Search results for "Exposure"

showing 10 items of 1125 documents

Noise-Induced Vascular Dysfunction, Oxidative Stress, and Inflammation Are Improved by Pharmacological Modulation of the NRF2/HO-1 Axis

2021

Vascular oxidative stress, inflammation, and subsequent endothelial dysfunction are consequences of traditional cardiovascular risk factors, all of which contribute to cardiovascular disease. Environmental stressors, such as traffic noise and air pollution, may also facilitate the development and progression of cardiovascular and metabolic diseases. In our previous studies, we investigated the influence of aircraft noise exposure on molecular mechanisms, identifying oxidative stress and inflammation as central players in mediating vascular function. The present study investigates the role of heme oxygenase-1 (HO-1) as an antioxidant response preventing vascular consequences following exposu…

0301 basic medicinePhysiologyClinical BiochemistryInflammationDiseaseRM1-950030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeenvironmental risk factorsBiochemistryArticleendothelial dysfunctionNRF203 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineoxidative stressInducerEndothelial dysfunctionMolecular BiologyDimethyl fumaratebusiness.industryaircraft noise exposureheme oxygenase-1Cell Biologymedicine.diseaseNoise030104 developmental biologychemistryinflammationTherapeutics. Pharmacologymedicine.symptombusinessOxidative stressHeminAntioxidants
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DNA methylation links prenatal smoking exposure to later life health outcomes in offspring

2019

Background Maternal smoking during pregnancy is associated with adverse offspring health outcomes across their life course. We hypothesize that DNA methylation is a potential mediator of this relationship. Methods We examined the association of prenatal maternal smoking with offspring blood DNA methylation in 2821 individuals (age 16 to 48 years) from five prospective birth cohort studies and perform Mendelian randomization and mediation analyses to assess whether methylation markers have causal effects on disease outcomes in the offspring. Results We identify 69 differentially methylated CpGs in 36 genomic regions (P value < 1 × 10−7) associated with exposure to maternal smoking in adolesc…

0301 basic medicinePhysiologyraskausDiseaseBioinformaticsEpigenesis Genetic/dk/atira/pure/core/keywords/icepCohort Studies0302 clinical medicinePregnancyGTP-Binding Protein gamma SubunitsEpidemiologySCHIZOPHRENIADiseaseLongitudinal StudiesProspective StudieskohorttitutkimusGenetics (clinical)Maternal smokingGenetics & HeredityRISK0303 health sciencesDNA methylationSmokingWIDEMethylationASSOCIATIONMiddle AgedDNA-metylaatio3. Good healthCausalityPREGNANCYOncologyMaternal ExposureSchizophreniaPrenatal Exposure Delayed Effects030220 oncology & carcinogenesisDNA methylationkausaliteettilifecourseLife course approachFemaleICEPLife Sciences & BiomedicineAdultTOBACCO-SMOKEMediation (statistics)medicine.medical_specialtyAdolescentOffspringBirth weightPersistenceYoung Adult03 medical and health sciencestupakointiterveysvaikutuksetMendelian randomizationGeneticsmedicineHumansMolecular BiologyMETAANALYSIS030304 developmental biologyPregnancyScience & TechnologyIDENTIFICATIONbusiness.industryMATERNAL CIGARETTE-SMOKINGResearchMediationLife courseMendelian Randomization Analysismedicine.diseaseBIRTH-WEIGHT030104 developmental biologyCpG Islandsbusiness030217 neurology & neurosurgeryGenome-Wide Association StudyDevelopmental Biology
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Release of copper-amended particles from micronized copper-pressure-treated wood during mechanical abrasion

2016

Background We investigated the particles released due to abrasion of wood surfaces pressure-treated with micronized copper azole (MCA) wood preservative and we gathered preliminary data on its in vitro cytotoxicity for lung cells. The data were compared with particles released after abrasion of untreated, water (0% MCA)-pressure-treated, chromated copper (CC)-pressure-treated wood, and varnished wood. Size, morphology, and composition of the released particles were analyzed. Results Our results indicate that the abrasion of MCA-pressure-treated wood does not cause an additional release of nanoparticles from the unreacted copper (Cu) carbonate nanoparticles from of the MCA formulation. Howev…

0301 basic medicinePreservativeCopper particlesAbrasion (mechanical)Cell SurvivalCytotoxicityIn vitro cytotoxicityBiomedical EngineeringNanoparticlechemistry.chemical_elementMedicine (miscellaneous)Pharmaceutical ScienceBioengineering010501 environmental sciences01 natural sciencesApplied Microbiology and BiotechnologyMass SpectrometryCell LineExposure03 medical and health sciencesPressureHumansCytotoxicity0105 earth and related environmental sciencesChemistryResearchtechnology industry and agricultureWaterCytotoxicity; Copper particles; Debris; Exposure; Inhalation; Wood dustMechanical abrasionCopperWood030104 developmental biologyInhalationA549 CellsMolecular MedicineNanoparticlesComposition (visual arts)DebrisReactive Oxygen SpeciesCopperWood dustNuclear chemistry
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Autophagy as a defense strategy against stress: focus on Paracentrotus lividus sea urchin embryos exposed to cadmium

2015

Autophagy is used by organisms as a defense strategy to face environmental stress. This mechanism has been described as one of the most important intracellular pathways responsible for the degradation and recycling of proteins and organelles. It can act as a cell survival mechanism if the cellular damage is not too extensive or as a cell death mechanism if the damage/stress is irreversible; in the latter case, it can operate as an independent pathway or together with the apoptotic one. In this review, we discuss the autophagic process activated in several aquatic organisms exposed to different types of environmental stressors, focusing on the sea urchin embryo, a suitable system recently in…

0301 basic medicineProgrammed cell deathAquatic Organismsfood.ingredientEmbryo NonmammalianStreMini ReviewApoptosis; Autophagy; Cadmium; Defense strategies; Sea urchin embryos; Stress; Biochemistry; Cell BiologyApoptosisBiochemistryParacentrotus lividusToxicology03 medical and health scienceschemistry.chemical_compoundfoodStress PhysiologicalDefense strategieParacentrotusAutophagyAnimalsSettore BIO/06 - Anatomia Comparata E CitologiaSea urchin embryobiologyMechanism (biology)AutophagyApoptosiCell BiologyEnvironmental exposureEnvironmental Exposurebiology.organism_classificationAdaptation PhysiologicalCell biology030104 developmental biologychemistryParacentrotusIntracellularToxicantCadmium
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Gene expression changes after parental exposure to metals in the sea urchin affect timing of genetic programme of embryo development

2021

Simple Summary Intergenerational and transgenerational effects, in which exposure to stressors in a parental generation affects the phenotype of the offspring have been connected to anthropic impacts on biological systems. Therefore, environmental stress experienced inside a generation, particularly during gametogenesis, may lead to erroneous patterns in their offspring just emerging at early developmental stages. In this scenario, the sea urchin embryo represents a suitable model for integrating analyses of gene expression through embryogenesis with developmental alteration induced by environmental stressors. Herein we provide pieces of evidence for the alteration of the gene regulatory ne…

0301 basic medicineSea urchinanimal structuresOffspringIntergenerational effectsSettore BIO/11 - Biologia Molecolare010501 environmental sciencesEmbryo development01 natural sciencesArticleGeneral Biochemistry Genetics and Molecular BiologyAndrology03 medical and health sciencesbiology.animalGene expressionParental exposureEpigeneticslcsh:QH301-705.5GeneSea urchin0105 earth and related environmental sciencesGeneral Immunology and MicrobiologybiologyEmbryogenesisEmbryoPhenotype030104 developmental biologyGene expression profileslcsh:Biology (General)embryonic structuressea urchin; redox homeostasis; parental exposure; intergenerational effects; embryo development; gene expression profilesGeneral Agricultural and Biological SciencesEmbryo development Gene expression profiles Intergenerational effects Parental exposure Redox homeostasis Sea urchinRedox homeostasis
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Genetically predicted longer telomere length is associated with increased risk of B-cell lymphoma subtypes

2016

International audience; Evidence from a small number of studies suggests that longer telomere length measured in peripheral leukocytes is associated with an increased risk of non-Hodgkin lymphoma (NHL). However, these studies may be biased by reverse causation, confounded by unmeasured environmental exposures and might miss time points for which prospective telomere measurement would best reveal a relationship between telomere length and NHL risk. We performed an analysis of genetically inferred telomere length and NHL risk in a study of 10 102 NHL cases of the four most common B-cell histologic types and 9562 controls using a genetic risk score (GRS) comprising nine telomere length-associa…

0301 basic medicineSerumMaleLymphomaanalysisChronic lymphocytic leukemiaFollicular lymphomaGlobal Health[ SDV.CAN ] Life Sciences [q-bio]/Cancerimmunologysurgery0302 clinical medicineEndocrinologyimmune system diseasessingle nucleotide polymorphismGermanyhemic and lymphatic diseasesLondon80 and overOdds RatiogeneticsProspective StudiesB-cell lymphomaAssociation Studies ArticleGenetics (clinical)Aged 80 and overeducation.field_of_studytelomereGenomeLeukemiaAge FactorsGeneral MedicineEnvironmental exposureGenomicsMiddle Agedb-cell lymphomasmall cell lymphomaItaly030220 oncology & carcinogenesisMedicineepidemiologyFemaleFranceRisk of B-cell lymphoma subtypesRiskAdultCanadaChinaLymphoma B-CellGenotypeAdolescentleukocytesetiologyPopulationPopulation[SDV.CAN]Life Sciences [q-bio]/CancerBiologyEnvironmentRisk AssessmentmethodsTime03 medical and health sciencesmedicineHumansFamilyGenetic Predisposition to DiseaseeducationMolecular BiologyAllelesOccupational HealthGenetic Association StudiesAgedB-CellInternational AgenciesOdds ratioEnvironmental Exposuremedicine.diseaseTelomereNon-Hodgkin's lymphoma030104 developmental biologyImmunologyphysiologyChronic DiseasepathologyLaboratoriesmetabolism
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Genetic and Environmental Effects on Telomere Length and Lung Function: A Twin Study.

2015

Background The purpose of the study was to estimate the heritability of leukocyte telomere length (LTL) and lung function and to examine whether LTL and lung function share genetic or environmental effects in common. Methods 386 monozygotic and dizygotic Finnish twin sisters (age 68.4±3.4 years) were included. Relative LTL was determined from peripheral blood DNA by qPCR. Lung function measures of FEV1, FVC, FEV1/FVC, and PEF were derived from spirometry. Genetic modeling was performed with MPlus statistical software. Results Univariate analysis revealed that in LTL, 62% (95% confidence interval 50-72) of the variance was explained by additive genetic and 38% (28-50) by unique environmental…

0301 basic medicineSpirometryAgingBivariate analysista3111Genetic correlation03 medical and health sciencesFEV1/FVC ratio0302 clinical medicineForced Expiratory VolumeLeukocytesTwins DizygoticMedicineHumansLungAgedmedicine.diagnostic_testbusiness.industryEnvironmental exposureta3142Environmental ExposureTwins Monozygoticrespiratory systemHeritabilityMiddle AgedTelomeretelomeresTwin studyConfidence intervalrespiratory tract diseases030104 developmental biology030228 respiratory systemSpirometrygenetic modelingFemaleGeriatrics and GerontologybusinessDemographyThe journals of gerontology. Series A, Biological sciences and medical sciences
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Circadian Rhythm Variations and Nutrition in Children

2018

AbstractCircadian rhythms are the changes in biological processes that occur on a daily basis. Among these processes are reactions involved in metabolic homeostasis. Circadian rhythms are structured by the central clock in the suprachiasmatic nucleus of the hypothalamus via the control of melatonin expression. Circadian rhythms are also controlled by the peripheral clocks, which are intracellular mechanisms composed of the clock genes, whose expression follows a circadian pattern. Circadian rhythms are impacted by signals from the environment called zeitgebers, or time givers, which include light exposure, feeding schedule and composition, sleeping schedule and pattern, temperature, and phy…

0301 basic medicineSuprachiasmatic nucleusMetabolic homeostasisBiologyMelatoninCLOCK03 medical and health sciences030104 developmental biology0302 clinical medicineHypothalamusPediatrics Perinatology and Child HealthZeitgebermedicineSurgeryCircadian rhythmNeuroscience030217 neurology & neurosurgerymedicine.drugLight exposureJournal of Child Science
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High Nutrient Concentration Can Induce Virulence Factor Expression and Cause Higher Virulence in an Environmentally Transmitted Pathogen

2016

Environmentally transmitted opportunistic pathogens shuttle between two substantially different environments: outside-host and within-host habitats. These environments differ from each other especially with respect to nutrient availability. Consequently, the pathogens are required to regulate their behavior in response to environmental cues in order to survive, but how nutrients control the virulence in opportunistic pathogens is still poorly understood. In this study, we examined how nutrient level in the outside-host environment affects the gene expression of putative virulence factors of the opportunistic fish pathogen Flavobacterium columnare. The impact of environmental nutrient concen…

0301 basic medicineVirulence Factors030106 microbiologyvirulence factorsSoil ScienceVirulenceBiologyReal-Time Polymerase Chain ReactionFlavobacteriumVirulence factorflavobacterium columnareMicrobiologyFish Diseases03 medical and health sciencesMicrobial ecologynutrientscolony typeAnimalsCollagenasesPathogenGeneEcology Evolution Behavior and SystematicsChondroitin LyasesEcologyHost (biology)RT-qPCREnvironmental Exposurechondroitinasebiology.organism_classificationcollagenase030104 developmental biologyFoodOncorhynchus mykissFlavobacterium columnareWater MicrobiologyBacteriaMicrobial Ecology
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VDBP, CYP27B1, and 25-Hydroxyvitamin D Gene Polymorphism Analyses in a Group of Sicilian Multiple Sclerosis Patients

2016

Multiple sclerosis (MS) is a chronic demyelinating disease of central nervous system regarded as one of the most common causes of neurological disability in young adults. The exact etiology of MS is not yet known, although epidemiological data indicate that both genetic susceptibility and environmental exposure are involved. A poor vitamin D status has been proposed as the most attractive environmental factor. Several evidence have highlighted the importance of mutations in vitamin D-regulating genes for vitamin D status. The purpose of our study was to assess the genetic variants of VDBP and CYP27B1 in MS patients and in a control group. A total of 192 subjects, including 100 MS patients a…

0301 basic medicineVitaminAdultMalemedicine.medical_specialtyMultiple SclerosisVitamin D-binding proteinBiologyBiochemistryVDBP polymorphism03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCYP27B1Internal medicineGenotypeMultiple SclerosimedicineVitamin D and neurologyGenetic predispositionGeneticsHumansVitamin DSicilyMolecular BiologyEcology Evolution Behavior and SystematicsGenetics25-Hydroxyvitamin D3 1-alpha-HydroxylasePolymorphism Genetic25(OH)DMultiple sclerosisVitamin D-Binding ProteinGeneral MedicineEnvironmental exposureMiddle Agedmedicine.diseaseEcology Evolution Behavior and Systematic030104 developmental biologyEndocrinologychemistryFemaleGene polymorphism030217 neurology & neurosurgeryHuman
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