Search results for "Estradiol"

showing 10 items of 203 documents

Low estradiol-to-testosterone ratio is associated with oligo-anovulatory cycles and atherogenic lipidic pattern in women with polycystic ovary syndro…

2010

The estradiol-to-testosterone (E2/T) ratio has been investigated in different diseases but few in vivo data are available with regard to its role in women with ovary syndrome (PCOS). The aim of this study was to evaluate the role of the E2/T ratio in the ovulatory function and metabolic pattern in such women.We retrospectively evaluated hyperandrogenemia, clinical hyperandrogenism, ovarian morphology, hypothalamo-hypophyseal axis and metabolic syndrome parameters in a cohort of 202 consecutive women affected by PCOS. An oral glucose tolerance test measured areas under the curve for insulin (AUC(2hIRI)), for glucose (AUC(2hglucose)), and the HOMA-IR and Matsuda index of insulin resistance we…

Adultmedicine.medical_specialtyAdolescentEndocrinology Diabetes and Metabolismmedia_common.quotation_subjectOvarySettore MED/13 - EndocrinologiaCohort StudiesYoung AdultEndocrinologyInsulin resistanceRisk FactorsInternal medicineFollicular phasePCOSmedicineHumansTestosteroneSicilyOvulationProgesteroneMenstrual cycleRetrospective Studiesmedia_commonEstradiolbusiness.industryOvaryHyperandrogenismObstetrics and GynecologyAtherosclerosismedicine.diseasePolycystic ovaryCholesterolEndocrinologymedicine.anatomical_structureFollicular PhaseFemaleMetabolic syndromeHyperandrogenismbusinessAlgorithmsAnovulationPolycystic Ovary SyndromeGynecological Endocrinology
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Estrogen Regulates the Satellite Cell Compartment in Females

2019

SUMMARY Skeletal muscle mass, strength, and regenerative capacity decline with age, with many measures showing a greater deterioration in females around the time estrogen levels decrease at menopause. Here, we show that estrogen deficiency severely compromises the maintenance of muscle stem cells (i.e., satellite cells) as well as impairs self-renewal and differentiation into muscle fibers. Mechanistically, by hormone replacement, use of a selective estrogen-receptor modulator (bazedoxifene), and conditional estrogen receptor knockout, we implicate 17β-estradiol and satellite cell expression of estrogen receptor α and show that estrogen signaling through this receptor is necessary to preven…

0301 basic medicineestrogeenitmedicine.medical_specialtyestradioliSatellite Cells Skeletal Musclemedicine.drug_classCellEstrogen receptorlihaksetBiologyGeneral Biochemistry Genetics and Molecular BiologyArticleBazedoxifene03 medical and health sciencesMice0302 clinical medicineInternal medicineestradiolmedicineAnimalsHumansquiescenceskeletal muscleReceptorlcsh:QH301-705.5lihassolutsukupuolihormonitSkeletal muscleEstrogensmedicine.diseaseMenopause030104 developmental biologymedicine.anatomical_structureEndocrinologymuscle stem cellsikääntyminenlcsh:Biology (General)EstrogenFemaleStem cellovarian hormones030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsmedicine.drugCell reports
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Isoflavonoid-based bone-sparing treatments exert a low activity on reproductive organs and on hepatic metabolism of estradiol in ovariectomized rats

2007

International audience; The use of soy isoflavones is a potential alternative to hormone replacement therapy in post-menopausal bone-loss prevention. Nevertheless, phytoestrogens can target other organs and may disrupt cell proliferation, or could modify endogenous steroid hormone metabolism. These mechanisms could be linked to an increased risk of developing cancer. We therefore studied the possible side effects of such treatments in an experimental model of menopause. Forty adult female Wistar rats were ovariectomized and fed with a genistein-, daidzein- or equol-supplemented diet at bone-sparing levels (10 mg/kg BW/day) for 3 months. The estrogenic effects were assessed by histological a…

GenisteinEstrogen receptorToxicologychemistry.chemical_compound0302 clinical medicineCytochrome P-450 Enzyme SystemBone Density[SDV.IDA]Life Sciences [q-bio]/Food engineeringESTROGEN RECEPTORS0303 health sciencesEstradiolfood and beveragesOrgan SizeEquolGenistein3. Good healthCYTOCHROME P450SOY ISOFLAVONEHormone receptor030220 oncology & carcinogenesisVaginaMicrosomes LiverFemaleMenopauseEQUOLmedicine.medical_specialtymedicine.drug_classOvariectomyPhytoestrogensBiology03 medical and health sciencesProliferating Cell Nuclear AntigenInternal medicinemedicineUTEROTROPHYAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRats Wistar030304 developmental biologyPharmacologyUterusDaidzeinIsoflavonesRatsDisease Models AnimalEndocrinologyGene Expression RegulationchemistryEstrogenESTRADIOL METABOLISMOsteoporosisPhytoestrogensSteroid hormone metabolism
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Inducibility of the avidin gene by progesterone is suppressed during estrogen-induced cytodifferentiation.

1992

Abstract We have studied epithelial differentiation of the chick oviduct as induced by diethylstilbestrol (DES) and 17β-estradiol (E 2 ). The proportion of goblet cells in the oviduct was slightly higher after E 2 than after DES treatment. Also avidin induction by progesterone was stronger following DES than E 2 priming. In the estrogen pretreated oviduct epithelium, avidin expression was induced by progesterone in the surface epithelial cells, protodifferentiated gland cells and tubular gland cells, but not in goblet cells. During prolonged estrogen treatment, however, the inducibility of avidin by progesterone ceased in tubular gland cells but not in surface epithelial cells. The estrogen…

medicine.medical_specialtymedicine.drug_classOvalbuminEndocrinology Diabetes and MetabolismClinical BiochemistryDiethylstilbestrolEstrogen receptorOviductsBiologyBiochemistryEpitheliumImmunoenzyme TechniquesEndocrinologystomatognathic systemInternal medicineProgesterone receptormedicineAnimalsTubular glandMolecular BiologyDiethylstilbestrolIn Situ HybridizationProgesteroneEstradiolCell DifferentiationEpithelial CellsCell BiologyAvidinEpitheliummedicine.anatomical_structureEndocrinologyGene Expression RegulationEstrogenbiology.proteinMolecular MedicineOviductChickenshormones hormone substitutes and hormone antagonistsmedicine.drugAvidinThe Journal of steroid biochemistry and molecular biology
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Therapeutic dosages of oral or transdermal estradiol did not modify sCD40L levels in postmenopausal women.

2008

The CD40/CD40L system is considered a crucial modulator of the inflammatory process underlying the progression and complication of atheroma plaques. The soluble fraction of CD40L (sCD40L) is a reliable indicator of the CD40/CD40L system. Our purpose was to investigate whether a therapeutic dose of estradiol, by either the oral or the transdermal route, was associated with changes in circulating levels of sCD40L. Forty-seven women completed a 4-week course of treatment with either oral estradiol valerate (2 mg/day, 20 women) or transdermal estradiol (50 microg/day, 27 women). Serum levels of sCD40L were measured by conventional enzyme-linked immunosorbent assay. Oral, but not transdermal est…

medicine.medical_specialtyDoseEndocrinology Diabetes and MetabolismCD40 LigandAdministration OralEnzyme-Linked Immunosorbent AssayPharmacologyAdministration CutaneousFollicle-stimulating hormoneEndocrinologyTherapeutic indexInternal medicinemedicineHumansCD40 AntigensTriglyceridesTransdermalmedicine.diagnostic_testEstradiolbusiness.industryTransdermal routeEstradiol valerateObstetrics and Gynecologymedicine.diseaseAtherosclerosisMenopausePostmenopauseEndocrinologyCholesterolFemaleFollicle Stimulating HormoneLipid profilebusinessmedicine.drugGynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology
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Dietary xenoestrogens differentially impair 3T3-L1 preadipocyte differentiation and persistently affect leptin synthesis

2008

International audience; Recent observations have highlighted adipogenesis alterations under exposure to several xenoestrogens at critical stages, and pointed at their possible involvement in the pathogenesis of obesity. However, it remains unclear whether these effects are mediated by classical estrogen receptor (ER) binding and subsequent transcriptional modulation. The aim of this study was to determine the (anti-)adipogenic impact of apigenin, bisphenol A, genistein and 17β-estradiol at the onset of adipose cell maturation, and to correlate it to their estrogenic potential. In steroid-free conditions, 3T3-L1 preadipocytes were induced to differentiate in the presence of xenoestrogens for…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryGene ExpressionAdipose tissueEstrogen receptorBiochemistryMicechemistry.chemical_compound0302 clinical medicineEndocrinologyAdipocyte[SDV.IDA]Life Sciences [q-bio]/Food engineeringAdipocytesApigeninESTROGEN RECEPTORS0303 health sciencesEstradiolReverse Transcriptase Polymerase Chain ReactionLeptinCell Differentiation[SDV.IDA] Life Sciences [q-bio]/Food engineeringGenisteinReceptors EstrogenAdipogenesis030220 oncology & carcinogenesisIntercellular Signaling Peptides and ProteinsMolecular Medicinehormones hormone substitutes and hormone antagonistsmedicine.medical_specialty[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringXENOESTROGENSEnzyme-Linked Immunosorbent AssayBiologyModels Biological03 medical and health sciencesLEPTINPhenols3T3-L1 CellsInternal medicinemedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRNA Messengerfas ReceptorBenzhydryl CompoundsMolecular Biology030304 developmental biology3T3-L1Leptin receptorCalcium-Binding ProteinsEstrogens3T3-L1Cell BiologyADIPOGENESISPPAR gammaSteroid hormoneEndocrinologychemistry
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Hormonal and metabolic adaptation to fasting: effects on the hypothalamic-pituitary-ovarianaxis and reproductive performance of rabbit does

2006

To assess the impact of acute caloric shortage on reproduction, rabbit does were either fed ad libitum (control, AL), or fasted for 24 (STF) or 48 h (LTF) before induction of ovulation with GnRH injection. Blood samples were collected during the last 3 h of fasting, and the following 4 h after GnRH injection, when feed was provided again, to measure plasma concentrations of LH, estradiol-17β, leptin, insulin, T3, corticosterone, glucose, and NEFA. Before re-feeding, plasma leptin, insulin, and T3 concentrations were lower (P ≤ 0.01) in both fasted groups than in controls, but then gradually increased following realimentation to match those of controls. During fasting, corticoster…

Blood GlucoseLHHypothalamo-Hypophyseal Systemmedicine.medical_specialtymedia_common.quotation_subjectmedicine.medical_treatmentPituitary-Adrenal SystemHypothalamic–pituitary–gonadal axisGonadotropin-releasing hormoneBiologyGonadotropin-Releasing HormoneRandom Allocationchemistry.chemical_compoundLEPTINEndocrinologyNEFAOvulation InductionFood AnimalsPregnancyRABBITCorticosteroneInternal medicineFASTINGmedicineAnimalsOvulationmedia_commonEstradiolReproductionLeptinInsulinOvaryEstradiol-17Luteinizing HormoneT3INSULINHormonesEndocrinologychemistryTriiodothyronineFemaleAnimal Science and ZoologyRabbitsCorticosteroneLuteinizing hormone
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The serum level of free testosterone is reduced in amyotrophic lateral sclerosis

2002

Sporadic amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder affecting upper and lower motoneurons. There is an approximately 2:1 higher incidence of ALS in men compared to women, and this has raised the hypothesis of an involvement of sex hormones in the etiopathogenesis of the disorder. In this work, the serum levels of dehydroepiandrosterone sulphate (DHEAS), 17-betaestradiol, free and total testosterone were measured in 35 patients with defined or probable ALS, according to the El-Escorial/WFN revisited criteria, and compared to those obtained from 57 disease controls, matched for age and gender to the ALS group. We found no differences between ALS cases and …

Malemedicine.medical_specialtyCentral nervous system diseaseDehydroepiandrosterone sulphateDegenerative diseaseSex hormone-binding globulinInternal medicinemedicineHumans17-βestradiol; Amyotrophic lateral sclerosis; Dehydroepiandrosterone sulphate; Motoneurons; Sex hormone binding globulin; TestosteroneTestosteroneAmyotrophic lateral sclerosisAged17-βestradiolAged 80 and overSex Characteristicsbiologybusiness.industryTestosterone (patch)Middle Agedmedicine.diseaseAmyotrophic lateral sclerosisPathophysiologySex hormone binding globulinMotoneuronsEndocrinologyNeurologybiology.proteinFemaleNeurology (clinical)businessSex characteristicsHormone
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Pregnenolone sulfate, a naturally occurring excitotoxin involved in delayed retinal cell death.

2002

The present study was designed to investigate the neurosteroid pregnenolone sulfate (PS), known for its ability to modulate NMDA receptors and interfere with acute excitotoxicity, in delayed retinal cell death. Three hours after exposure of the isolated and intact retina to a 30-min PS pulse, DNA fragmentation as assessed by genomic DNA gel electrophoresis and a modified in situ terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end-labeling (TUNEL) method appeared concurrently with an increase in superoxide dismutase (SOD) activity and thiobarbituric acid-reactive substances (TBARS) levels. At 7 h, the increased amount of DNA laddering was accompanied by a higher number of TUN…

Malemedicine.medical_specialtyNeurotoxinsExcitotoxicityApoptosisDNA FragmentationDNA ladderingBiologymedicine.disease_causeBiochemistryReceptors N-Methyl-D-AspartateThiobarbituric Acid Reactive SubstancesRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundAdjuvants ImmunologicSuperoxidesInternal medicinemedicineTBARSIn Situ Nick-End LabelingAnimalsCycloheximideRats WistarProgesteroneProtein Synthesis InhibitorsTUNEL assayEstradiolL-Lactate DehydrogenaseDehydroepiandrosterone SulfateSuperoxide DismutaseRatsEndocrinologychemistryApoptosisPregnenolonePregnenoloneDNA fragmentationLipid PeroxidationPregnenolone sulfateReactive Oxygen Speciesmedicine.drugJournal of neurochemistry
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ERa dimerization: a key factor for the weak estrogenic activity of an ERa modulator unable to compete with estradiol in binding assays

2016

PMID: 27400858; International audience; AbstractEstrothiazine (ESTZ) is a weak estrogen sharing structural similarities with coumestrol. ESTZ failed to compete with [3H]17β-estradiol ([3H]17β-E2) for binding to the estrogen receptor α (ERα), questioning its ability to interact with the receptor. However, detection by atomic force spectroscopy (AFS) of an ESTZ-induced ERα dimerization has eliminated any remaining doubts. The effect of the compound on the proliferation of ERα-positive and negative breast cancer cells confirmed the requirement of the receptor. The efficiency of ESTZ in MCF-7 cells was weak without any potency to modify the proliferation profile of estradiol and coumestrol. Gro…

0301 basic medicinemedicine.medical_specialtyTranscription Geneticmedicine.drug_class[SDV]Life Sciences [q-bio]ThiazinesEstrogen receptorBreast NeoplasmsPhytoestrogensCoumestrol[ CHIM ] Chemical SciencesBiochemistry[SPI.MAT]Engineering Sciences [physics]/Materials03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineHumans[CHIM]Chemical SciencesBinding site[SPI.NANO]Engineering Sciences [physics]/Micro and nanotechnologies/MicroelectronicsReceptorMolecular BiologyEstrogen receptor beta[SPI.ACOU]Engineering Sciences [physics]/Acoustics [physics.class-ph]Binding Sites[ SDV ] Life Sciences [q-bio]EstradiolSpectrophotometry AtomicEstrogen Receptor alphaCell BiologyCell biologyTranscription Factor AP-1030104 developmental biologyEndocrinologychemistryMechanism of actionEstrogen030220 oncology & carcinogenesisMCF-7 CellsFemalemedicine.symptomDimerizationEstrogen receptor alphaProtein Binding
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