Search results for "Gene Expression"

showing 10 items of 4085 documents

The 18S ribosomal RNA m 6 A methyltransferase Mettl5 is required for normal walking behavior in Drosophila

2020

RNA modifications have recently emerged as an important layer of gene regulation. N6-methyladenosine (m6A) is the most prominent modification on eukaryotic messenger RNA and has also been found on noncoding RNA, including ribosomal and small nuclear RNA. Recently, several m6A methyltransferases were identified, uncovering the specificity of m6A deposition by structurally distinct enzymes. In order to discover additional m6A enzymes, we performed an RNAi screen to deplete annotated orthologs of human methyltransferase-like proteins (METTLs) in Drosophila cells and identified CG9666, the ortholog of human METTL5. We show that CG9666 is required for specific deposition of m6A on 18S ribosomal …

AdenosineBiochimiem 6 AMettl5WalkingBiologyBiochemistryRibosome18S ribosomal RNA03 medical and health sciences0302 clinical medicineGene expressionRNA Ribosomal 18SGeneticsAnimalsHumansRNA methyltransferase[SDV.BDD]Life Sciences [q-bio]/Development BiologyMolecular Biology030304 developmental biologyBehavior0303 health sciencesMessenger RNAbehaviorBiologie moléculaireRNA[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyMethyltransferasesm6ARibosomal RNANon-coding RNARibosome[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry Molecular Biology/Biomolecules [q-bio.BM]3. Good healthCell biologyribosomeRNA RibosomalDrosophilaBiologie030217 neurology & neurosurgerySmall nuclear RNAReportsEMBO reports
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Expression of the pea S -adenosylmethionine decarboxylase gene is involved in developmental and environmental responses

2002

A cDNA, able to complement the S-adenosyl-L-methionine decarboxylase (SAMdC; EC 4.1.1.50)-defective yeast strain Y342, has been isolated from pea (Pisum sativum L.). Expression of the SAMdC gene was characterised during pea development. Northern analysis showed a differential expression of the pea SAMdC gene in vegetative and reproductive tissues. The highest SAMdC mRNA levels were found in undifferentiated callus and tissues with high rates of cell division, and at the onset of fruit development. SAMdC expression was also induced in senescing ovaries, probably in relation to an accumulation of spermine during ovary senescence. Finally, the levels of SAMdC transcripts in leaves and shoots w…

Adenosylmethionine DecarboxylaseDNA ComplementaryCarboxy-lyasesMolecular Sequence DataSpermineSaccharomyces cerevisiaePlant ScienceEnvironmentBiologyGene Expression Regulation EnzymologicPisumchemistry.chemical_compoundOzoneGene Expression Regulation PlantGene expressionGeneticsAmino Acid SequenceGeneSequence Homology Amino AcidReproductionGenetic Complementation TestPeasGene Expression Regulation Developmentalfood and beveragesSequence Analysis DNAbiology.organism_classificationCell biologyPlant LeavesBiochemistrychemistryAdenosylmethionine decarboxylaseFruitCallusMutationSperminePolyamineSequence AlignmentCell DivisionPlant ShootsPlanta
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Chicken adaptive response to low energy diet: main role of the hypothalamic lipid metabolism revealed by a phenotypic and multi-tissue transcriptomic…

2019

AbstractBackgroundProduction conditions of layer chicken can vary in terms of temperature or diet energy content compared to the controlled environment where pure-bred selection is undertaken. The aim of this study was to better understand the long-term effects of a 15%-energy depleted diet on egg-production, energy homeostasis and metabolism via a multi-tissue transcriptomic analysis. Study was designed to compare effects of the nutritional intervention in two layer chicken lines divergently selected for residual feed intake.ResultsChicken adapted to the diet in terms of production by significantly increasing their feed intake and decreasing their body weight and body fat composition, whil…

Adipose tissueadaptationEnergy homeostasisTranscriptome0302 clinical medicinehypothalamusBeta oxidation2. Zero hunger0303 health sciencesprise alimentaireEndocannabinoid systemAdaptation PhysiologicalCell biologyAlimentation et NutritionBody Composition[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]BiotechnologyResearch Articlelcsh:QH426-470FADS1FADS2lcsh:BiotechnologychickenpouletBiologyModels Biological03 medical and health sciencesQuantitative Trait Heritablelipidlcsh:TP248.13-248.65GeneticsAnimalsFood and Nutritionlipide030304 developmental biologyCaloric RestrictionNeurosciencesLipid metabolismLipid MetabolismDietlcsh:GeneticsGene Expression RegulationNeurons and Cognitionfeed intakeEnergy MetabolismChickenstranscriptome;lipid;feed intake;adaptation;hypothalamus;chickentranscriptome[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Neuroimmune Activation and Myelin Changes in Adolescent Rats Exposed to High-Dose Alcohol and Associated Cognitive Dysfunction: A Review with Referen…

2014

Aims: The aim of the study was to assess whether intermittent ethanol administration to adolescent rats activates innate immune response and TLRs signalling causing myelin disruption and long-term cognitive and behavioural deficits. Methods: We used a rat model of intermittent binge-like ethanol exposure during adolescence. Results: Binge-like ethanol administration to adolescent rats increased the gene expression of TLR4 and TLR2 in the prefrontal cortex (PFC), as well as inflammatory cytokines TNF alpha and IL-1 beta. Up-regulation of TLRs and inflammatory mediators were linked with alterations in the levels of several myelin proteins in the PFC of adolescent rats. These events were assoc…

AdolescentAlcohol DrinkingGene ExpressionPrefrontal CortexBinge drinkingImpulsivityProinflammatory cytokineMyelinmedicineAnimalsHumansPrefrontal cortexMyelin SheathNeuroinflammationInnate immune systemEthanolGeneral MedicineImmunity InnateToll-Like Receptor 2RatsToll-Like Receptor 4medicine.anatomical_structureImmunologyTLR4Inflammation Mediatorsmedicine.symptomCognition DisordersPsychologySignal TransductionAlcohol and Alcoholism
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Genetic proof for the transient nature of the Th17 phenotype

2010

IL-17-producing CD4(+) T cells (Th17) have been classified as a new T helper cell subset. Using an IL-17 fate mapping mouse strain, which genetically fixes the memory of IL-17 expression, we demonstrate that IL-17A/F-expressing T helper cells generated either in vitro or in vivo are not a stable T-cell subset. Upon adoptive transfer of IL-17F-reporter-positive Th17 cells to RAG-deficient or WT animals, encephalitogenic Th17 cells partially lose IL-17 expression and upregulate IFN-γ. Additionally, we show that Th1 cells can convert in vivo to IL-17A/IFN-γ-coexpressing cells in the mesenteric lymph nodes (mLN). Our data classify IL-17A and IL-17F as cytokines produced transiently in response …

Adoptive cell transferEncephalomyelitis Autoimmune ExperimentalGenes RAG-1TransgeneImmunologyReceptors Antigen T-CellMice TransgenicBiologyLymphocyte ActivationInterferon-gammaMiceInterleukin 21AntigenGenes ReporterT-Lymphocyte SubsetsIn vivomedicineAnimalsImmunology and AllergyCytotoxic T cellMesenteric lymph nodesMice KnockoutIntegrasesCell DifferentiationT helper cellTh1 CellsAdoptive TransferCell biologyMice Inbred C57BLmedicine.anatomical_structureGene Expression RegulationImmunologyTh17 CellsEuropean Journal of Immunology
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Stat3 and Gfi-1 Transcription Factors Control Th17 Cell Immunosuppressive Activity via the Regulation of Ectonucleotidase Expression

2012

International audience; Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that in vitro Th17 cells generated with the cytokines IL-6 and TGF-β expressed CD39 and CD73 ectonucleotidases, leading to adenosine release and the subsequent suppression of CD4(+) and CD8(+) T cell effector functions. The IL-6-mediated activation of the transcription factor Stat3 and the TGF-β-driven downregulation of Gfi-1 transcription factor were both essential for the expression of ectonucleotidases during Th17 cell differentiation. Stat3 supported whereas Gfi-1 repressed CD39 and CD73 expression by binding to thei…

Adoptive cell transferMESH : Transcription FactorsCellular differentiationMESH: Th17 CellsT-LymphocytesCellMESH : Promoter Regions GeneticMESH : RNA Small InterferingMESH: Mice KnockoutMice0302 clinical medicineTransforming Growth Factor betaMESH: RNA Small InterferingMESH : STAT3 Transcription FactorImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyEctonucleotidaseMESH: AnimalsRNA Small InterferingSTAT3MESH: Lymphocytes Tumor-InfiltratingPromoter Regions GeneticMESH: Antigens CD5'-NucleotidaseRegulation of gene expressionMice Knockout0303 health sciencesMESH : Gene Expression RegulationApyraseMESH: STAT3 Transcription FactorMESH: Transcription FactorsMESH: Gene Expression RegulationMESH : Mice TransgenicCell biologyMESH : Lymphocytes Tumor-InfiltratingDNA-Binding ProteinsMESH : ApyraseInfectious Diseasesmedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : DNA-Binding ProteinsMESH: ApyraseSTAT3 Transcription Factor[SDV.IMM] Life Sciences [q-bio]/ImmunologyMESH : Interleukin-6MESH: Mice TransgenicT cellImmunologyMice TransgenicMESH : Mice Inbred C57BLBiology03 medical and health sciencesLymphocytes Tumor-InfiltratingMESH: Mice Inbred C57BLAntigens CDMESH: Promoter Regions GeneticMESH : 5'-NucleotidaseMESH : MicemedicineMESH : Antigens CDMESH : Th17 CellsAnimalsTranscription factorMESH: MiceMESH: Transforming Growth Factor beta030304 developmental biologyMESH : T-LymphocytesBinding SitesInterleukin-6MESH: Interleukin-6Mice Inbred C57BLMESH: T-LymphocytesMESH : Transforming Growth Factor betaMESH: Binding SitesGene Expression Regulationbiology.proteinMESH : Mice KnockoutTh17 CellsMESH : AnimalsMESH: 5'-NucleotidaseMESH: DNA-Binding ProteinsMESH : Binding Sites030215 immunologyTranscription FactorsImmunity
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Coexpression of TGF-β1 and IL-10 Enables Regulatory T Cells to Completely Suppress Airway Hyperreactivity

2008

Abstract In allergic airway disease, Treg may play an important role in the modulation of airway hyperreactivity (AHR) and inflammation. We therefore investigated the therapeutic potential of Treg in an Ag-dependent murine asthma model. We here describe that AHR can be completely suppressed by adoptive transfer of Treg overexpressing active TGF-β1. Using mice with impaired TGF-β signaling in T cells, we could demonstrate that TGF-β signaling in recipient effector T cells or transferred Treg themselves is not required for the protective effects on AHR. However, the expression of IL-10 by Treg was found to be essential for the suppression of AHR, since Treg overexpressing active TGF-β1 but de…

Adoptive cell transferTransgeneImmunologyGene ExpressionMice Transgenicchemical and pharmacologic phenomenaInflammationT-Lymphocytes RegulatoryTransforming Growth Factor beta1MiceRespiratory HypersensitivitymedicineAnimalsImmunology and AllergyAsthmaInflammationbusiness.industryEffectorhemic and immune systemsrespiratory systemmedicine.diseaseAdoptive TransferAirway hyperreactivityInterleukin-10respiratory tract diseasesDisease Models AnimalInterleukin 10Immunologymedicine.symptombusinessSignal TransductionTransforming growth factorThe Journal of Immunology
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IL-21 regulates experimental colitis by modulating the balance between Treg and Th17 cells

2007

Regulatory T (T(reg)) cells play a key role in the maintenance of the immune system homeostasis. T(reg) cells can be generated in the periphery under control of TGF-beta, a cytokine involved in the negative control of the immune system. However, TGF-beta cooperates with IL-6 in the generation of Th17 cells, a novel class of effector cells involved in numerous inflammatory diseases, including colitis. Therefore, TGF-beta emerges as a mediator of both anti-inflammatory and pro-inflammatory processes, depending on the local cytokine milieu. Here we demonstrate that IL-21, a type-1 cytokine produced by T cells and involved in the pathogenesis of immune-mediated diseases, prevents the TGF-beta-d…

Adoptive cell transfermedicine.medical_treatmentImmunologyCellGene Expressionchemical and pharmacologic phenomenaMice SCIDBiologyT-Lymphocytes RegulatoryMiceImmune systemT-Lymphocyte SubsetsTransforming Growth Factor betaFoxP3IL-21medicineAnimalsImmunology and AllergyRNA MessengerIL-2 receptorTranscription factorSettore MED/12 - GastroenterologiaMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionInterleukinsInterleukin-17FOXP3Forkhead Transcription Factorshemic and immune systemsColitisFlow CytometryAdoptive TransferCell biologyColitis; FoxP3; IL-21; Treg cells; TGF-βTGF-βDisease Models Animalmedicine.anatomical_structureCytokineImmunologyTreg cellsHomeostasisEuropean Journal of Immunology
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Evolution of lactase persistence: an example of human niche construction

2011

Niche construction is the process by which organisms construct important components of their local environment in ways that introduce novel selection pressures. Lactase persistence is one of the clearest examples of niche construction in humans. Lactase is the enzyme responsible for the digestion of the milk sugar lactose and its production decreases after the weaning phase in most mammals, including most humans. Some humans, however, continue to produce lactase throughout adulthood, a trait known as lactase persistence. In European populations, a single mutation (−13910*T) explains the distribution of the phenotype, whereas several mutations are associated with it in Africa and the Middle …

Adult0106 biological sciencesAsiaNatural selectionmedicine.medical_treatmentLactoseBiology010603 evolutionary biology01 natural sciencesGene Expression Regulation EnzymologicGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesGene FrequencyLactase persistencemedicineAnimalsHumansComputer SimulationNeolithicAlleleDomesticationLactaseddc:599.9030304 developmental biology2. Zero hungerGenetics0303 health sciencesGenetic VariationLactaseArticlesBiological EvolutionEuropeDomestic animalsDairyingLactase persistenceNiche constructionGenetics PopulationMilkAfricaTraitLocal environmentCattleNiche constructionGeneral Agricultural and Biological SciencesSingle mutationPhilosophical Transactions of the Royal Society B: Biological Sciences
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Single-cell RNA sequencing of oocytes from ovarian endometriosis patients reveals a differential transcriptomic profile associated with lower quality

2018

Abstract STUDY QUESTION Do oocytes from women with ovarian endometriosis (OE) have a different transcriptomic profile than those from healthy women? SUMMARY ANSWER Oocytes from endometriosis patients, independently of whether they came from the affected ovary, exhibited a differential transcriptomic profile compared to oocytes from healthy egg donors. WHAT IS KNOWN ALREADY Studies of endometriosis have sought to determine whether OE affects oocyte quality. While many reports indicate that oocytes recovered from endometriotic ovaries may be affected by the disease, other studies have found no significant differences among oocyte/embryo quality and fertilization, implantation and pregnancy ra…

Adult0301 basic medicineAdolescentEndometriosisEndometriosisOvaryBiologyAndrologyTranscriptomeYoung Adult03 medical and health sciences0302 clinical medicineHuman fertilizationmedicineHumansOvarian Diseases030219 obstetrics & reproductive medicineSequence Analysis RNAGene Expression ProfilingRehabilitationEmbryogenesisObstetrics and Gynecologymedicine.diseaseOocyte030104 developmental biologymedicine.anatomical_structureReproductive MedicineCase-Control StudiesOocytesOvarian EndometriosisFemaleSingle-Cell AnalysisTranscriptomeEmbryo qualityHuman Reproduction
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