Search results for "GLUTAMATE"

showing 10 items of 434 documents

Full Domain Closure of the Ligand-binding Core of the Ionotropic Glutamate Receptor iGluR5 Induced by the High Affinity Agonist Dysiherbaine and the …

2009

The prevailing structural model for ligand activation of ionotropic glutamate receptors posits that agonist efficacy arises from the stability and magnitude of induced domain closure in the ligand-binding core structure. Here we describe an exception to the correlation between ligand efficacy and domain closure. A weakly efficacious partial agonist of very low potency for homomeric iGluR5 kainate receptors, 8,9-dideoxyneodysiherbaine (MSVIII-19), induced a fully closed iGluR5 ligand-binding core. The degree of relative domain closure, approximately 30 degrees , was similar to that we resolved with the structurally related high affinity agonist dysiherbaine and to that of l-glutamate. The ph…

Models MolecularAgonistStereochemistrymedicine.drug_classGlutamic AcidKainate receptorCrystallography X-RayLigandsBiochemistryPartial agonistCell LineReceptors Kainic AcidmedicineHumansComputer SimulationAmino AcidsReceptorMolecular BiologyAlanineBinding SitesChemistryMechanisms of Signal TransductionGlutamate receptorHydrogen BondingCell BiologyBridged Bicyclo Compounds HeterocyclicLigand (biochemistry)Protein Structure TertiaryProtein SubunitsIonotropic glutamate receptorProtein BindingIonotropic effectJournal of Biological Chemistry
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The genetic contribution of the NO system at the glutamatergic post-synapse to schizophrenia: Further evidence and meta-analysis

2013

NO is a pleiotropic signaling molecule and has an important role in cognition and emotion. In the brain, NO is produced by neuronal nitric oxide synthase (NOS-I, encoded by NOS1) coupled to the NMDA receptor via PDZ interactions; this protein-protein interaction is disrupted upon binding of NOS1 adapter protein (encoded by NOS1AP) to NOS-I. As both NOS1 and NOS1AP were associated with schizophrenia, we here investigated these genes in greater detail by genotyping new samples and conducting a meta-analysis of our own and published data. In doing so, we confirmed association of both genes with schizophrenia and found evidence for their interaction in increasing risk towards disease. Our stron…

NOS1Glutamic AcidNitric Oxide Synthase Type IPolymorphism Single NucleotidePrefrontal cortexNitric oxideAssociationSynapsechemistry.chemical_compoundGlutamatergicSDG 3 - Good Health and Well-beingmedicineAnimalsHumansGenetic Predisposition to DiseasePharmacology (medical)Promoter Regions GeneticPrefrontal cortexBiological PsychiatryAdaptor Proteins Signal TransducingPharmacologyGlutamate receptorComputational BiologyNitric oxideCognitionmedicine.diseasePsychiatry and Mental healthPost-synapseNeurologychemistrySchizophreniaSynapsesRC0321SchizophreniaNeurology (clinical)GlutamatePsychologyRC0321 Neuroscience. Biological psychiatry. NeuropsychiatryNeuroscienceSignal TransductionEuropean Neuropsychopharmacology
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Estimation of ambient GABA levels in layer I of the mouse neonatal cortex in brain slices

2010

GABAergic synapses on Cajal–Retzius neurons in layer I of the murine neocortex experience GABAB receptor (GABABR)-mediated tonic inhibition. Extracellular GABA concentration ([GABA]o) that determines the strength of GABABR-mediated inhibition is controlled by GABA transporters (GATs). In this study, we hypothesized that the strength ofpresynaptic GABABR activation reflects [GABA]o in the vicinity of synaptic contacts. Slices obtained from two age groups were used, namely postnatal days (P)2–3 and P5–7. GABAergic postsynaptic currents (IPSCs) were recorded using the whole-cell patch-clamp technique. Minimal electrical stimulation in layer I was applied to elicit evoked IPSCs (eIPSCs) using a…

NeocortexPhysiologyGlutamate decarboxylaseStimulationBiologyGABAB receptormedicine.anatomical_structurenervous systemmedicineExcitatory postsynaptic potentialBiophysicsGABAergicGABA Uptake InhibitorsPatch clampNeuroscienceThe Journal of Physiology
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NG2-expressing cells in the nervous system: role of the proteoglycan in migration and glial–neuron interaction

2005

The NG2 glycoprotein is a type I membrane protein expressed in the developing and adult central nervous system (CNS) by subpopulations of glia including oligodendroglial precursor cells (OPCs), and in the developing CNS additionally by pericytes. In the mouse CNS, expression of NG2 protein is already observed at embryonic day 13 and peaks between postnatal days 8 and 12. NG2+ cells persist in grey and white matter in adult mouse brain: cells in the developing and adult brain show clear differences in migration, cell-cycle length and lineage restriction. Several groups have provided evidence that subpopulations of NG2+ cells can generate neurons in vivo. Neuronal stimulation in the developin…

Nervous systemHistologyPDZ domainNeurexinAMPA receptorReviewCell CommunicationBiologyCell MovementmedicineCell AdhesionAnimalsHumansAntigensMolecular BiologyEcology Evolution Behavior and SystematicsNeuronsNG2 proteoglycanStem CellsGlutamate receptorBrainCell BiologyCell biologyOligodendrogliamedicine.anatomical_structurenervous systemImmunologyProteoglycansNeuronAnatomyNeurogliaDevelopmental BiologyAstrocyte
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NG2-expressing cells in the nervous system revealed by the NG2-EYFP-knockin mouse.

2008

The NG2 glycoprotein is a type I membrane protein expressed by immature cells in the developing and adult mouse. NG2+ cells of the embryonic and adult brain have been principally viewed as oligodendrocyte precursor cells but have additionally been considered a fourth glial class. They are likely to be a heterogeneous population. In order to facilitate studies on the function of NG2+ cells and to characterize these cells in situ, we generated an enhanced yellow fluorescent protein (EYFP) “knockin mouse.” EYFP-expressing cells in heterozygous knockin mice expressed the NG2 protein in all regions and at all ages studied. The EYFP+ cells did not express markers of mature glia, developing or mat…

Nervous systemYellow fluorescent proteinTransgenePopulationHippocampusS100 Calcium Binding Protein beta SubunitHippocampusNervous SystemMiceEndocrinologyBacterial ProteinsGlutamate-Ammonia LigaseGeneticsmedicineAnimalsGene Knock-In TechniquesNerve Growth FactorsAntigenseducationPromoter Regions GeneticCells CulturedNeuronseducation.field_of_studyMicrogliabiologyS100 ProteinsBrainGene Expression Regulation DevelopmentalCell BiologyEmbryonic stem cellCell biologyLuminescent ProteinsOligodendrogliamedicine.anatomical_structurenervous systemMembrane proteinAstrocytesImmunologybiology.proteinProteoglycansMicrogliaGenesis (New York, N.Y. : 2000)
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Neurofilament light chain levels reflect outcome in a patient with glutamic acid decarboxylase 65 antibody–positive autoimmune encephalitis under imm…

2020

Neurological immune-mediated side effects are rare but often severe complications of immune checkpoint inhibitor (ICI) treatment. This report describes a severe case of nivolumab/ipilimumab-associated glutamic acid decarboxylase 65-positive autoimmune encephalitis. It proposes neurofilament light chain levels, a biomarker indicating axonal damage, in the cerebrospinal fluid and serum as a putative novel biomarker for this diagnostically and therapeutically challenging entity with an often unfavorable outcome. Additionally, we provide an overview of previous reports of patients developing autoimmune encephalitis under ICI treatment.

NeurofilamentGlutamate decarboxylaseIntermediate Filaments610 MedizinHashimoto Disease03 medical and health sciences0302 clinical medicineCerebrospinal fluid610 Medical sciencesHumansMedicine030212 general & internal medicineImmune Checkpoint InhibitorsAutoimmune encephalitisbiologyGlutamate Decarboxylasebusiness.industrymedicine.diseaseIpilimumabNivolumabNeurologyImmunologybiology.proteinEncephalitisBiomarker (medicine)Neurology (clinical)AntibodyNivolumabbusiness030217 neurology & neurosurgeryEncephalitisEuropean Journal of Neurology
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Role of AMPA receptor desensitization in short term depression : lessons from retinogeniculate synapses

2021

Repetitive synapse activity induces various forms of short-term plasticity. The role of presynaptic mechanisms such as residual Ca2+ and vesicle depletion in short-term facilitation and short-term depression is well established. On the other hand, the contribution of postsynaptic mechanisms such as receptor desensitization and saturation to short-term plasticity is less well known and often ignored. In this review, I will describe short-term plasticity in retinogeniculate synapses of relay neurons of the dorsal lateral geniculate nucleus (dLGN) to exemplify the synaptic properties that facilitate the contribution of AMPA receptor desensitization to short-term plasticity. These include high …

Neuronal PlasticityPhysiologyChemistryGlutamate receptor610 MedizinExcitatory Postsynaptic PotentialsGeniculate BodiesAMPA receptorPlasticitySynaptic TransmissionSynapseDesensitization (telecommunications)Postsynaptic potentialUnipolar brush cell610 Medical sciencesSynapsesReceptors AMPANeuroscienceCalyx of Held
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Changes in NMDA-receptor function in the first week following laser-induced lesions in rat visual cortex.

2011

Focal brain injuries are accompanied by processes of functional reorganization that partially compensate the functional loss. In a previous study, extracellular recordings at the border of a laser-induced lesion in the visual cortex of rats showed an enhanced synaptic plasticity, which was mediated by the activity of NR2B-contaning NMDA-receptors (NMDARs) shedding light on the potential cellular mechanisms underlying this reorganization. Given the potentially important contribution of NMDARs in processes of functional reorganization, in the present study, we used the same lesion model to further investigate lesion-induced changes in function and localization of NMDARs in the vicinity of the…

Neuronal Plasticitymusculoskeletal neural and ocular physiologyCognitive NeuroscienceLasersBiologyReceptors N-Methyl-D-AspartateRatsLesionCellular and Molecular NeuroscienceGlutamatergicmedicine.anatomical_structureVisual cortexnervous systemCerebral cortexSynaptic plasticityNeuroplasticitymedicineNMDA receptorAnimalsmedicine.symptomRats WistarReceptorNeuroscienceVisual CortexCerebral cortex (New York, N.Y. : 1991)
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NG2 cells: Properties, progeny and origin

2010

The NG2 proteoglycan is a type 1-transmembrane protein expressed by a range of cell types within and outside the mammalian nervous system. NG2-expressing (NG2) cells are found in grey and white matter tracts of the developing and adult CNS and have previously been assumed to represent oligodendrocyte precursor cells: new work using transgenic mice has shown that NG2 cells generate oligodendrocytes, protoplasmic astrocytes and in some instances neurons in vivo. NG2 cells express GABAA receptors and the AMPA subtype of glutamate receptors. They make intimate contact to neurons prior to myelinating axons and also form electron-dense synaptic specialisations with axons in the cerebellum, cortex…

NeuronsCerebellumCell typeStem CellsGeneral NeuroscienceModels NeurologicalPDZ domainGlutamate receptorAMPA receptorBiologyArticlemedicine.anatomical_structurenervous systemSynapsesmedicineAnimalsHumansProteoglycansNeurology (clinical)AntigensReceptorLong-term depressionNeurogliaNeuroscienceIon channel linked receptorsBrain Research Reviews
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