitivity could play a critical role in the pathogenesis of NCSE. This is the first report of NCSE documented by electroencephalogram (EEG) in a child under 12 years of age on TGB treatment. © 2003 Elsevier Sci…

MaleTiagabinemedicine.medical_treatmentNipecotic AcidsStatus epilepticusNon-convulsive status epilepticuElectroencephalographyCentral nervous system diseaseEpilepsyStatus EpilepticusDevelopmental NeuroscienceRefractorymedicineHumansTiagabineEpilepsymedicine.diagnostic_testbusiness.industryElectroencephalographyGeneral Medicinemedicine.diseaseAnticonvulsantnervous systemEl NiñoChild PreschoolAnesthesiaPediatrics Perinatology and Child HealthAnticonvulsantsEpilepsies PartialNeurology (clinical)medicine.symptombusinessmedicine.drugBrain and Development
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Continuous intra-amygdalar infusion of GABA in the amygdala kindling model of epilepsy in rat.

2003

Objective: To explore the effect of continuous intra-amygdalar infusion of GABA in the amygdala kindling model of epilepsy in rat. Methods: An electrode and cannula complex was implanted in adult male Wistar rats, the electrode being targeted to the left basolateral amygdala. The animals were subjected to a standard kindling procedure. Osmotic minipumps filled with either GABA or mannitol were connected to cannulas and allowed to infuse during 7 days. Kindling experiments measuring after-discharge and seizure thresholds, seizure severity and duration, and behavioral toxicity were performed before, during and after the drug infusion period. Results: Both after-discharge and seizure threshold…

MaleTime FactorsCentral nervous systemPharmacologyAmygdalachemistry.chemical_compoundEpilepsyBasal gangliamedicineKindling NeurologicAnimalsRats WistarNeurotransmittergamma-Aminobutyric AcidEpilepsyDose-Response Relationship Drugbusiness.industryKindlingInfusion Pumps Implantablemedicine.diseaseAmygdalaElectric StimulationRatsDisease Models Animalmedicine.anatomical_structurenervous systemNeurologychemistryAnesthesiaToxicityAnticonvulsantsNeurology (clinical)businessBasolateral amygdalaEpilepsy research
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Antiepileptic effect of dimethyl sulfoxide in a rat model of temporal lobe epilepsy.

2012

Dimethyl sulfoxide (DMSO) is an amphipathic molecule widely used to solubilize water-insoluble compounds. In many studies it was reported that DMSO is capable of affecting several biological processes, thus resulting in a potential cause for the misinterpretation of experimental data. Recent papers showed that DMSO modified the brain bioelectric activity in animal models of epilepsy. In an in vivo model of temporal lobe epilepsy in the rat, we examined the effects of different doses (10%, 50% and 100%) of DMSO on the maximal dentate activation (MDA). The results show that DMSO induced a dose-dependent significant reduction of the electrically induced paroxysmal activity.

MaleTreatment outcomeRat modelAction PotentialsPharmacologySettore BIO/09 - FisiologiaTemporal lobeEpilepsychemistry.chemical_compoundIn vivomedicineAnimalsHumansDimethyl SulfoxideRats WistarTemporal lobe epilepsyDose-Response Relationship DrugChemistryDimethyl sulfoxideGeneral Neurosciencemedicine.diseaseRatsDose–response relationshipDisease Models AnimalMaximal dentate activationTreatment OutcomeBiochemistryCerebellar NucleiEpilepsy Temporal LobeSolubilizationAnticonvulsantsNeuroscience letters
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The endocannabinoid system controls key epileptogenic circuits in the hippocampus.

2006

SummaryBalanced control of neuronal activity is central in maintaining function and viability of neuronal circuits. The endocannabinoid system tightly controls neuronal excitability. Here, we show that endocannabinoids directly target hippocampal glutamatergic neurons to provide protection against acute epileptiform seizures in mice. Functional CB1 cannabinoid receptors are present on glutamatergic terminals of the hippocampal formation, colocalizing with vesicular glutamate transporter 1 (VGluT1). Conditional deletion of the CB1 gene either in cortical glutamatergic neurons or in forebrain GABAergic neurons, as well as virally induced deletion of the CB1 gene in the hippocampus, demonstrat…

MaleVesicular glutamate transporter 1HUMDISEASEHippocampusGene ExpressionHippocampal formationHippocampusMembrane Potentialschemistry.chemical_compoundMice0302 clinical medicineReceptor Cannabinoid CB1Premovement neuronal activitygamma-Aminobutyric Acid0303 health sciencesKainic AcidbiologyBehavior AnimalReverse Transcriptase Polymerase Chain Reactionmusculoskeletal neural and ocular physiologyGeneral NeurosciencePyramidal CellsCalcium Channel BlockersEndocannabinoid systemlipids (amino acids peptides and proteins)psychological phenomena and processesmedicine.drugKainic acidNeuroscience(all)MorpholinesGlutamic AcidMice TransgenicNaphthalenesMOLNEUROgamma-Aminobutyric acid03 medical and health sciencesGlutamatergicCannabinoid Receptor ModulatorsmedicineAnimals030304 developmental biologyAnalysis of VarianceEpilepsyBenzoxazinesMice Inbred C57BLnervous systemchemistryCalcium-Calmodulin-Dependent Protein KinasesVesicular Glutamate Transport Protein 1biology.proteinNerve NetSYSNEUROCalcium-Calmodulin-Dependent Protein Kinase Type 2Neuroscience030217 neurology & neurosurgeryEndocannabinoidsNeuron
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Mutations in SLC13A5 Cause Autosomal-Recessive Epileptic Encephalopathy with Seizure Onset in the First Days of Life

2014

International audience; Epileptic encephalopathy (EE) refers to a clinically and genetically heterogeneous group of severe disorders characterized by seizures, abnormal interictal electro-encephalogram, psychomotor delay, and/or cognitive deterioration. We ascertained two multiplex families (including one consanguineous family) consistent with an autosomal-recessive inheritance pattern of EE. All seven affected individuals developed subclinical seizures as early as the first day of life, severe epileptic disease, and profound developmental delay with no facial dysmorphism. Given the similarity in clinical presentation in the two families, we hypothesized that the observed phenotype was due …

Male[SDV]Life Sciences [q-bio]Genes Recessive[SDV.GEN] Life Sciences [q-bio]/GeneticsBiologymedicine.disease_causeCompound heterozygosity03 medical and health sciencesEpilepsy0302 clinical medicineSeizures[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyReportmedicineGeneticsRecessiveHumansIctalGenetics(clinical)[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Genetics (clinical)Exome sequencing030304 developmental biologySubclinical infectionGenetics0303 health sciencesMutation[SDV.GEN]Life Sciences [q-bio]/GeneticsBrain Diseases[SDV.MHEP] Life Sciences [q-bio]/Human health and pathology[ SDV ] Life Sciences [q-bio]SymportersGenetic heterogeneityCitrate transportmedicine.disease3. Good healthPedigree[SDV] Life Sciences [q-bio]Genes[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Mutation[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Female[ SDV.GEN ] Life Sciences [q-bio]/Genetics030217 neurology & neurosurgery[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyThe American Journal of Human Genetics
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Inhibitory effects of N-valproyl-L-tryptophan on high potassium, low calcium and low magnesium-induced CA1 hippocampal epileptiform bursting activity…

2012

N-valproyl-l-tryptophan (VPA-Tryp), new antiepileptic drug, was tested on CA1 hippocampal epileptiform bursting activity obtained by increasing potassium and lowering calcium and magnesium concentrations in the fluid perfusing rat brain slices. Each slice was treated with a single concentration (0.2, 0.5, 1 or 2 mM) of Valproate (VPA) or VPA-Tryp. Both burst duration and interburst frequency during and after treatment were off-line compared with baseline values. For both parameters, the latency and the length of statistically significant response periods as well as the magnitude of drug-induced responses were calculated. VPA-Tryp evoked fewer and weaker early excitatory effects than VPA on …

Maleantiepileptic drug valproic acidPotassiumchemistry.chemical_elementAction PotentialsCalciumHippocampal formationPharmacologyIn Vitro TechniquesInhibitory postsynaptic potentialSettore BIO/09 - Fisiologiaamino-acidic derivativeBurstingmedicineReaction Timehippocampal epilepsyAnimalsDrug InteractionsMagnesiumRats WistarCA1 Region HippocampalBiological PsychiatryValproic AcidAnalysis of VarianceDose-Response Relationship DrugMagnesiumDipeptidesElectric StimulationRatsPsychiatry and Mental healthNeurologychemistrySettore CHIM/09 - Farmaceutico Tecnologico ApplicativoExcitatory postsynaptic potentialPotassiuminterictal burstslipids (amino acids peptides and proteins)AnticonvulsantsNeurology (clinical)medicine.drugJournal of neural transmission (Vienna, Austria : 1996)
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Aplasia of the retinal vessels combined with optic nerve hypoplasia, neonatal epileptic seizures, and lactic acidosis due to mitochondrial complex I …

1992

A newborn male with mitochondrial complex I deficiency suffered from neonatal epileptic seizures, which later developed into infantile spasms. The infant was blind due to aplasia of the retinal vessels and hypoplasia of the optic nerve. There was congenital lactic acidosis, which persisted in later life. The boy was microcephalic and retarded. Muscular hypotonia later shifted to spasticity. Succinic acid was increased in urine. We assume that the aplasia of the retinal vessels is due to damage of the retinal ganglion cells caused by the mitochondrial disease in the first 3 to 4 months of pregnancy.

Malecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyCongenital lactic acidosisRetinal ganglionInternal medicineMedicineHumansNADH NADPH OxidoreductasesOptic nerve hypoplasiaRetinaElectron Transport Complex IEpilepsybusiness.industryInfant NewbornBrainRetinal VesselsOptic NerveAplasiamedicine.diseaseHypoplasiaMitochondriabody regionsEndocrinologymedicine.anatomical_structureLactic acidosisPediatrics Perinatology and Child HealthOptic nerveAcidosis LacticbusinessTomography X-Ray ComputedEuropean journal of pediatrics
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Effects of electrical stimulation in vestibular cortex areas in humans.

2010

The case of a patient with focal epilepsy is reported who underwent presurgical evaluation by stereotactic intracranial electroencephalographic (EEG) recording. A subdural semi-grid electrode, consisting of three multi-channel strip electrodes, was implanted over the temporal lobe and temporo-occipital region; one multi-channel depth electrode was applied towards the posterior insular cortex. During electrical stimulation and EEG monitoring eye movements were recorded by 3-D video-oculography. Stimulation of the medial temporal gyrus induced blurring of vision and horizontal nystagmus. Stimulation of the superior temporal gyrus with low intensities also induced blurring of vision and a simi…

Malegenetic structuresEye MovementsNystagmusElectroencephalographyInsular cortexTemporal lobeSuperior temporal gyrusYoung AdultGyrusNystagmus PhysiologicMonitoring IntraoperativePreoperative CaremedicineHumansPostural BalanceBrain MappingEpilepsymedicine.diagnostic_testEye movementElectroencephalographyReflex Vestibulo-OcularVestibular cortexeye diseasesElectric StimulationTemporal LobeElectrophysiologymedicine.anatomical_structureNeurologyNeurology (clinical)Vestibule Labyrinthmedicine.symptomPsychologyNeurosciencePsychomotor PerformanceJournal of the neurological sciences
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