Search results for "GABA"

showing 10 items of 390 documents

Role of GABAergic antagonism in the neuroprotective effects of bilobalide

2006

Bilobalide, a constituent of Ginkgo biloba, has neuroprotective properties. Its mechanism of action is unknown but it was recently found to block GABA(A) receptors. The goal of this study was to test the potential role of a GABAergic mechanism for the neuroprotective activity of bilobalide. In rat hippocampal slices exposed to NMDA, release of choline indicates breakdown of membrane phospholipids. NMDA-induced choline release was almost completely blocked in the presence of bilobalide (10 microM) and under low-chloride conditions. Bicuculline (100 microM), a competitive antagonist at GABA(A) receptors, reduced NMDA-induced choline release to a small extent (-23%). GABA (100 microM) partiall…

MaleN-MethylaspartateBrain EdemaCyclopentanesIn Vitro TechniquesPharmacologyBicucullineInhibitory postsynaptic potentialHippocampusArticlegamma-Aminobutyric acidCholineGABA AntagonistsRats Sprague-Dawleychemistry.chemical_compoundBilobalideExcitatory Amino Acid AgonistsmedicineAnimalsPicrotoxinDrug InteractionsFuransMolecular Biologygamma-Aminobutyric AcidChemistryGABAA receptorGeneral NeuroscienceBicucullineGABA receptor antagonistBridged Bicyclo Compounds HeterocyclicRatsGinkgolidesNeuroprotective Agentsnervous systemNonlinear DynamicsMechanism of actionArea Under CurveGABAergicNeurology (clinical)medicine.symptomSynaptosomesDevelopmental Biologymedicine.drugBrain Research
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Lateral habenula and hippocampus: A complex interaction raphe cells-mediated

1997

The study has shown an excitatory influence exerted by lateral habenula (LH) on hippocampal pyramidal cells. The modulatory influence is paradoxically serotonine-mediated; in fact all LH stimulation effects were abolished by intrahippocampal iontophoretic methysergide application. The data suggest the involvement of dorsal raphe nucleus. In fact, the dorsal raphe nucleus stimulation caused on hippocampus an expected inhibitory effect antagonized by intrahippocampal iontophoretic methysergide application. In the context of this neural structure we have highlighted a disinhibitory relation between two types of cells: slow serotonergic efferent neurones and fast GABAergic interneurones. The di…

MaleN-MethylaspartateMethysergideCell CommunicationBicucullineGABA AntagonistsDorsal raphe nucleusmedicineAnimalsRats WistarBiological PsychiatryNeuronsHabenulaRapheChemistryPyramidal CellsIontophoresisBicucullineGABA receptor antagonistElectric StimulationRatsPsychiatry and Mental healthHabenula2-Amino-5-phosphonovaleratenervous systemNeurologyRaphe NucleiGABAergicNeurology (clinical)Raphe nucleiNeurosciencemedicine.drugJournal of Neural Transmission
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Intensity of GABA-evoked responses is modified by nitric oxide-active compounds in the subthalamic nucleus of the rat: a microiontophoretic study.

2009

We have previously described modulatory effects of nitric oxide (NO)-active drugs on subthalamic nucleus (STN) neurons. In this study, the effects of microiontophoretically applied NO-active compounds on GABA-evoked responses were investigated in subthalamic neurons extracellularly recorded from anesthetized rats: 45 of 62 cells were excited by S-nitroso-glutathione (SNOG), an NO donor, whereas 28 of 43 neurons were inhibited by N-nitro-L-arginine methyl ester (L-NAME), a NOS inhibitor. Nearly all neurons responding to SNOG and/or L-NAME showed significant inhibitory responses to the administration of iontophoretic GABA. In these cells, the changes induced by NO-active drugs in the magnitud…

MaleNOS inhibitormedicine.drug_classBiophysicsAction PotentialsGlutamic AcidPharmacologyNeurotransmissionInhibitory postsynaptic potentialBicucullineNitric OxideSettore BIO/09 - FisiologiaNitric oxideGABA AntagonistsCellular and Molecular Neurosciencechemistry.chemical_compoundSubthalamic NucleusmedicineAnimalsDrug InteractionsNitric Oxide DonorsEnzyme InhibitorsRats Wistargamma-Aminobutyric AcidNeuronssubthalamic nucleus GABA SNOG L-NAMEIontophoresisBicucullineIontophoresisReceptor antagonistElectric StimulationRatsSubthalamic nucleusNG-Nitroarginine Methyl Esternervous systemchemistryS-Nitrosoglutathionemedicine.drugJournal of neuroscience research
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The gamma(2)-MSH peptide mediates a central analgesic effect via a GABA-ergic mechanism that is independent from activation of melanocortin receptors.

2001

Using the latency for tail-flick after thermal stimulation we have assessed the effects of alpha-, gamma(1)- and gamma(2)-MSH on nociceptive threshold in the mice. Intracisternal injections of gamma(2)-MSH induced a distinct analgesia, while gamma(1)-MSH in the same doses gave only a minor analgesia. Intracisternal alpha-MSH instead gave a short-term hyperalgesia. The effect of gamma(2)-MSH was not blocked by any of the MC(4)/MC(3)receptor antagonist HS014, naloxone or by the prior intracisternal administrations of gamma(1)-MSH. However, the gamma(2)-MSH analgesic response was completely attenuated by treating animals with the GABA(A)antagonist bicuculline. The gamma(2)-MSH analgesic effect…

MaleNarcotic Antagonists(+)-NaloxonePharmacologyGABA Antagonistschemistry.chemical_compoundMiceEndocrinologyDrug Interactionsgamma-Aminobutyric AcidAnalgesicsMice Inbred BALB Cintegumentary systemMuscimolNaloxoneReceptors MelanocortinNociceptorsGeneral MedicineReceptor antagonistNeurologyHyperalgesiamedicine.symptomhormones hormone substitutes and hormone antagonistsmedicine.drugPain ThresholdTailendocrine systemmedicine.medical_specialtyanimal structuresmedicine.drug_classCatalepsyBicucullinePeptides CyclicCellular and Molecular Neurosciencegamma-MSHMelanocortin receptorInternal medicinemedicineAnimalsGABA ModulatorsGABA AgonistsCatalepsyDiazepamEthanolEndocrine and Autonomic SystemsAntagonistCentral Nervous System DepressantsBicucullinemedicine.diseaseEndocrinologyMuscimolchemistryReceptors Corticotropinalpha-MSHNeuropeptides
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Inhibition of different GABA transporter systems is required to attenuate epileptiform activity in the CA3 region of the immature rat hippocampus

2014

GABA transporters (GATs) are an essential element of the GABAergic system, which regulate excitability in the central nervous system and are thus used as targets for anticonvulsive therapy. However, in the immature nervous system the functions of the GABAergic system and the expression profile of GATs are distinct from the adult situation, obscuring to predict how different GAT isoforms influence epileptiform activity. Therefore we analyzed the effects of subtype specific GAT inhibitors on repetitive epileptiform discharges using field potential and whole-cell patch-clamp recordings in the CA3 region of hippocampal slices of immature (postnatal days 4-7) rats. These experiments revealed tha…

MaleNervous systemGABA Plasma Membrane Transport Proteinsgenetic structuresTiagabineCentral nervous systemAction PotentialsHippocampusHippocampal formationPharmacologyGABA AntagonistsOrgan Culture TechniquesSeizuresmedicineAnimalsGABA transporter4-AminopyridineRats WistarbiologyChemistryNeural InhibitionTransporterCA3 Region Hippocampaleye diseasesRatsmedicine.anatomical_structureAnimals Newbornnervous systemNeurologybiology.proteinGABAergicGABA Uptake InhibitorsNeurology (clinical)medicine.drugEpilepsy Research
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GABAergic basal forebrain afferents innervate selectively GABAergic targets in the main olfactory bulb

2010

In this work we have analyzed the targets of the GABAergic afferents to the main olfactory bulb originating in the basal forebrain of the rat. We combined anterograde tracing of 10 kD biotinylated dextran amine (BDA) injected in the region of the horizontal limb of the diagonal band of Broca that projects to the main olfactory bulb, with immunocytochemical detection of GABA under electron microscopy or vesicular GABA transporter (vGABAt) under confocal fluorescent microscopy. GABAergic afferents were identified as double labeled BDA-GABA boutons. Their targets were identified by their ultrastructure and GABA content. We found that GABAergic afferents from the basal forebrain were distribute…

MaleOlfactory systemVesicular Inhibitory Amino Acid Transport ProteinsPeriglomerular cellOlfactionBiologyProsencephalonNeural PathwaysmedicineAnimalsNeurons AfferentRats Wistargamma-Aminobutyric AcidBasal forebrainGeneral NeuroscienceOlfactory tubercleGranule cellOlfactory BulbRatsOlfactory bulbNeuroanatomical Tract-Tracing Techniquesmedicine.anatomical_structurenervous systemGABAergicFemaleNeuroscienceNeuroscience
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Parvalbumin-containing interneurons do not innervate granule cells in the olfactory bulb

2001

Combining pre-embedding parvalbumin immunostaining and post-embedding immunogold detection of GABA in the olfactory bulb, we investigated whether the parvalbumin-containing GABAergic interneurons of the external plexiform layer exclusively innervate principal cells, or whether they also establish inhibitory synapses upon GABAergic local neurons such as granule cells. Our results demonstrate that the parvalbumin-containing cells do not contact GABAergic interneurons in the neuropil of the external plexiform layer. On the contrary, their postsynaptic elements were always non-GABAergic principal cells. Although classically it has been accepted that the interneurons of the external plexiform la…

MaleOlfactory systemgenetic structuresInterneuronInhibitory postsynaptic potentialInterneuronsPostsynaptic potentialNeural PathwaysNeuropilmedicineAnimalsRats WistarMicroscopy Immunoelectrongamma-Aminobutyric Acidbiologymusculoskeletal neural and ocular physiologyGeneral NeuroscienceOlfactory BulbRatsOlfactory bulbSmellParvalbuminsmedicine.anatomical_structurenervous systemSynapsesbiology.proteinGABAergicNeuroscienceParvalbuminNeuroreport
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Intergenerational continuity in parents’ and adolescents’ externalizing problems: The role of life events and their interaction with GABRA2.

2015

We examine whether parental externalizing behavior has an indirect effect on adolescent externalizing behavior via elevations in life events, and whether this indirect effect is further qualified by an interaction between life events and adolescents’ GABRA2 genotype (rs279871). We use data from 2 samples: the Child Development Project (CDP; n = 324) and FinnTwin12 (n = 802). In CDP, repeated measures of life events, mother-reported adolescent externalizing, and teacher-reported adolescent externalizing were used. In FinnTwin12, life events and externalizing were assessed at age 14. Parental externalizing was indexed by measures of antisocial behavior and alcohol problems or alcohol dependen…

MaleParentsExternalizationAdolescentGenotypeTwinsPolymorphism Single NucleotideArticleDevelopmental psychologyLife Change EventsLife eventsmedicineHumansGenetic Predisposition to DiseaseParent-Child RelationsChildta515AllelesBiological PsychiatryAggressionAntisocial personality disorderAlcohol dependenceAntisocial Personality DisorderReceptors GABA-Amedicine.diseaseModerationChild developmentTwin studyGene-environment interactionExternalizingAggressionAlcoholismIntergenerational continuityClinical PsychologyPsychiatry and Mental healthGABRA2FemaleGene-Environment Interactionmedicine.symptomPsychologyPsychopathologyJournal of Abnormal Psychology
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Functional Synaptic Projections onto Subplate Neurons in Neonatal Rat Somatosensory Cortex

2002

Subplate neurons (SPn) play an important role in the formation of thalamocortical connections during early development and show glutamatergic and GABAergic spontaneous synaptic activity. We characterized these synaptic inputs by performing whole-cell recordings from SPn in somatosensory cortical slices of postnatal day 0-3 rats. At -70 mV, electrical stimulation of the thalamocortical afferents elicited in 68% of the SPn a monosynaptic CNQX-sensitive postsynaptic current (PSC). These fast PSCs were mediated by AMPA receptors, because they were prolonged by cyclothiazide and blocked by GYKI 52466. On membrane depolarization, thalamocortical stimulation elicited in 50% of the cells an additio…

MalePatch-Clamp TechniquesAction PotentialsStimulationAMPA receptorBiologyIn Vitro TechniquesSomatosensory systemReceptors N-Methyl-D-AspartateMembrane PotentialsGABA AntagonistsThalamusSubplatemedicineAnimalsReceptors AMPAARTICLERats Wistargamma-Aminobutyric AcidNeuronsAfferent PathwaysGeneral NeuroscienceLysineCell MembraneExcitatory Postsynaptic PotentialsDepolarizationSomatosensory CortexReceptors GABA-AElectric StimulationRatsmedicine.anatomical_structurenervous systemAnimals NewbornSynapsesGABAergicNMDA receptorCyclothiazideNeuroscienceExcitatory Amino Acid Antagonistsmedicine.drug
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Membrane-Derived Phospholipids Control Synaptic Neurotransmission and Plasticity

2015

Synaptic communication is a dynamic process that is key to the regulation of neuronal excitability and information processing in the brain. To date, however, the molecular signals controlling synaptic dynamics have been poorly understood. Membrane-derived bioactive phospholipids are potential candidates to control short-term tuning of synaptic signaling, a plastic event essential for information processing at both the cellular and neuronal network levels in the brain. Here, we showed that phospholipids affect excitatory and inhibitory neurotransmission by different degrees, loci, and mechanisms of action. Signaling triggered by lysophosphatidic acid (LPA) evoked rapid and reversible depress…

MalePatch-Clamp TechniquesQH301-705.5NeurotransmissionBiologyInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyMicePregnancySynaptic augmentationMetaplasticityAnimalsRats WistarBiology (General)Motor Neuronsrho-Associated KinasesNeuronal PlasticityGeneral Immunology and MicrobiologyCalcineurinGeneral NeuroscienceReceptors GABA-ACell biologySynaptic fatigueBiochemistrySynapsesSynaptic plasticityExcitatory postsynaptic potentialFemalelipids (amino acids peptides and proteins)Synaptic signalingLysophospholipidsrhoA GTP-Binding ProteinGeneral Agricultural and Biological SciencesResearch Article
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