Search results for "Fatty acids."

showing 10 items of 706 documents

Changes in lipid metabolism and antioxidant defense status in spontaneously hypertensive rats and Wistar rats fed a diet enriched with fructose and s…

2005

Abstract Objective Larger doses of fructose and saturated fat have been associated with oxidative stress and development of hypertension. The effects of modest amounts of fructose and saturated fatty acids on oxidative stress are unknown. Methods To increase knowledge on this question, 10-wk-old spontaneously hypertensive rats and Wistar rats were fed for 8 wk with a control diet or an experimental diet enriched with fructose (18%) and saturated fatty acids (11%; FS diet). The total antioxidant status of organs and red blood cells was assayed by monitoring the rate of free radical-induced red blood cell hemolysis. Sensitivity of very low-density lipoprotein and low-density lipoprotein (VLDL…

Malemedicine.medical_specialtyErythrocytesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentSaturated fatGlutathione reductaseBlood PressureFructoseBiologyHemolysisThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationRandom Allocationchemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsRats WistarHypertriglyceridemiachemistry.chemical_classificationNutrition and DieteticsGlutathione peroxidaseFatty AcidsLipid MetabolismAscorbic acidRatsOxidative StressEndocrinologychemistrySaturated fatty acidlipids (amino acids peptides and proteins)Lipid PeroxidationPolyunsaturated fatty acidNutrition
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Increased erythrocytes n-3 and n-6 polyunsaturated fatty acids is significantly associated with a lower prevalence of steatosis in patients with type…

2011

Summary Background & aims Non-alcoholic fatty liver disease (NAFLD) is commonly associated with obesity, metabolic syndrome and type 2 diabetes. Although dietary fat contributes substantially to the accumulation of liver fat, the role of individual fatty acids in this accumulation is unclear. Objective In this study, we set out to determine whether liver fat content (LFC), was associated with red blood cell fatty acid (RBC-FA) composition in people with type 2 diabetes. Design, settings, and participants One hundred and sixty-two type 2 diabetic patients were included in this study. LFC was measured using 1H-MR Spectroscopy. RBC-FA composition was measured by gas chromatography. Results One…

Malemedicine.medical_specialtyErythrocytesPalmitic AcidCritical Care and Intensive Care MedicineFatty Acids Monounsaturatedchemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseFatty Acids Omega-6Internal medicineFatty Acids Omega-3PrevalencemedicineHumansPalmitoleic acidProspective StudiesTriglyceridesAgedchemistry.chemical_classificationNutrition and Dieteticsbusiness.industryFatty liverFatty acidMiddle Agedmedicine.diseaseDietary FatsFatty LiverCross-Sectional StudiesEndocrinologyDiabetes Mellitus Type 2chemistryDocosahexaenoic acidDietary SupplementsSaturated fatty acidFemaleArachidonic acidSteatosisbusinessPolyunsaturated fatty acidClinical Nutrition
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Omega-6 and omega-3 polyunsaturated fatty acid levels are reduced in whole blood of Italian patients with a recent myocardial infarction: the AGE-IM …

2014

Abstract Objective The relationship between whole blood fatty acids and myocardial infarction (MI) risk has not been analyzed in detail, especially in Mediterranean countries. The AGE-IM (Acidi Grassi Essenziali e Infarto Miocardico) study was planned to examine the relationships between MI, whole blood fatty acids and the diet in an Italian cohort. Methods 119 Patients with a recent MI and 103 control subjects were enrolled in the study. The whole blood fatty acid composition was determined; information on anthropometrics, biochemical parameters and blood pressure values were also obtained. Diet composition was assessed using a validated food frequency questionnaire from 86 cases and 72 co…

Malemedicine.medical_specialtyInfarctionPhysiologyBlood PressureCohort StudiesFatty Acids MonounsaturatedRisk FactorsFatty Acids Omega-6Fatty Acids Omega-3MedicineHumansMyocardial infarctionWhole bloodAgedchemistry.chemical_classificationAnthropometrybusiness.industryFatty AcidsFatty acidAnthropometryMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareSurgeryDietBlood pressurePolyunsaturated fatty acidmyocardial infarctionchemistryItalyCase-Control StudiesCohortFemaleCardiology and Cardiovascular MedicinebusinessPolyunsaturated fatty acid
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Hepatic steatosis is not due to impaired fatty acid oxidation capacities in C57BL/6J mice fed the conjugated trans-10,cis-12-isomer of linoleic acid.

2004

Decreased body fat mass and liver steatosis have been reported in mice fed diets containing the conjugated linoleic acid trans-10,cis-12-C18:2 (CLA2), but not in those fed diets containing cis-9,trans-11-C18:2 (CLA1). Because the decrease in fatty acid (FA) oxidation may cause fat accumulation, we questioned whether the effects of both CLAs on enzyme activities and mRNA expression were related to liver FA oxidation. To address this question, 7-wk-old male C57BL/6J mice were fed for 4 wk a diet supplemented with 1% CLA1, CLA2, or cis-9-C18:1 (control) esterified as triacylglycerols. In CLA2-fed mice, the proportions of CLA2 in the total FA of liver lipids were substantially lower than those …

Malemedicine.medical_specialtyLinoleic acidConjugated linoleic acidMedicine (miscellaneous)Mitochondria LiverBiologychemistry.chemical_compoundMiceDietary Fats UnsaturatedInternal medicinemedicineAnimalsLinoleic Acids ConjugatedCarnitineRNA MessengerEnzyme InhibitorsUnsaturated fatty acidTriglycerideschemistry.chemical_classificationNutrition and DieteticsCarnitine O-PalmitoyltransferaseEsterificationReverse Transcriptase Polymerase Chain ReactionFatty liverFatty AcidsFatty acidmedicine.diseaseFatty LiverMalonyl Coenzyme AMice Inbred C57BLEndocrinologychemistryBiochemistryLiverCarnitine palmitoyltransferase IOxidation-ReductionPolyunsaturated fatty acidmedicine.drug
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N-3 fatty acids modulate Th1 and Th2 dichotomy in diabetic pregnancy and macrosomia.

2006

We assessed the implication of Th (helper)-cells and the modulation of the Th1/Th2 dichotomy by n-3 polyunsaturated fatty acids (PUFA) in type I diabetic pregnancy (DP) and macrosomia. Female gestant rats fed a standard diet or n-3 PUFA regimen were rendered diabetic by administration of five low doses of streptozotocin. The macrosomic (MAC) offspring were sacrificed at the age of 90 days. The mRNAs of IL-2 and IFN-gamma (Th1 cytokines) and IL-4 (Th2 cytokine) were downregulated in the pancreas and spleen of diabetic pregnant rats. The levels of IL-10 mRNA, another Th2 cytokine, were unchanged in the spleen or upregulated in the pancreas of these animals. Feeding an n-3 PUFA diet to rats wi…

Malemedicine.medical_specialtyOffspringImmunologyPregnancy in DiabeticsSpleenBiologyFetal MacrosomiaTh2 CellsDownregulation and upregulationPregnancyInternal medicineDiabetes mellitusFatty Acids Omega-3medicineImmunology and AllergyAnimalsRNA MessengerRats Wistarchemistry.chemical_classificationPregnancyFatty AcidsTh1 CellsStreptozotocinmedicine.diseaseDietRatsmedicine.anatomical_structureEndocrinologyCholesterolDiabetes Mellitus Type 1chemistryCytokinesFemalePancreasSpleenmedicine.drugPolyunsaturated fatty acidJournal of autoimmunity
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Fatty Acid Profiles in Demented Patients: Identification of Hexacosanoic Acid (C26:0) as a Blood Lipid Biomarker of Dementia

2014

Background: Several lipid metabolism alterations have been described in the brain and plasma of Alzheimer’s disease (AD) patients, suggesting a relation between lipid metabolism alteration and dementia. 15 16 Objective: We attempted to identify blood fatty acids as biomarkers of dementia. 17 Methods: Fatty acid profiles were established using gas chromatography with or without mass spectrometry on matched plasma and red blood cells (RBCs) of demented patients diagnosed with AD, vascular dementia, or other dementia, and compared with a control group of elderly individuals. The severity of dementia was evaluated with the Mini-Mental State Examination test. 18 19 20 Results: Fatty acid analysi…

Malemedicine.medical_specialtyPathologyChromatography GasErythrocytesBlood lipidsNeuropsychological TestsBiologyMass Spectrometrychemistry.chemical_compoundInternal medicinemedicineHumansDementiaVascular dementiaAgedAged 80 and overchemistry.chemical_classificationFatty acid metabolismGeneral NeuroscienceFatty AcidsFatty acidLipid metabolismGeneral MedicineMiddle AgedPeroxisomemedicine.diseasePsychiatry and Mental healthClinical PsychologyEndocrinologyROC CurvechemistryBiomarker (medicine)DementiaFemaleGeriatrics and GerontologyMental Status ScheduleJournal of Alzheimer's Disease
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The Effect of Indobufen on the Activities of Selected Rat Liver Phase I and Phase II Drug Metabolizing Enzymes, Peroxisomal β-oxidation and Hepatic G…

1994

Abstract Oral administration of indobufen to male rats for three days at daily doses of 5, 10 and 20 mg kg−1 resulted in no changes in liver total glutathione, cytosolic glutathione S-transferases or microsomal epoxide hydrolase. Reduced glutathione appeared slightly diminished to about 84% of control at the highest dose level. Microsomal cytochrome P450-dependent ethoxyresorufin O-de-ethylase and pentoxyresorufin de-alkylase activities were decreased to 64% (not significantly) and 67% of control at the lowest dose level. 6α- and 7α-Hydroxytestosterone activities were decreased to 67 and 68% of control at the highest dose level. Cyanide-insensitive peroxisomal fatty acid β-oxidation was inc…

Malemedicine.medical_specialtyPharmaceutical ScienceIsoindolesBiologyMicrobodieschemistry.chemical_compoundCytochrome P-450 Enzyme SystemOral administrationInternal medicinemedicineAnimalsGlutathione TransferaseEpoxide HydrolasesPharmacologychemistry.chemical_classificationIndobufenDose-Response Relationship DrugFatty AcidsFatty acidCytochrome P450Rats Inbred StrainsGlutathionePeroxisomeGlutathionePhenylbutyratesRatsEndocrinologyLiverchemistryMicrosomal epoxide hydrolaseMicrosomebiology.proteinOxidation-Reductionmedicine.drugJournal of Pharmacy and Pharmacology
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Paracrine Activation of Hepatic CB1 Receptors by Stellate Cell-Derived Endocannabinoids Mediates Alcoholic Fatty Liver

2008

SummaryAlcohol-induced fatty liver, a major cause of morbidity, has been attributed to enhanced hepatic lipogenesis and decreased fat clearance of unknown mechanism. Here we report that the steatosis induced in mice by a low-fat, liquid ethanol diet is attenuated by concurrent blockade of cannabinoid CB1 receptors. Global or hepatocyte-specific CB1 knockout mice are resistant to ethanol-induced steatosis and increases in lipogenic gene expression and have increased carnitine palmitoyltransferase 1 activity, which, unlike in controls, is not reduced by ethanol treatment. Ethanol feeding increases the hepatic expression of CB1 receptors and upregulates the endocannabinoid 2-arachidonoylglycer…

Malemedicine.medical_specialtyPhysiologyHUMDISEASEArachidonic AcidsGlyceridesMiceCarnitine palmitoyltransferase 1PiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid Receptor ModulatorsParacrine CommunicationmedicineAnimalsReceptorDiet Fat-RestrictedMolecular BiologyCells CulturedMice KnockoutCarnitine O-PalmitoyltransferaseEthanolChemistryLipogenesisFatty AcidsFatty liverCell Biologymedicine.diseaseEndocannabinoid systemCoculture TechniquesUp-RegulationMice Inbred C57BLDisease Models AnimalLipoprotein LipaseEndocrinologyLiverLipogenesisHepatocytesHepatic stellate cellPyrazoleslipids (amino acids peptides and proteins)Alcoholic fatty liverFatty Acid SynthasesRimonabantSteatosisSterol Regulatory Element Binding Protein 1Oxidation-ReductionEndocannabinoidsFatty Liver AlcoholicCell Metabolism
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Visceral obesity and metabolic syndrome: two faces of the same medal?

2009

In this review, we have analyzed the role of visceral obesity in the occurrence of metabolic syndrome (MetS). MetS is a common metabolic disorder that has been related recently to the increasing prevalence of obesity. The disorder is defined in various ways, but in the near future a new definition(s) should be applicable worldwide. The pathophysiology has been largely attributed, in the past years, to insulin resistance, although several epidemiological and pathophysiological data now indicate visceral obesity as a main factor in the occurrence of all the components of MetS. In view of this, relationships among visceral obesity, free fatty acids, dyslipidemia and insulin resistance have bee…

Malemedicine.medical_specialtySettore MED/09 - Medicina InternaAdipokineFatty Acids NonesterifiedBioinformaticsInsulin resistanceAdipokinesInternal medicineEpidemiologyInternal MedicineMedicineHumansDyslipidemiasMetabolic SyndromeAdiponectinbusiness.industryMetabolic disordermedicine.diseaseObesityEndocrinologyObesity AbdominalEmergency MedicineFemaleAdiponectinMetabolic syndromeInsulin ResistancebusinessDyslipidemiaMetabolic syndrome - Visceral obesity - Adipocytokines - AdiponectinInternal and emergency medicine
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Effects of fluvastatin slow-release (XL 80 mg) versus simvastatin (20 mg) on the lipid triad in patients with type 2 diabetes.

2005

The lipid triad is the association of small, dense (sd) low-density lipoprotein (LDL), low high-density lipoprotein (HDL), and hypertriglyceridemia, all of which play a role in coronary artery disease in patients with type 2 diabetes. Although statins have demonstrated clear positive effects on cardiovascular morbidity/mortality in patients with diabetes and on single components of the lipid triad, it remains controversial whether they affect all components of the triad in these patients. Therefore, we performed a single-center, parallel-group, prospective, randomized, open-label, blinded-endpoint (PROBE)-type comparison of fluvastatin extended-release (XL) 80 mg (n=48) and simvastatin 20 m…

Malemedicine.medical_specialtySimvastatinIndolesHDLApolipoprotein BSmall dense LDLType 2 diabetesTriglycerideLDLFatty Acids MonounsaturatedFluvastatin XLInternal medicineDiabetes mellitusType 2 diabetes mellitusmedicineHumansPharmacology (medical)Prospective StudiesFluvastatinAgedHypertriglyceridemiabiologybusiness.industryAnticholesteremic AgentsApoA-IHypertriglyceridemianutritional and metabolic diseasesType 2 Diabetes MellitusGeneral MedicineMiddle Agedmedicine.diseaseLipoproteins LDLEndocrinologyDiabetes Mellitus Type 2SimvastatinDelayed-Action Preparationsbiology.proteinlipids (amino acids peptides and proteins)FemaleApoBbusinessLipoproteins HDLFluvastatinmedicine.drugLipoproteinAdvances in therapy
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