Search results for "Estrès oxidatiu"

showing 7 items of 17 documents

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome

2010

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome BACKGROUND & AIMS: Oxidative stress has a key role in atherosclerosis, cancer and other chronic diseases. Some bioactive compounds in nuts have been implicated in antioxidant activities. OBJECTIVE: We assessed how nut consumption affected several markers of oxidation and endothelial function (EF) in metabolic syndrome (MetS) patients. PATIENTS AND METHODS: A randomized, controlled, parallel feeding trial was conducted on 50 MetS adults who were recommended a healthy diet supplemented or not with 30 g of mixed nuts (Nut and Control groups, respectively) every day for 12 weeks. The plasma anti…

MaleAntioxidantmedicine.medical_treatmentantioxidant capacityIsoprostanesCritical Care and Intensive Care Medicinemedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundendothelial functionMedicineNutschemistry.chemical_classificationMetabolic SyndromeBioquímica y tecnologíaNutrition and Dieteticsdigestive oral and skin physiologyfood and beveragesArteriesMiddle Aged0261-5614Lipoproteins LDLBiochemistry and technologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineFatty Acids UnsaturatedPolyunsaturated fatty acidAdultmedicine.medical_specialtyEndotheliumAdolescentDNA damageEstrès oxidatiuFruita seca -- Aspectes nutritiusBioquímica i biotecnologiaYoung AdultLipid oxidationInternal medicineHumansAgedbusiness.industryDeoxyguanosineEndoteli vascularmedicine.diseaseDietOxidative StressEndocrinologychemistryDNA damageVascular ResistanceEndothelium VascularMetabolic syndromebusinessOxidative stressBiomarkers
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Different brain oxidative and neuroinflammation status in rats during prolonged abstinence depending on their ethanol relapse-like drinking behavior:…

2022

Rationale: Accumulating evidence suggests that chronic alcohol consumption is associated with excessive oxidative damage and neuroinflammatory processes and these events have been associated to early alcohol withdrawal. In the present research we wonder if brain oxidative stress and neuroinflammation remains altered during prolonged withdrawal situations and whether these alterations can be correlated with relapse behavior in alcohol consumption. The effects of alcohol reintroduction were also evaluated Methods: We have used a model based on the alcohol deprivation effect (ADE) within a cohort of wild-type male Wistar rats. Two subpopulations were identified according to the alcohol relapse…

MalePharmacologyAlcohol Efectes fisiològicsAlcohol DrinkingEthanolEstrès oxidatiuPrefrontal CortexToxicologyRatsSubstance Withdrawal SyndromeAlcoholismOxidative StressPsychiatry and Mental healthAlcohol relapseAlcohol deprivation effectNeuroinflammationRecurrenceOxidative stressNeuroinflammatory DiseasesAnimalsPharmacology (medical)Rats WistarOxidation-ReductionCraving
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ALLOPURINOL BLOCKS AORTIC ANEURYSM IN A MOUSE MODEL OF MARFAN SYNDROME VIA REDUCING AORTIC OXIDATIVE STRESS

2022

ABSTRACTBackgroundIncreasing evidence indicates that redox stress participates in MFS aortopathy, though its mechanistic contribution is little known. We reported elevated reactive oxygen species (ROS) formation and NADPH oxidase NOX4 upregulation in MFS patients and mouse aortae. Here we address the contribution of xanthine oxidoreductase (XOR), which catabolizes purines into uric acid and ROS in MFS aortopathy.Methods and ResultsIn aortic samples from MFS patients, XOR protein expression, revealed by immunohistochemistry, increased in both the tunicae intima and media of the dilated zone. In MFS mice (Fbn1C1041G/+), aortic XOR mRNA transcripts and enzymatic activity of the oxidase form (X…

Marfan syndromemedicine.medical_specialtyEstrès oxidatiuAortic aneurysmsAllopurinolAllopurinolBiochemistryMarfan SyndromeMicechemistry.chemical_compoundAortic aneurysmMetal·loproteïnasesPhysiology (medical)medicine.arteryInternal medicinemedicineAnimalsAortaAortaNADPH oxidasebiologybusiness.industryConnective tissues diseasesNOX4Enzyme inhibitorsHydrogen Peroxidemedicine.diseaseMetalloproteinasesAortic AneurysmÀcid úricDisease Models AnimalOxidative StressEndocrinologyInhibidors enzimàticschemistryXanthine dehydrogenaseOxidative stressbiology.proteinUric acidMalalties del teixit connectiuAneurismes aòrticsReactive Oxygen SpeciesbusinessOxidation-ReductionUric acidmedicine.drug
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Nonsense-mediated mRNA decay controls the changes in yeast ribosomal protein pre-mRNAs levels upon osmotic stress.

2013

The expression of ribosomal protein (RP) genes requires a substantial part of cellular transcription, processing and translation resources. Thus, the RP expression must be tightly regulated in response to conditions that compromise cell survival. In Saccharomyces cerevisiae cells, regulation of the RP gene expression at the transcriptional, mature mRNA stability and translational levels during the response to osmotic stress has been reported. Reprogramming global protein synthesis upon osmotic shock includes the movement of ribosomes from RP transcripts to stress-induced mRNAs. Using tiling arrays, we show that osmotic stress yields a drop in the levels of RP pre-mRNAs in S. cerevisiae cell…

OsmosisTranscription GeneticNonsense-mediated decaylcsh:MedicineYeast and Fungal ModelsMolecular cell biologyGene Expression Regulation FungalGene expressionProtein biosynthesisRNA PrecursorsRNA Processing Post-Transcriptionallcsh:ScienceOligonucleotide Array Sequence AnalysisCellular Stress ResponsesRegulation of gene expressionMultidisciplinarybiologyProtein translationExonsGenomicsCell biologyFunctional GenomicsMitogen-activated protein kinaseResearch ArticleRibosomal ProteinsSaccharomyces cerevisiae ProteinsOsmotic shockEstrès oxidatiuSaccharomyces cerevisiaeGenes FungalDNA transcriptionSaccharomyces cerevisiaeModels BiologicalGenètica molecularSaccharomycesModel OrganismsRibosomal proteinStress PhysiologicalBiologylcsh:RRNA stabilitybiology.organism_classificationMolecular biologyIntronsNonsense Mediated mRNA DecayKineticsRNA processingbiology.proteinlcsh:QGene expressionGenome Expression AnalysisProteïnesPloS one
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Phosphodiesterase inhibition induces retinal degeneration, oxidative stress and inflammation in cone-enriched cultures of porcine retina.

2013

nherited retinal degenerations affecting both rod and cone photoreceptors constitute one of the causes 74 of incurable blindness in the developed world. Cyclic guanosine monophosphate (cGMP) is crucial in the 75 phototransduction and, mutations in genes related to its metabolism are responsible for different retinal 76 dystrophies. cGMP-degrading phosphodiesterase 6 (PDE6) mutations cause around 4e5% of the retinitis 77 pigmentosa, a rare form of retinal degeneration. The aim of this study was to evaluate whether phar- 78 macological PDE6 inhibition induced retinal degeneration in cone-enriched cultures of porcine retina 79 similar to that found in murine models. PDE6 inhibition was induced…

Retinal degenerationgenetic structuresPurinonesPhosphodiesterase InhibitorsSwineEstrès oxidatiuApoptosisBiologyRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundOrgan Culture TechniquesRetinitis pigmentosamedicineIn Situ Nick-End LabelingAnimalsNeurociènciesCyclic GMPRetinaCalpainCaspase 3Retinal DegenerationPhosphodiesteraseRetinalmedicine.diseaseMolecular biologySensory SystemsOphthalmologyOxidative Stressmedicine.anatomical_structurechemistryBiochemistryRetinal Cone Photoreceptor CellsSwine Miniaturesense organsZaprinastRetinal DystrophiesRetinitis PigmentosaVisual phototransduction
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Antioxidant Therapies and Oxidative Stress in Friedreich´s Ataxia: The Right Path or Just a Diversion?

2020

Friedreich's ataxia is the commonest autosomal recessive ataxia among population of European descent. Despite the huge advances performed in the last decades, a cure still remains elusive. One of the most studied hallmarks of the disease is the increased production of oxidative stress markers in patients and models. This feature has been the motivation to develop treatments that aim to counteract such boost of free radicals and to enhance the production of antioxidant defenses. In this work, we present and critically review those 'antioxidant' drugs that went beyond the disease's models and were approved for its application in clinical trials. The evaluation of these trials highlights some …

reactive oxygen speciesFriedreich’s ataxia; clinical trials; oxidative stress; antioxidant therapies; reactive oxygenspecies; scavengers; antioxidant response; mitochondrial metabolism; ferroptosisclinical trialsmitochondrial metabolismantioxidant responseEstrès oxidatiulcsh:RM1-950Friedreich’s ataxiaReviewFriedreich´s ataxia590 Tiere (Zoologie)Antioxidantsferroptosisscavengerslcsh:Therapeutics. Pharmacology570 Biowissenschaften Biologieddc:590oxidative stressantioxidant therapiesddc:570Antioxidants
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Neuronal effects of Sugammadex in combination with Rocuronium or Vecuronium.

2017

Rocuronium (ROC) and Vecuronium (VEC) are the most currently used steroidal non-depolarizing neuromuscular blocking (MNB) agents. Sugammadex (SUG) rapidly reverses steroidal NMB agents after anaesthesia. The present study was conducted in order to evaluate neuronal effects of SUG alone and in combination with both ROC and VEC. Using MTT, CASP-3 activity and Western-blot we determined the toxicity of SUG, ROC or VEC in neurons in primary culture. SUG induces apoptosis/necrosis in neurons in primary culture and increases cytochrome C (CytC), apoptosis-inducing factor (AIF), Smac/Diablo and Caspase 3 (CASP-3) protein expression. Our results also demonstrated that both ROC and VEC prevent these…

vecuroniumNecrosisEstrès oxidatiuPrimary Cell CulturerocuroniumCaspase 3NeuronesPharmacologySugammadexSugammadex03 medical and health sciences0302 clinical medicine030202 anesthesiologymedicineAnimalsHumansAndrostanolsRocuroniumCell damageNeuronsVecuronium BromideDose-Response Relationship DrugCaspase 3business.industryapoptosis.Apoptosis Inducing FactorCytochromes c030208 emergency & critical care medicineGeneral Medicinemedicine.diseaseneuronRatsDrug Combinationsmedicine.anatomical_structureGene Expression RegulationApoptosisToxicityNeuronNeuromuscular Blocking Agentsmedicine.symptombusinessResearch Papergamma-Cyclodextrinsmedicine.drug
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