Search results for "Equilibrium"

showing 10 items of 1021 documents

On the fractional probabilistic Taylor's and mean value theorems

2016

In order to develop certain fractional probabilistic analogues of Taylor's theorem and mean value theorem, we introduce the nth-order fractional equilibrium distribution in terms of the Weyl fractional integral and investigate its main properties. Specifically, we show a characterization result by which the nth-order fractional equilibrium distribution is identical to the starting distribution if and only if it is exponential. The nth-order fractional equilibrium density is then used to prove a fractional probabilistic Taylor's theorem based on derivatives of Riemann-Liouville type. A fractional analogue of the probabilistic mean value theorem is thus developed for pairs of nonnegative rand…

Generalized Taylor’s formulaMean value theoremSurvival bounded order01 natural sciencesStochastic ordering010104 statistics & probabilityCharacterization of exponential distribution; Fractional calculus; Fractional equilibrium distribution; Generalized Taylor’s formula; Mean value theorem; Survival bounded orderFOS: MathematicsCharacterization of exponential distributionApplied mathematics0101 mathematicsMathematicsComputer Science::Information RetrievalApplied MathematicsProbability (math.PR)010102 general mathematicsProbabilistic logic60E99 26A33 26A24Fractional calculusFractional equilibrium distributionFractional calculusExponential functionDistribution (mathematics)Bounded functionMean value theorem (divided differences)Random variableAnalysisMathematics - Probability
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Evidence for a common origin of most Friedreich ataxia chromosomes in the Spanish population

1996

Haplotype analysis is a powerful approach to understand the spectrum of mutations accounting for a disease in a homogeneous population. We show that haplotype variation for 10 markers linked to the Friedreich ataxia locus (FRDA) argues in favor of an important mutation homogeneity in the Spanish population, and positions the FRDA locus in the region where it has been recently isolated. We also report the finding of a new single nucleotide polymorphism called FAD1. The new marker shows a very strong linkage disequilibrium with Friedreich ataxia (FA) in both the Spanish and French populations. suggesting the existence of an ancient and widespread FRDA mutations. Inclusion of FAD1 in the exten…

Genetic MarkersLinkage disequilibriumAtaxiaMolecular Sequence DataPopulationNerve Tissue ProteinsSingle-nucleotide polymorphismLocus (genetics)BiologyLinkage DisequilibriumTrinucleotide RepeatsGeneticsmedicineHumanseducationPhylogenyGenetics (clinical)Adaptor Proteins Signal TransducingGeneticseducation.field_of_studyPolymorphism GeneticBase SequenceHaplotypeIntronChromosome MappingIntronsHaplotypesFriedreich AtaxiaSpainGenetic markerMutationFrancemedicine.symptom
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Suggestive evidence for association of D2S2188 marker (2q31.1) with autism in 143 Sicilian (Italian) TRIO families

2005

We have screened 143 Sicilian (Italian) families with one autistic child to verify, by a linkage disequilibrium approach, the involvement of the 2q31.1 region in the cause of the disease in these families. Our study design includes the use of intrafamilial association to prevent a population stratification bias and ethnic homogeneity of the sample. The results of our analysis provided suggestive evidence of the occurrence of transmission disequilibrium between autism and the D2S2188 polymorphism in Sicilian TRIO families, a finding which provides further and independent support to the hypothesis of the existence of a susceptibility gene (or genes) for autism on chromosome 2q.

Genetic MarkersLinkage disequilibriumDisequilibriumEthnic groupautism ds2188 pcrDiseaseBiologyPopulation stratificationSettore BIO/13 - Biologia ApplicataPolymorphism (computer science)GeneticsmedicineHumansFamilyAutistic DisorderSicilyBiological PsychiatryGenetics (clinical)GeneticsPolymorphism GeneticChromosome Mappingmedicine.diseaselanguage.human_languagePsychiatry and Mental healthChromosomes Human Pair 2languageAutismmedicine.symptomSicilianPsychiatric Genetics
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Sex-dependent genetic markers of CYP3A4 expression and activity in human liver microsomes

2007

Objective: To find genetic markers of the individual cytochrome P450 (CYP)3A expression. Methods: A large collection of liver samples phenotyped for CYP3A expression and activity was genotyped for CYP3A variants. Data were analyzed for associations between CYP3A phenotypes and genotypes, and for evidence of recent selection. Results: We report associations between the hepatic CYP3A4 protein expression level, as well as its enzymatic activity, measured as verapamil N-dealkylation, and genetic polymorphisms from two regions within the CYP3A gene cluster. One region is defined by several variants, mostly located within CYP3A7, the other by a single nucleotide polymorphism in intron 7 of CYP3A…

Genetic MarkersMaleGene ExpressionSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideLinkage Disequilibrium03 medical and health sciences0302 clinical medicineCytochrome P-450 Enzyme SystemGene FrequencyPolymorphism (computer science)Gene expressionGenotypeGene clusterGeneticsCytochrome P-450 CYP3AHumansAllele frequencyCYP3A7030304 developmental biologyPharmacologyGeneticsSex Characteristics0303 health sciencesMolecular biologyGenetic markerMultigene Family030220 oncology & carcinogenesisLinear ModelsMicrosomes LiverMolecular MedicineFemalePharmacogenomics
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The copy number variant involving part of the α7 nicotinic receptor gene contains a polymorphic inversion.

2008

The alpha7 nicotinic acetylcholine receptor gene (CHRNA7) is located at 15q13-q14 in a region that is strongly linked to the P50 sensory gating deficit, an endophenotype of schizophrenia and bipolar disorder. Part of the gene is a copy number variant, due to a duplication of exons 5-10 and 3' sequence in CHRFAM7A, which is present in many but not all humans. Maps of this region show that the two genes are in opposite orientation in the individual mainly represented in the public access human DNA sequence database (Build 36), suggesting that an inversion had occurred since the duplication. We have used fluorescent in situ hybridization to investigate this putative inversion. Analysis of inte…

Genetic MarkersMaleLinkage disequilibriumBipolar DisorderPan troglodytesalpha7 Nicotinic Acetylcholine ReceptorReceptors NicotinicLinkage DisequilibriumExonGene duplicationGeneticsSettore MED/48 -Scienze Infermierist. e Tecn. Neuro-Psichiatriche e Riabilitat.AnimalsHumansCopy-number variationGeneSettore MED/25 - PsichiatriaGenetics (clinical)Sequence DeletionSegmental duplicationChromosomal inversionGeneticsChromosomes Human Pair 15Polymorphism GeneticBase SequencebiologyCHRNA7Chromosome Mappinginversion schizophrenia bipolar disorder 15q13–q14 CHRNA7 segmental duplicationChromosome InversionSchizophreniabiology.proteinFemale
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Clonal population structure of the chestnut blight fungus in expanding ranges in southeastern Europe.

2008

Expanding populations are often less genetically diverse at their margins than at the centre of a species' range. Established, older populations of the chestnut blight fungus, Cryphonectria parasitica, are more variable for vegetative compatibility (vc) types than in expanding populations in southeastern Europe where C. parasitica has colonized relatively recently. To test whether vc types represent clones, we genotyped 373 isolates of C. parasitica from southern Italy, Romania, Bulgaria, Macedonia, Greece and Turkey using 11 sequence-characterized amplified region (SCAR) markers. Ten SCAR loci and six vegetative incompatibility (vic) loci were polymorphic in these samples. These population…

Genetic MarkersMating typeLinkage disequilibriumPopulationZoologyLinkage DisequilibriumTreesAscomycotaChestnut blightBotanyGenetic variationGeneticsCryphonectriaeducationDNA FungalEcology Evolution Behavior and SystematicsPlant Diseaseseducation.field_of_studybiologyfungiHaplotypeGenetic VariationHippocastanaceaebiology.organism_classificationGenes Mating Type FungalEuropeGenetics PopulationHaplotypesFounder effectMolecular ecology
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Data for nine autosomal STRs markers from Valencia (East Mediterranean coast of the Iberian Peninsula)

2000

Nine STRs loci have been typed in a sample from Valencia, a population from the East Mediterranean coast of the Iberian Peninsula.

Genetic MarkersQuality ControlPopulationPolymerase Chain ReactionLinkage DisequilibriumWhite PeoplePathology and Forensic MedicineGene FrequencyPeninsulaHumansEast mediterraneaneducationValenciaInterneteducation.field_of_studygeographygeography.geographical_feature_categorybiologyMediterranean Regionbiology.organism_classificationDNA FingerprintingArchaeologySpainTandem Repeat SequencesPopulation dataLawSoftwareForensic Science International
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Conduct disorder and ADHD: evaluation of conduct problems as a categorical and quantitative trait in the international multicentre ADHD genetics stud…

2008

Contains fulltext : 71374.pdf (Publisher’s version ) (Closed access) Attention-deficit/hyperactivity disorder (ADHD) is typically characterized by inattention, excessive motor activity, impulsivity, and distractibility. Individuals with ADHD have significant impairment in family and peer relations, academic functioning, and show high co-morbidity with a wide range of psychiatric disorders including oppositional defiant disorder (ODD), conduct disorder (CD), anxiety disorder, depression, substance abuse, and pervasive developmental disorder (PDD). Family studies suggest that ADHD + CD represents a specific subtype of the ADHD disorder with familial risk factors only partly overlapping with t…

Genetics and epigenetic pathways of disease [NCMLS 6]2804 Cellular and Molecular NeuroscienceMedizinPoison controlComorbidityNeuroinformatics [DCN 3]Linkage DisequilibriumCohort Studies2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]Genetics(clinical)ChildGenetics (clinical)Oligonucleotide Array Sequence Analysis0303 health sciencesAntisocial Personality Disorder10058 Department of Child and Adolescent Psychiatry3. Good healthPedigreeEuropePsychiatry and Mental healthConduct disorderAttention Deficit and Disruptive Behavior DisordersADHD -- conduct disorder -- genome wide association study -- genetic association information networkmedicine.symptomPsychologyFunctional Neurogenomics [DCN 2]Anxiety disorderAlgorithmsClinical psychologyConduct DisorderGenetic Markers2716 Genetics (clinical)Quantitative Trait LociContext (language use)610 Medicine & healthChild Behavior DisordersImpulsivityPolymorphism Single NucleotideMental health [NCEBP 9]behavioral disciplines and activitiesGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]mental disordersmedicinePervasive developmental disorderHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und Jugendalters030304 developmental biologyPsychiatric Status Rating ScalesGenome HumanAntisocial personality disordermedicine.diseaseComorbidityGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivity030217 neurology & neurosurgeryGenome-Wide Association Study
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No association between two polymorphisms of the serotonin transporter gene and combined type attention deficit hyperactivity disorder.

2008

Contains fulltext : 71091.pdf (Publisher’s version ) (Closed access) Several independent studies have reported association between serotonin transporter gene (SLC6A4) polymorphisms and attention deficit hyperactivity disorder (ADHD). Five studies found evidence for association between the long-allele of a 44-bp insertion/deletion polymorphism (5-HTTLPR) and ADHD. Another two studies corroborated this finding while a further six studies did not find such an association. For a second polymorphism within the gene, a variable number tandem repeat (VNTR) within intron 2, one study demonstrated that the 12/12 genotype was significantly less frequent in ADHD cases compared to controls, while a sec…

Genetics and epigenetic pathways of disease [NCMLS 6]MedizinNeuroinformatics [DCN 3]0302 clinical medicinePolymorphism (computer science)GenotypePerception and Action [DCN 1]Childhealth care economics and organizationsSerotonin transporterGenetics (clinical)GeneticsSerotonin Plasma Membrane Transport Proteins0303 health sciencesDepression030305 genetics & heredityTransmission disequilibrium testhumanitiesVariable number tandem repeatPsychiatry and Mental healthChild Preschool/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFunctional Neurogenomics [DCN 2]medicine.medical_specialtyAdolescenteducationBiologybehavioral disciplines and activitiesMental health [NCEBP 9]Genomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceSDG 3 - Good Health and Well-beingCognitive neurosciences [UMCN 3.2]Internal medicinemental disordersmedicineAttention deficit hyperactivity disorderHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersAssociation (psychology)Gene030304 developmental biologyFamily HealthPolymorphism Geneticbusiness.industryMood Disordersmedicine.diseaseMoodEndocrinologyMood disordersGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivitybiology.proteinbusiness030217 neurology & neurosurgery
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Low frequency and rare coding variation contributes to multiple sclerosis risk

2018

AbstractMultiple sclerosis is a common, complex neurological disease, where almost 20% of risk heritability can be attributed to common genetic variants, including >230 identified by genome-wide association studies (Patsopoulos et al., 2017). Multiple strands of evidence suggest that the majority of the remaining heritability is also due to the additive effects of individual variants, rather than epistatic interactions between these variants, or mutations exclusive to individual families. Here, we show in 68,379 cases and controls that as much as 5% of this heritability is explained by low-frequency variation in gene coding sequence. We identify four novel genes driving MS risk independe…

Genetics0303 health sciencesLinkage disequilibriumMultiple sclerosisDiseaseBiologyHeritabilitymedicine.disease3. Good health03 medical and health sciences0302 clinical medicinemedicineEpistasisCoding regionGene030217 neurology & neurosurgery030304 developmental biologyGenetic association
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