Search results for "GENI"

showing 10 items of 6843 documents

The Fontan-Operation: From Intra- to Extracardiac Procedure

2003

Purpose For treatment of univentricular heart, the Fontan operation has been established as the definitive palliation. The current controversy is mainly based on the high incidence of arrhythmias after an intra-atrial lateral tunnel Fontan operation. Methods From January 1995 until April 2002. 46 children underwent a Fontan-type operation with or without a small fenestration. In 33 patients (group I) an Intracardiac tunnel and in 13 patients (group II) an extracardiac conduit procedure was performed. Principal findings There was no perioperative mortality. All patients showed postoperative a significant increase of arterial oxygen saturation, from 76 to 86% after surgery with fenestration, …

AdultHeart Defects CongenitalMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAdolescentmedicine.medical_treatmentGroup iiExtracardiac conduitFontan ProcedureIntracardiac injectionFontan procedureIntraoperative PeriodmedicineHumansRadiology Nuclear Medicine and imagingcardiovascular diseasesChildCardiopulmonary Bypassbusiness.industryInfantPerioperativeSurgical InjuryUniventricular heartSurgeryOxygensurgical procedures operativeChild Preschoolcardiovascular systemFemaleSurgeryHigh incidencebusinessCardiology and Cardiovascular MedicineFollow-Up StudiesCardiovascular Surgery
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Microdeletion 22q11 in complex cardiovascular malformations.

1997

Besides DiGeorge, velocardiofacial and conotruncal anomaly face syndromes, some of the isolated congenital heart diseases have also been associated with a chromosomal deletion in 22q11. These disease entities, which had originally been considered to have a different genetic background, are now included in the CATCH-22 microdeletion complex. CATCH 22 is an acronym for cardiac defect, abnormal facies, thymic hypoplasia or aplasia and T-cell deficiency, cleft palate, hypoparathyroidism, and hypocalcemia. In the present study, we focused on the complex cardiovascular defects (CCVD) and screened 40 patients for a microdeletion of 22q11 by fluorescence in situ hybridization using the D22S75 DNA p…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyAdolescentChromosomes Human Pair 22Persistent truncus arteriosusBiologyDouble outlet right ventricleDuctus arteriosusInternal medicineConotruncal defectGeneticsmedicineHumansChildGenetics (clinical)In Situ Hybridization FluorescenceTetralogy of FallotInfant NewbornInfantAplasiamedicine.diseasemedicine.anatomical_structureEndocrinologyGreat arteriesThymic hypoplasiaChild PreschoolCardiologyFemaleChromosome DeletionHuman genetics
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Phenotypic analysis of individuals with Costello syndrome due to HRAS p.G13C.

2011

Costello syndrome is characterized by severe failure-to-thrive, short stature, cardiac abnormalities (heart defects, tachyarrhythmia, and hypertrophic cardiomyopathy (HCM)), distinctive facial features, a predisposition to papillomata and malignant tumors, postnatal cerebellar overgrowth resulting in Chiari 1 malformation, and cognitive disabilities. De novo germline mutations in the proto-oncogene HRAS cause Costello syndrome. Most mutations affect the glycine residues in position 12 or 13, and more than 80% of patients share p.G12S. To test the hypothesis that subtle genotype-phenotype differences exist, we report the first cohort comparison between 12 Costello syndrome individuals with p…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyAdolescentrasopathy.RASopathyShort statureProto-Oncogene MasArticleProto-Oncogene Proteins p21(ras)Young AdultGermline mutationSettore MED/38 - Pediatria Generale E SpecialisticaCostello syndromePregnancyInternal medicineNeoplasmsGeneticsMedicineHumansHRASChildGenetics (clinical)business.industryloose anagen hairCostello SyndromeMacrocephalyHypertrophic cardiomyopathyBrainInfantgenotype–phenotype correlationmedicine.diseaseDermatologyMagnetic Resonance ImagingMusculoskeletal AbnormalitiesEndocrinologyPhenotypeChild PreschoolFaceMutationFemalemedicine.symptombusinessMultifocal atrial tachycardiaAmerican journal of medical genetics. Part A
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Cardiomyopathy due to PRDM16 mutation: First description of a fetal presentation, with possible modifier genes

2020

PRDM16 (positive regulatory domain 16) is localized in the critical region for cardiomyopathy in patients with deletions of chromosome 1p36, as defined by Gajecka et al., American Journal of Medical Genetics, 2010, 152A, 3074-3083, and encodes a zinc finger transcription factor. We present the first fetal case of left ventricular non-compaction (LVNC) with a PRDM16 variant. The third-trimester obstetric ultrasound revealed a hydropic fetus with hydramnios and expanded hypokinetic heart. After termination of pregnancy, foetopathology showed a eutrophic fetus with isolated cardiomegaly. Endocardial fibroelastosis was associated with non-compaction of the myocardium of the left ventricle. Exom…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyCardiomyopathyBiologyLabor PresentationGenetic HeterogeneityPregnancyExome SequencingGeneticsmedicineHumansMissense mutationGenetic Predisposition to DiseaseGenetics (clinical)Exome sequencingGeneticsFetusGenes ModifierGenetic heterogeneityInfant NewbornEndocardial fibroelastosisMiddle AgedFetal Presentationmedicine.diseasePedigreeDNA-Binding ProteinsMutationMedical geneticsFemaleCardiomyopathiesTranscription FactorsAmerican Journal of Medical Genetics Part C: Seminars in Medical Genetics
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Quantitative criteria for the diagnosis of the congenital absence of pericardium by cardiac magnetic resonance

2015

Congenital absence of the left ventricular pericardium (LCAP) is a rare and poorly known cardiac malformation. Cardiac Magnetic Resonance (CMR) is generally used for the diagnosis of LCAP because of its high soft tissue contrast, multiplanarity and cine capability, but the diagnosis is usually made by only qualitative criteria. The aim of the present study was to establish quantitative criteria for the accurate diagnosis of LCAP on CMR.We enrolled nine consecutive patients affected by LCAP (mean age 26±8years, 7 males), 13 healthy controls, 13 patients with dilated cardiomyopathy (DCM), 12 patients with hypertrophic cardiomyopathy (HCM) and 13 patients with right ventricular overload (RVO).…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyMagnetic Resonance SpectroscopyCardiac magnetic resonanceLeft congenital absence of the pericardium030204 cardiovascular system & hematologyVolume change030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansPericardiumRadiology Nuclear Medicine and imagingcardiovascular diseasesbusiness.industryHealthy subjectsHypertrophic cardiomyopathyReproducibility of ResultsDilated cardiomyopathyMean ageGeneral MedicineSteady-state free precession imagingmedicine.diseasemedicine.anatomical_structurecardiovascular systemCardiologyFemaleRadiologybusinessCardiac magnetic resonancePericardiumHumanEuropean Journal of Radiology
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Ventricular tachycardia in non-compaction of left ventricle: Is this a frequent complication?

2007

Background: Isolated left ventricular non-compaction is the result of incomplete myocardial morphogenesis, leading to persistence of the embryonic myocardium. The condition is recognized by an excessively prominent trabecular meshwork and deep intertrabecular recesses of the left ventricle. Whether these intertrabecular recesses are a favorable substrate for ventricular arrhythmias is unclear. Some reports have found that the fatal ventricular arrhythmias may occur in approximately half of the patients. In this report we investigated about this association. Methods and Results: In total we evaluated a continuous series of 238 patients affected by non-compaction. Periodic Holter monitoring w…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyVentricular tachycardiaElectrocardiographyVentricular arrhythmiasIsolated left ventricular non-compaction; Malignant; Ventricular arrhythmias; Ventricular tachycardiaRisk FactorsInternal medicinemedicineHumansRegistriesisolated left ventricular non-compaction ventricular arrhythmias ventricular tachycardia malignantcardiovascular diseasesRisk factorRetrospective StudiesMALIGNANCYIsolated left ventricular non-compactionMalignantbusiness.industryVentricular tachycardiaGeneral MedicineMiddle Agedmedicine.diseasemedicine.anatomical_structureItalyVentricleAnesthesiaChild PreschoolVentricular fibrillationCardiologyTachycardia Ventricularcardiovascular systemFemaleTrabecular meshworkCardiology and Cardiovascular MedicinebusinessComplicationHolter monitoringVENTRICULAR ARRHYTHMIAS.
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Double inlet left ventricular main chamber, subaortic small left sided right ventricle and interrupted aortic arch type A. What operation is indicate…

1987

A case of a 23 year old female patient who suffered from the complex congenital heart lesion of a double inlet left ventricular main chamber, subaortic small left sided right ventricle and interrupted aortic arch type A is reported. With equally high blood pressures, the perfusion in the upper half of the body was maintained through the ascending aorta while the lower half and the lungs were supplied through the pulmonary artery and a patent ductus arteriosus (PDA). Angiographically, the bulbo-ventricular foramen appeared to be nonrestrictive. However, distinct signs of muscular subaortic stenosis were detected. The hemodynamic status principally allowed surgical correction when this became…

AdultHeart Defects CongenitalPulmonary and Respiratory MedicineAortic archmedicine.medical_specialtyHeart VentriclesAorta ThoracicAfterloadInternal medicinemedicine.arteryDuctus arteriosusAscending aortaMethodsmedicineHumansAortabusiness.industryPalliative CareInterrupted aortic arch type Amedicine.anatomical_structureDescending aortaPulmonary arterycardiovascular systemCardiologyFemaleSurgeryCardiology and Cardiovascular MedicinebusinessThe Thoracic and Cardiovascular Surgeon
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Genotype and phenotype analysis of Friedreich's ataxia compound heterozygous patients

2000

Friedreich's ataxia is caused by mutations in the FRDA gene that encodes frataxin, a nuclear-encoded mitochondrial protein. Most patients are homozygous for the expansion of a GAA triplet repeat within the FRDA gene, but a few patients show compound heterozygosity for a point mutation and the GAA-repeat expansion. We analyzed DNA samples from a cohort of 241 patients with autosomal recessive or isolated spinocerebellar ataxia for the GAA triplet expansion. Patients heterozygous for the GAA expansion were screened for point mutations within the FRDA coding region. Molecular analyses included the single-strand conformation polymorphism analysis, direct sequencing, and linkage analysis with FR…

AdultHeterozygotecongenital hereditary and neonatal diseases and abnormalitiesAtaxiaGenotypeGenetic LinkageDNA Mutational AnalysisGenes RecessiveCompound heterozygosityLoss of heterozygosityTrinucleotide RepeatsIron-Binding ProteinsGenotypeGeneticsmedicineHumansPoint MutationAge of OnsetAlleleChildAllelesPolymorphism Single-Stranded ConformationalGenetics (clinical)Family HealthGeneticsbiologynutritional and metabolic diseasesmedicine.diseasePedigreePhosphotransferases (Alcohol Group Acceptor)PhenotypeFriedreich AtaxiaChild PreschoolFrataxinbiology.proteinSpinocerebellar ataxiamedicine.symptomTrinucleotide Repeat ExpansionTrinucleotide repeat expansionMicrosatellite Repeats
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Extensive clinical experience: relative prevalence of different androgen excess disorders in 950 women referred because of clinical hyperandrogenism.

2005

CONTEXT: We undertook this study to estimate the prevalence of the various androgen excess disorders using the new criteria suggested for the diagnosis of polycystic ovary syndrome (PCOS). SETTING: The study was performed at two endocrine departments at the University of Palermo (Palermo, Italy). PATIENTS: The records of all patients referred between 1980 and 2004 for evaluation of clinical hyperandrogenism were reevaluated. All past diagnoses were reviewed using the actual diagnostic criteria. To be included in this study, the records of the patients had to present the following available data: clinical evaluation of hyperandrogenism, body weight and height, testosterone (T), free T, dehyd…

AdultHirsutismAdolescent17-alpha-HydroxyprogesteroneBody WeightOvaryAlopeciaBody HeightBody Mass IndexPelvisDiagnosis DifferentialPolycystic ovary syndrome HyperandrogenismNeoplasmsAcne VulgarisAndrogensHumansFemaleTestosteroneHyperandrogenismProgesteroneAnovulationPolycystic Ovary SyndromeRetrospective StudiesUltrasonography
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Pituitary-adrenal responses to corticotropin-releasing factor in late onset 21-hydroxylase deficiency

1990

Intravenous corticotropin-releasing factor (CRF) and adrenocorticotropin hormone (ACTH) were administered in patients with adult onset 21-hydroxylase deficiency to compare their diagnostic capability as well as to investigate hypothalamic-pituitary-adrenal function in this disorder. Responses of 17-hydroxyprogesterone, which were markedly elevated compared with controls, were identical with CRF and ACTH. However, intravenous ACTH resulted in higher androstenedione levels in comparison to CRF. Adrenocorticotropin hormone also resulted in decreased cortisol responses, confirming a defect in steroidogenesis, a finding that was not evident with CRF. Plasma ACTH responses to CRF were similar in …

AdultHirsutismendocrine systemmedicine.medical_specialtyAdolescentCorticotropin-Releasing HormoneLate onsetPeptide hormoneDecreased cortisolAdrenocorticotropic HormoneInternal medicineAdrenal GlandsHydroxyprogesteronesmedicineHumansTestosteroneIn patientAndrostenedioneAdrenal Hyperplasia Congenitalbiologybusiness.industry17-alpha-HydroxyprogesteroneAndrostenedione21-HydroxylaseObstetrics and GynecologyDiagnostic testEndocrinologyReproductive MedicinePituitary GlandSteroid Hydroxylasesbiology.proteinFemalebusinesshormones hormone substitutes and hormone antagonistsHormoneFertility and Sterility
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