Search results for "FAT"

showing 10 items of 4644 documents

Studies on the mechanism of PMN activation II. by triggering the alternative pathway of complement activation

1982

By means of cobra venom factor (CVF) it is demonstrated that the stimulation of hexosemonophosphate shunt (HMPS) of human polymorphonuclear leukocytes (PMN) by zymosan (Z) and dextran sulfate (DS) is caused by at least two modes of activation: (a) via activation caused by phagocytosis, (b) via activated alternative pathway of complement activation (APC). Active factors of APC presented with phagocytizable objects strongly enhance activation of PMN. The effect of APC can be observed in serum-containing as well as in serum-free cultures. It can be demonstrated that in serum-free cultures the factors of APC participating in the activation of PMN are supplied by monocytes. By the use of synthet…

medicine.medical_specialtyNeutrophilsPhagocytosisComplement Pathway AlternativeDose-Response Relationship ImmunologicStimulationMonocyteschemistry.chemical_compoundPhagocytosisInternal medicinemedicineHumansComplement ActivationHematologyChemistryDextran SulfateZymosanZymosanDextransComplement C3HematologyGeneral MedicinePeptide FragmentsCell biologyComplement systemDextran sulfateBiochemistryComplement C3aAlternative complement pathwayCobra venom factorBlut
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Placebo and Nocebo Effects Across Symptoms: From Pain to Fatigue, Dyspnea, Nausea, and Itch

2019

Contains fulltext : 208540.pdf (Publisher’s version ) (Open Access) Placebo and nocebo effects are, respectively, the helpful and harmful treatment effects that do not arise from active treatment components. These effects have thus far been researched most often in pain. It is not yet clear to what extent these findings from pain can be generalized to other somatic symptoms. This review investigates placebo and nocebo effects in four other highly prevalent symptoms: dyspnea, fatigue, nausea, and itch. The role of learning mechanisms (verbal suggestions, conditioning) in placebo and nocebo effects on various outcomes (self-reported, behavioral, and physiological) of these different somatic s…

medicine.medical_specialtyNoceboNausealcsh:RC435-571Reviewplacebo and nocebo effectsPlacebo03 medical and health sciences0302 clinical medicinesuggestionconditioninglcsh:PsychiatryHeart ratemedicinepainitchLung functionPsychiatrybusiness.industrydyspneanausea030227 psychiatry3. Good healthNocebo EffectPsychiatry and Mental healthPhysical therapyInflammatory diseases Radboud Institute for Health Sciences [Radboudumc 5]fatigueActive treatmentmedicine.symptombusiness030217 neurology & neurosurgery
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n-3 Fatty Acids Modulate T-Cell Calcium Signaling in Obese Macrosomic Rats

2004

Objective: We investigated the effects of a diet containing EPAX-7010, rich in PUFAs such as eicosapentaenoic acid [20:5(n-3)] and docosahexaenoic acid [22:6(n-3)], i.e., a PUFA/EPAX regimen, on T-cell activation in diabetic pregnant rats and their obese pups. Research Methods and Procedures: Mild hyperglycemia in pregnant rats was induced by intraperitoneal injection of streptozotocin on Day 5 of gestation. T-cell blastogenesis was assayed by using 3H-thymidine, whereas intracellular free calcium concentrations ([Ca2+]i) were measured by using Fura-2 in diabetic pregnant rats and their obese offspring. Results: Concavalin-A-stimulated T-cell proliferation was decreased in both pregnant dia…

medicine.medical_specialtyOffspringT-LymphocytesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentIntraperitoneal injectionPregnancy in DiabeticsMedicine (miscellaneous)Gestational AgeLymphocyte ActivationDiabetes Mellitus ExperimentalFetal MacrosomiaEndocrinologyDietary Fats UnsaturatedPregnancyDiabetes mellitusInternal medicineFatty Acids Omega-3Concanavalin AmedicineAnimalsObesityRats Wistarchemistry.chemical_classificationCalcium metabolismbusiness.industryIonomycinPublic Health Environmental and Occupational Healthmedicine.diseaseStreptozotocinEicosapentaenoic acidRatsEndocrinologychemistryDocosahexaenoic acidThapsigarginCalciumFemalebusinessSpleenSignal TransductionFood SciencePolyunsaturated fatty acidmedicine.drugObesity Research
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Perinatal high-fat diet increases hippocampal vulnerability to the adverse effects of subsequent high-fat feeding

2015

Epidemiological observations report an increase in fat consumption associated with low intake of n-3 relative to n-6 polyunsaturated fatty acids (PUFAs) in women of childbearing age. However, the impact of these maternal feeding habits on cognitive function in the offspring is unknown. This study aims to investigate the impact of early exposure to a high-fat diet (HFD) with an unbalanced n-6/n-3 PUFAs ratio on hippocampal function in adult rats. Furthermore, we explored the effects of perinatal HFD combined with exposure to HFD after weaning. Dams were fed a control diet (C, 12% of energy from lipids, n-6/n-3 PUFAs ratio: 5) or HFD (HF, 39% of energy from lipids, n-6/n-3 PUFAs ratio: 39) th…

medicine.medical_specialtyOffspringhippocampusEndocrinology Diabetes and MetabolismWeaningHippocampal formationBiologyDiet High-FatmemoryEndocrinologyPregnancyFatty Acids Omega-6Internal medicineLactation[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyFatty Acids Omega-3medicineAnimalsLactationWeaningwater mazeTaqMan low-density arrayRats WistarMaze LearningOmega 3 fatty acidBiological PsychiatrySpatial Memory2. Zero hungerchemistry.chemical_classificationPerinatal ExposureEndocrine and Autonomic Systemsomega-6 fatty acidomega-3 fatty acidfood and beveragesRatsPsychiatry and Mental healthEndocrinologymedicine.anatomical_structureAnimals NewbornchemistryPrenatal Exposure Delayed Effects[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Omega-6 fatty acidFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]lipids (amino acids peptides and proteins)[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPolyunsaturated fatty acid
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Towards a Rapid Screening of Liver Grafts at the Operating Room Using Mid-Infrared Spectroscopy

2020

The estimation of steatosis in a liver graft is mandatory prior to liver transplantation, as the risk of graft failure increases with the level of infiltrated fat. However, the assessment of liver steatosis before transplantation is typically based on a qualitative or semiquantitative characterization by visual inspection and palpation and histological analysis. Thus, there is an unmet need for transplantation surgeons to have access to a diagnostic tool enabling an in situ fast classification of grafts prior to extraction. In this study, we have assessed an attenuated total reflection-Fourier transform infrared (ATR-FTIR) spectroscopic method compatible with the requirements of an operatio…

medicine.medical_specialtyOperating RoomsGraft failureTime FactorsSpectrophotometry Infraredmedicine.medical_treatmentgraft failureassessmentLiver transplantation010402 general chemistry01 natural sciencesPalpationMid infrared spectroscopyAnalytical Chemistryliver steatosisLiver steatosisstomatognathic systemliver graftmedicinesteatosisHumansmedicine.diagnostic_testliver transplantationChemistryPhysics010401 analytical chemistrymedicine.disease0104 chemical sciences3. Good healthLiver TransplantationLiver graftTransplantationAtomic Molecular and Optical Physicslipids (amino acids peptides and proteins)RadiologySteatosisinfiltrated fat
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IRS-2 deficiency impairs NMDA receptor-dependent long-term potentiation

2011

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License.-- et al.

medicine.medical_specialtyPatch-Clamp TechniquesCognitive Neurosciencemedicine.medical_treatmentBlotting WesterneducationHippocampusComputingMilieux_LEGALASPECTSOFCOMPUTINGNeurotransmissionBiologyHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionMiceCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsImmunoprecipitationlong-term potentiationMice Knockoutsynaptic plasticitydiabetesInsulinDiabetesLong-term potentiationArticlesNMDA receptorIRS2insulin receptor signalingSynaptic fatigueEndocrinologynervous systemSynaptic plasticityInsulin Receptor Substrate ProteinsNMDA receptorFemaleNeuroscienceCerebral Cortex
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Enfermedad del hígado graso no alcohólico, asociación con la enfermedad cardiovascular y tratamiento ( I ). Enfermedad del hígado graso no alcohólico…

2017

Non-alcoholic fatty liver disease (NAFLD) comprises a series of histologically lesions similar to those induced by alcohol consumption in people with very little or no liver damage. The importance of NAFLD is its high prevalence in the Western world and, from the point of view of the liver, in its gradual progression from steatosis to steatohepatitis, cirrhosis, and liver cancer. During the last decade it has been observed that NAFLD leads to an increased cardiovascular risk with acceleration of arteriosclerosis and events related to it, being the main cause of its morbidity and mortality. This review, updated to January 2016, consists of two parts, with the first part analysing the associa…

medicine.medical_specialtyPathologyCirrhosisHigh prevalencebusiness.industryFatty livernutritional and metabolic diseasesArteriosclerosis030204 cardiovascular system & hematologymedicine.diseaseGastroenterologydigestive system diseases03 medical and health sciences0302 clinical medicineInternal medicinemedicine030211 gastroenterology & hepatologyPharmacology (medical)Liver damageSteatosisSteatohepatitisCardiology and Cardiovascular MedicineLiver cancerbusinessClínica e Investigación en Arteriosclerosis
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Enfermedad del hígado graso no alcohólico, asociación con la enfermedad cardiovascular y tratamiento (II). Tratamiento de la enfermedad del hígado gr…

2017

Disease nonalcoholic fatty liver disease (NAFLD) comprises a series of histologically similar to those induced by alcohol consumption in people with very little or no liver damage same. The importance of NAFLD is its high prevalence in our Western societies, from the point of view liver in its progressive evolution from steatosis to steatohepatitis, cirrhosis and liver cancer. During the last decade it has been observed that NAFLD leads to an increased cardiovascular risk with accelerated atherosclerosis and cardiovascular events, the leading cause of morbidity and mortality. This updated January 2016 revision consists of two parts. In this second part, the treatment of NAFLD and its influe…

medicine.medical_specialtyPathologyCirrhosisbusiness.industryDisease030204 cardiovascular system & hematologymedicine.diseaseGastroenterologydigestive system diseases03 medical and health sciencesLiver disease0302 clinical medicineInternal medicineNonalcoholic fatty liver diseasemedicine030211 gastroenterology & hepatologyPharmacology (medical)Liver damageSteatosisSteatohepatitisCardiology and Cardiovascular MedicineLiver cancerbusinessClínica e Investigación en Arteriosclerosis
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In vivo imaging of colitis and colon cancer development in mice using high resolution chromoendoscopy

2005

Background: Mouse models of colitis and cancer are indispensable for our understanding of the pathogenesis of these diseases. In the past, mice had to be sacrificed in order to analyse colitis activity and tumour development. We have developed a safe method for high resolution endoscopic monitoring of living mice. Methods: Mice developing colitis or colonic tumours were anaesthetised using avertine and repeatedly examined by endoscopy. A novel miniendoscope (1.9 mm outer diameter), denoted Coloview, was introduced via the anus and the colon was carefully insufflated with an air pump before analysis of the colonic mucosa. An extra working channel allowed the introduction of biopsy forceps or…

medicine.medical_specialtyPathologyColorectal cancerAzoxymethaneColonoscopyMice Inbred StrainsSeverity of Illness IndexGastroenterologyChromoendoscopyMiceIntestinal mucosaInternal medicineBiopsymedicineAnimalsIntestinal MucosaColitisGrading (tumors)Colonoscopesmedicine.diagnostic_testbusiness.industryDextran SulfateInflammatory Bowel DiseaseGastroenterologyCancerColonoscopyColitismedicine.diseaseDisease Models AnimalCell Transformation NeoplasticColonic NeoplasmsDisease ProgressionbusinessGut
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Genetic Deletion of JNK1 and JNK2 Aggravates the DSS-Induced Colitis in Mice

2007

The c-Jun N-terminal kinases (JNKs) are considered as novel targets for therapy of inflammatory bowel diseases (IBD). However, the relevant JNK isoforms have to be elucidated. Here, we analyze the individual contribution of the JNK1 and JNK2 isoforms in a dextran sulfate sodium (DSS) model of experimental colitis. JNK1 and JNK2 knockout mice (JNK1 ko, JNK2 ko) and their wild-type controls (WT1, WT2) received three cycles of DSS treatment, each consisting of 1.7% DSS for 5 days, followed by 5 days with water. Animals were daily evaluated by a disease activity index (DAI) comprising measurement of body weight, estimation of stool consistency, and test for occult blood/gross rectal bleeding. A…

medicine.medical_specialtyPathologyCryptApoptosisMice TransgenicInflammatory bowel diseaseGastroenterologyProinflammatory cytokineMiceCecumImmune systemInternal medicineWeight LossAnimalsMitogen-Activated Protein Kinase 9MedicineMitogen-Activated Protein Kinase 8Single-Blind MethodIntestinal MucosaColitisCrosses Geneticbusiness.industryDextran SulfateColitismedicine.diseaseMice Inbred C57BLmedicine.anatomical_structureApoptosisChronic DiseaseKnockout mouseSurgeryGastrointestinal HemorrhagebusinessJournal of Investigative Surgery
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