Search results for "GABAergic"

showing 10 items of 131 documents

Cannabinoid CB1 Receptor Calibrates Excitatory Synaptic Balance in the Mouse Hippocampus

2015

The endocannabinoid system negatively regulates the release of various neurotransmitters in an activity-dependent manner, thereby influencing the excitability of neuronal circuits. In the hippocampus, cannabinoid type 1 (CB1) receptor is present on both GABAergic and glutamatergic axon terminals. CB1 receptor-deficient mice were previously shown to have increased hippocampal long-term potentiation (LTP). In this study, we have investigated the consequences of cell-type-specific deletion of the CB1 receptor on the induction of hippocampal LTP and on CA1 pyramidal cell morphology. Deletion of CB1 receptor in GABAergic neurons in GABA-CB1-KO mice leads to a significantly decreased hippocampal …

Long-Term PotentiationHippocampusHippocampal formationBiologyHippocampusSynaptic TransmissionMiceGlutamatergicReceptor Cannabinoid CB1medicineAnimalsAxonMice KnockoutNeuronal Plasticitymusculoskeletal neural and ocular physiologyGeneral NeuroscienceExcitatory Postsynaptic Potentialsfood and beveragesLong-term potentiationArticlesEndocannabinoid systemMice Inbred C57BLmedicine.anatomical_structurenervous systemSynapsesSynaptic plasticityGABAergiclipids (amino acids peptides and proteins)NeuroscienceThe Journal of Neuroscience
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Neuroprotective action of diazepam at very low and moderate doses in Alzheimer's disease model rats

2018

Abstract Early manifestations of Alzheimer's disease (AD) include neuroinflammation, disrupted neurotransmission and cognitive deficits. Impairment of the GABAergic system is essentially involved in the pathogenesis of AD. Traditionally, agonists of GABAA receptors at doses above 1 mg/kg are known to possess memory impairing effects. However, we have previously found that GABAA receptor GABA site ligand muscimol at very low doses acted contrary – enhanced spatial learning/memory, as well as prevented neuroinflammation and augmented neurotransmission in AD model rats. Therefore, in the present study we focused on the assessment of the effects of non-sedative – very low (0.05 mg/kg) and moder…

Male0301 basic medicineAllosteric modulatormedicine.drug_classSynaptophysinNeurotransmissionPharmacologyHippocampusNeuroprotectionRandom Allocation03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compound0302 clinical medicineAlzheimer DiseasemedicineAnimalsGliosisRats Wistargamma-Aminobutyric AcidCerebral CortexPharmacologyMemory DisordersBenzodiazepineDiazepamDose-Response Relationship DrugGlutamate DecarboxylaseGABAA receptorAcetylcholineNeuroprotective Agents030104 developmental biologyGene Expression RegulationMuscimolchemistryAstrocytesSynaptic plasticityGABAergic030217 neurology & neurosurgeryNeuropharmacology
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Memory-enhancing and brain protein expression-stimulating effects of novel calcium antagonist in Alzheimer’s disease transgenic female mice

2016

The prevalence of Alzheimer's disease (AD) is higher in females than in males, and causes more severe cognitive, memory and behavioral impairments. Previously, in male transgenic (Tg) APPSweDI mice, we reported that the novel lipophilic 1,4-dihydropyridine (DHP) derivative AP-12 crossed the blood-brain barrier, blocked neuronal and vascular calcium channels, changed brain protein expression and improved behavior. In this study, we used female Tg APPSweDI mice to assess the effects of AP-12 on behavior, and brain protein expression, with a particular focus on those of the GABAergic system. The results showed that in female Tg mice, similar to male Tg mice, AP-12 improved spatial learning/mem…

Male0301 basic medicineCingulate cortexDihydropyridinesmedicine.medical_specialtyElevated plus mazeVesicular Inhibitory Amino Acid Transport ProteinsHippocampusMice TransgenicWater mazeBiologyHippocampal formationGyrus CinguliHippocampusArticleAmyloid beta-Protein PrecursorMice03 medical and health sciences0302 clinical medicineAlzheimer DiseaseMemoryInternal medicineNeuroplasticitymedicineAnimalsGABAergic NeuronsMaze LearningPharmacologyAmyloid beta-PeptidesNeuronal PlasticityGlutamate DecarboxylaseCalcium Channel BlockersUp-RegulationDisease Models Animal030104 developmental biologyEndocrinologyAnti-Anxiety AgentsBlood-Brain BarrierSynaptic plasticityGABAergicCalciumFemale030217 neurology & neurosurgeryPharmacological Research
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Pathology-selective antiepileptic effects in the focal freeze-lesion rat model of malformation of cortical development

2021

Malformations of cortical development (MCD) represent a group of rare diseases with severe clinical presentation as epileptic and pharmacoresistant encephalopathies. Morphological studies in tissue from MCD patients have revealed reduced GABAergic efficacy and increased intracellular chloride concentration in neuronal cells as important pathophysiological mechanisms in MCD. Also, in various animal models, alterations of GABAergic inhibition have been postulated as a predominant factor contributing to perilesional hyperexcitability. Along with this line, the NKCC1 inhibitor bumetanide has been postulated as a potential drug for treatment of epilepsy, mediating its antiepileptic effect by red…

Male0301 basic medicinePathologymedicine.medical_specialtyZonisamideInhibitory postsynaptic potentialCryosurgeryLesion03 medical and health sciencesEpilepsyOrgan Culture Techniques0302 clinical medicineSodium Potassium Chloride Symporter InhibitorsDevelopmental NeuroscienceSeizuresmedicineAnimals4-AminopyridineRats WistarBumetanideCerebral Cortexbusiness.industryCarbamazepinemedicine.diseaseRatsMicrogyrusMalformations of Cortical Development030104 developmental biologyNeurologyGABAergicAnticonvulsantsmedicine.symptombusiness030217 neurology & neurosurgeryBumetanidemedicine.drugExperimental Neurology
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Neuroligin-2 Expression in the Prefrontal Cortex is Involved in Attention Deficits Induced by Peripubertal Stress

2016

Emerging evidence indicates that attention deficits, which are frequently observed as core symptoms of neuropsychiatric disorders, may be elicited by early life stress. However, the mechanisms mediating these stress effects remain unknown. The prefrontal cortex (PFC) has been implicated in the regulation of attention, including dysfunctions in GABAergic transmission, and it is highly sensitive to stress. Here, we investigated the involvement of neuroligin-2 (NLGN-2), a synaptic cell adhesion molecule involved in the stabilization and maturation of GABAergic synapses, in the PFC in the link between stress and attention deficits. First, we established that exposure of rats to stress during th…

Male0301 basic medicineSerial reaction timeCell Adhesion Molecules NeuronalGenetic VectorsGlutamate decarboxylasePrefrontal CortexNerve Tissue ProteinsNeuropsychological Testsgamma-Aminobutyric acidRandom Allocation03 medical and health sciences0302 clinical medicineContinuous performance taskCortex (anatomy)medicineAnimalsAttentionRNA MessengerSexual MaturationRats WistarPrefrontal cortexgamma-Aminobutyric AcidPharmacologymedicine.diagnostic_testCognitionDependovirusImmunohistochemistryDisease Models AnimalPsychiatry and Mental health030104 developmental biologymedicine.anatomical_structureAttention Deficit Disorder with HyperactivityGABAergicOriginal ArticlePsychologyNeuroscienceStress Psychological030217 neurology & neurosurgerymedicine.drugNeuropsychopharmacology
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Phencyclidine inhibits the activity of thalamic reticular gamma-aminobutyric acidergic neurons in rat brain.

2014

Póster presentado en el IX Simposi de Neurobiologia Experimental, celebrado los días 22 y 23 de octubre de 2014 en Barcelona y organizado por la Societat Catalana de Biologia del Institut d'Estudis Catalans

MaleAction PotentialsPhencyclidinePrefrontal CortexLocal field potentialGABA AntagonistsThalamusthalamocortical networksNeural PathwaysmedicinePremovement neuronal activityAnimalsNMDA receptor antagonistsAntipsychotic drugsGABAergic NeuronsRats WistarPrefrontal cortexReceptorPhencyclidineClozapineBiological PsychiatryClozapineAnalysis of VarianceChemistryRatsschizophreniaElectrophysiologyParvalbuminspsychotic symptomsExcitatory postsynaptic potentialHallucinogensNeurosciencemedicine.drugBiological psychiatry
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Effects of 8-OH-DPAT on open field performance of young and aged rats prenatally exposed to diazepam: a tool to reveal 5-HT1A receptor function

2003

Central GABAergic and serotoninergic systems interact with one another and are implicated in controlling different behaviours. A gentle early long-lasting handling can prevent the deficits in locomotion and exploration in open field (O.F.) in 3-month-old male rats prenatally exposed to diazepam (DZ). Purpose of this study was to extend the research to older handled rats prenatally exposed to DZ and to assess the activity of 5-HT1A receptors (Rs), evaluating the performance in O.F. at 3 and 18 months of age following 8-OH-DPAT administration. A single daily s.c. injection of DZ (1.5 mg/kg) from gestation day 14 to gestation day 20 induced in aged, but not in young rats, a decrease in total d…

MaleAgingmedicine.medical_specialtySettore BIO/14 - FARMACOLOGIARats Prenatal diazepam Long-lasting handling Aging 8-OH-DPAT Open field testMotor ActivityHandling PsychologicalSerotonergicOpen fieldchemistry.chemical_compoundPregnancyInternal medicinemedicineAnimalsPharmacology (medical)Rats WistarReceptorgamma-Aminobutyric AcidBiological PsychiatrydiazepamPharmacology8-Hydroxy-2-(di-n-propylamino)tetralinBehavior Animal8-OH-DPATin utero treatmentRatsSerotonin Receptor AgonistsPsychiatry and Mental healthEndocrinologyAnti-Anxiety AgentsNeurologychemistryPrenatal Exposure Delayed EffectsReceptors Serotonin5-HT1a receptorsGABAergicGestation5-HT1A receptorSettore MED/26 - NeurologiaFemaleNeurology (clinical)PsychologyReceptors Serotonin 5-HT1Diazepammedicine.drugEuropean Neuropsychopharmacology
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A restricted population of CB1 cannabinoid receptors with neuroprotective activity.

2014

The CB1 cannabinoid receptor, the main molecular target of endocannabinoids and cannabis active components, is the most abundant G protein-coupled receptor in the mammalian brain. Of note, CB1 receptors are expressed at the synapses of two opposing (i.e., GABAergic/inhibitory and glutamatergic/excitatory) neuronal populations, so the activation of one and/or another receptor population may conceivably evoke different effects. Despite the widely reported neuroprotective activity of the CB1 receptor in animal models, the precise pathophysiological relevance of those two CB1 receptor pools in neurodegenerative processes is unknown. Here, we first induced excitotoxic damage in the mouse brain b…

MaleCannabinoid receptorPopulationNeurotoxinsExcitotoxicityGlutamic AcidBiologymedicine.disease_causeNeuroprotectionGlutamatergicMiceOrgan Culture TechniquesReceptor Cannabinoid CB1medicineAnimalsHumansGABAergic NeuronsReceptoreducationCaenorhabditis elegans ProteinsAgedCerebral CortexMice KnockoutNeuronseducation.field_of_studyMultidisciplinaryIntegrasesmusculoskeletal neural and ocular physiologyNeurodegenerative DiseasesBiological SciencesMiddle AgedReceptors GABA-AEndocannabinoid systemCorpus Striatumnervous systemGABAergiclipids (amino acids peptides and proteins)FemaleNeurosciencepsychological phenomena and processesEndocannabinoidsSynaptosomesProceedings of the National Academy of Sciences of the United States of America
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γ1- and γ2-melanocyte stimulating hormones induce central anxiogenic effects and potentiate ethanol withdrawal responses in the elevated plus-maze te…

2008

Little is known about the endogenous functions of gamma1- and gamma2-melanocyte stimulating hormones (gamma1- and gamma2-MSH). Although gamma-MSHs bind to melanocortin receptor subtypes 3 and 4, we have previously shown that these peptides also influence non-melanocortinergic processes, such as dopaminergic and GABAergic. The aim of this study was to determine the effects of gamma1- and gamma2-MSH (at doses 0.3, 1 and 2 nmol/mouse/5 microl) on the anxiety levels in mice in elevated plus maze. Three experimental paradigms were performed to assess the effects of peptides on: a) ethanol withdrawal; b) acute ethanol-induced anxiolytic action; c) peptides per se. We used ethanol as the model sub…

MaleElevated plus mazemedicine.medical_specialtyMelanocyte-stimulating hormonemedicine.drug_classClinical BiochemistryAnxietyToxicologyBiochemistryAnxiolyticMiceBehavioral NeuroscienceMelanocortin receptorInternal medicinemedicineAnimalsMelanocyte-Stimulating HormonesMaze LearningBiological PsychiatryPharmacologyMice Inbred ICRDose-Response Relationship DrugEthanolDopaminergicSubstance Withdrawal SyndromeEndocrinologyAnxiogenicGABAergicPsychologyHormonePharmacology Biochemistry and Behavior
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Hippocampal GABAergic Synapses Possess the Molecular Machinery for Retrograde Nitric Oxide Signaling

2007

Nitric oxide (NO) plays an important role in synaptic plasticity as a retrograde messenger at glutamatergic synapses. Here we describe that, in hippocampal pyramidal cells, neuronal nitric oxide synthase (nNOS) is also associated with the postsynaptic active zones of GABAergic symmetrical synapses terminating on their somata, dendrites, and axon initial segments in both mice and rats. The NO receptor nitric oxide-sensitive guanylyl cyclase (NOsGC) is present in the brain in two functional subunit compositions: α1β1and α2β1. The β1subunit is expressed in both pyramidal cells and interneurons in the hippocampus. Using immunohistochemistry andin situhybridization methods, we describe that the …

MaleInterneuronReceptors Cytoplasmic and NuclearNitric Oxide Synthase Type IBiologyNitric OxideHippocampusSynapseMiceSoluble Guanylyl CyclasemedicineAnimalsHumansRNA MessengerRats Wistargamma-Aminobutyric AcidMice Knockoutmusculoskeletal neural and ocular physiologyGeneral NeuroscienceArticlesAxon initial segmentRatsMice Inbred C57BLmedicine.anatomical_structurenervous systemGuanylate CyclaseSynaptic plasticitySynapsesbiology.proteinRetrograde signalingGABAergicSoluble guanylyl cyclaseNeuroscienceParvalbuminSignal Transduction
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