Search results for "Fragility"

showing 10 items of 75 documents

Very Long Term Stabilization of a 2D Magnet down to the Monolayer for Device Integration

2020

2D materials have recently demonstrated a strong potential for spintronic applications. This has been further reinforced by the discovery of ferromagnetic 2D layers. Nevertheless, the fragility of ...

010302 applied physics[PHYS]Physics [physics]Materials scienceSpintronicsNanotechnology02 engineering and technology021001 nanoscience & nanotechnology01 natural sciencesElectronic Optical and Magnetic MaterialsTerm (time)FragilityFerromagnetismMagnet0103 physical sciencesMonolayerMaterials ChemistryElectrochemistry[PHYS.COND]Physics [physics]/Condensed Matter [cond-mat]0210 nano-technologyComputingMilieux_MISCELLANEOUS
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Loss-of-Function Mutations in UNC45A Cause a Syndrome Associating Cholestasis, Diarrhea, Impaired Hearing, and Bone Fragility

2018

International audience; Despite the rapid discovery of genes for rare genetic disorders, we continue to encounter individuals presenting with syndromic manifestations. Here, we have studied four affected people in three families presenting with cholestasis, congenital diarrhea, impaired hearing, and bone fragility. Whole-exome sequencing of all affected individuals and their parents identified biallelic mutations in Unc-45 Myosin Chaperone A (UNC45A) as a likely driver for this disorder. Subsequent in vitro and in vivo functional studies of the candidate gene indicated a loss-of-function paradigm, wherein mutations attenuated or abolished protein activity with concomitant defects in gut dev…

0301 basic medicineDiarrheaMaleCandidate geneAdolescentBone fragilityArticleBone and Bones03 medical and health sciencesYoung AdultCholestasisLoss of Function MutationGCUNC-45MyosinGeneticsMedicineAnimalsHumansFamilyLymphocytes[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsHearing LossGeneGenetics (clinical)Loss functionZebrafishCholestasisbusiness.industryInfant NewbornIntracellular Signaling Peptides and ProteinsSyndromeFibroblastsmedicine.disease3. Good healthPedigreeDiarrhea030104 developmental biologyPhenotype[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsConcomitantChild PreschoolImmunologyFemalemedicine.symptombusinessGastrointestinal Motility
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FANCD2 modulates the mitochondrial stress response to prevent common fragile site instability

2021

Common fragile sites (CFSs) are genomic regions frequently involved in cancer-associated rearrangements. Most CFSs lie within large genes, and their instability involves transcription- and replication-dependent mechanisms. Here, we uncover a role for the mitochondrial stress response pathway in the regulation of CFS stability in human cells. We show that FANCD2, a master regulator of CFS stability, dampens the activation of the mitochondrial stress response and prevents mitochondrial dysfunction. Genetic or pharmacological activation of mitochondrial stress signaling induces CFS gene expression and concomitant relocalization to CFSs of FANCD2. FANCD2 attenuates CFS gene transcription and pr…

0301 basic medicineGenome instabilitymusculoskeletal diseasesTranscription GeneticQH301-705.5RegulatorMedicine (miscellaneous)MitochondrionBiology[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyGeneral Biochemistry Genetics and Molecular BiologyOxidative PhosphorylationArticle03 medical and health sciences0302 clinical medicineTranscription (biology)Stress Physiologicalhemic and lymphatic diseasesGene expressionFANCD2HumansBiology (General)GeneUbiquitinsChromosomal fragile siteChromosome Fragile SitesChromosome FragilityFanconi Anemia Complementation Group D2 ProteinDNA damage and repair[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyHCT116 CellsCell biologyMitochondriaSettore BIO/18 - Genetica030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisUnfolded Protein ResponseGeneral Agricultural and Biological SciencesDNA Damage
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Role of vitamin D supplementation in the management of musculoskeletal diseases: update from an European Society of Clinical and Economical Aspects o…

2022

© The Author(s) 2022. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds…

AgingBone Density Conservation Agents610 Medicine & healthVitaminsFragility fractureVitamin D DeficiencyFractures BoneFalls Fragility fracture Osteoarthritis Vitamin DDietary SupplementsOsteoarthritisHumansOsteoporosisFallsGeriatrics and GerontologyVitamin D610 Medicine & healthAgedCalcifediolAging clinical and experimental research
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Osteoporosi e diabete mellito. [Osteoporosis and diabetes]

2004

Diabetes mellitus and osteoporosis are chronic diseases with an elevated and growing incidence in the elderly. Recent epidemiological studies have demonstrated an elevated risk of hip, humerus and foot fractures in elder diabetic subjects. While type 1 diabetes is generally associated with a mild reduction in bone mineral density (BMD), type 2 diabetes, more prevalent in old subjects, is frequently linked to a normal or high BMD. Studies on experimental models of diabetes have suggested an altered bone structure that may help to explain the elevated risk of fractures observed in these animals and may as well help to explain the paradox of an incremented risk of fractures in type 2 diabetic …

AgingDiabetes mellituFractureBone mineral densityFragility
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Novel formulations of oral bisphosphonates in the treatment of osteoporosis

2022

AbstractOral bisphosphonates are a key intervention in the treatment of osteoporosis and in reducing the risk of fragility fractures. Their use is supported by over 3 decades of evidence; however, patient adherence to oral bisphosphonates remains poor in part due to complex dosing instructions and adverse events, including upper gastrointestinal symptoms. This problem has led to the development of novel oral bisphosphonate formulations. Buffered, effervescent alendronate is dissolved in water and so seeks to reduce upper gastro-intestinal adverse events, and gastro-resistant risedronate aims to reduce the complexity of dosing procedure (e.g. fasting prior to consumption) whilst still mainta…

AgingDiphosphonatesAlendronateAlendronate/adverse effects610 Medicine & healthRisedronic Acid/therapeutic useDiphosphonates/adverse effectsFractures BoneHumansOsteoporosisAlendronate Bisphosphonates Fragility fracture Osteoporosis Risedronate TherapyGeriatrics and Gerontology610 Medizin und GesundheitBoneFracturesRisedronic AcidOsteoporosis/drug therapy
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Melatonin protects human red blood cells from oxidative hemolysis: new insights into the radical-scavenging activity.

1999

Antioxidant activity of melatonin in human erythrocytes, exposed to oxidative stress by cumene hydroperoxide (cumOOH), was investigated. CumOOH at 300 microM progressively oxidized a 1% suspension of red blood cells (RBCs), leading to 100% hemolysis in 180 min. Malondialdehyde and protein carbonyls in the membrane showed a progressive increase, as a result of the oxidative damage to membrane lipids and proteins, reaching peak values after 30 and 40 min, respectively. The membrane antioxidant vitamin E and the cytosolic reduced glutathione (GSH) were totally depleted in 20 min. As a consequence of the irreversible oxidative damage to hemoglobin (Hb), hemin accumulated into the RBC membrane d…

AntioxidantLysisErythrocytesmedicine.medical_treatmentIn Vitro Techniquesmedicine.disease_causeHemolysisMelatoninchemistry.chemical_compoundEndocrinologymedicineBenzene DerivativesHumansDimethyl SulfoxideMannitolMelatoninChemistryHydroxyl RadicalErythrocyte MembraneGlutathioneFree Radical ScavengersMalondialdehydeGlutathioneRed blood cellOsmotic FragilityOxidative Stressmedicine.anatomical_structureBiochemistryHeminHydroxyl radicalLipid PeroxidationOxidative stressmedicine.drugJournal of pineal research
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Stochastic Vulnerability Assessment of Masonry Structures: Concepts, Modeling and Restoration Aspects

2019

A methodology aiming to predict the vulnerability of masonry structures under seismic action is presented herein. Masonry structures, among which many are cultural heritage assets, present high vulnerability under earthquake. Reliable simulations of their response to seismic stresses are exceedingly difficult because of the complexity of the structural system and the anisotropic and brittle behavior of the masonry materials. Furthermore, the majority of the parameters involved in the problem such as the masonry material mechanical characteristics and earthquake loading characteristics have a stochastic-probabilistic nature. Within this framework, a detailed analytical methodological approac…

Artificial Neural Networkfailure criteriaComputer scienceRestoration mortarStructural system0211 other engineering and technologiesVulnerability020101 civil engineering02 engineering and technologylcsh:Technology0201 civil engineeringlcsh:Chemistryfragility analysisFragilitySeismic assessmentVulnerability assessmentForensic engineeringGeneral Materials ScienceMasonry structurelcsh:QH301-705.5InstrumentationArtificial Neural NetworksmonumentsFluid Flow and Transfer Processes021110 strategic defence & security studieslcsh:Tbusiness.industryProcess Chemistry and TechnologyGeneral EngineeringProbabilistic logicMonumentMasonrylcsh:QC1-999Computer Science ApplicationsCultural heritageSettore ICAR/09 - Tecnica Delle Costruzionilcsh:Biology (General)lcsh:QD1-999restoration mortarslcsh:TA1-2040Fragility analysiseismic assessmentlcsh:Engineering (General). Civil engineering (General)businessdamage indexlcsh:Physicsmasonry structuresstochastic modelingApplied Sciences
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IS THERE A RELATIONSHIP BETWEEN MILD-MODERATE BACK PAIN AND FRAGILITY FRACTURES? ORIGINAL INVESTIGATION

2020

Introduction: Fragility fractures are the cause of 43 thousand deaths: 50% are caused by femur fractures, 28% by vertebral fractures and 22% by various fractures in women, in men the percentages are 47%, 39% and 14% respectively. Vertebral fractures are the most common, 30-50% women over age 50 will experience one of them. Worldwide, is estimated that a new vertebral fracture occurs every 22 seconds. Additionally, vertebral fractures strongly predict the risk of both further fractures and re-fractures, in any skeletal segment. Vertebral fractures are often asymptomatic. Early diagnosis of these is extremely important. The aim of the study is to show, within a population of women over-50 in …

Back pain fragility fractures vertebral osteoporosis.
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Piranesi Prix de Roma. INTERNATIONAL SEMINAR OF STUDIES ON ARCHITECTURE FOR ARCHAEOLOGY

2018

Buffer Zone/Fragility Heritage/ArcheologySettore ICAR/14 - Composizione Architettonica E Urbana
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