Search results for "FOXP3"

showing 10 items of 140 documents

Crosstalk of regulatory T cells and tolerogenic dendritic cells prevents contact allergy in subjects with low zone tolerance

2012

Background Allergic contact dermatitis is one of the most common occupational diseases. A main protective mechanism in those who do not develop allergic contact dermatitis is tolerance induction by repeated exposure to low doses of contact allergen, which is termed low zone tolerance (LZT). The mechanisms that determine the tolerance induction in subjects with LZT are still elusive. Objective We performed analysis of the role of CD4 + CD25 + forkhead box protein 3 (FOXP3)–positive regulatory T (Treg) cells and dendritic cells (DCs) in mice with LZT. Methods Mechanisms of tolerance induction were analyzed in a murine model of LZT by using FOXP3 and IL-10 reporter mice, as well as mice that a…

Receptors CCR7Adoptive cell transferImmunologyMice Transgenicchemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationT-Lymphocytes RegulatoryMiceImmune ToleranceAnimalsImmunology and AllergyIL-2 receptorInterleukin-2 Receptor alpha SubunitFOXP3Forkhead Transcription Factorshemic and immune systemsDendritic CellsDendritic cellCD11c AntigenInterleukin-10Tolerance inductionInterleukin 10CTLA-4Dermatitis Allergic ContactImmunologyCD8Journal of Allergy and Clinical Immunology
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Regulatory T cells selectively preserve immune privilege of self-antigens during viral central nervous system infection.

2012

Abstract Regulatory T cells (Tregs) are important for the attenuation of immune reactions. During viral CNS infections, however, an indiscriminate maintenance of CNS immune privilege through Treg-mediated negative regulation could prevent autoimmune sequelae but impair the control of viral replication. We analyzed in this study the impact of Tregs on the development of acute viral encephalomyelitis, T cell-mediated antiviral protection, and prevention of CNS autoimmunity following intranasal infection with the gliatropic mouse hepatitis virus strain A59. To assess the contribution of Tregs in vivo, we specifically depleted CD4+Foxp3+ T cells in a diphtheria toxin-dependent manner. We found …

Receptors CXCR3T cellImmunologychemical and pharmacologic phenomenaAutoimmunityBiologyCD8-Positive T-Lymphocytesmedicine.disease_causeCXCR3Lymphocyte ActivationAutoantigensT-Lymphocytes RegulatoryLymphocyte DepletionAutoimmunity03 medical and health sciencesMice0302 clinical medicineCentral Nervous System InfectionsImmune privilegeImmunitymedicineImmunology and AllergyAnimalsHumansEncephalomyelitisAdministration Intranasal030304 developmental biologyCell Proliferation0303 health sciencesImmunity CellularMice Inbred BALB CMurine hepatitis virusFOXP3hemic and immune systemsForkhead Transcription Factors3. Good healthmedicine.anatomical_structureViral replicationImmunologyAcute DiseaseCD4 AntigensLymph NodesCoronavirus InfectionsCD8030215 immunologyJournal of immunology (Baltimore, Md. : 1950)
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Cutting Edge: Trans-Signaling via the Soluble IL-6R Abrogates the Induction of FoxP3 in Naive CD4+CD25− T Cells

2007

Abstract Chronic inflammatory diseases may develop when regulatory T cells (Tregs) fail to control the balance between tolerance and immunity. Alternatively, activated immune cells might prevent the induction or activation of Tregs in such diseases. In this study, we demonstrate that trans-signaling into T cells via the soluble IL-6 receptor completely abrogates the de novo induction of adaptive Tregs. Mechanistically, IL-6 trans-signaling augmented the expression of the TGF-β signaling inhibitor SMAD7. Consequently, SMAD7 overexpression in T cells using newly created transgenic mice rendered CD4+CD25− T cells resistant to the induction of FoxP3. Finally, IL-6 trans-signaling inhibited Treg…

Regulatory T cellImmunologyMice Transgenicchemical and pharmacologic phenomenaMice SCIDBiologyT-Lymphocytes RegulatoryAutoimmune DiseasesSmad7 ProteinMiceInterleukin 21Immune systemTransforming Growth Factor betaImmunitymedicineAnimalsHumansImmunology and AllergyCytotoxic T cellIL-2 receptorInflammationMice Inbred BALB CInterleukin-6ZAP70FOXP3Forkhead Transcription FactorsColitisReceptors Interleukin-6Cell biologyDisease Models Animalmedicine.anatomical_structureGene Expression RegulationChronic DiseaseImmunologySignal TransductionThe Journal of Immunology
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Large scale preparation of human MHC class II+ integrin beta(1)+ Tregs.

2010

Abstract The human CD4 + CD25 + FoxP3 + regulatory T cell population (Tregs) contains both MHC class II + and MHC class II − cells. MHC class II + Tregs belong to the integrin α 4 β 1 + subpopulation and exclusively execute contact-dependent suppressive activity. Here we present a method optimized for isolation of these MHC class II expressing Tregs from large leukaphereses products using magnetic microbeads that achieves a reproducible purity of more than 90% and enables the use of this small-sized Treg population in pre-clinical application and basic research.

Regulatory T cellImmunologyPopulationIntegrinchemical and pharmacologic phenomenaIntegrin alpha4beta1T-Lymphocytes RegulatoryT-Lymphocyte SubsetsmedicineImmune ToleranceImmunology and AllergyHumansIL-2 receptorLeukapheresiseducationCells CulturedMHC class IIeducation.field_of_studybiologyImmunomagnetic SeparationHistocompatibility Antigens Class IIInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsForkhead Transcription FactorsT lymphocyteMHC restrictionFlow CytometryCell biologyHigh-Throughput Screening Assaysmedicine.anatomical_structureImmunologyCD4 Antigensbiology.proteinJournal of immunological methods
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DRB1*0401-restricted human T cell clone specific for the major proinsulin73-90 epitope expresses a down-regulatory T helper 2 phenotype.

2006

Recently, we have identified proinsulin (P-Ins) 73-90 as an immunodominant T cell epitope of HLA-DRB1*0401 (DR4) subjects with β-islet cell autoimmunity and of HLA-DR4/CD4 double-transgenic mice immunized with human P-Ins. We have compared the fine specificities of one human CD4 T cell clone and two mouse T cell hybridoma clones recognizing this epitope, and, although these three clones all recognized the same core region (LALEGSLQK), there were major differences in how they interacted with the peptide (p)/HLA complex, reflecting the fact that human P-Ins is a foreign antigen in the mouse and an autoantigen in the type 1 diabetes patient. The human T cell clone was forkhead transcription f…

Regulatory T cellT cellT-LymphocytesMolecular Sequence DataClone (cell biology)Mice TransgenicHuman leukocyte antigenBiologyEpitopeEpitopesMiceAntigenT-Lymphocyte SubsetsmedicineCytotoxic T cellAnimalsHumansAmino Acid SequenceAmino AcidsMultidisciplinaryFOXP3Forkhead Transcription FactorsHLA-DR AntigensBiological SciencesMolecular biologyPeptide Fragmentsmedicine.anatomical_structurePhenotypeHLA-DRB1 ChainsProinsulinProceedings of the National Academy of Sciences of the United States of America
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Increased regulatory T-cell frequencies in patients with advanced melanoma correlate with a generally impaired T-cell responsiveness and are restored…

2009

Naturally occurring CD4(+) CD25(+) regulatory T-cell (Treg) activity is assumed to facilitate tumor development and progression. To elucidate the possible role of Tregs in the course of melanoma progression, we analysed the frequency of Tregs in the peripheral blood of patients at melanoma stages I-IV and in patients at melanoma stage IV that underwent dendritic cell (DC)-based immunotherapy. Using CD25, Foxp3, CD127 and HLA-DR as Treg associated markers, we observed increased Treg frequencies in patients at the late melanoma stage (stage IV) when compared to healthy donors. Accumulation of Tregs in patients with progressed melanoma correlated with a general reduction of T-cell responsivene…

Regulatory T cellbusiness.industryT cellmedicine.medical_treatmentMelanomaFOXP3hemic and immune systemschemical and pharmacologic phenomenaDermatologyImmunotherapyDendritic cellmedicine.diseaseBiochemistrymedicine.anatomical_structureTumor progressionImmunologymedicineIL-2 receptorbusinessMolecular BiologyExperimental Dermatology
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Different profile and distribution of antigen specific T cells induced by intranasal and intrarectal immunization with rotavirus 2/6-VLP with and wit…

2013

International audience; In this study, we compared both the profile and distribution of antigen specific primed T cells after intrarectal (IR) and intranasal (IN) immunization with rotavirus (RV) 2/6-VLP, alone or in the presence of LT-R192G, in order to highlight the differences between the two routes and the impact of the adjuvant. Adult BALB/c mice were immunized once with 2/6-VLP with or without adjuvant and the T cell response was analyzed in lymphoid tissues after in vitro restimulation with the antigen. IN, but not IR, immunization of mice with 2/6-VLP alone induced antigen-specific IL-10 and IL-17 secreting T cells. IL-10-, in contrast to IL-17-, secreting T cells did not migrate to…

Rotavirusmedicine.medical_treatmentT-Lymphocytes[SDV]Life Sciences [q-bio]Priming (immunology)DistributionPHENOTYPEPROTECTSEnterotoxins0302 clinical medicineCell MovementINFECTIONMesenteric lymph nodesHEAT-LABILE TOXINIMMUNE-RESPONSEIL-2 receptorAntigens Viral0303 health sciencesB-LymphocytesMice Inbred BALB CIntrarectalEscherichia coli ProteinsVaccinationFOXP3CHOLERA-TOXINLT-R192G3. Good healthInfectious Diseasesmedicine.anatomical_structureIntranasal030220 oncology & carcinogenesisMolecular MedicineFemaleAdjuvantLymphoid TissueT cellBacterial ToxinsSpleenBiologyMUCOSAL VACCINESRotavirus Infections03 medical and health sciencesCross-PrimingAntigenAdjuvants ImmunologicAdministration RectalVIRUS-LIKE PARTICLESmedicineAnimalsVaccines Virus-Like ParticleImmunity MucosalAdministration Intranasal030304 developmental biologyGeneral VeterinaryGeneral Immunology and MicrobiologyInterleukinsPublic Health Environmental and Occupational HealthRotavirus VaccinesT cellMICEImmunologyCHALLENGE
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Protein kinase CK2 governs the molecular decision between encephalitogenic T H 17 cell and T reg cell development

2016

T helper 17 (TH17) cells represent a discrete TH cell subset instrumental in the immune response to extracellular bacteria and fungi. However, TH17 cells are considered to be detrimentally involved in autoimmune diseases like multiple sclerosis (MS). In contrast to TH17 cells, regulatory T (Treg) cells were shown to be pivotal in the maintenance of peripheral tolerance. Thus, the balance between Treg cells and TH17 cells determines the severity of a TH17 cell-driven disease and therefore is a promising target for treating autoimmune diseases. However, the molecular mechanisms controlling this balance are still unclear. Here, we report that pharmacological inhibition as well as genetic ablat…

STAT3 Transcription Factor0301 basic medicineEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisCellMice Transgenicchemical and pharmacologic phenomenaBiologySeverity of Illness IndexT-Lymphocytes RegulatoryMice03 medical and health sciences0302 clinical medicineImmune systemmedicineAnimalsHumansIL-2 receptorPhosphorylationCasein Kinase IISTAT3MultidisciplinaryCell growthInterleukin-17Experimental autoimmune encephalomyelitisGranulocyte-Macrophage Colony-Stimulating FactorFOXP3Peripheral toleranceForkhead Transcription Factorshemic and immune systemsReceptors Interleukinmedicine.diseasePeptide FragmentsMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureGene Expression RegulationImmunologybiology.proteinCancer researchTh17 CellsMyelin-Oligodendrocyte GlycoproteinSignal Transduction030215 immunologyProceedings of the National Academy of Sciences
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Induction of tolerogenic lung CD4+ T cells by local treatment with a pSTAT-3 and pSTAT-5 inhibitor ameliorated experimental allergic asthma.

2010

Signal transducer and activator of transcription (STAT)-3 inhibitors play an important role in regulating immune responses. Galiellalactone (GL) is a fungal secondary metabolite known to interfere with the binding of phosphorylated signal transducer and activator of transcription (pSTAT)-3 as well of pSTAT-6 dimers to their target DNA in vitro. Intra nasal delivery of 50 μg GL into the lung of naive Balb/c mice induced FoxP3 expression locally and IL-10 production and IL-12p40 in RNA expression in the airways in vivo. In a murine model of allergic asthma, GL significantly suppressed the cardinal features of asthma, such as airway hyperresponsiveness, eosinophilia and mucus production, after…

STAT3 Transcription Factormedicine.medical_treatmentImmunologyCD11cSuppressor of Cytokine Signaling ProteinsT-Lymphocytes RegulatoryLactonesMiceImmune systemIn vivomedicineSTAT5 Transcription FactorImmunology and AllergyAnimalsIndoleamine-Pyrrole 23-DioxygenaseAnti-Asthmatic AgentsLungAdministration IntranasalCells CulturedMice Inbred BALB CbiologyChemistryFOXP3General MedicineDendritic CellsT-Lymphocytes Helper-Inducerrespiratory systemAsthmaReceptors Interleukin-3CD11c Antigenrespiratory tract diseasesOvalbuminInterleukin 10CytokineSuppressor of Cytokine Signaling 3 ProteinImmunologySTAT proteinCancer researchbiology.proteinFemaleInterleukin-4T-Box Domain Proteins
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Transforming growth factor   induced FoxP3+ regulatory T cells suppress Th1 mediated experimental colitis

2006

Background and aims: The imbalance between effector and regulatory T cells plays a central role in the pathogenesis of inflammatory bowel diseases. In addition to the thymus, CD4+CD25+ regulatory T cells can be induced in the periphery from a population of CD25− T cells by treatment with transforming growth factor β (TGF-β). Here, we analysed the in vivo function of TGF-β induced regulatory T (Ti-Treg) cells in experimental colitis. Methods: Ti-Treg cells were generated in cell culture in the presence or absence of TGF-β and tested for their regulatory potential in experimental colitis using the CD4+CD62L+ T cell transfer model. Results: Ti-Treg cells significantly suppressed Th1 mediated c…

T cellGastroenterologyFOXP3hemic and immune systemschemical and pharmacologic phenomenaBiologyNatural killer T cellCell biologyInterleukin 21medicine.anatomical_structureImmunologymedicineCytotoxic T cellIL-2 receptorAntigen-presenting cellInterleukin 3Gut
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