Search results for "Flow cytometry"

showing 10 items of 814 documents

Radial Scar Versus Tubular Carcinoma of the Breast

1995

Summary The present study is focused on the differential diagnosis between radial scar (RS) and tubular carcinoma (TC) using morphometrical and cytophotometrical analysis (static and flow cytometry) of a number of histologically well-established RS cases, (17 lesions) compared with 6 early infiltrating small TC with sclerotic stroma and pseudo-RS fields. One case displayed both RS and TC foci in contiguity. Mean nuclear area was larger in the group of tubular carcinomas (51.0 μ 2 ) than in the cases o f radial sclerosis (38.30 μ 2 ). We also found a larger number of aneuploid cases in tubular carcinomas measured by image cytometry, but both types o f lesions were diploid when measured by fl…

Pathologymedicine.medical_specialtymedicine.diagnostic_testRadial scarNuclear areaTubular CarcinomasCell BiologyBiologymedicine.diseasePathology and Forensic MedicineFlow cytometryStromamedicineImage CytometryTubular carcinomaDifferential diagnosisPathology - Research and Practice
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THU0260 IL-25 Axis Is Activated and Associated with An ILC2 and M2 Inflammatory State in Patients with Primary Sjogren's Syndrome

2016

Background Type 2 innate lymphoid cells (ILC2) and M2 polarized macrophages are activated and produce cytokines in response to IL-25/IL-17RB, although the relevance of this axis to immune responses in primary Sjoren9s syndrome (pSS) is unknown. Objectives We sought to investigate the role of the IL-25/IL-17RB axis and ILC2 and M2 macrophages in patients with pSS. Methods Expression analysis of IL-17B, IL-25, IL-33 and IL-17RB was performed by TaqMan real-time PCR and immunohistochemistry on salivary glands from 50 patients and 20 controls. Analysis of IL-17RB expression and the frequencies of natural type 2 innate lymphoid cells (nILC2), inflammatory ILC2 (iILC2), and M2-polarized macrophag…

Pathologymedicine.medical_specialtymedicine.diagnostic_testSalivary glandbusiness.industryImmunologyInnate lymphoid cellStimulationPeripheral blood mononuclear cellGeneral Biochemistry Genetics and Molecular BiologyFlow cytometryImmune systemmedicine.anatomical_structureRheumatologyImmunologymedicineImmunology and AllergyImmunohistochemistrybusinessEx vivoAnnals of the Rheumatic Diseases
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OP0081 Tissue Deficiency of The Atypical Chemokine Receptor D6 Is Associated with The Selective Increase of Gut-Derived Pro-Inflammatory CXCR1HIGHLY6…

2016

Background Gut derived innate lymphoid cells of type 3 (ILC)3 are increased in number in the circulation and inflamed tissues of AS patients. Factors influencing the maintainace of ILC3 in an activated status are not clear. The atypical chemokine receptor D6 is a decoy and scavenger receptor for most inflammatory CC chemokines and acts preventing exacerbated inflammatory reactions. Mice lacking D6 expression in the non-hematopoietic compartment display a significant increase of pro-inflammatory monocytes in the peripheral blood and in secondary lymphoid tissues. The role of D6 in human inflammatory disorders has not been inverstigated. Objectives To evaluate whether modulation of D6 express…

Pathologymedicine.medical_specialtymedicine.diagnostic_testbusiness.industryMonocyteImmunologyInnate lymphoid cellC-C chemokine receptor type 7IleumGeneral Biochemistry Genetics and Molecular BiologyFlow cytometrymedicine.anatomical_structureRheumatologyImmunologymedicineImmunology and AllergyMacrophageBone marrowScavenger receptorbusinessAnnals of the Rheumatic Diseases
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Molecular in vivo imaging of gastric cancer in a human-murine xenograft model: targeting epidermal growth factor receptor

2012

Background The prognosis of gastric cancer depends on early diagnosis. Targeted therapies against epidermal growth factor receptors (EGFRs) are currently emerging for the treatment of gastric cancer. Objective To specifically visualize gastric cancer by using monoclonal antibodies targeting EGFR1 as molecular probes for in vivo molecular confocal laser endomicroscopy (mCLE) in a human-murine xenograft model. Design Prospective in vivo animal study. Setting Animal laboratory. Interventions Human gastric carcinoma xenografts were examined in 26 nude mice by using mCLE after injection of fluorescently labeled antibodies. Nine mice received low-dose anti-EGFR1 antibodies, 7 mice cetuximab, and …

Pathologymedicine.medical_specialtymedicine.drug_classCetuximabMice NudeAntineoplastic AgentsAntibodies Monoclonal HumanizedMonoclonal antibodyAntibodiesMiceStomach NeoplasmsEpidermal growth factorIn vivomedicineAnimalsHumansRadiology Nuclear Medicine and imagingEpidermal growth factor receptorFluorescent DyesMicroscopy ConfocalbiologyCetuximabbusiness.industryCarcinomaGastroenterologyAntibodies MonoclonalCancerFlow Cytometrymedicine.diseaseMolecular ImagingErbB ReceptorsDisease Models Animalbiology.proteinImmunohistochemistrybusinessPreclinical imagingmedicine.drugGastrointestinal Endoscopy
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A New Algorithm and Panel Construction for Pediatric Leukemia Immunophenotyping Using 10-Color Flow Cytometry

2012

Abstract Abstract 4799 Background: Rapid identification and quantification of abnormal cell populations in minimal specimen are crucial for diagnosis and longitudinal minimal residual disease (MRD) testing of childhood leukemia. So far, most standard immunophenotypic analyses are performed using antibody panels with up to five-colors and require high cell numbers. For infant and pediatric specimen, high-level multicolor analyses is highly desirable to gather sufficient data for initial diagnostic and follow up monitoring of pathologic populations. Objective: In this study, we aimed to establish a newly defined pediatric multicolor flow cytometric panel algorithm with high reliability yet mi…

Pediatric leukemiamedicine.diagnostic_testChildhood leukemiabusiness.industrymedicine.drug_classImmunologyCell BiologyHematologyMonoclonal antibodymedicine.diseaseBiochemistryMinimal residual diseaseFlow cytometryImmunophenotypingMedicineStage (cooking)businessCytometryAlgorithmBlood
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Expression of membrane C1q in human monocyte-derived macrophages is developmentally regulated and enhanced by interferon-γ

2001

The present study investigated when during "in vitro" maturation macrophages (MPhi) express membrane C1q (mC1q), and whether cell activation affects expression and function of mC1q. Although C1q mRNA was repeatedly detected in freshly isolated monocytes using reverse transcriptase-polymerase chain reaction, C1q protein was observed only in developing MPhi from day 1 to 4 on using immunodetection and flow cytometry. However, the quantity of mC1q and other MPhi membrane proteins differed strikingly in cells from different donors. We report here for the first time that CD14(+) and CD14(-) mC1q-bearing MPhi can develop, and that interferon-gamma increases mC1q display at the cell surface, and m…

PhagocytosisCD14CellLipopolysaccharide ReceptorsBiophysicsMonocyte/macrophageComplementEnzyme-Linked Immunosorbent AssayBiologyLymphocyte ActivationBiochemistryFlow cytometryInterferon-gammaPhagocytosisStructural BiologyGeneticsmedicineHumansMolecular BiologyCells CulturedC1qMessenger RNAmedicine.diagnostic_testComplement C1qMacrophagesCell DifferentiationCell BiologyFlow CytometryPrecipitin TestsMolecular biologyIn vitromedicine.anatomical_structureGene Expression RegulationMembrane proteinDifferentiationCell activationFEBS Letters
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Mast cell-derived mediators promote murine neutrophil effector functions

2013

Mast cells are able to trigger life-saving immune responses in murine models for acute inflammation. In such settings, several lines of evidence indicate that the rapid and protective recruitment of neutrophils initiated by the release of mast cell-derived pro-inflammatory mediators is a key element of innate immunity. Herein, we investigate the impact of mast cells on critical parameters of neutrophil effector function. In the presence of activated murine bone marrow-derived mast cells, neutrophils freshly isolated from bone marrow rapidly lose expression of CD62L and up-regulate CD11b, the latter being partly driven by mast cell-derived TNF and GM-CSF. Mast cells also strongly enhance neu…

PhagocytosisImmunologyApoptosisInflammation610 Medicine & healthmast cellsBiology142-005 142-005Neutrophil ActivationlungMiceImmune systemPhagocytosisneutrophilsmedicineAnimalsImmunology and AllergyCells CulturedMice Knockout2403 ImmunologyInnate immune systemTumor Necrosis Factor-alpharodentGranulocyte-Macrophage Colony-Stimulating FactorPneumoniaGeneral MedicineFlow CytometryMast cellMice Mutant StrainsCell biologycell activationMice Inbred C57BLInterleukin 33medicine.anatomical_structureinflammationImmunology2723 Immunology and AllergyTumor necrosis factor alphamedicine.symptomCell activation
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Cytotoxicity of a naturally occurring furoquinoline alkaloid and four acridone alkaloids towards multi-factorial drug-resistant cancer cells

2015

Abstract Introduction Chemotherapy is one of the preferred mode of treatment of malignancies, but is complicated by the expression of diverse resistance mechanisms of cancer cells. Methods In the present study, we investigated the cytotoxicity of five alkaloids including a furoquinoline montrofoline (1) and four acridones namely 1-hydroxy-4-methoxy-10-methylacridone (2), norevoxanthine (3), evoxanthine (4), 1,3-dimethoxy-10-methylacridone (5) against 9 drug-sensitive and multidrug-resistant (MDR) cancer cell lines. The resazurin reduction assay was used to evaluate the cytotoxicity of these compounds, whilst caspase-Glo assay was used to detect caspase activation. Cell cycle, mitochondrial …

Pharmaceutical ScienceApoptosisPharmacologyBiologyFuroquinoline alkaloidFlow cytometryInhibitory Concentration 50chemistry.chemical_compoundAlkaloidsCell Line TumorDrug DiscoverymedicineHumansCytotoxic T cellCytotoxicityMembrane Potential MitochondrialPharmacologyMolecular Structuremedicine.diagnostic_testCell Cyclemedicine.diseaseAntineoplastic Agents PhytogenicMolecular biologyDrug Resistance MultipleAcridoneLeukemiaComplementary and alternative medicinechemistryDrug Resistance NeoplasmApoptosisCaspasesCancer cellMolecular MedicineReactive Oxygen SpeciesAcridonesPhytomedicine
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The influence of leflunomide on cell cycle, IL-2-receptor (IL-2-R) and its gene expression

1994

Leflunomide is a novel immunomodulatory drug shown to be very effective in animal models of autoimmune diseases and transplantation rejection, as well as in human rheumatoid arthritis. Leflunomide's main metabolite, A77 1726, has been shown to be reversibly antiproliferativein vitro. Pursuing this, we performed cell cycle analysis by flow cytometry of a B-cell lymphoma line and found that at concentrations >2.5 μM cells accumulated in the early S-phase. In order to determine A77 1726's effects on cell activation, human peripheral blood lymphocytes (PBL) were cultured in the presence of PHA or OKT 3 antibody. Flow cytometric evaluation of IL-2 and transferrin receptor expression exhibited a …

Pharmacologymedicine.diagnostic_testImmunologyTransferrin receptorBiologyCell cycleToxicologyMolecular biologyFlow cytometryTransplantationGene expressionmedicinePharmacology (medical)IL-2 receptorCell activationLeflunomidemedicine.drugAgents and Actions
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Sodium-glucose cotransporter type 2 inhibitors prevent ponatinib-induced endothelial senescence and disfunction: A potential rescue strategy

2021

Background: Ponatinib (PON), a third-generation tyrosine kinase inhibitor (TKI), has proven cardiovascular toxicity, with no known preventing agents usable to limit such side effect. Sodium-glucose cotransporter type 2 (SGLT2) inhibitors are a new class of glucose-lowering agents, featuring favorable cardiac and vascular effects. Aims: We assessed the effects of the SGLT2 inhibitors empagliflozin (EMPA) and dapagliflozin (DAPA) on human aortic endothelial cells (HAECs) and underlying vasculo-protective mechanisms in an in vitro model of PON-induced endothelial toxicity. Methods and results: We exposed HAECs to PON or vehicle (DMSO) in the presence or absence of EMPA (100 and 500 nmol/L) or …

Physiologymedicine.drug_classCellPharmacologyAutophagy; Ponatinib; Sodium-glucose cotransporter type 2 (SGLT2) inhibitors; Tyrosine kinase inhibitors; Vascular toxicityTyrosine-kinase inhibitorFlow cytometrychemistry.chemical_compoundmedicineAutophagyHumansViability assayDapagliflozinCellular SenescencePharmacologyTyrosine kinase inhibitorsMatrigelmedicine.diagnostic_testChemistrySodiumImidazolesEndothelial CellsEndothelial stem cellPyridazinesmedicine.anatomical_structureGlucoseDiabetes Mellitus Type 2Sodium-glucose cotransporter type 2 (SGLT2) inhibitorsToxicityPonatinibMolecular MedicineVascular toxicity
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