Search results for "Genodermatosis"

showing 7 items of 7 documents

Oxidative stress and mitochondrial dysfunction in Kindler syndrome

2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License.-- et al.

Premature agingMaleKeratinocytesAdolescentComputingMilieux_LEGALASPECTSOFCOMPUTINGMitochondrionmedicine.disease_causePathogenesisKindler syndrome03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBlistermedicineHumansGenetics(clinical)Pharmacology (medical)Photosensitivity DisordersGenodermatosisChildGenetics (clinical)Cells CulturedPeriodontal Diseases030304 developmental biologyAged 80 and overMedicine(all)0303 health sciencesintegumentary systemResearchGeneral MedicineGlutathioneMiddle Agedmedicine.diseaseMalondialdehydeMolecular biology3. Good healthMitochondriaOxidative StresschemistryOxidative stress030220 oncology & carcinogenesisChild PreschoolFemaleSkin cancerEpidermolysis BullosaKindlin1Oxidative stressOrphanet Journal of Rare Diseases
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Autosomal-recessive SASH1 variants associated with a new genodermatosis with pigmentation defects, palmoplantar keratoderma and skin carcinoma

2014

SASH1 (SAM and SH3 domain-containing protein 1) is a tumor suppressor gene involved in the tumorigenesis of a spectrum of solid cancers. Heterozygous SASH1 variants are known to cause autosomal-dominant dyschromatosis. Homozygosity mapping and whole-exome sequencing were performed in a consanguineous Moroccan family with two affected siblings presenting an unclassified phenotype associating an abnormal pigmentation pattern (hypo- and hyperpigmented macules of the trunk and face and areas of reticular hypo- and hyperpigmentation of the extremities), alopecia, palmoplantar keratoderma, ungueal dystrophy and recurrent spinocellular carcinoma. We identified a homozygous variant in SASH1 (c.1849…

AdultMaleSkin NeoplasmsDNA Mutational AnalysisMutation MissenseGenes RecessiveConsanguinityBiologyArticleConsanguinityKeratoderma PalmoplantarGeneticsmedicineHumansExomeGenetic Predisposition to DiseaseGenetics (clinical)Pigmentation disorderSkinFamily HealthGeneticsSiblingsTumor Suppressor ProteinsHomozygoteGenodermatosisSequence Analysis DNAFibroblastsmedicine.diseaseDisease gene identificationHyperpigmentationPedigreePalmoplantar keratodermaFemaleSkin cancermedicine.symptomSkin CarcinomaPigmentation DisordersEuropean Journal of Human Genetics
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Depressed indurated plaque with elastorrhexis as a distinctive lesion in Buschke‐Ollendorff syndrome

2020

Buschke-Ollendorff syndrome (BOS) is a rare autosomal dominant genodermatosis caused by heterozygous mutations in LEMD3 and characterized by connective tissue nevi and sclerotic bone lesions known as osteopoikilosis. We report a family with three individuals affected by BOS, two of whom manifested clinical and histopathological peculiarities, presenting with a depressed indurated plaque as the main cutaneous manifestation instead of the classic connective tissue nevi. Notable elastorrhexis was present in both biopsies.

AdultMalePathologymedicine.medical_specialtyDermatologySkin DiseasesLesionBuschke–Ollendorff syndrome030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicinemedicineHumansChildSclerotic boneOsteopoikilosisConnective tissue nevusbusiness.industryGenodermatosisSkin Diseases Geneticmedicine.diseasehumanities030220 oncology & carcinogenesisPediatrics Perinatology and Child HealthFemaleOsteopoikilosisDermatopathologymedicine.symptombusinessPediatric Dermatology
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Zinsser-Cole-Engmann syndrome: A rare case report with literature review

2014

Zinsser-Cole-Engmann syndrome, more commonly known as Dyskeratosis Congenita, is a heritable genodermatosis having an estimated incidence of 1 in 1 million people. It is important for an oral physician to be aware of this condition as oral leukoplakia occurs in this condition as part of a classic triad along with reticulate skin pigmentation and nail dystrophy. Besides these, there may be myriad multisystem involvement as well. These individuals have a high predilection for developing malignancies as well as other grave life-threatening conditions. Timely diagnosis and management of these cases may help improve their morbidity and mortality, for which oral physicians can play a major role i…

Progeriamedicine.medical_specialtyPathologyOral Medicine and Pathologybusiness.industryIncidence (epidemiology)GenodermatosisCase ReportOdontologíaDisease:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseCiencias de la saludDermatologystomatognathic diseasesUNESCO::CIENCIAS MÉDICASZinsser-Cole-Engmann syndromeRare casemedicinebusinessGeneral DentistryDyskeratosis congenitaLeukoplakiaJournal of Clinical and Experimental Dentistry
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A novel mutation of the extracellular matrix protein 1 gene (ECM1) in a patient with lipoid proteinosis (Urbach-Wiethe disease) from Sicily

2005

Summary Background  Lipoid proteinosis (LP), also known as Urbach–Wiethe disease, is a rare autosomal recessive disorder characterized by a hoarse voice, warty skin infiltration and scarring. Mutations within the extracellular matrix protein 1 (ECM1) gene cause LP. Objectives  We report the molecular analysis of the ECM1 gene in a Sicilian patient with LP in order to extend the mutation spectrum of this genodermatosis. Methods  We studied a 32-year-old female born from consanguineous parents who was diagnosed at the age of 11 years as having LP. She has a clinical phenotype corresponding to Urbach–Wiethe disease characterized by papules/nodules, indurated plaques and sometimes ulcerated les…

AdultPathologymedicine.medical_specialtySettore MED/09 - Medicina InternaBiopsyMolecular Sequence DataNonsense mutationDermatologyBiologyUrbach–Wiethe diseasemedicine.disease_causePolymerase Chain ReactionFrameshift mutationExtracellular matrix protein 1ExonmedicineHumanseducationSicilyGeneExtracellular Matrix Proteinseducation.field_of_studyMutationBase SequenceGenodermatosisSkin Diseases Geneticmedicine.diseasePedigreeECM1 gene lipoid proteinosis mutationSettore MED/03 - Genetica MedicaCodon NonsenseLipoid Proteinosis of Urbach and WietheSettore MED/26 - NeurologiaFemaleBritish Journal of Dermatology
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Cowden's syndrome diagnosed through oral lesions : a case report

2021

Cowden's syndrome (CS), also known as multiple hamartoma syndrome, is a rare autosomal dominant genodermatosis first described in 1963. It has a high penetrance in both sexes and variable phenotypes. Its origin is a PTEN (phosphatase and tensin homologue) gene mutation and affects multiple organs of endodermal, ectodermal, and mesodermal origin, resulting in the development of hamartomatous mucocutaneus lesions and an increased risk for malignancies in breast, thyroid, endometrium, kidney, colon, rectum, among other organs. The diagnosis of CS is based mainly on clinical findings and oral cavity manifestations are frequent, occurring in 80-90% of patients. This include oral and labial papil…

medicine.medical_specialtyOral Medicine and Pathologybiologybusiness.industryThyroidGenodermatosisRectumMultiple hamartoma syndromeCase ReportGene mutationmedicine.diseaseDermatologyPenetrancestomatognathic diseasesmedicine.anatomical_structuremedicinebiology.proteinPTENmedia_common.cataloged_instancebusinessGeneral DentistryDental surgeonUNESCO:CIENCIAS MÉDICASmedia_common
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Birt–Hogg–Dubé Syndrome: An Update

2012

Birt-Hogg-Dube syndrome is a rare autosomal dominant genodermatosis that is characterized by the presence of fibrofolliculomas and/or trichodiscomas, pulmonary cysts, spontaneous pneumothorax, and renal tumors. The most common histological types found in renal tumors from patients with the syndrome are oncocytoma-chromophobe carcinoma hybrids and pure forms of chromophobe carcinoma, oncocytic carcinoma, and clear cell or papillary cell carcinoma. The syndrome is linked to mutations in the FLCN gene, which encodes folliculin and is preferentially expressed in the skin, kidney, and lung. The syndrome can exhibit a high degree of clinical variability, and the skin lesions that are a warning si…

Pathologymedicine.medical_specialtyHistologyAdenomabusiness.industryGenodermatosisDermatologymedicine.diseaseBirt–Hogg–Dubé syndromePathology and Forensic MedicineGermline mutationPneumothoraxCarcinomaMedicineFolliculinbusinessClear cellActas Dermo-Sifiliográficas (English Edition)
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