Search results for "Erythrocyte Membrane"

showing 10 items of 59 documents

Involvement of carboxyl groups in chloride transport and reversible DIDS binding to band 3 protein in human erythrocytes

2011

AbstractNoncovalent DIDS binding to Band 3 (AE1) protein in human erythrocyte membranes, modified by non-penetrating, water soluble 1-ethyl-3-(4-azonia-4,4-dimethylpentyl)-carbodiimide iodide (EAC), was studied at 0°C in the presence of 165 mM KCl. Under experimental conditions applied up to (48 ± 5) % of irreversible chloride self-exchange inhibition was observed. The apparent dissociation constant, KD, for “DIDS-Band 3” complex, determined from the chloride transport experiments, was (34 ± 3) nM and (80 ± 12) nM for control and EAC-treated resealed ghosts, respectively. The inhibition constant, Ki, for DIDS was (35 ± 6) nM and (60 ± 8) nM in control and EAC-treated ghosts, respectively. T…

ErythrocytesIodideDissociation constant44'-Diisothiocyanostilbene-22'-Disulfonic AcidTritiumBiochemistryChloridechemistry.chemical_compoundCarbodiimideChloridesAnion Exchange Protein 1 ErythrocytemedicineHumansBinding siteErythrocyte membraneMolecular BiologyBand 3Ion transporterchemistry.chemical_classificationStilbenedisulfonateBinding SitesIon TransportbiologyCooperative bindingCell BiologyDissociation constantBand 3CrystallographyCarbodiimidesKineticsBiochemistrychemistryDIDSbiology.proteinmedicine.drugResearch ArticleProtein BindingCellular & Molecular Biology Letters
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Exposure to malondialdehyde induces an early redox unbalance preceding membrane toxicity in human erythrocytes.

2002

This work investigated the oxidative injury to human red blood cells (RBCs) by the exposure to exogenous malondialdehyde (MDA), in a physiological environment. When a 10% RBC suspension was incubated in autologous plasma, in the presence of 50 microM MDA, 30% of MDA entered into the cells. A time-course study showed that MDA caused early (30-120 min) and delayed (3-18 h) effects. MDA caused a fast depletion of reduced glutathione, and loss of the glucose-6-phosphate dehydrogenase activity, followed by a decrease of HbO2. Accumulation of methemoglobin, and formation of small amounts of hemichrome were later evident. Also, an HbO2-derived fluorescent product was measured in the membrane. The …

ErythrocytesTime FactorsOxidative phosphorylationmedicine.disease_causeBiochemistryHemolysisMethemoglobinchemistry.chemical_compoundHemoglobinsMalondialdehydemedicineHumansMethemoglobinHemichromeCell MembraneErythrocyte MembraneGeneral MedicineGlutathionemedicine.diseaseMalondialdehydeMolecular biologyHemolysisOxygenSpectrometry FluorescencechemistryBiochemistryGlucose-6-PhosphatasePotassiumElectrophoresis Polyacrylamide GelOxidation-ReductionIntracellularOxidative stressFree radical research
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The autosomal recessive (Becker) form of myotonia congenita

1979

In the last two decades, two genetically distinct forms of myotonia congenita have been identified--an autosomal dominant and an autosomal recessive form. The purpose of this review is to describe the features that enable us to distinguish between these two forms in the absence of sufficient genetic data. Thus far, it can be concluded that the only probable difference between the two forms is in the fatty-acid pattern of muscle phospholipids. Clinical, histologic, ultrastructural, and electromyographic investigation may prove helpful, but they alone cannot provide a reliable means of identifying the genotype in an individual patient.

GeneticsPhysiologyMyotonia congenitaGenetic dataHeterozygote advantageBiologymedicine.diseaseCellular and Molecular NeuroscienceErythrocyte membranePhysiology (medical)GenotypemedicineNeurology (clinical)Autosomal recessive formMuscle & Nerve
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A lytic mechanism based on soluble phospholypases A2 (sPLA2) and b-galactoside specific lectins is exerted by Ciona intestinalis (ascidian) unilocula…

2011

Abstract Hemocytes from the ascidian Ciona intestinalis exert in vitro Ca 2+ -dependent cytotoxic activity toward mammalian erythrocytes and K562 cells. To examine the lytic mechanism, hemocyte populations were separated (B1–B6 bands) through a Percoll discontinuous density gradient, the hemocyte cytotoxic activity (HCA) and the lytic activity of the hemocyte lysate supernatant (HLS) were assayed. In addition the separated hemocytes were cultured and the cell-free culture medium (CFM) assayed after 3 h culture. Results support that unilocular refractile hemocytes (URGs), enriched in B5, are cytotoxic. The B5-HLS contains lysins and the activity of B5-CFM shows that lysins can be released in…

HemocytesPhospholipase A2 Inhibitorsmedicine.medical_treatmentLysinDibucaineSettore BIO/05 - ZoologiaAquatic ScienceBiologyFucoseCell membranechemistry.chemical_compoundmedicineEnvironmental ChemistryAnimalsHumansCiona intestinalisLectins C-TypeEnzyme InhibitorsProteaseErythrocyte MembraneGeneral Medicinebiology.organism_classificationCytotoxicity Tests Immunologicbeta-GalactosidaseGalactosideCiona intestinalisPhospholipases A2medicine.anatomical_structurechemistryBiochemistryLytic cycleInvertebrate immunity Ciona intestinalis Hemocyte Cytotoxicity Soluble phospholipase A2 Rabbit erythrocyte K562QuinacrineCaspasesImmunologyMicroscopy Electron ScanningRabbitsK562 CellsPercoll
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Histidine residues near the N terminus of staphylococcal alpha-toxin as reporters of regions that are critical for oligomerization and pore formation.

1994

Chemical modification of histidine residues in staphylococcal alpha-toxin leads to loss of functional activity. Site-directed mutants of the toxin in which each of the four histidine residues was replaced by several amino acids were therefore produced. The mutant proteins were purified and characterized. Exchange of H-259 or H-144 was sometimes tolerated without reduction in hemolytic activity. These histidine residues are thus not essential for toxin function. Exchange of H-35 and H-48, however, had marked effects. H-35 mutant toxins bound with high affinity to rabbit erythrocytes but displayed faulty oligomerization and were unable to form pores. H-48 mutant toxins also had severely impai…

ImmunologyMutantBacterial ToxinsBiologyHemolysin Proteinsmedicine.disease_causeMicrobiologyHemolysisHemolysin ProteinsStructure-Activity RelationshipmedicineStructure–activity relationshipAnimalsHistidineHistidinechemistry.chemical_classificationToxinErythrocyte Membranebiology.organism_classificationAmino acidN-terminusInfectious DiseaseschemistryBiochemistryMutagenesis Site-DirectedParasitologyRabbitsBacteriaResearch Article
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Complement pore genesis observed in erythrocyte membranes by fluorescence microscopic single-channel recording

1991

The formation and opening of single complement pores could be directly observed in erythrocyte ghosts by confocal laser-scanning microscopy employing the recently introduced method of fluorescence microscopic single-channel recording. Resealed sheep erythrocyte ghosts were incubated with human complement. By limiting the concentration of C8, the eighth component of complement, the fraction of cells rendered permeable for the small polar fluorescent probe Lucifer Yellow was varied between 0.50 and 0.90. Under each condition the flux rate, k, of Lucifer Yellow was determined for a substantial number of ghosts. By analysing the sample population distribution of k the flux rate k1 of ghosts wit…

Lucifer yellowPhotolysisSheepScanning electron microscopeConfocalErythrocyte MembraneAnalytical chemistryComplement System ProteinsCell BiologyModels TheoreticalIsoquinolinesBiochemistryFluorescenceKineticschemistry.chemical_compoundMonomerMembraneMicroscopy FluorescencechemistryMicroscopyFluorescence microscopeAnimalsMolecular BiologyFluorescent DyesResearch ArticleBiochemical Journal
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Increased eryptosis in smokers is associated with the antioxidant status and C-reactive protein levels

2018

Abstract Cigarette smoking has been linked with oxidative stress and inflammation. In turn, eryptosis, the suicidal erythrocyte death similar to apoptosis that can be triggered by oxidative stress, has been associated with chronic inflammatory diseases including atherosclerosis. However, the link between smoking and eryptosis has not been explored so far. The aim of the present study was to determine the level of eryptotic erythrocytes in healthy male smokers (n = 21) compared to non-smokers (n = 21) and assess its relationship with systemic inflammation (CRP) as well as with antioxidant defense (GSH) and their resistance to ex-vivo induced hemolysis. Smoking caused an increase in phosphati…

Male0301 basic medicineAntioxidantHealth Statusmedicine.medical_treatmentEryptosismedicine.disease_causeSystemic inflammationToxicologyAntioxidantsLeukocyte Countchemistry.chemical_compound0302 clinical medicineCigarette smokingSettore BIO/10 - BiochimicaSmokersbiologySmokingMiddle AgedGlutathioneHemolysismedicine.symptomAdultmedicine.medical_specialtyHemolysiInflammationPhosphatidylserinesIn Vitro TechniquesHemolysisC-reactive proteinYoung Adult03 medical and health sciencesInternal medicinemedicineHumansbusiness.industryErythrocyte MembraneC-reactive proteinGlutathionemedicine.diseaseOxidative StressCross-Sectional Studies030104 developmental biologyEndocrinologychemistryApoptosisbiology.proteinOxidative strebusinessEryptosi030217 neurology & neurosurgeryOxidative stress
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Effect of the extract of Ginkgo biloba (EGb 761) on the circulating and cellular profiles of polyunsaturated fatty acids: correlation with the anti-o…

2000

Abstract Ginkgo biloba extract (EGb 761) has beneficial effects on cognitive functions in aging patients, and on various pathologies, including cardiovascular diseases. Although the extract is known to have antioxidant properties and improve membrane fluidity, the cellular mechanisms underlying these effects have not been determined. Here, we examined the in vivo effects of EGb 761 on circulating and cellular lipids. EGb 761 treatment induced significant increases in the levels of circulating polyunsaturated fatty acids (PUFAs), and a decrease in the saturation index SI (saturated/polyunsaturated species). Plasma triglycerides and cholesterol were not affected, while phospholipids were slig…

MaleChromatography GasErythrocytesAntioxidantmedicine.medical_treatmentClinical BiochemistryPharmacologymedicine.disease_causeAntioxidantschemistry.chemical_compoundmedicineMembrane fluidityAnimalsGinkgoalesRats WistarPhospholipidsTriglycerideschemistry.chemical_classificationPlants MedicinalbiologyPlant ExtractsGinkgo bilobaCholesterolCell MembraneErythrocyte MembraneFatty AcidsGinkgo bilobaHydrogen PeroxideCell Biologybiology.organism_classificationEicosapentaenoic acidRatsOxidative StressCholesterolEicosapentaenoic AcidchemistryBiochemistryFatty Acids Unsaturatedlipids (amino acids peptides and proteins)Oxidative stressPolyunsaturated fatty acidProstaglandins, Leukotrienes and Essential Fatty Acids (PLEFA)
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Erythrocyte Phospholipid and Polyunsaturated Fatty Acid Composition in Diabetic Retinopathy

2014

Background: Long chain polyunsaturated fatty acids (LCPUFAs) including docosahexaenoic acid and arachidonic acid are suspected to play a key role in the pathogenesis of diabetes. LCPUFAs are known to be preferentially concentrated in specific phospholipids termed as plasmalogens. This study was aimed to highlight potential changes in the metabolism of phospholipids, and particularly plasmalogens, and LCPUFAs at various stages of diabetic retinopathy in humans. Methodology and Principal Findings: We performed lipidomic analyses on red blood cell membranes from controls and mainly type 2 diabetes mellitus patients with or without retinopathy. The fatty acid composition of erythrocytes was det…

MaleOrganes des senslcsh:MedicineType 2 diabetesBiochemistrySeverity of Illness Indexchemistry.chemical_compoundMELLITUS0302 clinical medicineMedicine and Health SciencesMedicineOXIDATIVE STRESSlcsh:ScienceRETINAPhospholipidschemistry.chemical_classification0303 health sciencesCOMPLICATIONSMultidisciplinaryINSULIN SENSITIVITYFatty AcidsDiabetic retinopathyMiddle AgedLipids3. Good healthDocosahexaenoic acid[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansFatty Acids UnsaturatedPhosphatidylcholinesRetinal DisordersArachidonic acidlipids (amino acids peptides and proteins)FemalePolyunsaturated fatty acidRetinopathyResearch ArticleEXPRESSIONAdultmedicine.medical_specialtySensory OrgansPlasmalogensPhospholipidMédecine humaine et pathologie030209 endocrinology & metabolism03 medical and health sciencesInternal medicineDiabetes mellitus[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyDiabetes MellitusHumans[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory OrgansRetinopathy030304 developmental biologyAgedDiabetic Retinopathybusiness.industryPhosphatidylethanolamineslcsh:RErythrocyte MembraneBiology and Life SciencesDOCOSAHEXAENOIC ACIDmedicine.diseaseLipid MetabolismENDOTHELIAL-CELLSDOCOSAHEXAENOIC ACID;INSULIN SENSITIVITY;ENDOTHELIAL-CELLS;OXIDATIVE STRESS;RETINA;LIPIDS;DEFORMABILITY;COMPLICATIONS;EXPRESSION;MELLITUSOphthalmologyEndocrinologyDiabetes Mellitus Type 1chemistryDiabetes Mellitus Type 2Metabolic DisordersCase-Control Studieslcsh:QHuman health and pathologybusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyDEFORMABILITYPLoS ONE
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Red Cell Filterability and Erythrocyte Membrane Microviscosity during Ticlopidine Treatment

1990

The effects of 10 days' treatment with 500 mg/day ticlopidine on the filterability of red blood cells in suspension and on the microviscosity of the red blood cell membrane was evaluated in 11 patients with vascular atherosclerosis. The results show ticlopidine to be effective in influencing the rheological measures of red cell filterability and membrane microviscosity: filterability was increased and microviscosity was decreased. Ticlopidine also decreased the extent of fluorescence polarization. These results are discussed regarding possible mechanisms of action of ticlopidine.

MaleTiclopidineArteriosclerosis030204 cardiovascular system & hematologyPharmacologyBiochemistryMicroviscosity03 medical and health sciences0302 clinical medicineErythrocyte DeformabilitymedicineHumansErythrocyte deformabilityTiclopidineRed CellViscositybusiness.industryErythrocyte MembraneBiochemistry (medical)Cell BiologyGeneral MedicineArteriosclerosismedicine.diseaseRed blood cellErythrocyte membranemedicine.anatomical_structureMechanism of actionBiochemistry030220 oncology & carcinogenesisFemalemedicine.symptomRheologybusinessmedicine.drugJournal of International Medical Research
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