Search results for "GII.4"

showing 9 items of 9 documents

Variant GII.4 noroviruses in Italian children

2009

Objectives: Among human noroviruses (NoVs), a few genogroup II strains of genotype 4 (GII.4) are dominant worldwide. GII.4 NoVs evolve rapidly and in 2006 two new epidemic variants have been identified. To investigate the circulation of GII.4 NoV variants in Italy a sequence analysis was performed on NoV strains obtained from children hospitalized for sporadic viral gastroenteritis in Palermo. Methods: A total of 465 faecal specimens were collected from children (< 5 years) hospitalized from January 2005 to December 2006. The presence of NoVs was detected by RT-PCR using primers JV12/JV13, targeting the region A of the RNA-dependent RNA-polymerase (RdRp) gene. NoV strains were genotyped by …

NoroviruSettore MED/07 - Microbiologia E Microbiologia ClinicagenotypingItalySettore MED/17 - Malattie InfettiveGII.4 genotype
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Analysis of early strains of the norovirus pandemic variant GII.4 Sydney 2012 identifies mutations in adaptive sites of the capsid protein.

2014

AbstractGlobal surveillance for norovirus identified in 2012 the emergence of a novel pandemic GII.4 variant, termed Sydney 2012. In Italy, the novel pandemic variant was identified as early as November 2011 but became predominant only in the winter season 2012–2013. Upon sequencing and comparison with strains of global origin, the early Sydney 2012 strains were found to differ from those spreading in 2012–2013 in the capsid (ORF2) putative epitopes B, C and D, segregating into a distinct phylogenetic clade. At least three residues (333, 340 and 393, in epitopes B, C and D, respectively) of the VP1 varied among Sydney 2012 strains of different clades. These findings suggest that the spread …

Settore MED/07 - Microbiologia E Microbiologia ClinicaEvolutionMolecular Sequence DataCapsid protein VP1 epitopes Evolution GII.4 Italy Norovirus Sydney 2012 variantBiologymedicine.disease_causeEpitopeSydney 2012 variantVirologyPandemicmedicineHumansAmino Acid SequenceCladePandemicsPhylogenyPhylogenetic treeNorovirusCapsid protein VP1 epitopesVirologyGastroenteritisCapsidItalyMutationNorovirusCapsid ProteinsSeasonsWinter seasonGII.4Virology
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Evidence for recombination between the pandemic GII.4 norovirus strains New Orleans 2009 and Sydney 2012

2013

ABSTRACT During 2012, a novel pandemic GII.4 norovirus variant, Sydney 2012, emerged worldwide. A signature of the variant was a GII.Pe ORF1, in association with GII.4 Apeldoorn 2008-like ORF2-ORF3 genes. We report the detection of recombinant GII.4 Sydney 2012 strains, possessing the ORF1 gene of the former pandemic variant New Orleans 2009.

Microbiology (medical)Settore MED/07 - Microbiologia E Microbiologia ClinicaNorovirus GII.4 Sydney 2012 New Orleans 2009 recombinationvirusesMolecular Sequence DataBiologymedicine.disease_causeOpen Reading Framesfluids and secretionsViral geneticsVirologyPandemicmedicineHumansChildPandemicsCaliciviridae InfectionsRecombination GeneticGeneticsNorovirusvirus diseasesSequence Analysis DNAVirologyChild PreschoolNorovirusRNA Viral
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Norovirus GII.4/Sydney/2012 in Italy, Winter 2012–2013

2013

To the Editor: Noroviruses (NoVs) are the major cause of acute gastroenteritis in children and adults; they are responsible for sporadic cases and outbreaks of gastroenteritis in various epidemiologic settings. NoVs can be classified genetically into at least 5 genogroups, GI to GV (1). Although >30 genotypes within genogroups GI, GII, and GIV can infect humans (2), a single genotype, GII.4, has been associated with most NoV-related outbreaks and sporadic cases of gastroenteritis worldwide (3). GII.4 NoV strains continuously undergo genetic/antigenic diversification and periodically generate novel strains through accumulation of punctate mutations or recombination. New GII.4 variants emerge…

Microbiology (medical)Settore MED/07 - Microbiologia E Microbiologia ClinicaLetterGenes ViralGenotypeEpidemiologySequence analysisviruseslcsh:MedicineBiologymedicine.disease_causeNorovirus GII.4 Italylcsh:Infectious and parasitic diseasesDisease Outbreaksfluids and secretionsGenotypemedicinePrevalencevariant Sydney 2012Humanslcsh:RC109-216virusesTypingviruses enteric diseasesLetters to the EditorCaliciviridae InfectionsIncidence (epidemiology)enteric infectionslcsh:RgenogroupsNorovirusvirus diseasesOutbreakVirologyGastroenteritisInfectious DiseasesCaliciviridae InfectionsItalyChild PreschoolNorovirussurveillanceMultilocus sequence typingSeasonsGII.4Multilocus Sequence TypingEmerging Infectious Diseases
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Epidemiological dynamics of norovirus GII.4 variant New Orleans 2009.

2015

Norovirus (NoV) is one of the major causes of diarrhoeal disease with epidemic, outbreak and sporadic patterns in humans of all ages worldwide. NoVs of genotype GII.4 cause nearly 80–90 % of all NoV infections in humans. Periodically, some GII.4 strains become predominant, generating major pandemic variants. Retrospective analysis of the GII.4 NoV strains detected in Italy between 2007 and 2013 indicated that the pandemic variant New Orleans 2009 emerged in Italy in the late 2009, became predominant in 2010–2011 and continued to circulate in a sporadic fashion until April 2013. Upon phylogenetic analysis based on the small diagnostic regions A and C, the late New Orleans 2009 NoVs circulati…

Settore MED/07 - Microbiologia E Microbiologia ClinicaGenotypeMolecular Sequence DataBiologymedicine.disease_causeGenomeFecesOpen Reading FramesPhylogeneticsVirologyPandemicGenotypemedicineHumansAmino Acid SequencePhylogenyCaliciviridae InfectionsRetrospective StudiesGeneticsnorovirus GII.4 variant New Orleans 2009 epidemiologyPhylogenetic treeNorovirusOutbreakNew OrleansVirologyGastroenteritisCaliciviridae InfectionsItalyNorovirusCapsid ProteinsSequence AlignmentThe Journal of general virology
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Antibodies against Lewis antigens inhibit the binding of human norovirus GII.4 virus-like particles to saliva but not to intestinal Caco-2 cells.

2016

BACKGROUND: Human noroviruses (NoVs) are the main cause of gastroenteritis worldwide. The most commonly detected NoV strains belong to the genetically diverse GII.4 genotype, with new pandemic variants emerging periodically. Despite extensive efforts, NoV investigation has been hampered by the lack of an effective in vitro cell culture system. However, NoV-derived recombinant virus-like particles (VLPs) resembling empty capsids are good surrogates for analysing NoV antigenicity and virus-ligand interactions. NoV VLPs have been reported to bind to histo-blood group antigens (HBGAs). We have analysed the ability of NoV VLPs derived from GI.1 genotype and from three GII.4 genotype variants, GI…

0301 basic medicineAdultMaleSalivaAntigenicitymedicine.drug_classSwinevirusesVirus AttachmentMonoclonal antibodyImmunofluorescenceVirusMicrobiology03 medical and health sciencesLewis Blood Group Antigensfluids and secretionsAntigenstomatognathic systemGII.4 genotypeHisto-blood group antigens (HBGAs)VirologymedicineAnimalsHumansChildSalivaAutoantibodiesbiologymedicine.diagnostic_testResearchMucinNorovirusvirus diseasesEpithelial CellsMiddle AgedVirology3. Good healthVirus-like particles (VLPs)030104 developmental biologyInfectious DiseasesHuman norovirus (NoV)Host-Pathogen Interactionsbiology.proteinReceptors VirusReceptor bindingFemaleAntibodyCaco-2 Cells
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Emerging GII.4 norovirus variants affect children with diarrhea in Palermo, Italy in 2006

2009

Although the genetic/antigenic heterogeneity of human noroviruses (NoVs) is impressive, a few genogroup II strains of genotype 4 (GII.4) are dominant worldwide. GII.4 NoVs evolve rapidly and in the last 15 years six epidemic variants have been identified. In 2005–2006, surveillance of sporadic viral gastroenteritis in children in Palermo, Italy, resulted in the detection of NoV strains in 20.9% of the patients admitted to hospital. By restriction fragment length polymorphism (RFLP) and sequence analysis of region A in the RNA-dependent RNA-polymerase (RdRp) gene, 59 NoV strains were successfully characterized. Eighty-one percent of the strains were characterized as GII.4, 14% as GIIb/Hilver…

NoroviruSettore MED/07 - Microbiologia E Microbiologia ClinicachildrenItalySettore MED/17 - Malattie InfettiveGII.4 genotypegastroenteriti
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Sorveglianza delle gastroenteriti da Norovirus in Italia: comparsa e diffusione della nuova variante GII.4 Sydney 2012

2013

In the 2012-2013 winter season, global surveillance for norovirus circulation evidenced the onset of a new norovirus GII.4 variant, termed Sydney 2012. In Italy, ISGEV hospital-based surveillance revealed that this variant already circulated at low frequency in the winter season 2011-2012 and emerged definitively only in the late 2012. This lag-time pattern mirrors the findings reported elsewhere and suggests that the novel variant circulated at low prevalence before spreading globally.

Settore MED/07 - Microbiologia E Microbiologia Clinicanorovirus surveillance Italy GII.4 variant Sydney 2012
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Genotyping of GII.4 and GIIb norovirus RT-PCR amplicons by RFLP analysis

2007

GII.4 and GIIb/Hilversum norovirus (NoV) strains appear to have a prominent epidemiological role in outbreaks or sporadic cases of human gastroenteritis. Sequence analysis, although laborious, is the reference method used for characterization of noroviruses. In this study a screening test is proposed to characterize GIIb and GII.4 NoVs based on restriction fragment length polymorphism (RFLP) analysis of amplicons obtained from the RNA-dependent RNA polymerase (RdRp) region. Virtual analysis of 793 RdRp sequences of GGI and GGII NoVs, retrieved from GenBank, and representative of global geographical origins on a long-time period, permitted the selection of four restriction enzymes, XmnI, Ahd…

Settore MED/07 - Microbiologia E Microbiologia ClinicaGenotypeSequence analysisvirusesBiologymedicine.disease_causeVirologyGenotypemedicineHumansTypingGenotypingCaliciviridae InfectionsGeneticsReverse Transcriptase Polymerase Chain ReactionNorovirusvirus diseasesInfantVirologyGastroenteritisRestriction enzymeGIIb/Hilversum strain GII.4 genotype Restriction fragment length polymorphism (RFLP)GenBankChild PreschoolNorovirusRNA ViralRestriction fragment length polymorphismPolymorphism Restriction Fragment Length
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