Search results for "Gastric"

showing 10 items of 536 documents

A pharmacological approach to gastric acid inhibition.

2005

Proton pump inhibitors have changed our approach to gastric acid-related diseases. They are much more potent acid inhibitors than H2-antagonists and exhibit a sophisticated mechanism of action. The present review analyses the general pharmacology of proton pump inhibitors and differentiates the specific characteristics of the various compounds that belong to this family. Special emphasis has been placed in describing the clinical implications of such differences and the potential importance for adverse effects.

Peptic UlcerChemistrymedicine.drug_classPharmacology toxicologyProton-pump inhibitorProton Pump InhibitorsAnti-Ulcer AgentsGastric AcidBiochemistryMechanism of actionmedicineGastric acidHumansPharmacology (medical)medicine.symptomAdverse effectDrugs
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Gastric evacuation rate of burbot fed single-fish meals at different temperatures

1997

The gastric evacuation rates of burbot Lota lota, fed a single meal of vendace, Coregonus albula, were measured in the laboratory at five temperatures (1·3, 2·6, 4·8, 9·4 and 12·6° C). Gastric evacuation rate increased exponentially with increasing temperatrure, but the results suggest that gastric evacuation rates of burbot at low temperatures are lower than those of other freshwater fish species. Temperature and the ratio of meal weight to burbot weight were the most important factors affecting gastric evacuation rate. There was no significant difference in gastric evacuation rate between three different prey species: vendace, perch Perca fluviatilis, and smelt Osmerus eperlanus.

PerchbiologyGastric emptyingbusiness.industrydigestive oral and skin physiologyAquatic ScienceGadidaebiology.organism_classificationFisheryAnimal scienceAquacultureFreshwater fishCoregonus albulaOsmerus eperlanusbusinessSmeltEcology Evolution Behavior and SystematicsJournal of Fish Biology
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Improvement of trospium-specific absorption models for fasted and fed states in humans

2014

The purpose of this study was to mechanistically interpret the oral absorption pattern of trospium in fasted and fed states by means of gastrointestinal simulation technology. A drug absorption model was built on the basis of experimental data. According to the generated model, low permeability across the intestinal epithelium, delayed gastric emptying time and a prolonged residence time in the small intestine are the key factors governing trospium absorption in the fasted state. Furthermore, in silico modelling provided a plausible explanation of the pronounced reduction in the oral bioavailability of trospium when administered with food. The simulation results support the decreased dissol…

PharmacologyAbsorption (pharmacology)medicine.medical_specialtyFOOD EFFECTChemistryPharmaceutical ScienceGeneral MedicineGastric emptying timePharmacologySmall intestineBioavailabilitymedicine.anatomical_structureEndocrinologyInternal medicinemedicineLow permeabilityPharmacology (medical)Viscous mediumFederal stateBiopharmaceutics & Drug Disposition
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The analgesic-antiinflammatory extract fromAraujia sericifera lacks ulcerogenic activity on stress-gastric damage model

1995

The present study evaluates the ulcerogenic activity of the hexane extract from dry fruits of Araujia sericifera in an experimental model of cold-restraint stress. Treatment of rats with 200 mg/kg (i.p.) of hexane extract did not affect the gastric lesions induced by cold-restraint stress. However, it was confirmed that a dose of 300 mg/kg (i.p.) of acetylsalicylic acid (ASA) which induced a similar analgesic-antiinflammatory effect, increased significantly the level of gastric damage. Thus, the hexane extract from the fruits of A. sericifera seems to possess analgesic-antiinflammatory properties which are not linked to gastrolesive effects.

PharmacologyAspirinbiologyTraditional medicineExperimental modelbusiness.industryStomachAnalgesicGastric lesionsbiology.organism_classificationHexanechemistry.chemical_compoundmedicine.anatomical_structurechemistryBiochemistryToxicitymedicineAraujia sericiferabusinessmedicine.drugPhytotherapy Research
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Low endotoxemia prevents the reduction of gastric blood flow induced by NSAIDs: role of nitric oxide

2003

1 The role of nitric oxide (NO) in the effects of low endotoxemia on gastric damage and blood flow has been evaluated in indomethacin-treated rats. 2 Pretreatment (-1 h) with endotoxin (40 micro g kg(-1)) reduced gastric damage induced by indomethacin (20 mg kg(-1)) in conscious rats. 3 Endotoxin prevented the reduction in gastric blood flow (laser Doppler flowmetry) induced by indomethacin in pentobarbital-anaesthetised rats. 4 Pretreatment with an NO-synthase (NOS) inhibitor (L-NAME, 1 mg kg(-1)) reversed the protective effect of endotoxin on gastric blood perfusion. 5 Endotoxin did not modify the expression of mRNA for endothelial NOS or inducible NOS in the gastric corpus when evaluated…

PharmacologyCalcium metabolismbiologyEndotheliumChemistryPharmacologyEndothelial NOSNitric oxideNitric oxide synthasechemistry.chemical_compoundmedicine.anatomical_structureIn vivoAnesthesiabiology.proteinGastric mucosamedicinePerfusionBritish Journal of Pharmacology
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Gastric mucosal resistance to acute injury in experimental portal hypertension

2001

The gastric mucosa of portal hypertensive rats exhibits important microvascular changes and a nitric oxide (NO)-dependent hyperemia. This study analyses whether portal hypertensive mucosa exhibits changes in its ability to withstand aggression. Portal hypertension was induced by partial portal vein ligation (PPVL) or common bile duct ligation (CBDL) and gastric damage was induced by oral administration of ethanol or aspirin. Experiments were performed in conscious or anaesthetized rats and some animals were pre-treated with the NO-synthesis inhibitor L-NAME. Conscious PPVL or CBDL rats showed an increased resistance to the damaging effects of ethanol. Oral administration of aspirin produced…

PharmacologyPentobarbitalAspirinmedicine.medical_specialtyCirrhosisbiologybusiness.industrybiology.organism_classificationmedicine.diseaseNitric oxideNitric oxide synthasechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryEnosInternal medicinemedicinebiology.proteinGastric mucosaPortal hypertensionbusinessmedicine.drugBritish Journal of Pharmacology
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Mechanisms of gastroprotection by transdermal nitroglycerin in the rat

1999

Nitric oxide (NO) donors prevent experimentally-induced gastric mucosal damage, but their clinical utility is limited by short duration of action or unsuitability of the pharmaceutical form employed. This study analyses the gastroprotection elicited by a clinically used mode of continuous administration of an NO donor, namely the nitroglycerin patch. Application to rats of a transdermal patch that releases doses of nitroglycerin comparable to those used in man (40, 80, 160 and 400 ng min(-1) rat(-1)) reduced gastric damage induced by indomethacin (25 mg kg(-1), p.o. or s.c.). The nitroglycerin patch (160 ng min(-1) rat(-1)) also diminished damage by oral administration (1 ml) of acidified b…

PharmacologyTransdermal patchStomachPharmacologyTaurocholic acidNitric oxidechemistry.chemical_compoundmedicine.anatomical_structurechemistryPharmacokineticsOral administrationGastric mucosamedicineIntravital microscopyBritish Journal of Pharmacology
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Studies on the gastric anti-ulcer activity of hypolaetin-8-glucoside

1988

The gastric anti-ulcer activity of hypolaetin-8-O-β-D-glucoside (H-8-G), a flavonoid isolated from Sideritis leucantha, has been studied in rats. This compound significantly reduced gastric lesions induced by ethanol or acetylsalicylic acid (ASA), with an increase in mucus production. In the pylorus-ligated rat, H-8-G did not affect the volume of gastric secretion but decreased its acidity and peptic activity. The anti-ulcer activity of this flavonoid does not depend on stimulation of cell proliferation in the rat gastric mucosa. Our results suggest that H-8-G activity is related to its cytoprotective effects [mediated by endogenous prostaglandins (PGs) and associated to an increase in gast…

Pharmacologychemistry.chemical_classificationEthanolbiologyCell growthPepticdigestive oral and skin physiologyFlavonoidStimulationEndogenyPharmacologybiology.organism_classificationdigestive system diseaseschemistry.chemical_compoundmedicine.anatomical_structurechemistryBiochemistryGastric mucosamedicineSideritis leucanthaPhytotherapy Research
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Gastric histamine methyltransferase: Different methylation rates for enantiomers of side-chain methylated histamine analogues using a highly purified…

1984

The inhibitor/activator and substrate properties of enantiomers of two methylated histamines (MH) were investigated using a histamine methyltransferase preparation which was purified 1207-fold from pig fundic mucosa by ultracentrifugation, ion-exchange chromatography on DEAE-cellulose and preparative electrofocusing. In 1-100 microM concentrations, S-alpha-MH and R-alpha-MH were acceptor substrates as good as histamine itself. When substrate concentrations were increased to 1 mM these substances were methylated to an even greater extent than histamine, since they did not exert substrate inhibition on HMT. Introduction of a further methyl-group into the N alpha-position reduced acceptor subs…

Pharmacologychemistry.chemical_classificationHistamine N-MethyltransferaseChromatographyHistamine N-methyltransferaseSwineActivator (genetics)ChemistryIsoelectric focusingMethylhistaminesImmunologySubstrate (chemistry)StereoisomerismMethyltransferasesMethylationToxicologyMethylationchemistry.chemical_compoundEnzymeBiochemistryGastric MucosaAnimalsPharmacology (medical)UltracentrifugeHistamineAgents and Actions
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Role of central glutamate receptors, nitric oxide and soluble guanylyl cyclase in the inhibition by endotoxin of rat gastric acid secretion

2000

1. This study examines the role of a central pathway involving glutamate receptors, nitric oxide (NO) and cyclic GMP in the acute inhibitory effects of low doses of peripheral endotoxin on pentagastrin-stimulated acid production. 2. Vagotomy or intracisternal (i.c.) microinjections of the NO-inhibitor, N(G)-nitro-L-arginine methyl esther (L-NAME; 200 microg rat(-1)) restored acid secretory responses in endotoxin (10 microg kg(-1), i.v.)-treated rats. 3. The acid-inhibitory effect of i.v. endotoxin (10 microg kg(-1), i.v.) was prevented by prior i.c. administration of the NMDA receptor antagonists, dizocilpine maleate (MK-801; 10 nmol rat(-1)) and D-2-amino-5-phosphono-valeric acid (AP-5; 20…

Pharmacologymedicine.medical_specialtyGlutamate receptorKainate receptorAMPA receptorchemistry.chemical_compoundEndocrinologychemistryMetabotropic glutamate receptorInternal medicinemedicineDNQXGastric acidNMDA receptorSoluble guanylyl cyclaseBritish Journal of Pharmacology
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