Search results for "Fatty acid metabolism"

showing 10 items of 37 documents

Membrane fluidity and fatty acid metabolism in kidney cells from rats fed purified eicosapentaenoic acid or purified docosahexaenoic acid

1998

Hagve T-A, Woldseth B, Brox J, Narce M, Poisson J-P. Membrane £uidity and fatty acid metabolism in kidney cells from rats fed puri¢ed eicosapentaenoic acid or puri¢ed docosahexaenoic acid. Scand J Clin Lab Invest 1998; 58: 187^194. Rats were given a supplement (1.5 ml/day) of purified eicosapentaenoic acid (EPA, 20:5,n-3), purified docosahexaenoic acid (DHA, 22:6,n-3)), or corn oil for 10 days. Membrane fluidity, measured as the steady-state fluorescence polarization of diphenylhexatriene (DPH), was approximately 20% lower in kidney cells from rats fed purified EPA than in cells from the DHA-fed or corn-oil fed animals. The level of 20:5(n-3) in kidney phospholipids was 18 times higher in r…

MaleDocosahexaenoic AcidsMembrane FluidityLinolenic acidLinoleic acidClinical BiochemistryBiologyKidneychemistry.chemical_compoundAnimalsRats WistarPhospholipidsUnsaturated fatty acidchemistry.chemical_classificationFatty acid metabolismCell MembraneFatty AcidsFatty acidGeneral MedicineDietary FatsEicosapentaenoic acidRatsEicosapentaenoic AcidchemistryBiochemistryDocosahexaenoic acidlipids (amino acids peptides and proteins)Arachidonic acidScandinavian Journal of Clinical and Laboratory Investigation
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A high-fat diet increases l-carnitine synthesis through a differential maturation of the Bbox1 mRNAs.

2013

International audience; l-carnitine is a key molecule in both mitochondrial and peroxisomal lipid metabolisms. l-carnitine is biosynthesized from gamma-butyrobetaine by a reaction catalyzed by the gamma-butyrobetaine hydroxylase (Bbox1). The aim of this work was to identify molecular mechanisms involved in the regulation of l-carnitine biosynthesis and availability. Using 3' RACE, we identified four alternatively polyadenylated Bbox1 mRNAs in rat liver. We utilized a combination of in vitro experiments using hybrid constructs containing the Bbox1 3' UTR and in vivo experiments on rat liver mRNAs to reveal specificities in the different Bbox1 mRNA isoforms, especially in terms of polyadenyla…

MaleUntranslated regionPolyadenylation[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMolecular Sequence DataBiologyCell Line03 medical and health scienceschemistry.chemical_compoundBiosynthesisCarnitineAnimalsRNA MessengerRats WistarMolecular BiologyDNA Primers030304 developmental biologychemistry.chemical_classification0303 health sciencesMessenger RNABase SequenceFatty acid metabolism030302 biochemistry & molecular biologyTranslation (biology)Cell BiologyPeroxisomeDietary FatsRatsEnzymeLiverchemistryBiochemistry[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Thyroid hormone controls carnitine status through modifications of gamma-butyrobetaine hydroxylase activity and gene expression.

2002

The carnitine system plays a key role in beta-oxidation of long-chain fatty acids by permitting their transport into the mitochondrial matrix. The effects of hypothyroidism and hyperthyroidism were studied on gamma-butyrobetaine hydroxylase (BBH), the enzyme responsible for carnitine biosynthesis in the rat. In rat liver, BBH activity was decreased in the hypothyroid state and increased in hyperthyroid animals. The modifications in BBH activity correlated with changes in the enzyme Vmax values. These changes were shown to be related to hepatic BBH mRNA abundance. Thyroid hormones are known to interact with lipid metabolism, in particular by increasing long-chain fatty acid oxidation through…

Maleendocrine systemmedicine.medical_specialtyThyroid Hormonesendocrine system diseasesgamma-Butyrobetaine DioxygenaseThyroid GlandBiologyGene Expression Regulation EnzymologicMixed Function OxygenasesCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicineCarnitinemedicineAnimalsCarnitineRNA MessengerMolecular BiologyBeta oxidationPharmacologychemistry.chemical_classificationFatty acid metabolismThyroidFatty acidLipid metabolismCell BiologyRatsKineticsEndocrinologymedicine.anatomical_structurechemistryBiochemistryLiverOrgan SpecificityCarnitine biosynthesisMolecular Medicinemedicine.drugHormoneCellular and molecular life sciences : CMLS
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Changes of peroxisomal fatty acid metabolism during cold acclimatization in hibernating jerboa (Jaculus orientalis)

2003

Abstract Jerboa (Jaculus orientalis) is a deep hibernator originating from sub-desert highlands and represents an excellent model to help to understand the incidence of seasonal variations of food intake and of body as well as environmental temperatures on lipid metabolism. In jerboa, hibernation processes are characterized by changes in the size of mitochondria, the number of peroxisomes in liver and in the expression of enzymes linked to fatty acid metabolism. In liver and kidney, cold acclimatization shows an opposite effect on the activities of the mitochondrial acyl-CoA dehydrogenase (–50%) and the peroxisomal acyl-CoA oxidase (AOX) (+50%), while in brown and white adipose tissues, bot…

Malemedicine.medical_specialtyAcclimatizationAdipose tissueRodentiaWhite adipose tissueBiologyFatty acid degradationBiochemistryAcclimatizationchemistry.chemical_compoundHibernationInternal medicineBrown adipose tissuePeroxisomesmedicineAnimalsRNA MessengerFatty acid metabolismFatty AcidsLipid metabolismGeneral MedicinePeroxisomeMitochondriaCold TemperatureEnzyme ActivationEndocrinologymedicine.anatomical_structureLiverchemistryBiochemistryAcyl-CoA OxidaseBody Temperature RegulationBiochimie
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Fatty Acid Profiles in Demented Patients: Identification of Hexacosanoic Acid (C26:0) as a Blood Lipid Biomarker of Dementia

2014

Background: Several lipid metabolism alterations have been described in the brain and plasma of Alzheimer’s disease (AD) patients, suggesting a relation between lipid metabolism alteration and dementia. 15 16 Objective: We attempted to identify blood fatty acids as biomarkers of dementia. 17 Methods: Fatty acid profiles were established using gas chromatography with or without mass spectrometry on matched plasma and red blood cells (RBCs) of demented patients diagnosed with AD, vascular dementia, or other dementia, and compared with a control group of elderly individuals. The severity of dementia was evaluated with the Mini-Mental State Examination test. 18 19 20 Results: Fatty acid analysi…

Malemedicine.medical_specialtyPathologyChromatography GasErythrocytesBlood lipidsNeuropsychological TestsBiologyMass Spectrometrychemistry.chemical_compoundInternal medicinemedicineHumansDementiaVascular dementiaAgedAged 80 and overchemistry.chemical_classificationFatty acid metabolismGeneral NeuroscienceFatty AcidsFatty acidLipid metabolismGeneral MedicineMiddle AgedPeroxisomemedicine.diseasePsychiatry and Mental healthClinical PsychologyEndocrinologyROC CurvechemistryBiomarker (medicine)DementiaFemaleGeriatrics and GerontologyMental Status ScheduleJournal of Alzheimer's Disease
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PPARα/HNF4α Interplay on Diversified Responsive Elements. Relevance in the Regulation of Liver Peroxisomal Fatty Acid Catabolism

2012

In mammals, the liver is the major organ of fatty acid catabolism. This pathway is involved in both mitochondria and peroxisome. While mitochondria breaks down fatty acids with short, medium and long carbon chains, peroxisomes are involved in the catabolism of very long and branched chain fatty acids, which are degraded by three enzymes: acyl-CoA oxidase, multifunctional enzyme and thiolase enzyme. The active pathway results mainly from a tight transcriptional control of these gene-encoding enzymes. Two major nuclear receptors that are highly expressed in this organ are involved in this control, e.g. PPARα (peroxisome proliferator-activated receptor, α isoform) and HNF4α (hepatic nuclear fa…

Pharmacologychemistry.chemical_classificationFatty acid metabolismCatabolismThiolaseFatty AcidsClinical BiochemistryPeroxisome proliferator-activated receptorMetabolismPeroxisomeBiologyResponse Elementschemistry.chemical_compoundGene Expression RegulationHepatocyte Nuclear Factor 4LiverHepatocyte nuclear factor 4BiochemistrychemistryNuclear receptorPeroxisomesAnimalsHumansPPAR alphaCurrent Drug Metabolism
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0204: Proteome-wide sex-related differences in response to mouse thoracic aortic constriction: molecular bio-signature of failing hearts

2014

Chronic pressure overload (PO) induces pathological left ventricular hypertrophy (LVH) leading to congestive heart failure (HF). Over-expression of FKBP12.6 (FK506 binding protein (K)) in mice should prevent Ca2+-leak during diastole and may improve overall cardiac function. In order to decipher molecular mechanisms involved in thoracic aortic constriction (TAC)-induced cardiac remodelling and the influence of gender and genotype, we performed a proteomic analysis using 2D-DIGE, mass spectrometry and bioinformatics techniques to identify alterations in characteristic biological networks. Wild type (W) and K mice of both genders underwent TAC. Thirty days post-TAC, the altered cardiac remode…

Pressure overloadCardiac function curvemedicine.medical_specialtyHuntingtinFatty acid metabolismbiologybusiness.industryDiastolemedicine.diseaseLeft ventricular hypertrophychemistry.chemical_compoundEndocrinologychemistryHeart failureInternal medicinebiology.proteinmedicineCardiology and Cardiovascular MedicinebusinessCalreticulinArchives of Cardiovascular Diseases Supplements
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Transcriptome responses to carbon tetrachloride and pyrene in the kidney and liver of juvenile rainbow trout (Oncorhynchus mykiss)

2005

Abstract We report the effects of the hepatotoxic compound carbon tetrachloride (CCl 4 ) and pyrene, a model polycyclic aromatic hydrocarbon, on the transcriptomes of juvenile rainbow trout kidneys and livers. Fish were exposed to sublethal doses for 4 days and expression of 1273 genes was measured using a cDNA microarray. Efforts were focused on differentiating between unspecific responses and those that can be regarded as molecular signatures of CCl 4 and pyrene toxicities. Expression profiles were analyzed in terms of Gene Ontology categories. Universal reactions to chemical toxicity were observed in metallothionein, HSP90 and mitochondrial proteins of oxidative phosphorylation, which we…

PyrenesbiologyFatty acid metabolismGene Expression ProfilingHealth Toxicology and MutagenesisAquatic SciencePeroxisomeKidneydigestive systemHsp90Transcriptomechemistry.chemical_compoundFatty acid desaturaseGene Expression RegulationLiverchemistryBiochemistryOncorhynchus mykissHeat shock proteinbiology.proteinAnimalsMetallothioneinPyreneCarbon TetrachlorideOligonucleotide Array Sequence AnalysisAquatic Toxicology
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Effect of Exercise on Fatty Acid Metabolism and Adipokine Secretion in Adipose Tissue

2019

Increased physical activity is an optimal way to maintain a good health. During exercise, triacylglycerols, an energy reservoir in adipose tissue, are hydrolyzed to free fatty acids (FAs) which are then released to the circulation, providing a fuel for working muscles. Thus, regular physical activity leads to a reduction of adipose tissue mass and improves metabolism. However, the reduction of lipid reservoir is also associated with many other interesting changes in adipose tissue FA metabolism. For example, a prolonged exercise contributes to a decrease in lipoprotein lipase activity and resultant reduction of FA uptake. This results in the improvement of mitochondrial function and upregul…

Settore BIO/17 - Istologia0301 basic medicinemedicine.medical_specialtyPhysiologyMini ReviewAdipokineAdipose tissue030209 endocrinology & metabolismInflammationmyokinelcsh:Physiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSettore BIO/10 - BiochimicaPhysiology (medical)AdipocyteInternal medicineMyokinemedicineadipose tissue beigingchemistry.chemical_classificationexerciseadipokinelcsh:QP1-981Fatty acid metabolismSettore BIO/16 - Anatomia UmanaMetabolismadipose tissueexercise adipose tissue fatty acid adipokine myokine adipose tissue beiging030104 developmental biologyEndocrinologychemistryfatty acidmedicine.symptomPolyunsaturated fatty acidFrontiers in Physiology
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Peroxisome-proliferator-activated receptors as physiological sensors of fatty acid metabolism: molecular regulation in peroxisomes

2001

The enzymes required for the beta-oxidation of fatty acyl-CoA are present in peroxisomes and mitochondria. Administration of hypolipidaemic compounds such as clofibrate to rodents leads to an increase in the volume and density of peroxisomes in liver cells. These proliferators also induce simultaneously the expression of genes encoding acyl-CoA oxidase, enoyl-CoA hydratase-hydroxyacyl-CoA dehydrogenase (multifunctional enzyme) and thiolase (3-ketoacyl-CoA thiolase). All these enzymes are responsible for long-chain and very-long-chain fatty acid beta-oxidation in peroxisomes. Similar results were observed when rat hepatocytes, or liver-derived cell lines, were cultured with a peroxisome prol…

Transcriptional ActivationGuinea PigsResponse elementReceptors Cytoplasmic and NuclearBiologyBiochemistryGene Expression Regulation EnzymologicMicechemistry.chemical_compoundPeroxisomesAnimalsAcetyl-CoA C-AcetyltransferasePhosphorylationTranscription factorProtein Kinase Cchemistry.chemical_classificationFatty acid metabolismThiolaseFatty AcidsFatty acidPeroxisomeRatsLiverchemistryBiochemistryAcetyl-CoA C-acetyltransferasePeroxisome proliferator-activated receptor alphaSignal TransductionTranscription FactorsBiochemical Society Transactions
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