Search results for "GLIOMA"

showing 10 items of 230 documents

Radiologie des gastrointestinalen Stromatumors (GIST). Gleichzeitig ein Beitrag zum Carney-Syndrom

2000

Gastrointestinal stromal tumors (GIST) represent an extremely rare group of tumors, which are mostly of smooth muscle origin like leiomyomas, leiomyosarcomas and leiomyoblastomas. With the introduction of immunohistochemical analysis an epitheloid and an autonomic nerve variant can be distinguished. The purpose of this review is to demonstrate the image morphological appearance of these rare tumors together with the pathology based upon a retrospective analysis of five of our own cases since 1997. There are no pathognomonic imaging findings for characterizing a gastrointestinal stromal tumor; however, it should be included in the differential diagnosis if one or multiple large, round or ova…

LeiomyosarcomaPathologymedicine.medical_specialtyGiSTbusiness.industrymedicine.diseaseGastric LeiomyosarcomaParagangliomaPathognomonicmedicineRadiology Nuclear Medicine and imagingStromal tumorDifferential diagnosisbusinessChondromaRöFo - Fortschritte auf dem Gebiet der Röntgenstrahlen und der bildgebenden Verfahren
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The impact of conventional and heavy ion irradiation on tumor cell migration in vitro.

2007

The influence of X-ray and (12)C heavy ion irradiation on tumor cell migration and of beta(3) and beta(1) integrin expression was investigated.Two different tumor cell lines (U87 glioma and HCT116 colon carcinoma cells) were irradiated with 1, 3, or 10 Gy X-rays or (12)C heavy ions. 24 h after irradiation a standardized Boyden Chamber assay for migration analysis was performed and cells were lysed for Western blotting.Radiation-induced influences were cell line- and radiation type-dependent. X-rays decreased HCT116 migration at higher doses and appear to increase U87 migration after 3 Gy. Heavy ions decreased migration of both cell lines dose-dependently. A trend of increased beta(3) and be…

LysisHeavy Ion RadiotherapyCell MovementGliomaCell Line TumorNeoplasmsmedicineTumor Cell MigrationHumansRadiology Nuclear Medicine and imagingIrradiationNeoplasm MetastasisneoplasmsRadiological and Ultrasound TechnologyChemistryIntegrin beta1RadiochemistryIntegrin beta3Dose-Response Relationship RadiationGliomaHeavy Ion Radiotherapymedicine.diseaseMolecular biologyIn vitroDose–response relationshipCell cultureColonic NeoplasmsInternational journal of radiation biology
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Prognostic Implications of the Complement Protein C1q in Gliomas

2019

The contribution of the complement system in the pathophysiology of brain cancers has been recently considered in light of its well-known involvement in carcinogenesis. Complement system represents an important component of the inflammatory response, which acts as a functional bridge between the innate and adaptive immune response. C1q, the first recognition subcomponent of the complement classical pathway, has recently been shown to be involved in a range of pathophysiological functions that are not dependent on complement activation. C1q is expressed in the microenvironment of various types of human tumors, including melanoma, prostate, mesothelioma, and ovarian cancers, where it can exer…

Male0301 basic medicinemedicine.disease_causePathogenesisbioinformatics analysis; C1q complement; gliomas; prognostic significance of C1q; survival probability0302 clinical medicinegliomaTumor MicroenvironmentImmunology and AllergyComplement C1qbioinformatics analysiOriginal ResearchSettore MED/27 - NeurochirurgiaBrain NeoplasmsMelanomaBioinformatics analysiGliomaPrognosisAcquired immune systemNeoplasm ProteinsGene Expression Regulation NeoplasticBioinformatics analysisFemalePrognostic significance of C1q.Databases Nucleic Acidlcsh:Immunologic diseases. Allergybioinformatics analysisImmunologyprognostic significance of C1qBiology03 medical and health sciencesClassical complement pathwayC1q complementGliomaBiomarkers TumormedicineHumanssurvival probabilitySurvival probabilityGliomasC1q complementComplement C1qmedicine.diseaseComplement systemgliomas030104 developmental biologyCancer researchlcsh:RC581-607Carcinogenesis030215 immunologyFrontiers in Immunology
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Maternal medication use and the risk of brain tumors in the offspring: The SEARCH international case-control study

2006

International audience; N-nitroso compounds (NOC) have been associated with carcinogenesis in a wide range of species, including humans. There is strong experimental data showing that nitrosamides (R(1)NNO.COR(2)), a type of NOC, are potent neuro-carcinogens when administered transplacentally. Some medications are a concentrated source of amides or amines, which in the presence of nitrites under normal acidic conditions of the stomach can form NOC. Therefore, these compounds, when ingested by women during pregnancy, may be important risk factors for tumors of the central nervous system in the offspring. The aim of the present study was to test the association between maternal use of medicat…

MaleCancer ResearchMESH: Maternal-Fetal ExchangeMESH: Pregnancy0302 clinical medicinePregnancyRisk FactorsMESH: Risk FactorsMESH: ChildRecall biasEpidemiologyMedicine030212 general & internal medicineAminesChildMaternal-Fetal Exchangeeducation.field_of_studyBrain NeoplasmsN-nitroso compoundsMESH: AminesMESH: InfantMESH: AmidesMESH: Case-Control StudiesMESH: Mothers3. Good healthOncologyChild Preschool030220 oncology & carcinogenesisMESH: Brain NeoplasmsFemaleDisease SusceptibilityAdultmedicine.medical_specialtyAdolescentOffspringcase-control studyPopulationMESH: Disease SusceptibilityMothers[SDV.CAN]Life Sciences [q-bio]/Cancerchildhood brain tumors03 medical and health sciencesInternal medicineGliomamaternal medicationHumansRisk factoreducationMESH: AdolescentPregnancyMESH: Humansbusiness.industryMESH: Child PreschoolCase-control studyInfantMESH: Adultmedicine.diseaseAmidesMESH: MaleCase-Control StudiesbusinessMESH: FemaleInternational Journal of Cancer
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Inhibition of DNA methylation sensitizes glioblastoma for tumor necrosis factor-related apoptosis-inducing ligand-mediated destruction.

2005

AbstractLife expectancy of patients affected by glioblastoma multiforme is extremely low. The therapeutic use of tumor necrosis factor–related apoptosis-inducing ligand (TRAIL) has been proposed to treat this disease based on its ability to kill glioma cell lines in vitro and in vivo. Here, we show that, differently from glioma cell lines, glioblastoma multiforme tumors were resistant to TRAIL stimulation because they expressed low levels of caspase-8 and high levels of the death receptor inhibitor PED/PEA-15. Inhibition of methyltransferases by decitabine resulted in considerable up-regulation of TRAIL receptor-1 and caspase-8, down-regulation of PED/PEA-15, inhibition of cell growth, and …

MaleCancer ResearchMethyltransferaseNudeDrug ResistanceApoptosisReceptors Tumor Necrosis FactorTNF-Related Apoptosis-Inducing LigandCASPASE-8 EXPRESSIONMiceNude mouseSIGNALING COMPLEXReceptorsAntineoplastic Combined Chemotherapy ProtocolsTumor Cells CulturedDNA Modification MethylasesIN-VIVOHeterologousCaspase 8CulturedMembrane GlycoproteinsbiologyIntracellular Signaling Peptides and ProteinsMiddle AgedTumor CellsGene Expression Regulation NeoplasticMALIGNANT GLIOMA-CELLSOncologyCaspasesDNA methylationAzacitidineTumor necrosis factor alphaFemalemedicine.drugSignal TransductionAdultBRAIN-TUMORSTransplantation HeterologousCHEMOTHERAPEUTIC-AGENTSDecitabineMice NudeDecitabineDRUG-INDUCED APOPTOSISDEATH RECEPTOR5-AZA-2'-DEOXYCYTIDINEIn vivoSettore MED/04 - PATOLOGIA GENERALEmedicineAnimalsHumansneoplasmsAgedTransplantationNeoplasticCell growthTumor Necrosis Factor-alphaHistocompatibility Antigens Class IDNA Methylationbiology.organism_classificationPhosphoproteinsReceptors TNF-Related Apoptosis-Inducing LigandGene Expression RegulationApoptosisDrug Resistance NeoplasmImmunologyCancer researchNeoplasmAdult; Aged; Animals; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Apoptosis Regulatory Proteins; Azacitidine; Caspase 8; Caspases; DNA Modification Methylases; Drug Resistance Neoplasm; Female; Glioblastoma; Histocompatibility Antigens Class I; Humans; Intracellular Signaling Peptides and Proteins; Male; Membrane Glycoproteins; Mice; Mice Nude; Middle Aged; Phosphoproteins; Receptors TNF-Related Apoptosis-Inducing Ligand; Receptors Tumor Necrosis Factor; Signal Transduction; TNF-Related Apoptosis-Inducing Ligand; Transplantation Heterologous; Tumor Cells Cultured; Tumor Necrosis Factor-alpha; DNA Methylation; Gene Expression Regulation Neoplastic; Cancer Research; OncologyTumor Necrosis FactorTRAIL-INDUCED APOPTOSISApoptosis Regulatory ProteinsGlioblastomaCancer research
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ERGO: A pilot study of ketogenic diet in recurrent glioblastoma

2014

Limiting dietary carbohydrates inhibits glioma growth in preclinical models. Therefore, the ERGO trial (NCT00575146) examined feasibility of a ketogenic diet in 20 patients with recurrent glioblastoma. Patients were put on a low-carbohydrate, ketogenic diet containing plant oils. Feasibility was the primary endpoint, secondary endpoints included the percentage of patients reaching urinary ketosis, progression-free survival (PFS) and overall survival. The effects of a ketogenic diet alone or in combination with bevacizumab was also explored in an orthotopic U87MG glioblastoma model in nude mice. Three patients (15%) discontinued the diet for poor tolerability. No serious adverse events attri…

MaleCancer ResearchStatement (logic)medicine.medical_treatmentSalvage treatmentPilot ProjectsBioinformaticsGastroenterologyMicegliomaClinical endpoint1306 Cancer ResearchglucoseArticlesMiddle AgedCombined Modality TherapyBevacizumabTreatment OutcomeTolerabilityOncologyketogenic dietFemale2730 OncologyDiet Ketogenicmedicine.drugAdultmedicine.medical_specialtyBevacizumabMice NudeAntineoplastic Agents610 Medicine & healthMistakeRecurrent GliomaBiologyAntibodies Monoclonal HumanizedInternal medicineGliomamedicineAnimalsHumansddc:610Adverse effectAgedbusiness.industryRecurrent glioblastomaGeneral surgeryKetosisNeoplasms Experimentalmedicine.diseaseSurvival Analysis10040 Clinic for NeurologySurgeryQuality of LifeNeoplasm Recurrence LocalKetosisGlioblastomabusinessmetabolismfeasibilityKetogenic dietInternational Journal of Oncology
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Clinical predictors and algorithm for the genetic diagnosis of pheochromocytoma patients.

2009

Abstract Purpose: Six pheochromocytoma susceptibility genes causing distinct syndromes have been identified; approximately one of three of all pheochromocytoma patients carry a predisposing germline mutation. When four major genes (VHL, RET, SDHB, SDHD) are analyzed in a clinical laboratory, costs are ∼$3,400 per patient. The aim of the study is to systematically obtain a robust algorithm to identify who should be genetically tested, and to determine the order in which genes should be tested. Experimental Design: DNA from 989 apparently nonsyndromic patients were scanned for germline mutations in the genes VHL, RET, SDHB, SDHC, and SDHD. Clinical parameters were analyzed as potential predic…

MaleCancer Researchendocrine system diseasesSDHBGenetic Carrier ScreeningAdrenal Gland NeoplasmsPheochromocytomamedicine.disease_causePheochromocytomaParagangliomaGermline mutationMedicineHumansGenetic Predisposition to DiseaseGenetic TestingGeneFamily HealthMutationbusiness.industryGenetic Carrier ScreeningCancerMiddle Agedmedicine.diseaseOncologyHead and Neck NeoplasmsMutationFemaleSDHDbusinessAlgorithmAlgorithmsClinical cancer research : an official journal of the American Association for Cancer Research
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Oral topotecan in children with recurrent or progressive high-grade glioma: a Phase I/II study by the German Society for Pediatric Oncology and Hemat…

2004

BACKGROUND Continuous oral treatment with topotecan may be more effective than the typical 1-day and 5-day treatment schedules. In previous studies of continuous treatment with topotecan, increased intestinal side effects were reported in adult patients; however, the experience in pediatric patients and patients with high-grade glioma is quite limited. METHODS Thirty-two pediatric patients with recurrent high-grade glioma (16 females and 16 males; median age, 9.5 years) were enrolled in the current Phase I/II study. Tumor locations included the cerebral cortex (n = 5), pons (n = 18), and other sites (n = 9). An injectable formulation of topotecan was administered orally, in ice-cold orange …

MaleCancer Researchmedicine.medical_specialtyAdolescentAdministration OralAntineoplastic AgentsGastroenterologyDrug Administration Schedule03 medical and health sciences0302 clinical medicinePharmacokineticsOral administrationInternal medicineGliomaMedicineHumansDosingChild030304 developmental biologyOrange juice0303 health sciencesbusiness.industryBrain NeoplasmsGliomamedicine.disease3. Good healthSurgeryTreatment OutcomeOncology030220 oncology & carcinogenesisChild PreschoolToxicityTopotecanFemalebusinessTopotecanProgressive diseasemedicine.drugCancer
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Glioblastoma-associated circulating monocytes and the release of epidermal growth factor.

1996

✓ Monocytes/macrophages frequently infiltrate malignant gliomas and play a central role in the tumor-associated immune response as they process tumor antigen and present it to T-lymphocytes. Findings have accumulated that peripheral blood monocytes leaving the cerebral circulation become microglial cells and vice versa and that monocytes/macrophages may stimulate malignant tumor growth by some unknown mechanism. Most malignant gliomas express growth factor receptors, for example epidermal growth factor receptor (EGFR). The aim of this study was to determine whether peripheral blood monocytes of glioma patients release EGF, the appropriate ligand of gliomacell membrane-bound EGFR. Long-term …

MaleEpidermal Growth Factorbusiness.industryBrain NeoplasmsGrowth factormedicine.medical_treatmentMonocyteMiddle Agedmedicine.diseaseImmunohistochemistryMonocytesmedicine.anatomical_structureGrowth factor receptorEpidermal growth factorCell cultureGliomaImmunologyCancer researchmedicineInterleukin 19MacrophageHumansFemalebusinessGlioblastomaJournal of neurosurgery
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Functional reorganization of the attentional networks in low-grade glioma patients: a longitudinal study.

2015

International audience; Right brain damage often provokes deficits of visuospatial attention. Although the spatial attention networks have been widely investigated in stroke patients as well as in the healthy brain, little is known about the impact of slow growing lesions in the right hemisphere. We here present a longitudinal study of 20 patients who have been undergoing awake brain surgery with per-operative line bisection testing. Our aim was to investigate the impact of tumour presence and of tumour resection on the functional (re)organization of the attention networks. We assessed patients' performance on lateralized target detection, visual exploration and line bisection before surger…

MaleLongitudinal studyMESH: Attention: physiologySettore M-PSI/02 - PSICOBIOLOGIA E PSICOLOGIA FISIOLOGICAcost function maskingAudiologyFunctional LateralityDevelopmental psychologyMESH: Nerve Net: pathologyphysiopathologyAttentionLongitudinal StudiesMESH: Space Perception: physiologyMESH: Longitudinal Studiesmedia_commonMESH: AgedMESH: Middle AgedBrain Neoplasmsbrain-tumor patientsright-hemisphere damageFunctional recoveryGliomaMiddle AgedMESH: Functional Laterality: physiologyNeuropsychology and Physiological Psychologymedicine.anatomical_structureMESH: Young Adultunilateral neglectFemalevisual neglect[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptomPsychologyAdultmedicine.medical_specialtyAdolescentCognitive Neurosciencemedia_common.quotation_subjectExperimental and Cognitive Psychologyspatial neglectMESH: Brain Neoplasms: complicationspathologyphysiopathologyLateralization of brain functionNeglectWhite matterPerceptual DisordersYoung AdultMESH: Perceptual Disorders: etiologypathologyphysiopathologymedicineDisconnection syndromeHumansSlow growing lesionNeglectAgeddisconnection syndromeMESH: AdolescentMESH: HumansHemispatial neglecthemispatial neglectMESH: AdultMESH: Glioma: complicationspathologyphysiopathologyFunctional recoveryMESH: Malesustained attentionUnilateral neglectSpace PerceptionNerve Netvisuospatial attentionMESH: Female
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