Search results for "GLUTATHIONE"

showing 10 items of 743 documents

Effect of a two-year supplementation with low doses of antioxidant vitamins and/or minerals in elderly subjects on levels of nutrients and antioxidan…

1997

Eighty-one elderly hospitalized subjects (65 years) were recruited for a double-blind placebo-controlled study to examine low dose supplementation of antioxidant vitamins and minerals on biological and functional parameters of free radical metabolism. Subjects were randomly assigned to one of the four treatment groups, daily receiving for 2 years: placebo group; mineral group: 20 mg zinc, 100 micrograms selenium; vitamin group: 120 mg vitamin C (Vit C), 6 mg beta-carotene (beta CA), 15 mg vitamin E (Vit E); mineral and vitamin group: Zn 20 mg, Se 100 micrograms, Vit C 120 mg, beta CA 6 mg, Vit E 15 mg.Fifty-seven subjects completed the study. A large frequency of Vit C, Zn and Se deficienci…

MaleVitaminmedicine.medical_specialtyErythrocytesAntioxidantmedicine.medical_treatmentNutritional StatusMedicine (miscellaneous)chemistry.chemical_elementAscorbic AcidThiobarbituric Acid Reactive SubstancesAntioxidantsSeleniumchemistry.chemical_compoundOral administrationInternal medicinemedicineHumansVitamin ENutritional Physiological PhenomenaAgedAged 80 and overGlutathione PeroxidaseMineralsNutrition and DieteticsVitamin CSuperoxide DismutaseVitamin EVitaminsMetabolismbeta CaroteneAscorbic acidGlutathioneZincEndocrinologychemistryBiochemistryFemaleSeleniumJournal of the American College of Nutrition
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Vitamin E deficiency and the susceptibility to lipid peroxidation of mouse cardiac and skeletal muscles

1984

Effects of a short-term vitamin E deficiency on some lipid peroxidative properties were investigated in mouse cardiac and skeletal muscles. The concentration of vitamin E decreased 35.8% in 5 weeks and 61.2% in 12 weeks in skeletal muscle. The corresponding decrease in cardiac muscle was 65.7% in 12 weeks. Simultaneously the susceptibility of muscle homogenates to in vitro lipid peroxidation increased with 48.6% (5 weeks) and 44.5% (12 weeks) in skeletal muscle and with 101.8% (12 weeks) in cardiac muscle. Highly significant negative correlations were observed between the concentration of vitamin E and in vitro lipid peroxidation in cardiac and skeletal muscles. Also the sensitivity to Fe2+…

MaleVitaminmedicine.medical_specialtyTime FactorsPhysiologymedicine.medical_treatmentMice Inbred StrainsBiologyLipofuscinLipid peroxidationMicechemistry.chemical_compoundInternal medicinemedicineAnimalsVitamin EVitamin E DeficiencyTocopherolchemistry.chemical_classificationMusclesMyocardiumGlutathione peroxidaseVitamin ECardiac muscleSkeletal muscleLipid Metabolismmedicine.anatomical_structureEndocrinologychemistryVitamin E deficiencyOxidation-ReductionActa Physiologica Scandinavica
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Xanthine oxidase is involved in exercise-induced oxidative stress in chronic obstructive pulmonary disease

1999

In the present study, we hypothesized that exhaustive exercise in patients with chronic obstructive pulmonary disease (COPD) results in glutathione oxidation and lipid peroxidation and that xanthine oxidase (XO) contributes to free radical generation during exercise. COPD patients performed incremental cycle ergometry until exhaustion with (n = 8) or without (n = 8) prior treatment with allopurinol, an XO inhibitor. Reduced (GSH) and oxidized glutathione (GSSG) and lipid peroxides [malondialdehyde (MDA)] were measured in arterial blood. In nontreated COPD patients, maximal exercise (approximately 75 W) resulted in a significant increase in the GSSG-to-GSH ratio (4. 6 +/- 0.9% at rest vs. 9.…

MaleXanthine OxidasePhysiologyAllopurinolRestPhysical ExertionPhysical exercisePharmacologymedicine.disease_causeLipid peroxidationchemistry.chemical_compoundAdenosine TriphosphatePhysiology (medical)MalondialdehydemedicineHumansLung Diseases ObstructiveXanthine oxidaseCOPDGlutathione DisulfideRespiratory diseaseGlutathioneMiddle Agedmedicine.diseaseGlutathionePathophysiologyOxidative StressBiochemistrychemistryExercise TestFemaleLipid PeroxidationOxidative stress
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Induction of cytochrome P450 and/or detoxication enzymes by various extracts or rosemary: description of specific patterns

2001

The ability of rosemary to modulate cytochrome P450 (CYP) and detoxication enzymes in rat liver was evaluated by comparing the effects of dried leaves and leaf extracts with different chemical compositions: essential oil (EO) containing monoterpenes, a dichloromethane extract (DCME) containing phenolic diterpenes and a water-soluble extract (WSE) containing phenolic compounds such as rosmarinic acid and flavonoids. Chemical analyses were done in order to characterize the composition of extracts. Male Wistar rats received the leaves or extracts of rosemary in their diet at 0.5% (w/w) for 2 weeks. The effects of such treatments were evaluated for CYP (1A, 2B, 2E1), glutathione S-transferase (…

Male[SDV]Life Sciences [q-bio]ReductaseToxicologychemistry.chemical_compoundCytosol0302 clinical medicineCytochrome P-450 Enzyme System[SDV.IDA]Life Sciences [q-bio]/Food engineeringCYTOCHROME P 450AnticarcinogenComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationGLUTATHIONE S-TRANSFERASE0303 health sciencesbiologyReverse Transcriptase Polymerase Chain ReactionChemistryRosmarinic acidOrgan SizeGeneral Medicine[SDV.IDA] Life Sciences [q-bio]/Food engineeringSpecific Pathogen-Free Organisms[SDV] Life Sciences [q-bio]LiverBiochemistryEnzyme Induction030220 oncology & carcinogenesisMicrosomes Liver[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringImmunoblottingChemopreventiondigestive system03 medical and health sciencesAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRNA MessengerRats Wistar030304 developmental biologyLamiaceaePlant ExtractsBody WeightROMARINCytochrome P450GlutathioneUDP-GLUCURONOSYLTRANSFERASENAD(P)H Dehydrogenase (Quinone)RatsEnzymeMicrosomebiology.proteinRATFood Science
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Complex I dysfunction and tolerance to nitroglycerin: an approach based on mitochondrial-targeted antioxidants.

2006

Nitroglycerin (GTN) tolerance was induced in vivo (rats) and in vitro (rat and human vessels). Electrochemical detection revealed that the incubation dose of GTN (5×10 −6 mol/L) did not release NO or modify O 2 consumption when administered acutely. However, development of tolerance produced a decrease in both mitochondrial O 2 consumption and the K m for O 2 in animal and human vessels and endothelial cells in a noncompetitive action. GTN tolerance has been associated with impairment of GTN biotransformation through inhibition of aldehyde dehydrogenase (ALDH)-2, and with uncoupling of mitochondrial respiration. Feeding rats with mitochondrial-targeted antioxidants (mitoquinone [MQ]) and i…

MaleantioxidantAntioxidantPhysiologyUbiquinonemedicine.medical_treatmentMuscle RelaxationVasodilator AgentsAldehyde dehydrogenasePharmacologyMitochondrionmedicine.disease_causeAntioxidantsMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundNitroglycerinDrug toleranceoxidative stressCyclic GMPchemistry.chemical_classificationbiologyAldehyde Dehydrogenase MitochondrialDrug ToleranceGlutathioneMitochondriamitochondriaBiochemistrycardiovascular systemCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyMuscle ContractionendotheliumIn Vitro TechniquesMitochondrial ProteinsOrganophosphorus CompoundsOxygen ConsumptionRespirationmedicineAnimalsHumansReactive oxygen speciesElectron Transport Complex IDose-Response Relationship DrugEndothelial CellsGlutathioneAldehyde DehydrogenasenitroglycerinRatsOxidative Stresschemistrybiology.proteinReactive Oxygen SpeciesOxidative stressCirculation research
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Glutathionyl-biliverdin IXα, a new heme catabolite in a marine annelid: Sex and cell specific accumulation.

2011

Abstract We have isolated and characterized the green pigment accumulating in coelomic cells (eleocytes) of the common clam worm Nereis virens by means of RP-HPLC and ESI-tandem mass spectrometry. This pigment is a novel biliverdin-glutathione conjugate in which the glutathione is linked to the biliverdin-backbone via a thioether bond. The yolk precursor vitellogenin, a female-specific high-density lipoprotein ( ρ  = 1179 kg/m 3 ) with a native molecular mass of ∼500 kDa and a subunit mass of ∼150 kDa, is capable of transporting this pigment as well as heme. The sex-independent large discoidal lipoprotein present in the coelomic fluid, which is sequestered by the eleocytes, could also be sh…

Malefood.ingredientProtein subunitHemeBiochemistrychemistry.chemical_compoundVitellogeninfoodYolkAnimalsHemeSex CharacteristicsBiliverdinMolecular massbiologyBiliverdinePolychaetaGeneral MedicineGlutathioneGlutathionechemistryBiochemistrybiology.proteinlipids (amino acids peptides and proteins)FemaleHemoglobinBiochimie
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Metabolic activity of fresh and cryopreserved cynomolgus monkey (Macaca fascicularis) hepatocytes

2000

1. The effect of cryopreservation on the metabolic capacity of monkey hepatocytes over 4 h in suspension and 24 h in culture was determined. Hepatocytes were diluted in a buffer containing 10% DMSO and frozen in a computer-controlled chamber. 2. Initial ethoxyresorufin and ethoxycoumarin O-deethylase (ECOD) activities were the same in fresh and cryopreserved (CP) hepatocytes. ECOD activity in suspensions declined over 4 h but was the same in fresh and CP hepatocytes. 3. The formation of testosterone hydroxy (OHT) metabolites (namely 6beta-OHT, 2beta-OHT, 16beta-OHT, 16alpha-OHT, 15beta-OHT, 2alpha-OHT and 6beta-OHT) was unaffected by cryopreservation. The loss of OHT activities over 4 h in …

Malegenetic structuresCell SurvivalHealth Toxicology and MutagenesisCell SeparationIn Vitro TechniquesBiologyToxicologyBiochemistryCryopreservationchemistry.chemical_compoundCytochrome P-450 Enzyme SystemCell AdhesionmedicineAnimalsCytotoxicityIncubationCells CulturedCryopreservationPharmacologychemistry.chemical_classificationL-Lactate DehydrogenaseGeneral MedicineGlutathioneGlutathioneMolecular biologyeye diseasesIn vitroMacaca fascicularisEnzymemedicine.anatomical_structurechemistryBiochemistryCell cultureHepatocyteSteroid HydroxylasesHepatocytesAryl Hydrocarbon HydroxylasesXenobiotica
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Changes in glutathione status and the antioxidant system in blood and in cancer cells associate with tumour growth in vivo

1999

The relationship among cancer growth, the glutathione redox cycle and the antioxidant system was studied in blood and in tumour cells. During cancer growth, the glutathione redox status (GSH/GSSG) decreases in blood of Ehrlich ascites tumour-bearing mice. This effect is mainly due to an increase in GSSG levels. Two reasons may explain the increase in blood GSSG: (a) the increase in peroxide production by the tumour that, in addition to changes affecting the glutathione-related and the antioxidant enzyme activities, can lead to GSH oxidation within the red blood cells; and (b) an increase of GSSG release from different tissues into the blood. GSH and peroxide levels are higher in the tumour …

Maleinorganic chemicalsmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentOxidative phosphorylationmedicine.disease_causeBiochemistryAntioxidantsLipid peroxidationMicechemistry.chemical_compoundfluids and secretionsIn vivoPhysiology (medical)Internal medicinemedicineAnimalsCarcinoma Ehrlich TumorHematologic TestsCancerGlutathionemedicine.diseaseGlutathioneOxidative StressEndocrinologyBiochemistrychemistryCancer cellCell DivisionOxidative stressFree Radical Biology and Medicine
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Blood Glutathione as an Index of Radiation-Induced Oxidative Stress in Mice and Humans

1997

Abstract The effect of x-rays on GSH and GSSG levels in blood was studied in mice and humans. An HPLC method that we recently developed was applied to accurately determine GSSG levels in blood. The glutathione redox status (GSH/GSSG) decreases after irradiation. This effect is mainly due to an increase in GSSG levels. Mice received single fraction radiotherapy, at total doses of 1.0 to 7.0 Gy. Changes in GSSG in mouse blood can be detected 10 min after irradiation and last for 6 h within a range of 2.0–7.0 Gy. The highest levels of GSSG (20.1 ± 2.9 μ M), a 4.7-fold increase as compared with controls) in mouse blood are found 2 h after radiation exposure (5 Gy). Breast and lung cancer patien…

Maleinorganic chemicalsmedicine.medical_specialtyLung NeoplasmsRadicalBreast NeoplasmsRadiation inducedOxidative phosphorylationGlucosephosphate Dehydrogenasemedicine.disease_causeBiochemistryMicechemistry.chemical_compoundfluids and secretionsPhysiology (medical)Internal medicinemedicineAnimalsHumansIrradiationRadiation InjuriesChromatography High Pressure LiquidGlutathione TransferaseGlutathione PeroxidaseGlutathione DisulfideChemistryDose-Response Relationship RadiationGlutathioneGlutathioneRedox statusSingle fractionOxidative StressGlutathione ReductaseEndocrinologyBiochemistryFemaleOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich’s Ataxia

2015

Friedreich's ataxia (FRDA), the most common inherited ataxia in the Caucasian population, is a multisystemic disease caused by a significant decrease in the frataxin level. To identify genes capable of modifying the severity of the symptoms of frataxin depletion, we performed a candidate genetic screen in a Drosophila RNAi-based model of FRDA. We found that genetic reduction in TOR Complex 1 (TORC1) signalling improves the impaired motor performance phenotype of FRDA model flies. Pharmacologic inhibition of TORC1 signalling by rapamycin also restored this phenotype and increased the lifespan and ATP levels. Furthermore, rapamycin reduced the altered levels of malondialdehyde + 4-hydroxyalke…

Malelcsh:MedicineGene Expressionmedicine.disease_causeAntioxidantsAnimals Genetically ModifiedAdenosine Triphosphate0302 clinical medicineRNA interferenceIron-Binding ProteinsMalondialdehydeDrosophila Proteinslcsh:ScienceAconitate HydrataseGenetics0303 health sciencesMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionGlutathione3. Good healthCell biologyDrosophila melanogasterRNA Interferencemedicine.symptomImmunosuppressive AgentsDrosophila ProteinResearch ArticleAtaxiaLongevityMotor ActivityBiologyAconitase03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologySirolimusAldehydesSuperoxide Dismutaselcsh:RAutophagyRepressor ProteinsDisease Models AnimalOxidative StressFriedreich AtaxiaFrataxinbiology.proteinlcsh:Q030217 neurology & neurosurgeryOxidative stressTranscription FactorsGenetic screenPLOS ONE
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